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The Journal of physiology, ISSN 0022-3751, 07/2009, Volume 587, Issue 13, pp. 3221 - 3231
Journal Article
American Journal of Psychiatry, ISSN 0002-953X, 04/2017, Volume 174, Issue 4, pp. 378 - 386
Journal Article
Molecular neurobiology, ISSN 0893-7648, 5/2016, Volume 53, Issue 4, pp. 2451 - 2467
Elevated plasma total homocysteine (Hcy) level is associated with an increased risk of Alzheimer’s disease (AD). During transsulfuration pathways, Hcy is... 
Cerebrovascular pathology | Neurology | Neurosciences | Biomedicine | Blood–brain barrier dysfunction | Alzheimer’s disease | Neurobiology | Homocysteine | Cell Biology | Dementia | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Memory - drug effects | Glial Fibrillary Acidic Protein - genetics | Cadherins - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Blood-Brain Barrier - physiopathology | Claudin-5 - metabolism | Microvessels - pathology | Male | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Antigens, CD - genetics | Alzheimer Disease - pathology | Antigens, CD - metabolism | Matrix Metalloproteinase 9 - metabolism | Cadherins - genetics | Synapses - drug effects | Alzheimer Disease - physiopathology | Hydrogen Sulfide - pharmacology | Cerebrovascular Circulation - drug effects | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Alzheimer Disease - drug therapy | Microvessels - drug effects | Claudin-5 - genetics | Cystathionine beta-Synthase - metabolism | Permeability | Blood-Brain Barrier - drug effects | Nerve Tissue Proteins - genetics | Blood-Brain Barrier - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Blood-Brain Barrier - pathology | Hydrogen Sulfide - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Intercellular Adhesion Molecule-1 - genetics | Avoidance Learning - drug effects | Alzheimer Disease - genetics | Dizocilpine Maleate - pharmacology | Hydrogen sulfide | Methyl aspartate | Brain | RNA | Neurons | Intermediate filament proteins | Alzheimer's disease | Index Medicus | cerebrovascular pathology | dementia | blood brain barrier dysfunction
Journal Article
Cell reports (Cambridge), ISSN 2211-1247, 03/2016, Volume 14, Issue 8, pp. 1930 - 1939
.... Here, we show that in vivo hippocampal synaptic plasticity in the CA3-CA1 circuit underlies contextual learning during inhibitory avoidance (IA) training... 
passive avoidance | fear conditioning | memory | ventral tegmental area | inhibitory avoidance | LTP | in vivo recording | Memory | Passive avoidance | Inhibitory avoidance | In vivo recording | Ventral tegmental area | Fear conditioning | CA1 Region, Hippocampal - cytology | Mesencephalon - cytology | Synaptic Transmission - physiology | CA1 Region, Hippocampal - drug effects | Electrodes | CA3 Region, Hippocampal - drug effects | Long-Term Potentiation - drug effects | Pyramidal Cells - physiology | Receptors, Dopamine D1 - agonists | Long-Term Potentiation - physiology | Mesencephalon - drug effects | Synaptic Transmission - drug effects | Pyramidal Cells - drug effects | Mesencephalon - physiology | Action Potentials - drug effects | Dopamine - metabolism | Microtomy | Pyramidal Cells - cytology | Synapses - drug effects | CA1 Region, Hippocampal - physiology | CA3 Region, Hippocampal - cytology | Conditioning (Psychology) - drug effects | Conditioning (Psychology) - physiology | Synapses - physiology | Tissue Culture Techniques | CA3 Region, Hippocampal - physiology | Mice, Inbred C57BL | Memory, Long-Term - drug effects | Synapses - ultrastructure | Avoidance Learning - physiology | Benzazepines - pharmacology | Memory, Long-Term - physiology | Action Potentials - physiology | Learning - physiology | Animals | Receptors, Dopamine D1 - physiology | Avoidance Learning - drug effects | Mice | Index Medicus
Journal Article
Journal Article
Molecular neurobiology, ISSN 1559-1182, 07/2016, Volume 54, Issue 6, pp. 4560 - 4583
...) and acetylcholinesterase (AChE) and enhance reactive oxygen species (ROS) generation associated with mitochondrial dysfunctions, ultrastructural changes, and learning deficits... 
Neurology | Cadmium | Neuroprotection | Neurosciences | Biomedicine | Neurobiology | Quercetin | MAP kinase signaling | Rat brain | Cell Biology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Memory - drug effects | Reactive Oxygen Species - metabolism | Rats, Wistar | Apoptosis - drug effects | Brain - enzymology | Male | Receptors, Muscarinic - metabolism | Hippocampus - drug effects | Membrane Potential, Mitochondrial - drug effects | Mitochondria - ultrastructure | Choline O-Acetyltransferase - metabolism | Acetylcholinesterase - genetics | Neuroprotective Agents - pharmacology | Brain - ultrastructure | Hippocampus - enzymology | Hippocampus - ultrastructure | Cadmium - toxicity | Brain - physiopathology | Cytochromes c - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Maze Learning - drug effects | Brain - drug effects | Animals | MAP Kinase Signaling System - drug effects | Quercetin - pharmacology | Protein Kinase C beta - metabolism | Avoidance Learning - drug effects | Hippocampus - physiopathology | Acetylcholinesterase - metabolism | Antioxidants | Brain | RNA | Choline | Body weight | Mitogens | Reactive oxygen species | Acetylcholine receptors (muscarinic) | Genes | Cognitive ability | Choline O-acetyltransferase | Kinases | Acetylcholinesterase | Proteins | Signal transduction | Neurotoxicity | Receptors | Mitochondria | Alterations | Cortex (frontal) | Rodents | Attenuation | Acetyltransferase | Water pollution effects | Rats | MAP kinase | Exposure | Gene expression | Molecular modelling | Hippocampus | Apoptosis | Integrity | Index Medicus
Journal Article