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1. Axoplasmic transport in physiology and pathology
by Weiss, D. G and Gorio, Alfredo
1982, ISBN 354011663X, ix, 192
Axonal transport | Nervous system | Diseases
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2. Full Text Neuron‐to‐neuron transmission of α‐synuclein fibrils through axonal transport
by Freundt, Eric C and Maynard, Nate and Clancy, Eileen K and Roy, Shyamali and Bousset, Luc and Sourigues, Yannick and Covert, Markus and Melki, Ronald and Kirkegaard, Karla and Brahic, Michel
Annals of Neurology, ISSN 0364-5134, 10/2012, Volume 72, Issue 4, pp. 517 - 524
Objective: The lesions of Parkinson disease spread through the brain in a characteristic pattern that corresponds to axonal projections. Previous observations... 
SLOW COMPONENT-B | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | PARKINSONS-DISEASE | Immunohistochemistry | Microfluidic Analytical Techniques | Peptide Fragments - metabolism | Synaptic Transmission - physiology | Neuroglia - physiology | alpha-Synuclein - physiology | Axonal Transport - physiology | Neurofibrils - physiology | Maleimides | Microscopy, Confocal | Animals | Amyloid beta-Peptides - metabolism | Neurons - physiology | Mice | Fluorescent Dyes | Microscopy, Fluorescence | Index Medicus | Peptide Fragments | Axonal Transport | Neurons | Neuroglia | Neurons and Cognition | Psychology and behavior | Neurobiology | Neurofibrils | Synaptic Transmission | Life Sciences | Amyloid beta-Peptides | Cognitive Sciences | alpha-Synuclein
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3. Full Text Pervasive Axonal Transport Deficits in Multiple Sclerosis Models
by Sorbara, Catherine Diamante and Wagner, Naomi Elizabeth and Ladwig, Anne and Nikić, Ivana and Merkler, Doron and Kleele, Tatjana and Marinković, Petar and Naumann, Ronald and Godinho, Leanne and Bareyre, Florence Martine and Bishop, Derron and Misgeld, Thomas and Kerschensteiner, Martin
Neuron, ISSN 0896-6273, 12/2014, Volume 84, Issue 6, pp. 1183 - 1190
Impaired axonal transport can contribute to axon degeneration and has been described in many neurodegenerative diseases. Multiple sclerosis (MS) is a common... 
MITOCHONDRIAL TRANSPORT | MARIE-TOOTH-DISEASE | KINESIN | DEMYELINATION | HEREDITARY SPASTIC PARAPLEGIA | IN-VIVO | MOUSE MODEL | NITRIC-OXIDE | NEURODEGENERATION | MUTATIONS | NEUROSCIENCES | Free Radical Scavengers - pharmacology | Organelles - physiology | Microtubules - physiology | Nerve Degeneration - physiopathology | Axonal Transport - physiology | Axonal Transport - drug effects | Axons - physiology | Multiple Sclerosis - physiopathology | Animals | Functional Neuroimaging | Spermine - pharmacology | Spinal Cord - physiology | Multiple Sclerosis - pathology | Mice | Nitric Oxide Donors - pharmacology | Spermine - analogs & derivatives | Disease Models, Animal | Multiple sclerosis | Genetic engineering | Cells | Mitochondria | Disease | Microscopy | Neurodegeneration | Rodents | Cytoskeleton | Mutation | Index Medicus
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4. Full Text Drosophila Miro Is Required for Both Anterograde and Retrograde Axonal Mitochondrial Transport
by Russo, Gary J and Louie, Kathryn and Wellington, Andrea and Macleod, Greg T and Hu, Fangle and Panchumarthi, Sarvari and Zinsmaier, Konrad E
Journal of Neuroscience, ISSN 0270-6474, 04/2009, Volume 29, Issue 17, pp. 5443 - 5455
Microtubule-based transport of mitochondria into dendrites and axons is vital for sustaining neuronal function. Transport along microtubule tracks proceeds in... 
CYTOPLASMIC DYNEIN | NEURONAL-ACTIVITY | CARGO TRANSPORT | KINESIN | PROTEIN | DYNACTIN COMPLEX | SYNAPSES | RECEPTOR | GTPASE | NEUROSCIENCES | MOTOR AXONS | Drosophila Proteins - physiology | Dyneins - physiology | Animals | Animals, Genetically Modified | Drosophila | Mitochondrial Proteins - physiology | Axonal Transport - physiology | Axons - physiology | Kinesin - physiology | Mitochondria - physiology | rho GTP-Binding Proteins - physiology | Kinesin - antagonists & inhibitors | Index Medicus | Axonal Transport | Mitochondria | Motility | Motor | Milton | GTPase | Miro
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5. Full Text Trk activation of the ERK1/2 kinase pathway stimulates intermediate chain phosphorylation and recruits cytoplasmic dynein to signaling endosomes for retrograde axonal transport
by Mitchell, David J and Blasier, Kiev R and Jeffery, Erin D and Ross, Mitchell W and Pullikuth, Ashok K and Suo, Dong and Park, Juyeon and Russell Smiley, W and Lo, Kevin W.-H and Shabanowitz, Jeffrey and Deppmann, Christopher D and Trinidad, Jonathan C and Hunt, Donald F and Catling, Andrew D and Pfister, K. Kevin
Journal of Neuroscience, ISSN 0270-6474, 10/2012, Volume 32, Issue 44, pp. 15495 - 15510
The retrograde transport of Trk-containing endosomes from the axon to the cell body by cytoplasmic dynein is necessary for axonal and neuronal survival. We... 
BETA-TUBULIN | ANTEROGRADE TRANSPORT | DYNACTIN | NEURONS | MOLECULAR MOTORS | NEUROTROPHIN RECEPTORS | IDENTIFICATION | NEUROSCIENCES | NERVE GROWTH-FACTOR | POSTTRANSLATIONAL MODIFICATION | LIGHT-CHAINS | RNA, Small Interfering - genetics | MAP Kinase Signaling System - physiology | Phosphorylation | Receptor, trkA - physiology | Endosomes - physiology | Organelles - physiology | Cell Membrane - physiology | PC12 Cells | Nerve Growth Factors - pharmacology | MAP Kinase Signaling System - genetics | Transfection | Nerve Growth Factor - physiology | Neurons - physiology | Plasmids - genetics | Cell Membrane - metabolism | Cell Survival - physiology | Electrophoresis, Polyacrylamide Gel | Rats | Axonal Transport - physiology | Cytoplasm - physiology | Blotting, Western | Dyneins - physiology | Animals | Signal Transduction - physiology | Enzyme Activation | Index Medicus
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6. Full Text The GTPase dMiro Is Required for Axonal Transport of Mitochondria to Drosophila Synapses
by Guo, Xiufang and Macleod, Greg T and Wellington, Andrea and Hu, Fangle and Panchumarthi, Sarvari and Schoenfield, Miriam and Marin, Leo and Charlton, Milton P and Atwood, Harold L and Zinsmaier, Konrad E
Neuron, ISSN 0896-6273, 2005, Volume 47, Issue 3, pp. 379 - 393
We have identified EMS-induced mutations in Miro (dMiro), an atypical mitochondrial GTPase that is orthologous to human Miro (hMiro). Mutant animals exhibit... 
MOTOR-NERVE TERMINALS | PROTEIN | STIMULATION | CYTOSOLIC CA2 | HEREDITARY SPASTIC PARAPLEGIA | MICROTUBULES | SYNAPTIC-TRANSMISSION | CYTOCHROME-OXIDASE | MUTATIONS | RELEASE | NEUROSCIENCES | Mitochondria - enzymology | Motor Activity - physiology | Calcium - metabolism | Cercopithecus aethiops | Homeostasis | Nerve Endings - metabolism | Presynaptic Terminals - ultrastructure | Drosophila Proteins - metabolism | Mitochondria - ultrastructure | Drosophila - physiology | Drosophila Proteins - physiology | Neurons - ultrastructure | Drosophila - genetics | Transport Vesicles - physiology | Muscles - ultrastructure | Synapses - physiology | rho GTP-Binding Proteins - genetics | Axonal Transport - physiology | Mitochondria - metabolism | rho GTP-Binding Proteins - physiology | Motor Neurons - metabolism | Animals | Larva | rho GTP-Binding Proteins - metabolism | Drosophila - growth & development | Drosophila Proteins - genetics | Mutation | Mitochondria - physiology | COS Cells | Neuromuscular Junction - physiology | Cell research | Neurosciences | Mitochondrial DNA | Snow | Neurons | Drosophila | Studies | Mitochondria | Insects | Microscopy | Genes | Software | Apoptosis | Defects | Index Medicus
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7. Full Text Human neural stem cells enhance structural plasticity and axonal transport in the ischaemic brain
by Andres, Robert H and Horie, Nobutaka and Slikker, William and Keren-Gill, Hadar and Zhan, Ke and Sun, Guohua and Manley, Nathan C and Pereira, Marta P and Sheikh, Lamiya A and McMillan, Erin L and Schaar, Bruce T and Svendsen, Clive N and Bliss, Tonya M and Steinberg, Gary K
Brain, ISSN 0006-8950, 2011, Volume 134, Issue 6, pp. 1777 - 1789
Stem cell transplantation promises new hope for the treatment of stroke although significant questions remain about how the grafted cells elicit their effects.... 
dendrites | APP | brain rewiring | stroke | transplantation | FUNCTIONAL RECOVERY | ENRICHED ENVIRONMENT | PRECURSOR | ADULT-RATS | STROKE RECOVERY | NEUROSCIENCES | CLINICAL NEUROLOGY | PYRAMIDAL NEURONS | NEURITE OUTGROWTH | GENE-EXPRESSION | GROWTH-FACTOR | Humans | Brain Infarction - pathology | Male | Neural Stem Cells - cytology | RNA, Messenger - metabolism | Rats, Nude | Cerebral Cortex - cytology | Psychomotor Performance - physiology | Neuronal Plasticity - physiology | Time Factors | Dendrites - physiology | Neural Stem Cells - transplantation | Biotin - metabolism | Fetus | Vibrissae - innervation | Disease Models, Animal | Brain Ischemia - complications | Dextrans - metabolism | Cell Survival | Cells, Cultured | Gene Expression Regulation | Neural Stem Cells - physiology | Rats | Axonal Transport - physiology | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Nerve Tissue Proteins - metabolism | Brain Infarction - etiology | Animals | Analysis of Variance | Corpus Callosum - pathology | Biotin - analogs & derivatives | Brain Ischemia - surgery | Index Medicus | Abridged Index Medicus | Original
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8. Full Text CNS axons globally increase axonal transport after peripheral conditioning
by Mar, Fernando M and Simões, Anabel R and Leite, Sérgio and Morgado, Marlene M and Santos, Telma E and Rodrigo, Inês S and Teixeira, Carla A and Misgeld, Thomas and Sousa, Mónica M
Journal of Neuroscience, ISSN 0270-6474, 2014, Volume 34, Issue 17, pp. 5965 - 5970
Despite the inability of CNS axons to regenerate, an increased regenerative capacity can be elicited following conditioning lesion to the peripheral branch of... 
Conditioning lesion | Axonal transport | Axon regeneration | CYCLIC-AMP | MICROTUBULES | REGENERATION | GANGLION-CELLS | axonal transport | SLOW COMPONENT-B | axon regeneration | SPINAL-CORD-INJURY | conditioning lesion | PROTEINS | LESION | NEUROSCIENCES | Rats, Wistar | Nerve Regeneration - physiology | Ganglia, Spinal - physiology | Rats | Axonal Transport - physiology | Mice, Transgenic | Axons - physiology | Animals | Lysosomes - metabolism | Neurons - physiology | Mice | Mitochondria - physiology | Cyclic AMP - metabolism | Synaptophysin - metabolism | Index Medicus | Brief Communications
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9. Full Text Deficits in axonal transport precede ALS symptoms in vivo
by Lynsey G. Bilsland and Erik Sahai and Gavin Kelly and Matthew Golding and Linda Greensmith and Giampietro Schiavo and Pietro De Camilli
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 47, pp. 20523 - 20528
ALS is a fatal neurodegenerative disease characterized by selective motor neuron death resulting in muscle paralysis. Mutations in superoxide dismutase 1... 
Axons | Speed | Mitochondria | Neuroscience | Nervous system diseases | Neurodegenerative diseases | Neurons | Sensory neurons | Amyotrophic lateral sclerosis | Sciatic nerve | Sensory neuron | Mitochondrial transport | Neurodegeneration | Motor neuron disease | motor neuron disease | TETANUS TOXIN | DYNEIN | MULTIDISCIPLINARY SCIENCES | neurodegeneration | AMYOTROPHIC-LATERAL-SCLEROSIS | NEURODEGENERATIVE DISEASES | sensory neuron | MOTOR-NEURON DEGENERATION | mitochondrial transport | RETROGRADE TRANSPORT | MOUSE MODEL | LINKED SOD1 MUTANTS | MOTONEURON DISEASE | HEAVY-CHAIN KIF5A | p75(NTR) neurotrophin receptor | Motor Neurons - physiology | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Axonal Transport - physiology | Mutation - genetics | Disease Progression | Motor Neurons - pathology | Animals | Sciatic Nerve - cytology | Mice | Superoxide Dismutase-1 | Kymography | Mitochondria - physiology | Development and progression | Research | Axonal transport | Observations | Neurophysiology | Proteins | Brain | Rodents | Mutation | Gene expression | Index Medicus | Motor neurons | Transgenic mice | Muscles | Superoxide dismutase | Retrograde transport | Paralysis | p75NTR neurotrophin receptor | Biological Sciences
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10. Full Text In vivo imaging of axonal transport of mitochondria in the diseased and aged mammalian CNS
by Yuji Takihara and Masaru Inatani and Kei Eto and Toshihiro Inoue and Alexander Kreymerman and Seiji Miyake and Shinji Ueno and Masatoshi Nagaya and Ayami Nakanishi and Keiichiro Iwao and Yoshihiro Takamura and Hirotaka Sakamoto and Keita Satoh and Mineo Kondo and Tatsuya Sakamoto and Jeffrey L. Goldberg and Junichi Nabekura and Hidenobu Tanihara
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2015, Volume 112, Issue 33, pp. 10515 - 10520
The lack of intravital imaging of axonal transport of mitochondria in the mammalian CNS precludes characterization of the dynamics of axonal transport of... 
Aging | Mammalian CNS | Mitochondrial axonal transport | Neurodegeneration | In vivo imaging | SYSTEM | mammalian CNS | in vivo imaging | OPTIC-NERVE | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | neurodegeneration | MECHANISMS | MODEL | mitochondrial axonal transport | EYE | RETINAL GANGLION-CELLS | AXONOPATHY | aging | GLAUCOMA | BRAIN | Optic Nerve - pathology | Retinal Ganglion Cells - physiology | Mice, Inbred C57BL | Central Nervous System - pathology | Axonal Transport - physiology | Male | Axons - physiology | Photons | Central Nervous System - physiology | Sclera - physiopathology | Animals | Biological Transport | Retina - cytology | Time Factors | Female | Glaucoma - pathology | Mice | Mice, Inbred BALB C | Imaging, Three-Dimensional | Mitochondria - physiology | Glaucoma - physiopathology | Disease Models, Animal | Axons | Mitochondria | Observations | Health aspects | Biological transport, Active | Central nervous system | Glaucoma | Scientific imaging | Mammals | Index Medicus | Biological Sciences
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11. Full Text LC3 Binding to the Scaffolding Protein JIP1 Regulates Processive Dynein-Driven Transport of Autophagosomes
by Fu, Meng-meng and Nirschl, Jeffrey J and Holzbaur, Erika L.F
Developmental Cell, ISSN 1534-5807, 06/2014, Volume 29, Issue 5, pp. 577 - 590
Autophagy is essential for maintaining cellular homeostasis in neurons, where autophagosomes undergo robust unidirectional retrograde transport along axons. We... 
KINESIN | STRUCTURAL BASIS | PHOSPHORYLATION | MAMMALIAN HOMOLOG | DISEASE | VESICLE TRANSPORT | DYNAMICS | INTERACTING PROTEIN-1 | AXONAL-TRANSPORT | DEVELOPMENTAL BIOLOGY | SELECTIVE AUTOPHAGY | CELL BIOLOGY | Dyneins - metabolism | Phosphorylation | Immunoprecipitation | Microtubule-Associated Proteins - metabolism | Dynactin Complex | Phagosomes | Axonal Transport - physiology | Mice, Transgenic | Autophagy - physiology | Kinesin - metabolism | Microtubule-Associated Proteins - physiology | Animals | Fluorescent Antibody Technique | Dual Specificity Phosphatase 1 - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Microscopy, Fluorescence | Dynein | Protein binding | Medical colleges | Phosphatases | Kinesin | Index Medicus | dynein | JIP1 | autophagy | kinesin | axonal transport | dynactin | LC3
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12. Full Text Sortilin associates with Trk receptors to enhance anterograde transport and neurotrophin signaling
by Tessarollo, Lino and Glerup, Simon and Fjorback, Anja W and Richner, Mette and Chao, Moses V and Willnow, Thomas E and Nykjaer, Anders and Skeldal, Sune and Kjolby, Mads and Jansen, Pernille and Vaegter, Christian B and Nyengaard, Jens R and Erdmann, Bettina and Lewin, Gary R
Nature Neuroscience, ISSN 1097-6256, 01/2011, Volume 14, Issue 1, pp. 54 - 61
Binding of target-derived neurotrophins to Trk receptors at nerve terminals is required to stimulate neuronal survival, differentiation, innervation and... 
NERVOUS-SYSTEM | TARGETED DISRUPTION | PROTEIN | GENE | BIOCHEMICAL-CHARACTERIZATION | AXONAL-TRANSPORT | DORSAL-ROOT GANGLION | NEUROSCIENCES | SENSORY NEURONS | MICE LACKING | BRAIN | Axonal Transport - genetics | Neurites - physiology | Receptor, Nerve Growth Factor - genetics | Superior Cervical Ganglion - pathology | Humans | Adaptor Proteins, Vesicular Transport - deficiency | Adaptor Proteins, Vesicular Transport - genetics | Receptors, Nerve Growth Factor - physiology | Ganglia, Spinal - cytology | Receptors, Nerve Growth Factor - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Receptor Cross-Talk - physiology | Cerebral Cortex - metabolism | HEK293 Cells | Cell Culture Techniques | Nerve Growth Factors - physiology | Neurites - drug effects | Adaptor Proteins, Vesicular Transport - physiology | Embryo, Mammalian - pathology | Nerve Growth Factor - pharmacology | Axonal Transport - physiology | Receptors, Nerve Growth Factor - genetics | Superior Cervical Ganglion - metabolism | Mice, Knockout | Hippocampus - metabolism | Animals | Signal Transduction - physiology | Mice | Ganglia, Spinal - metabolism | Physiological aspects | Neural transmission | Cell receptors | Research | Neurons | Neurotrophic functions | Index Medicus
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13. Full Text Glycoproteins gE and gI Are Required for Efficient KIF1A-Dependent Anterograde Axonal Transport of Alphaherpesvirus Particles in Neurons
by Radomir Kratchmarov and Tal Kramer and Todd M. Greco and Matthew P. Taylor and Toh Hean Ch'ng and Ileana M. Cristea and Lynn W. Enquist
Journal of Virology, ISSN 0022-538X, 09/2013, Volume 87, Issue 17, pp. 9431 - 9440
Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CYTOPLASMIC TAIL | RAT VISUAL-SYSTEM | NERVOUS-SYSTEM | HERPES-SIMPLEX-VIRUS | II MEMBRANE-PROTEIN | VIROLOGY | PSEUDORABIES VIRUS | TO-CELL SPREAD | TRIGEMINAL GANGLIA | US9 | TRANSNEURONAL SPREAD | Lipoproteins - genetics | Recombinant Fusion Proteins - physiology | PC12 Cells | Swine | Neurons - physiology | Viral Proteins - physiology | Alphaherpesvirinae - pathogenicity | Phosphoproteins - physiology | Alphaherpesvirinae - genetics | Cell Line | Neurons - virology | Viral Envelope Proteins - physiology | Viral Envelope Proteins - genetics | Virion - physiology | Cells, Cultured | Herpesvirus 1, Suid - physiology | Rats | Viral Proteins - genetics | Axonal Transport - physiology | Herpesvirus 1, Suid - pathogenicity | Phosphoproteins - genetics | Lipoproteins - physiology | Host-Pathogen Interactions |