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Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2018, Volume 20, Issue 3, pp. 307 - 319
Reactive oxygen species (ROS) contribute to tissue damage and remodelling mediated by the inflammatory response after injury. Here we show that ROS, which... 
STEM-CELLS | PHAGOCYTOSIS | ACTIVATION | SUPEROXIDE-PRODUCTION | ROS | KINASE | PTEN | NEURONS | MECHANISMS | OUTGROWTH | CELL BIOLOGY | Ganglia, Spinal - injuries | Sciatic Nerve - injuries | Axons - enzymology | Reactive Oxygen Species - metabolism | Exosomes - pathology | Sciatic Nerve - physiopathology | Spinal Cord Injuries - genetics | Spinal Cord Injuries - pathology | Endocytosis | CX3C Chemokine Receptor 1 - metabolism | Ganglia, Spinal - enzymology | Peripheral Nerve Injuries - physiopathology | Spinal Cord Injuries - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Dyneins - metabolism | Cell Line | NADPH Oxidase 2 - deficiency | Macrophages - pathology | Signal Transduction | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Endosomes - pathology | Nerve Regeneration | Macrophages - enzymology | Mice, Knockout | Animals | Peripheral Nerve Injuries - enzymology | NADPH Oxidase 2 - metabolism | Axons - pathology | Ganglia, Spinal - pathology | Exosomes - enzymology | Endosomes - enzymology | Spinal Cord Injuries - physiopathology | NADPH Oxidase 2 - genetics | Nerve Degeneration | Peripheral Nerve Injuries - genetics | Sciatic Nerve - enzymology | Axons | Regeneration | Reactive oxygen species | NADP (Coenzyme) | Nervous system | Cellular signal transduction | Research | Gene expression | Oxidase | Recovery of function | Spinal injuries | CX3CR1 protein | Inflammatory response | AKT protein | Exosomes | Macrophages | Inactivation | NAD(P)H oxidase | Signal transduction | Dorsal root ganglia | Neurodegeneration | Dieback | CYBB protein | Degeneration | Cell body | Oxygen | Deactivation | Inflammation | Ganglia | 1-Phosphatidylinositol 3-kinase | Signaling | Dynein | PTEN protein | Sciatic nerve | Endosomes | Index Medicus
Journal Article
Glia, ISSN 0894-1491, 09/2018, Volume 66, Issue 9, pp. 1960 - 1971
Myelinating glial cells (MGCs), oligodendrocytes (OLs) in the central nervous system (CNS) and Schwann cells (SCs) in the peripheral nervous system (PNS),... 
NG2 | neurofilament | PLP | axon integrity | sciatic nerve | RANVIER | MECHANISMS | AMYOTROPHIC-LATERAL-SCLEROSIS | MICRORNAS | MATURATION | NEUROSCIENCES | OLIGODENDROCYTE DIFFERENTIATION | SUPPORT | METABOLISM | INTEGRITY | ABLATION | Motor Activity - physiology | Optic Nerve - pathology | Axons - enzymology | Male | Paralysis - pathology | Atrophy | Spinal Cord - pathology | DEAD-box RNA Helicases - deficiency | Female | Myelin Sheath - enzymology | Paralysis - enzymology | Ataxia - pathology | Nerve Degeneration - enzymology | Optic Nerve - enzymology | Ribonuclease III - genetics | Myelin Sheath - pathology | Sciatic Nerve - pathology | White Matter - enzymology | Mice, Inbred C57BL | Spinal Cord - enzymology | Mice, Transgenic | Ribonuclease III - deficiency | Disease Progression | Nerve Degeneration - pathology | White Matter - pathology | DEAD-box RNA Helicases - genetics | Animals | Ataxia - enzymology | Axons - pathology | Sciatic Nerve - enzymology | Physiological aspects | Enzymes | Recombination | Peripheral nerves | Central nervous system | Motors | Nervous system | Glial cells | Clonal deletion | Sheaths | Myelin proteolipid protein | Demyelination | Neurodegeneration | Rodents | Oligodendrocytes | Nerves | Deletion | Ataxia | Degeneration | Paralysis | Peripheral nervous system | Cell survival | Myelin | Schwann cells | Tamoxifen | Survival | Axons | Myelination | Mice | Index Medicus
Journal Article
Trends in Neurosciences, ISSN 0166-2236, 2012, Volume 35, Issue 11, pp. 700 - 709
Caspase-3 has been identified as a key mediator of neuronal programmed cell death. This protease plays a central role in the developing nervous system and its... 
Neurology | synaptic plasticity | synaptic loss | Alzheimer's disease | long-term depression | neuronal differentiation | long-term potentiation | Neuronal differentiation | Synaptic loss | Long-term depression | Long-term potentiation | Synaptic plasticity | ALZHEIMERS-DISEASE | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | PROGRAMMED CELL-DEATH | MITOCHONDRIAL-MEMBRANE PERMEABILIZATION | PRECURSOR PROTEIN | HUNTINGTONS-DISEASE | PARKINSONS-DISEASE | Nerve Net - embryology | Mitochondria - enzymology | Caspase Inhibitors - pharmacology | Humans | Huntington Disease - pathology | Neurons - cytology | Nerve Net - enzymology | Molecular Targeted Therapy | Alzheimer Disease - pathology | Caspase 3 - physiology | Nervous System - growth & development | Nervous System - embryology | Huntington Disease - enzymology | Disease Models, Animal | Nerve Net - growth & development | Long-Term Synaptic Depression - physiology | Nerve Tissue Proteins - physiology | Nervous System - enzymology | Parkinson Disease - pathology | Embryonic Development - physiology | Neural Tube - physiology | Alzheimer Disease - enzymology | Receptors, N-Methyl-D-Aspartate - physiology | Mice, Knockout | Animals | Caspase Inhibitors - therapeutic use | Neurons - enzymology | Parkinson Disease - enzymology | Apoptosis Regulatory Proteins - physiology | Mice | Apoptosis - physiology | Enzyme Activation | Nervous system diseases | Neurosciences | Neural networks | Proteases | Neurons | Depression, Mental | Apoptosis | Nervous system | Mitochondria | Pharmacology | Neurodegeneration | Index Medicus | Neurodegenerative diseases | Proteinase | Central nervous system | Caspase-3 | Neurological diseases | Axons | Neural tube | Plasticity (synaptic) | Reviews | Differentiation
Journal Article
Journal Article
The Journal of cell biology, 11/2017, Volume 216, Issue 11, p. 3655
Axon degeneration is an early event and pathological in neurodegenerative conditions and nerve injuries. To discover agents that suppress neuronal death and... 
Axons - enzymology | Apoptosis - drug effects | Crush Injuries - genetics | Motor Neurons - pathology | Wallerian Degeneration | Neuroprotective Agents - pharmacology | Time Factors | Superoxide Dismutase-1 - metabolism | Transcription, Genetic | Cytoprotection | Disease Models, Animal | Signal Transduction | Adrenergic Fibers - pathology | Sciatic Neuropathy - pathology | Genotype | Mice, Transgenic | Mitochondria - pathology | Motor Neurons - enzymology | Rats, Sprague-Dawley | Sciatic Neuropathy - enzymology | Apoptosis Regulatory Proteins - metabolism | Phenotype | Axons - pathology | Neurons - enzymology | Anilides - pharmacology | Sensory Receptor Cells - enzymology | Mutation | Mitochondria - enzymology | Neurons - pathology | Phosphorylation | Sciatic Nerve - injuries | Receptor, trkA - antagonists & inhibitors | Adrenergic Fibers - drug effects | Adrenergic Fibers - enzymology | Quinolines - pharmacology | Dose-Response Relationship, Drug | Sciatic Neuropathy - genetics | Apoptosis Regulatory Proteins - genetics | Crush Injuries - pathology | Sciatic Nerve - drug effects | Neurons - drug effects | Sciatic Neuropathy - drug therapy | Motor Neurons - drug effects | Sensory Receptor Cells - pathology | Sciatic Nerve - pathology | Mice, Inbred C57BL | Cells, Cultured | Axons - drug effects | Crush Injuries - enzymology | Mitochondria - drug effects | Animals | Sensory Receptor Cells - drug effects | Receptor, trkA - metabolism | Superoxide Dismutase-1 - genetics | Receptor, trkA - genetics | Protein Kinase Inhibitors - pharmacology | Crush Injuries - drug therapy | Sciatic Nerve - enzymology
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2009, Volume 106, Issue 46, pp. 19599 - 19604
Journal Article
Journal Article
Journal Article
Neuron, ISSN 0896-6273, 2001, Volume 29, Issue 1, pp. 73 - 84
The EphA4 receptor tyrosine kinase regulates the formation of the corticospinal tract (CST), a pathway controlling voluntary movements, and of the anterior... 
NEURAL DEVELOPMENT | CARDIOVASCULAR DEVELOPMENT | TRANSMEMBRANE LIGANDS | TYROSINE KINASE | COMMISSURAL AXONS | EPHRINB LIGANDS | CENTRAL-NERVOUS-SYSTEM | SAM DOMAIN | CELL-CONTACT | NEUROSCIENCES | FAMILY | Axons - enzymology | Fetal Proteins - deficiency | Ephrin-A4 | Molecular Sequence Data | Fetal Proteins - metabolism | Brain Stem - cytology | Prosencephalon - enzymology | RNA, Messenger - biosynthesis | Brain Stem - enzymology | In Situ Hybridization | Mice, Mutant Strains | Receptor Protein-Tyrosine Kinases - deficiency | Ephrin-B2 | Pyramidal Tracts - enzymology | Motor Cortex - enzymology | Membrane Proteins - genetics | Pyramidal Tracts - embryology | Protein Structure, Tertiary - genetics | Signal Transduction - genetics | Organ Specificity | Receptor Protein-Tyrosine Kinases - metabolism | Mice, Knockout | Membrane Proteins - biosynthesis | Temporal Lobe - embryology | Animals | Receptor Protein-Tyrosine Kinases - genetics | Prosencephalon - cytology | Brain Stem - embryology | Fetal Proteins - genetics | Mice | Motor Cortex - embryology | Prosencephalon - embryology | Pyramidal Tracts - cytology | Temporal Lobe - cytology | Motor Cortex - cytology | Receptor, EphA4 | ephrins | EphA4 protein | Index Medicus | Prosencephalon/cytology/embryology/enzymology | Neurosciences | Medical and Health Sciences | Medicin och hälsovetenskap | Fetal Proteins/deficiency/genetics/metabolism | Motor Cortex/cytology/embryology/enzymology | Receptor Protein-Tyrosine Kinases/deficiency/genetics/metabolism | Research Support; Non-U.S. Gov't | Neurovetenskaper | Basic Medicine | Signal Transduction/genetics | Axons/enzymology | Protein Structure; Tertiary/genetics | Mice; Knockout | Pyramidal Tracts/cytology/embryology/enzymology | Brain Stem/cytology/embryology/enzymology | Receptor; EphA4 | Temporal Lobe/cytology/embryology | Mice; Mutant Strains | Medicinska och farmaceutiska grundvetenskaper | Membrane Proteins/biosynthesis/genetics | RNA; Messenger/biosynthesis
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2012, Volume 32, Issue 49, pp. 17540 - 17553
Journal Article