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PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, pp. e0173676 - e0173676
Autophagy is a catabolic mechanism to degrade cellular components to maintain cellular energy levels during starvation, a condition where PPAR alpha may be... 
INSULIN SIGNALING TRANSDUCTION | GLUCOSE-HOMEOSTASIS | FIBROBLAST-GROWTH-FACTOR-21 | DEACETYLATION | METABOLISM | PATHWAY | STEATOSIS | MULTIDISCIPLINARY SCIENCES | RESISTANCE | RECEPTORS | FOXO1 | TOR Serine-Threonine Kinases - metabolism | Autophagy-Related Protein 7 - metabolism | Fibroblast Growth Factors - genetics | Autophagy-Related Protein 5 - genetics | fas Receptor - metabolism | Autophagy - drug effects | Fibroblast Growth Factors - metabolism | TOR Serine-Threonine Kinases - genetics | Liver - drug effects | fas Receptor - genetics | Autophagy - genetics | Proto-Oncogene Proteins c-akt - metabolism | Forkhead Box Protein O1 - metabolism | Signal Transduction | Liver - metabolism | PPAR alpha - genetics | Ubiquitin-Conjugating Enzymes - genetics | Stearoyl-CoA Desaturase - genetics | Mice, Knockout | Triglycerides - metabolism | Sequestosome-1 Protein - genetics | Ubiquitin-Conjugating Enzymes - metabolism | Cysteine Endopeptidases - genetics | Autophagy-Related Protein 5 - metabolism | Beclin-1 - genetics | Stearoyl-CoA Desaturase - metabolism | Mice | PPAR alpha - metabolism | Autophagy-Related Proteins - antagonists & inhibitors | Blood Glucose - metabolism | Autophagy-Related Proteins - metabolism | Forkhead Box Protein O1 - genetics | Autophagy-Related Protein 5 - antagonists & inhibitors | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Fenofibrate - pharmacology | Cysteine Endopeptidases - metabolism | Autophagy-Related Proteins - genetics | Sequestosome-1 Protein - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Autophagy-Related Protein 7 - antagonists & inhibitors | Mice, Inbred C57BL | Autophagy-Related Protein 7 - genetics | Gene Expression Regulation - drug effects | Animals | Sterol Regulatory Element Binding Protein 1 - genetics | PPAR alpha - agonists | Ubiquitin-Conjugating Enzymes - antagonists & inhibitors | Beclin-1 - metabolism | Transcription factors | Adipose tissue | Liver | Body weight | AKT protein | Biochemistry | Glucose | Assaying | Proteins | Signal transduction | Temperature effects | Fibroblasts | Physiology | Acetylation | Inhibition | Growth factors | Hepatotoxicity | Activation analysis | Methanol | Starvation | Ethanol | AMP | Metabolism | Insulin | Fatty acids | Studies | Acetaminophen | Food intake | Weight reduction | Animal welfare | Circulation | Drugs | Biotechnology | Drug abuse | Laboratories | Centrifugation | Glass | Homeostasis | Activation | Biology | Kinases | AMP-activated protein kinase | Autophagy | Nutrient status | Rodents | Nutrients | Oxidation | Heart diseases | Age | Epinephrine | AKT1 protein | Fasting | Chloroform | Diabetes mellitus | Cardiomyocytes | Acclimatization | Triglycerides | Pharmacology | Nitrogen | Calories | Medicine | Nuclear fuels | Protein kinase | Insulin resistance | Diabetes | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 05/2016, Volume 6, Issue 1, pp. 27071 - 27071
Until now, there is not yet antitumor drug with dramatically improved efficacy on non-small cell lung cancer (NSCLC). Marine organisms are rich source of novel... 
SESQUITERPENE AMINOQUINONE | OXIDATIVE STRESS | THERAPY | INHIBITION | MECHANISM | MULTIDISCIPLINARY SCIENCES | ISOMALABARICANE TRITERPENES | SMENOSPONGINE | PROGRESSION | AAPTAMINE | MARINE SPONGE | Cyclin D1 - metabolism | RNA-Binding Proteins - genetics | Reactive Oxygen Species | Triterpenes - pharmacology | Microtubule-Associated Proteins - genetics | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Humans | Cyclin-Dependent Kinase Inhibitor p27 - agonists | Gene Expression Regulation, Neoplastic | Autophagy-Related Protein 5 - genetics | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Autophagy - drug effects | Proto-Oncogene Proteins c-akt - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Cyclin D1 - antagonists & inhibitors | TOR Serine-Threonine Kinases - genetics | Antineoplastic Agents - isolation & purification | Antineoplastic Agents - pharmacology | Porifera - chemistry | Proto-Oncogene Proteins c-akt - metabolism | Protein-Serine-Threonine Kinases - metabolism | A549 Cells | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | G1 Phase Cell Cycle Checkpoints - genetics | Phosphatidylinositol 3-Kinases - genetics | Poly(ADP-ribose) Polymerases - metabolism | Animals | Poly(ADP-ribose) Polymerases - genetics | Cyclin D1 - genetics | Autophagy-Related Protein 5 - metabolism | Triterpenes - isolation & purification | Cell Proliferation - drug effects | G1 Phase Cell Cycle Checkpoints - drug effects | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | RNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Index Medicus
Journal Article
eLife, ISSN 2050-084X, 11/2016, Volume 5, Issue 2016
Selective autophagy is mediated by cargo receptors that link the cargo to the isolation membrane via interactions with Atg8 proteins. Atg8 proteins are... 
MOLECULAR-DYNAMICS | ATG12-ATG5 CONJUGATE | SWISS-MODEL WORKSPACE | STRUCTURAL BASIS | MEMBRANE | IN-VIVO | BIOLOGY | SCAFFOLD PROTEIN | UBIQUITIN-LIKE PROTEINS | SACCHAROMYCES-CEREVISIAE | LIPIDATION | Saccharomyces cerevisiae - genetics | Vesicular Transport Proteins - metabolism | Humans | Transcription Factor TFIIIA - genetics | Autophagy-Related Protein 5 - chemistry | Autophagy-Related Protein 5 - genetics | Autophagy-Related Protein 8 Family - metabolism | Transcription Factor TFIIIA - metabolism | Autophagy-Related Proteins - chemistry | Saccharomyces cerevisiae - metabolism | Biological Transport | Cloning, Molecular | Escherichia coli - metabolism | Protein Interaction Domains and Motifs | Autophagy - genetics | Autophagy-Related Protein 8 Family - genetics | Nuclear Proteins - genetics | Autophagy-Related Proteins - genetics | Binding Sites | Sequestosome-1 Protein - metabolism | Recombinant Proteins - metabolism | Amino Acid Sequence | Autophagy-Related Protein 12 - genetics | Protein Structure, Secondary | Signal Transduction | Vesicular Transport Proteins - genetics | Gene Expression Regulation | Receptors, Cell Surface - metabolism | Recombinant Proteins - chemistry | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Recombinant Proteins - genetics | Autophagy-Related Protein 8 Family - chemistry | Saccharomyces cerevisiae Proteins - genetics | Autophagy-Related Protein 12 - metabolism | Sequestosome-1 Protein - genetics | Escherichia coli - genetics | Saccharomyces cerevisiae Proteins - metabolism | Autophagy-Related Protein 5 - metabolism | Protein Binding | Molecular Docking Simulation | HeLa Cells | Autophagy-Related Proteins - metabolism | Receptors, Cell Surface - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Saccharomyces cerevisiae Proteins - chemistry | Observations | Autophagy (Cytology) | Ubiquitin | Proteins | Enzymes | Membranes | Peptides | Laboratories | Kinases | Autophagy | Phagocytosis | Binding sites | Recruitment | Index Medicus
Journal Article
Cell Death and Disease, ISSN 2041-4889, 2017, Volume 8, Issue 3, pp. e2716 - e2716
Journal Article
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 04/2018, Volume 27, Issue 8, pp. 1332 - 1342
Journal Article
Molecular Cell, ISSN 1097-2765, 07/2017, Volume 67, Issue 1, pp. 84 - 95.e5
Journal Article
The FEBS Journal, ISSN 1742-464X, 05/2017, Volume 284, Issue 9, pp. 1324 - 1337
SIRT 6 is a pivotal regulator of lipid metabolism. It is also closely connected to cardiovascular diseases, which are the main cause of death in diabetic... 
atherosclerosis | foam cells | autophagy | cholesterol efflux | 6 | SIRT | SIRT6 | ACTIVATION | CHROMATIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | LOW-DENSITY-LIPOPROTEIN | INDUCTION | PROTECTS | HYPERTROPHY | DEACETYLATION | METABOLISM | GENE-EXPRESSION | Autophagy-Related Protein 5 - antagonists & inhibitors | Microtubule-Associated Proteins - genetics | Foam Cells - ultrastructure | Microtubule-Associated Proteins - metabolism | Humans | MicroRNAs - metabolism | Autophagy-Related Protein 5 - genetics | ATP Binding Cassette Transporter, Sub-Family G, Member 1 - metabolism | Autophagy | Foam Cells - cytology | Recombinant Fusion Proteins - metabolism | ATP Binding Cassette Transporter 1 - metabolism | RNA Interference | Gene Expression Regulation, Developmental | Sirtuins - genetics | Foam Cells - metabolism | Macrophages - immunology | Recombinant Proteins - metabolism | Macrophages - ultrastructure | Microscopy, Electron, Transmission | Lysosome-Associated Membrane Glycoproteins - metabolism | Lipoproteins, LDL - adverse effects | Recombinant Proteins - chemistry | Macrophages - cytology | Cholesterol - metabolism | Macrophages - metabolism | Lipoproteins, LDL - antagonists & inhibitors | Autophagy-Related Protein 5 - metabolism | Cell Line, Tumor | Lysosome-Associated Membrane Glycoproteins - genetics | Luminescent Proteins - genetics | Foam Cells - immunology | Mutation | ATP Binding Cassette Transporter, Sub-Family G, Member 1 - genetics | Sirtuins - antagonists & inhibitors | Sirtuins - metabolism | ATP Binding Cassette Transporter 1 - genetics | Amino Acid Substitution | Luminescent Proteins - metabolism | Diabetics | Macrophages | Low density lipoproteins | Cholesterol | Atherosclerosis | Lipoproteins (low density) | Diabetes mellitus | Lipids | Metabolism | Electron microscopy | ABCA1 protein | Transmission electron microscopy | Arteriosclerosis | ATP-binding protein | Lipid metabolism | Cardiovascular diseases | Phagocytosis | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2017, Volume 292, Issue 34, pp. 14050 - 14065
Nonalcoholic fatty liver disease (steatosis) is the most prevalent liver disease in the Western world. One of the advanced pathologies is nonalcoholic... 
Non-alcoholic Fatty Liver Disease - pathology | Transcription Factor CHOP - genetics | Liver - pathology | Autophagy-Related Protein 5 - antagonists & inhibitors | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Vesicular Transport Proteins - metabolism | eIF-2 Kinase - metabolism | Humans | Endoplasmic Reticulum - metabolism | Autophagy-Related Protein 5 - genetics | Green Fluorescent Proteins - genetics | Liver - physiopathology | Recombinant Fusion Proteins - metabolism | Endoplasmic Reticulum - pathology | Liver - immunology | RNA Interference | eIF-2 Kinase - genetics | Biomarkers - metabolism | Green Fluorescent Proteins - metabolism | Liver - metabolism | Non-alcoholic Fatty Liver Disease - immunology | Carrier Proteins - antagonists & inhibitors | Gene Expression Regulation | Transcription Factor CHOP - antagonists & inhibitors | Non-alcoholic Fatty Liver Disease - metabolism | Non-alcoholic Fatty Liver Disease - physiopathology | Unfolded Protein Response | Disease Progression | eIF-2 Kinase - antagonists & inhibitors | Hep G2 Cells | Protein Transport | Carrier Proteins - genetics | Carrier Proteins - metabolism | Biopsy | Endoplasmic Reticulum Stress | Endoplasmic Reticulum - immunology | Autophagy-Related Protein 5 - metabolism | Recombinant Fusion Proteins - genetics | Transcription Factor CHOP - metabolism | Index Medicus | eukaryotic initiation factor 2 (eIF2) | autophagy | eukaryotic translation initiation | translation control | unfolded protein response (UPR) | Protein Synthesis and Degradation
Journal Article
Diabetes, ISSN 0012-1797, 11/2016, Volume 65, Issue 11, pp. 3262 - 3275
Journal Article
Gastroenterology, ISSN 0016-5085, 01/2019, Volume 156, Issue 1, pp. 203 - 217.e20
Journal Article
Nature, ISSN 0028-0836, 11/2018, Volume 563, Issue 7732, pp. 569 - 573
Autophagy captures intracellular components and delivers them to lysosomes, where they are degraded and recycled to sustain metabolism and to enable survival... 
ARGININOSUCCINATE SYNTHETASE EXPRESSION | HOMEOSTASIS | MELANOMA | CANCERS | DEPRIVATION | MULTIDISCIPLINARY SCIENCES | METABOLOMIC ANALYSIS | SUPPRESSION | DEFICIENCY | CYCLE | AGE | Neoplasm Transplantation | Liver - enzymology | Autophagy-Related Protein 7 - metabolism | Arginase - metabolism | Male | Autophagy-Related Protein 5 - genetics | Hepatocytes - metabolism | Arginine - administration & dosage | Allografts | Neoplasms - blood | Neoplasms - genetics | Autophagy - genetics | Arginase - blood | Cell Proliferation - genetics | Ornithine - metabolism | Carcinogenesis - genetics | Autophagy-Related Protein 7 - genetics | Carcinogenesis - drug effects | Animals | Arginine - blood | Arginine - pharmacology | Autophagy-Related Protein 5 - deficiency | Cell Proliferation - drug effects | Mice | Neoplasms - pathology | Dietary Supplements | Autophagy-Related Protein 7 - deficiency | Hepatocytes - enzymology | Autophagy (Cytology) | Arginine | Growth | Health aspects | Cancer cells | Tumors | Adipose tissues | Enzymes | Glycogen | Analysis | Liver | Dietary supplements | Physiological aspects | Cancer | Animal models | Adipose tissue | Lysosomes | Arginase | Biosynthesis | Malignancy | Gene deletion | Polyamines | Auxotrophs | Autophagy | Degradation | Proteins | Clonal deletion | Metabolites | Deletion | Tumorigenesis | Supplementation | Phenotypes | Starvation | Melanoma | Muscles | Argininosuccinate synthase | Regression analysis | Tumor cell lines | Metabolism | Survival | Intolerance | Diet | Hepatocytes | Cell lines | Stem cells | Ornithine | Phagocytosis | Apoptosis | Index Medicus
Journal Article