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Publication DateJournal of the National Cancer Institute, 04/2017, Volume 109, Issue 4
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Loading RightsGastrointestinal endoscopy, 01/2016, Volume 83, Issue 1, p. 274
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Loading RightsExpert Review of Clinical Immunology, ISSN 1744-666X, 2014, Volume 10, Issue 9, pp. 1171 - 1184
Neonates have little immunological memory and a developing immune system, which increases their vulnerability to infectious agents. Recent advances in the...
IL-27 macrophages | cord blood | RTE | TCR | T cell | Treg cell | neonates | chemokines | adaptive immunity | BCR | B cell | CD103 dendritic cell | innate immunity | CD5 cell | NK-cell | CD71 erythroid cell | toll-like receptor | γδ-T cell | T follicular cell | neutrophils | Th 17 | dendritic cell | B-reg cell | ERYTHROID-CELLS | IMMUNOLOGY | gamma delta-T cell | REGULATORY T-CELLS | HUMAN CORD BLOOD | RECENT THYMIC EMIGRANTS | TH1 RESPONSES | DENDRITIC CELLS | LOW EXPRESSION | HUMAN NEWBORNS | B-CELL | Adaptive Immunity | T-Lymphocyte Subsets - immunology | Infant, Newborn - immunology | Humans | Immune System - growth & development | Immunity, Innate | Animals | Immune System - immunology | Adult | B-Lymphocyte Subsets - immunology | Models, Animal | Mice | Animals, Newborn - immunology
IL-27 macrophages | cord blood | RTE | TCR | T cell | Treg cell | neonates | chemokines | adaptive immunity | BCR | B cell | CD103 dendritic cell | innate immunity | CD5 cell | NK-cell | CD71 erythroid cell | toll-like receptor | γδ-T cell | T follicular cell | neutrophils | Th 17 | dendritic cell | B-reg cell | ERYTHROID-CELLS | IMMUNOLOGY | gamma delta-T cell | REGULATORY T-CELLS | HUMAN CORD BLOOD | RECENT THYMIC EMIGRANTS | TH1 RESPONSES | DENDRITIC CELLS | LOW EXPRESSION | HUMAN NEWBORNS | B-CELL | Adaptive Immunity | T-Lymphocyte Subsets - immunology | Infant, Newborn - immunology | Humans | Immune System - growth & development | Immunity, Innate | Animals | Immune System - immunology | Adult | B-Lymphocyte Subsets - immunology | Models, Animal | Mice | Animals, Newborn - immunology
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Journal of Clinical Investigation, ISSN 0021-9738, 09/2017, Volume 127, Issue 9, pp. 3462 - 3471
BACKGROUND. Targeting CD30 with monoclonal antibodies in Hodgkin lymphoma (HL) and anaplastic large cell lymphoma (ALCL) has had profound clinical success....
MEDICINE, RESEARCH & EXPERIMENTAL | SOLUBLE CD30 | THERAPY | B-CELL | CYTOKINE RELEASE SYNDROME | DISEASE | BRENTUXIMAB VEDOTIN | CLASSICAL HODGKINS LYMPHOMA | STEM-CELL TRANSPLANTATION | ANTITUMOR-ACTIVITY | CYTOTOXIC T-LYMPHOCYTES | Lymphoma, Large-Cell, Anaplastic - immunology | Receptors, Antigen, T-Cell - chemistry | Lymphoma, Large-Cell, Anaplastic - therapy | Humans | Middle Aged | Neoplasm Recurrence, Local | Immunophenotyping | Male | Treatment Outcome | Antineoplastic Agents - chemistry | Ki-1 Antigen - metabolism | Molecular Targeted Therapy | Immunoconjugates - administration & dosage | Disease Progression | Dose-Response Relationship, Drug | Young Adult | T-Lymphocytes - cytology | Hodgkin Disease - therapy | Immunoconjugates - therapeutic use | Adult | Female | Hodgkin Disease - immunology | CD28 Antigens - chemistry | Transplantation Conditioning | Antigens | Lymphocytes | Health aspects | Immunoglobulins | Leukemia | T cell receptors | Lymphocytes T | Malignancy | CD28 antigen | Kinases | Patients | Cancer therapies | Chemotherapy | CD30 antigen | Immunotherapy | Peripheral blood | Monoclonal antibodies | Antitumor activity | Lymphomas | Anaplastic large-cell lymphoma | Drug dosages | Hodgkin's disease | Index Medicus | Abridged Index Medicus
MEDICINE, RESEARCH & EXPERIMENTAL | SOLUBLE CD30 | THERAPY | B-CELL | CYTOKINE RELEASE SYNDROME | DISEASE | BRENTUXIMAB VEDOTIN | CLASSICAL HODGKINS LYMPHOMA | STEM-CELL TRANSPLANTATION | ANTITUMOR-ACTIVITY | CYTOTOXIC T-LYMPHOCYTES | Lymphoma, Large-Cell, Anaplastic - immunology | Receptors, Antigen, T-Cell - chemistry | Lymphoma, Large-Cell, Anaplastic - therapy | Humans | Middle Aged | Neoplasm Recurrence, Local | Immunophenotyping | Male | Treatment Outcome | Antineoplastic Agents - chemistry | Ki-1 Antigen - metabolism | Molecular Targeted Therapy | Immunoconjugates - administration & dosage | Disease Progression | Dose-Response Relationship, Drug | Young Adult | T-Lymphocytes - cytology | Hodgkin Disease - therapy | Immunoconjugates - therapeutic use | Adult | Female | Hodgkin Disease - immunology | CD28 Antigens - chemistry | Transplantation Conditioning | Antigens | Lymphocytes | Health aspects | Immunoglobulins | Leukemia | T cell receptors | Lymphocytes T | Malignancy | CD28 antigen | Kinases | Patients | Cancer therapies | Chemotherapy | CD30 antigen | Immunotherapy | Peripheral blood | Monoclonal antibodies | Antitumor activity | Lymphomas | Anaplastic large-cell lymphoma | Drug dosages | Hodgkin's disease | Index Medicus | Abridged Index Medicus
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Loading RightsAnnual Review of Immunology, ISSN 0732-0582, 2009, Volume 27, Issue 1, pp. 393 - 422
Tuberculosis is primarily a disease of the lung, and dissemination of the disease depends on productive infection of this critical organ. Upon aerosol...
B cell | Inflammation | Regulation | Memory | T cell | Phagocyte | memory | VACCINIA VIRUS ANKARA | DENDRITIC CELLS | IFN-GAMMA | MYCOBACTERIUM-BOVIS BCG | CALMETTE-GUERIN VACCINATION | IMMUNOLOGY | phagocyte | TOLL-LIKE RECEPTOR-2 | C-TYPE LECTINS | regulation | inflammation | T-REGULATORY CELLS | IN-VIVO | CHRONIC PULMONARY TUBERCULOSIS | T-Lymphocyte Subsets - immunology | B-Lymphocytes - microbiology | Cytokines - metabolism | Receptors, Pattern Recognition - immunology | Humans | Tuberculosis - microbiology | Mycobacterium tuberculosis - pathogenicity | Tuberculosis - immunology | Immunity, Cellular - immunology | Receptors, Pattern Recognition - metabolism | Macrophages - metabolism | Animals | B-Lymphocytes - immunology | T-Lymphocyte Subsets - metabolism | Immunologic Memory | T-Lymphocyte Subsets - microbiology | Tuberculosis - metabolism | B-Lymphocytes - metabolism | Macrophages - immunology | Macrophages - microbiology | Cytokines - immunology | Physiological aspects | Development and progression | Tuberculosis | Immune response | Properties | Immunological research
B cell | Inflammation | Regulation | Memory | T cell | Phagocyte | memory | VACCINIA VIRUS ANKARA | DENDRITIC CELLS | IFN-GAMMA | MYCOBACTERIUM-BOVIS BCG | CALMETTE-GUERIN VACCINATION | IMMUNOLOGY | phagocyte | TOLL-LIKE RECEPTOR-2 | C-TYPE LECTINS | regulation | inflammation | T-REGULATORY CELLS | IN-VIVO | CHRONIC PULMONARY TUBERCULOSIS | T-Lymphocyte Subsets - immunology | B-Lymphocytes - microbiology | Cytokines - metabolism | Receptors, Pattern Recognition - immunology | Humans | Tuberculosis - microbiology | Mycobacterium tuberculosis - pathogenicity | Tuberculosis - immunology | Immunity, Cellular - immunology | Receptors, Pattern Recognition - metabolism | Macrophages - metabolism | Animals | B-Lymphocytes - immunology | T-Lymphocyte Subsets - metabolism | Immunologic Memory | T-Lymphocyte Subsets - microbiology | Tuberculosis - metabolism | B-Lymphocytes - metabolism | Macrophages - immunology | Macrophages - microbiology | Cytokines - immunology | Physiological aspects | Development and progression | Tuberculosis | Immune response | Properties | Immunological research
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Loading RightsImmunity, ISSN 1074-7613, 10/2013, Volume 39, Issue 4, pp. 758 - 769
The vast majority of currently licensed human vaccines work on the basis of long-term protective antibody responses. It is now conceivable that an...
B-CELLS | T-FOLLICULAR HELPER | COMMITMENT | BCL6 | PROTECTION | DYNAMICS | REGULATORS | IMMUNOLOGY | EXPRESSION | PROGRAM | LYMPHOCYTES | Germinal Center - immunology | Humans | Molecular Sequence Data | Gene Expression Profiling | T-Lymphocytes, Helper-Inducer - virology | HIV Infections - immunology | HIV Infections - pathology | Immunity, Humoral | T-Lymphocytes, Helper-Inducer - immunology | B-Lymphocytes - virology | HIV Antibodies - biosynthesis | T-Lymphocytes, Helper-Inducer - pathology | B-Lymphocytes - pathology | Amino Acid Sequence | Gene Expression | Receptors, CXCR5 - genetics | Signal Transduction | HIV Infections - virology | Germinal Center - pathology | Receptors, CXCR5 - immunology | HIV-1 - immunology | B-Lymphocytes - immunology | Antibodies, Neutralizing - biosynthesis | Germinal Center - virology | Receptors, CXCR3 - genetics | Receptors, CXCR3 - immunology | Immunologic Memory | Programmed Cell Death 1 Receptor - immunology | Programmed Cell Death 1 Receptor - genetics | Antigens | Hypotheses | Acquired immune deficiency syndrome--AIDS | Lymphocytes | Infections | Vaccines | Mutation | Gene expression | Viral infections | Immune system
B-CELLS | T-FOLLICULAR HELPER | COMMITMENT | BCL6 | PROTECTION | DYNAMICS | REGULATORS | IMMUNOLOGY | EXPRESSION | PROGRAM | LYMPHOCYTES | Germinal Center - immunology | Humans | Molecular Sequence Data | Gene Expression Profiling | T-Lymphocytes, Helper-Inducer - virology | HIV Infections - immunology | HIV Infections - pathology | Immunity, Humoral | T-Lymphocytes, Helper-Inducer - immunology | B-Lymphocytes - virology | HIV Antibodies - biosynthesis | T-Lymphocytes, Helper-Inducer - pathology | B-Lymphocytes - pathology | Amino Acid Sequence | Gene Expression | Receptors, CXCR5 - genetics | Signal Transduction | HIV Infections - virology | Germinal Center - pathology | Receptors, CXCR5 - immunology | HIV-1 - immunology | B-Lymphocytes - immunology | Antibodies, Neutralizing - biosynthesis | Germinal Center - virology | Receptors, CXCR3 - genetics | Receptors, CXCR3 - immunology | Immunologic Memory | Programmed Cell Death 1 Receptor - immunology | Programmed Cell Death 1 Receptor - genetics | Antigens | Hypotheses | Acquired immune deficiency syndrome--AIDS | Lymphocytes | Infections | Vaccines | Mutation | Gene expression | Viral infections | Immune system
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Loading RightsFEBS Journal, ISSN 1742-464X, 02/2016, Volume 283, Issue 3, pp. 413 - 424
Hypoxia and inflammation have been associated with a number of pathological conditions, in particular inflammatory diseases. While hypoxia is mainly associated...
hypoxia | macrophages | HIF | immune response | inflammation | IKK | κB | B cells | T cells | neutrophils | NF-κB | Immune response | Neutrophils | Hypoxia | Inflammation | Macrophages | ACTIVATION | CELL-DEVELOPMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-I | NF-kappa B | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | SIGNALING PATHWAY | FACTOR 2-ALPHA | GENE-EXPRESSION | T-CELLS | INNATE IMMUNITY | T-Lymphocyte Subsets - immunology | Humans | Neutrophils - immunology | Immunity - immunology | NF-kappa B - metabolism | Hypoxia-Inducible Factor 1 - metabolism | Macrophages - metabolism | Animals | B-Lymphocyte Subsets - metabolism | T-Lymphocyte Subsets - metabolism | B-Lymphocyte Subsets - immunology | Neutrophils - metabolism | Macrophages - immunology | State‐of‐the‐Art Review | NF‐κB | State‐of‐the‐Art Reviews
hypoxia | macrophages | HIF | immune response | inflammation | IKK | κB | B cells | T cells | neutrophils | NF-κB | Immune response | Neutrophils | Hypoxia | Inflammation | Macrophages | ACTIVATION | CELL-DEVELOPMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-I | NF-kappa B | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | SIGNALING PATHWAY | FACTOR 2-ALPHA | GENE-EXPRESSION | T-CELLS | INNATE IMMUNITY | T-Lymphocyte Subsets - immunology | Humans | Neutrophils - immunology | Immunity - immunology | NF-kappa B - metabolism | Hypoxia-Inducible Factor 1 - metabolism | Macrophages - metabolism | Animals | B-Lymphocyte Subsets - metabolism | T-Lymphocyte Subsets - metabolism | B-Lymphocyte Subsets - immunology | Neutrophils - metabolism | Macrophages - immunology | State‐of‐the‐Art Review | NF‐κB | State‐of‐the‐Art Reviews
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Loading RightsScience, ISSN 0036-8075, 3/2009, Volume 323, Issue 5922, pp. 1722 - 1725
Patten recognition receptors, which recognize pathogens or components of injured cells (danger), trigger activation of the innate immune system. Whether and...
Receptors | Immune response | Cytokines | Hepatocytes | Splenocytes | Liver | Antibodies | Heat shock proteins | Ligands | Reports | Physiological regulation | SYSTEM | ACTIVATION | PROTEIN | AUTOIMMUNITY | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOBILITY GROUP BOX-1 | HMGB1 | HEAT-STABLE ANTIGEN | T-CELL GROWTH | ISCHEMIA-REPERFUSION | Liver - pathology | Immunoprecipitation | Receptors, Pattern Recognition - immunology | CD24 Antigen - metabolism | Dendritic Cells - immunology | Humans | HMGB1 Protein - immunology | Receptors, Antigen, B-Cell - metabolism | Necrosis - immunology | Lectins - metabolism | Receptors, Pattern Recognition - metabolism | Liver - immunology | HMGB1 Protein - chemistry | HMGB1 Protein - metabolism | CD24 Antigen - genetics | Protein Structure, Tertiary | Lipopolysaccharides - toxicity | Cytokines - metabolism | Signal Transduction | Acetaminophen - toxicity | Receptors, Cell Surface - metabolism | Mutant Proteins - metabolism | Inflammation - immunology | Immunity, Innate | HSP70 Heat-Shock Proteins - metabolism | Necrosis - chemically induced | Animals | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - metabolism | Transcription Factor RelA - metabolism | Mutant Proteins - chemistry | HSP90 Heat-Shock Proteins - metabolism | Mice | Research | Properties | Cell adhesion molecules | Proteins | Immunology | Cellular biology | Molecular biology | Rodents | Index Medicus
Receptors | Immune response | Cytokines | Hepatocytes | Splenocytes | Liver | Antibodies | Heat shock proteins | Ligands | Reports | Physiological regulation | SYSTEM | ACTIVATION | PROTEIN | AUTOIMMUNITY | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOBILITY GROUP BOX-1 | HMGB1 | HEAT-STABLE ANTIGEN | T-CELL GROWTH | ISCHEMIA-REPERFUSION | Liver - pathology | Immunoprecipitation | Receptors, Pattern Recognition - immunology | CD24 Antigen - metabolism | Dendritic Cells - immunology | Humans | HMGB1 Protein - immunology | Receptors, Antigen, B-Cell - metabolism | Necrosis - immunology | Lectins - metabolism | Receptors, Pattern Recognition - metabolism | Liver - immunology | HMGB1 Protein - chemistry | HMGB1 Protein - metabolism | CD24 Antigen - genetics | Protein Structure, Tertiary | Lipopolysaccharides - toxicity | Cytokines - metabolism | Signal Transduction | Acetaminophen - toxicity | Receptors, Cell Surface - metabolism | Mutant Proteins - metabolism | Inflammation - immunology | Immunity, Innate | HSP70 Heat-Shock Proteins - metabolism | Necrosis - chemically induced | Animals | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - metabolism | Transcription Factor RelA - metabolism | Mutant Proteins - chemistry | HSP90 Heat-Shock Proteins - metabolism | Mice | Research | Properties | Cell adhesion molecules | Proteins | Immunology | Cellular biology | Molecular biology | Rodents | Index Medicus
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Loading RightsJournal of Autoimmunity, ISSN 0896-8411, 2014, Volume 50, Issue 100, pp. 107 - 122
Abstract Regulatory B cells (Breg) have attracted increasing attention for their roles in maintaining peripheral tolerance. Interleukin 33 (IL-33) is a...
Allergy and Immunology | Regulatory B cell | IBD | IL-33 | Mucosal inflammation | IL-10 | Breg | IMMUNE-RESPONSE | DENDRITIC CELLS | AMELIORATES EXPERIMENTAL COLITIS | CD4(+) T-CELLS | INTERLEUKIN-33 | ALLERGIC AIRWAY INFLAMMATION | IMMUNOLOGY | INTERLEUKIN-10-DEFICIENT MICE | BOWEL-DISEASE | INTESTINAL INFLAMMATION | ST2 RECEPTOR | Antigens, CD - immunology | Colitis - genetics | Colitis - pathology | Gastric Mucosa - pathology | Adoptive Transfer | Antigens, CD - genetics | B-Lymphocytes, Regulatory - transplantation | Interleukins - immunology | Gastric Mucosa - drug effects | Female | Interleukins - pharmacology | Colitis - immunology | Gene Expression | Interleukin-10 - deficiency | Interleukin-33 | Lymphocyte Activation | Injections, Intraperitoneal | B-Lymphocytes, Regulatory - immunology | Mice, Knockout | Gastric Mucosa - immunology | Animals | Interleukin-10 - genetics | Mice | B-Lymphocytes, Regulatory - drug effects | Interleukin-10 - immunology | Immunoglobulin E | Interleukins | Health aspects | Gastrointestinal diseases | Breg, regulatory B cell | IBD, inflammatory bowel disease | expanded Breg | BregIL-33, IL-33-induced
Allergy and Immunology | Regulatory B cell | IBD | IL-33 | Mucosal inflammation | IL-10 | Breg | IMMUNE-RESPONSE | DENDRITIC CELLS | AMELIORATES EXPERIMENTAL COLITIS | CD4(+) T-CELLS | INTERLEUKIN-33 | ALLERGIC AIRWAY INFLAMMATION | IMMUNOLOGY | INTERLEUKIN-10-DEFICIENT MICE | BOWEL-DISEASE | INTESTINAL INFLAMMATION | ST2 RECEPTOR | Antigens, CD - immunology | Colitis - genetics | Colitis - pathology | Gastric Mucosa - pathology | Adoptive Transfer | Antigens, CD - genetics | B-Lymphocytes, Regulatory - transplantation | Interleukins - immunology | Gastric Mucosa - drug effects | Female | Interleukins - pharmacology | Colitis - immunology | Gene Expression | Interleukin-10 - deficiency | Interleukin-33 | Lymphocyte Activation | Injections, Intraperitoneal | B-Lymphocytes, Regulatory - immunology | Mice, Knockout | Gastric Mucosa - immunology | Animals | Interleukin-10 - genetics | Mice | B-Lymphocytes, Regulatory - drug effects | Interleukin-10 - immunology | Immunoglobulin E | Interleukins | Health aspects | Gastrointestinal diseases | Breg, regulatory B cell | IBD, inflammatory bowel disease | expanded Breg | BregIL-33, IL-33-induced
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Loading RightsCurrent Opinion in Immunology, ISSN 0952-7915, 2016, Volume 39, pp. 59 - 67
Highlights • Competition for Tfh drives GC B cell proliferation, mutation and affinity maturation. • Relationship between IgA memory, the microbiota and...
Allergy and Immunology | IMMUNE-RESPONSE | MAINTENANCE | IMMUNOLOGICAL MEMORY | FOLLICULAR-HELPER-CELLS | BONE-MARROW | DYNAMICS | BCL6 EXPRESSION | DIFFERENTIATION | IMMUNOLOGY | ANTIBODY-RESPONSES | T-CELLS | Animals | B-Lymphocytes - immunology | Immunity, Humoral | Germinal Center - immunology | Humans | Antibody Formation | Immunologic Memory | Lymphocyte Activation - physiology | Plasma Cells - immunology | Immunoglobulin A | Epigenetic inheritance | B cells | Genetics | Microbiota (Symbiotic organisms) | T cells
Allergy and Immunology | IMMUNE-RESPONSE | MAINTENANCE | IMMUNOLOGICAL MEMORY | FOLLICULAR-HELPER-CELLS | BONE-MARROW | DYNAMICS | BCL6 EXPRESSION | DIFFERENTIATION | IMMUNOLOGY | ANTIBODY-RESPONSES | T-CELLS | Animals | B-Lymphocytes - immunology | Immunity, Humoral | Germinal Center - immunology | Humans | Antibody Formation | Immunologic Memory | Lymphocyte Activation - physiology | Plasma Cells - immunology | Immunoglobulin A | Epigenetic inheritance | B cells | Genetics | Microbiota (Symbiotic organisms) | T cells
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Loading RightsImmunity, ISSN 1074-7613, 08/2018, Volume 49, Issue 2, pp. 211 - 224
Various immune mechanisms are deployed in the mucosa to confront the immense diversity of resident bacteria. A substantial fraction of the commensal microbiota...
FC-ALPHA-RI | MEMORY B-CELLS | CLASS SWITCH RECOMBINATION | FOXP3(+) T-CELLS | PEYERS-PATCHES | IMMUNOLOGY | GUT IGA | MUCOSAL IMMUNE-SYSTEM | SEGMENTED FILAMENTOUS BACTERIA | PLASMA-CELLS | SECRETORY IMMUNOGLOBULIN-A | Bacteria | Microbiota (Symbiotic organisms) | T cells | Immunoglobulin A | Analysis | Antigens | Immunoglobulins | Mucosa | Antibodies | Homeostasis | Lymphocytes T | Vaccines | Mammals | Immunity | Reptiles & amphibians | Microbiota | Immunology | Lymphocytes | Rodents | Immunity (humoral) | Immune system
FC-ALPHA-RI | MEMORY B-CELLS | CLASS SWITCH RECOMBINATION | FOXP3(+) T-CELLS | PEYERS-PATCHES | IMMUNOLOGY | GUT IGA | MUCOSAL IMMUNE-SYSTEM | SEGMENTED FILAMENTOUS BACTERIA | PLASMA-CELLS | SECRETORY IMMUNOGLOBULIN-A | Bacteria | Microbiota (Symbiotic organisms) | T cells | Immunoglobulin A | Analysis | Antigens | Immunoglobulins | Mucosa | Antibodies | Homeostasis | Lymphocytes T | Vaccines | Mammals | Immunity | Reptiles & amphibians | Microbiota | Immunology | Lymphocytes | Rodents | Immunity (humoral) | Immune system
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