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The American journal of surgical pathology, ISSN 0147-5185, 2010, Volume 34, Issue 3, pp. 327 - 340
B-cell lymphomas with concurrent IGH-BCL2 and MYC rearrangements, also known as "double-hit'' lymphomas (DHL), are rare neoplasms characterized by highly... 
BCL2 | Cytogenetics | High-grade B-cell lymphoma | MYC | Burkitt lymphoma | Diffuse large B-cell lymphoma | high-grade B-cell lymphoma | TRANSFORMATION | SURGERY | TRANSLOCATION | T(14/18) | T(14-18) | C-MYC | FOLLICULAR LYMPHOMA | diffuse large B-cell lymphoma | PATHOLOGY | cytogenetics | POLYMERASE-CHAIN-REACTION | ACUTE LYMPHOBLASTIC-LEUKEMIA | EXPRESSION | Predictive Value of Tests | Humans | Lymphoma, B-Cell - therapy | Middle Aged | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Male | Lymphoma, B-Cell - genetics | Burkitt Lymphoma - mortality | Burkitt Lymphoma - pathology | Genes, Immunoglobulin Heavy Chain | Young Adult | Lymphoma, Large B-Cell, Diffuse - therapy | Time Factors | Karyotyping | Polymerase Chain Reaction | Aged, 80 and over | Burkitt Lymphoma - genetics | Adult | Female | Retrospective Studies | Child | Burkitt Lymphoma - therapy | Lymphoma, B-Cell - mortality | World Health Organization | Burkitt Lymphoma - classification | Lymphoma, Large B-Cell, Diffuse - pathology | Risk Assessment | Kaplan-Meier Estimate | Proportional Hazards Models | Antineoplastic Combined Chemotherapy Protocols | Terminology as Topic | Immunophenotyping | In Situ Hybridization, Fluorescence | Treatment Outcome | Lymphoma, B-Cell - classification | Lymphoma, Large B-Cell, Diffuse - mortality | Gene Rearrangement, B-Lymphocyte, Heavy Chain | Lymphoma, Large B-Cell, Diffuse - classification | Adolescent | Lymphoma, B-Cell - pathology | Aged | Proto-Oncogene Proteins c-myc - genetics | Neoplasm Staging | Lymphoma, Large B-Cell, Diffuse - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics
Journal Article
Molecular cancer, ISSN 1476-4598, 2018, Volume 17, Issue 1, pp. 57 - 23
textabstractBruton's tyrosine kinase (BTK) is a non-receptor kinase that plays a crucial role in oncogenic signaling that is critical for proliferation and... 
Chronic lymphocytic leukemia | Tumor microenvironment | B cell receptor signaling | Leukemia | Chemokine receptor | B cell development | Bruton's tyrosine kinase | Lymphoma | Ibrutinib | SIGNALING PATHWAYS | BIOCHEMISTRY & MOLECULAR BIOLOGY | X-LINKED AGAMMAGLOBULINEMIA | FACTOR-KAPPA-B | ATRIAL-FIBRILLATION | BTK INHIBITOR IBRUTINIB | TEC KINASE | ONCOLOGY | THERAPEUTIC TARGET | CHRONIC LYMPHOCYTIC-LEUKEMIA | TUMOR-ASSOCIATED MACROPHAGES | BETA-GAMMA-SUBUNITS | Leukemia, B-Cell - metabolism | Humans | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Tumor Microenvironment | Leukemia, B-Cell - etiology | Receptors, Antigen, B-Cell - metabolism | Molecular Targeted Therapy | Biomarkers, Tumor | Cell Differentiation - genetics | Agammaglobulinaemia Tyrosine Kinase - metabolism | Cell Transformation, Neoplastic - genetics | Leukemia, B-Cell - drug therapy | Agammaglobulinaemia Tyrosine Kinase - chemistry | B-Lymphocytes - pathology | Gene Expression Regulation, Neoplastic - drug effects | Lymphoma, B-Cell - etiology | Bone Marrow | B-Lymphocytes - metabolism | Lymphoma, B-Cell - drug therapy | Lymphoma, B-Cell - metabolism | Lymphopoiesis - ethics | Leukemia, B-Cell - pathology | Treatment Outcome | Cell Transformation, Neoplastic - metabolism | Animals | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Lymphoma, B-Cell - pathology | Agammaglobulinaemia Tyrosine Kinase - genetics | Protein Kinase Inhibitors - pharmacology | Care and treatment | Usage | Lymphomas | Research | Chemokines | Cancer | Bruton’s tyrosine kinase
Journal Article
Blood, ISSN 1528-0020, 2008, Volume 112, Issue 5, pp. 1600 - 1609
Journal Article
Immunological reviews, ISSN 0105-2896, 2011, Volume 244, Issue 1, pp. 115 - 133
Members of the tumor necrosis factor receptor superfamily (TNFRSF) participate prominently in B‐cell maturation and function. In particular, B‐cell activating... 
BAFF | B cell | CD40 | BCMA | NF‐κB | TACI | NF-κB | FORKHEAD TRANSCRIPTION FACTOR | SYSTEMIC-LUPUS-ERYTHEMATOSUS | HUMORAL IMMUNE-RESPONSES | COMMON VARIABLE IMMUNODEFICIENCY | IMMUNOLOGY | KINASE-C-DELTA | GERMINAL CENTER FORMATION | NF-?B | ACTIVATING FACTOR-RECEPTOR | NON-HODGKIN-LYMPHOMA | X-LINKED IMMUNODEFICIENCY | NF-KAPPA-B | Cell Proliferation | Humans | NF-kappa B - immunology | NF-kappa B - metabolism | Transmembrane Activator and CAML Interactor Protein - metabolism | Signal Transduction - immunology | CD40 Antigens - metabolism | B-Cell Activating Factor - immunology | B-Lymphocyte Subsets - immunology | CD40 Antigens - genetics | B-Cell Activation Factor Receptor - genetics | B-Lymphocytes - metabolism | B-Cell Activation Factor Receptor - immunology | B-Cell Maturation Antigen - immunology | Tumor Necrosis Factor Ligand Superfamily Member 13 - immunology | Gene Expression | Mice, Transgenic | Cell Survival - immunology | Immunity, Innate | Tumor Necrosis Factor Ligand Superfamily Member 13 - metabolism | Transmembrane Activator and CAML Interactor Protein - genetics | B-Cell Activation Factor Receptor - metabolism | Animals | B-Cell Activating Factor - metabolism | B-Cell Maturation Antigen - metabolism | B-Lymphocytes - immunology | B-Lymphocyte Subsets - metabolism | NF-kappa B - genetics | B-Cell Activating Factor - genetics | Neoplasms - immunology | Autoimmune Diseases | Tumor Necrosis Factor Ligand Superfamily Member 13 - genetics | Mice | B-Cell Maturation Antigen - genetics | Transmembrane Activator and CAML Interactor Protein - immunology | CD40 Antigens - immunology | Autoimmunity | Antigens | Tumor necrosis factor | Tumors | Necrosis
Journal Article
British Journal of Haematology, ISSN 0007-1048, 11/2016, Volume 175, Issue 4, pp. 631 - 640
Summary Rearrangement of MYC is associated with a poor prognosis in patients with diffuse large B cell lymphoma (DLBCL) and B cell lymphoma unclassifiable... 
fluorescence in situ hybridization | B cell lymphoma unclassifiable | diffuse large B cell lymphoma | chemotherapy | MYC | SURVIVAL | TRANSLOCATION | IMMUNOCHEMOTHERAPY | R-CHOP | CLASSIFICATION | TRANSPLANTATION | IMPACT | fluorescence insitu hybridization | BURKITT-LYMPHOMA | FEATURES INTERMEDIATE | HEMATOLOGY | EXPRESSION | Doxorubicin - therapeutic use | Prognosis | Humans | Lymphoma, B-Cell - diagnosis | Middle Aged | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Genes, myc | Lymphoma, B-Cell - genetics | Cyclophosphamide - adverse effects | Cyclophosphamide - therapeutic use | Adult | Female | Lymphoma, B-Cell - drug therapy | Lymphoma, B-Cell - mortality | Lymphoma, Large B-Cell, Diffuse - drug therapy | Prednisone - adverse effects | Proportional Hazards Models | Treatment Outcome | Lymphoma, Large B-Cell, Diffuse - mortality | Lymphoma, Large B-Cell, Diffuse - diagnosis | Gene Amplification | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Bone Marrow - pathology | Gene Rearrangement | Vincristine - therapeutic use | Biomarkers | Vincristine - adverse effects | Aged | Neoplasm Staging | Antibodies, Monoclonal, Murine-Derived - adverse effects | Lymphoma, Large B-Cell, Diffuse - genetics | Doxorubicin - adverse effects | Prednisone - therapeutic use | Antibodies, Monoclonal, Murine-Derived - therapeutic use | Lymphomas | Gene amplification | B cell lymphoma | CHEMOTHERAPY | Gene rearrangement
Journal Article
Nature (London), ISSN 1476-4687, 2018, Volume 560, Issue 7718, pp. 387 - 391
B cell receptor (BCR) signalling has emerged as a therapeutic target in B cell lymphomas, but inhibiting this pathway in diffuse large B cell lymphoma (DLBCL)... 
PATHOGENESIS | SURVIVAL | B-CELL LYMPHOMA | INHIBITION | MULTIDISCIPLINARY SCIENCES | KINASE | GENE-EXPRESSION | IBRUTINIB | TARGETS | REQUIRES | SUBGROUPS | Pyrazoles - therapeutic use | TOR Serine-Threonine Kinases - metabolism | Humans | Toll-Like Receptor 9 - genetics | NF-kappa B - metabolism | Lymphoma, Large B-Cell, Diffuse - metabolism | Receptors, Antigen, B-Cell - metabolism | TOR Serine-Threonine Kinases - antagonists & inhibitors | Multiprotein Complexes - metabolism | Drug Design | Female | Tumor Cells, Cultured | Pyrazoles - pharmacology | Lymphoma, Large B-Cell, Diffuse - pathology | Carcinogenesis - genetics | Myeloid Differentiation Factor 88 - genetics | Receptors, Antigen, B-Cell - antagonists & inhibitors | Signal Transduction - genetics | Pyrimidines - pharmacology | CRISPR-Cas Systems - genetics | Xenograft Model Antitumor Assays | Multiprotein Complexes - chemistry | Animals | Signal Transduction - drug effects | Biopsy | Pyrimidines - therapeutic use | Proteomics | Receptors, Antigen, B-Cell - genetics | Mice | Mutation | Toll-Like Receptor 9 - metabolism | Lymphoma, Large B-Cell, Diffuse - genetics | Myeloid Differentiation Factor 88 - metabolism | Lymphomas | Genetic aspects | B cells | Health aspects | TOR protein | Protein kinase C | Toxicity | Genomes | B-cell receptor | Kinases | Gene sequencing | Proteins | Signal transduction | Dependence | Toll-like receptors | CRISPR | NF-κB protein | Therapeutic applications | Autoantigens | Gene expression | Lymphoma | Bruton's tyrosine kinase | TLR9 protein | Signaling | Inhibitors | Lymphocytes B | Bcl-10 protein | MyD88 protein | Syk protein | Scientific imaging | Mass spectrometry | Cancer | Tumors | B-cell lymphoma | Life Sciences | Immunology
Journal Article