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Nature communications, ISSN 2041-1723, 11/2018, Volume 9, Issue 1, pp. 4822 - 12
Pathogen immune responses are profoundly attenuated in fetuses and premature infants, yet the mechanisms underlying this developmental immaturity remain... 
SYSTEMATIC ANALYSIS | PHAGOCYTOSIS | CANDIDA-ALBICANS | INTERFERON-GAMMA | RECOGNITION | NEWBORN | MULTIDISCIPLINARY SCIENCES | PRETERM | HUMAN MONOCYTES | UNDER-5 MORTALITY | DECTIN-1 | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - deficiency | Monocytes - cytology | Humans | Transcription Factors - deficiency | Tumor Necrosis Factor-alpha - genetics | Lectins, C-Type - immunology | Protein Biosynthesis - immunology | CARD Signaling Adaptor Proteins - deficiency | PPAR gamma - immunology | Monocytes - immunology | Transcriptome - immunology | Tumor Necrosis Factor-alpha - deficiency | Lectins, C-Type - genetics | TOR Serine-Threonine Kinases - deficiency | CARD Signaling Adaptor Proteins - genetics | Interleukins - genetics | TOR Serine-Threonine Kinases - genetics | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - immunology | Gene Expression Regulation, Developmental | Microarray Analysis | Interleukins - immunology | PPAR gamma - deficiency | Tumor Necrosis Factor-alpha - immunology | Candida parapsilosis - immunology | Adult | Lectins, C-Type - deficiency | Transcription Factors - immunology | Infant, Newborn | PPAR gamma - genetics | B-Cell CLL-Lymphoma 10 Protein - genetics | CARD Signaling Adaptor Proteins - immunology | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - genetics | B-Cell CLL-Lymphoma 10 Protein - immunology | Transcription Factors - genetics | Candida albicans - immunology | Immunity, Innate | B-Cell CLL-Lymphoma 10 Protein - deficiency | Monocytes - drug effects | TOR Serine-Threonine Kinases - immunology | Infant, Premature | Lipopolysaccharides - pharmacology | Interleukins - deficiency | Primary Cell Culture | TOR protein | Neonates | Pathogens | Immune response | Transcription | Cytokines | Fetuses | DNA damage | Ontogeny | Infants | Gestation | Metabolism | Gene expression | Monocytes | Glycolysis | Metabolic pathways | Peroxisome proliferator-activated receptors | Human behavior | Deoxyribonucleic acid--DNA | Phagocytosis | Immune system
Journal Article
Cellular Microbiology, ISSN 1462-5814, 01/2018, Volume 20, Issue 1, pp. e12788 - n/a
The neutrophil‐specific innate immune receptor CEACAM3 functions as a decoy to capture Gram‐negative pathogens, such as Neisseria gonorrhoeae, that exploit... 
CEACAM3 | Bcl10 | Neisseria gonorrhoeae | neutrophil | TLR4 | Malt1 | INDUCED ACTIVATION | HUMAN NEUTROPHILS | INNATE | MICROBIOLOGY | CELL BIOLOGY | BACTERIAL PATHOGENS | EPITHELIAL-CELLS | CELL ANTIGEN RECEPTOR | MOUSE MODEL | TOLL-LIKE RECEPTORS | NEISSERIA-GONORRHOEAE | NF-KAPPA-B | Antigens, CD - immunology | B-Cell CLL-Lymphoma 10 Protein - genetics | Gonorrhea - microbiology | GPI-Linked Proteins - immunology | Carcinoembryonic Antigen - immunology | Gonorrhea - immunology | Humans | Mice, Inbred C57BL | Cells, Cultured | Neutrophils - immunology | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - genetics | Toll-Like Receptor 4 - immunology | B-Cell CLL-Lymphoma 10 Protein - immunology | Cell Adhesion Molecules - immunology | rac GTP-Binding Proteins - immunology | Mice, Knockout | Animals | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - immunology | rac GTP-Binding Proteins - genetics | Neisseria gonorrhoeae - immunology | Mice | Bacterial infections | B cells | Bifurcations | Inflammatory response | Proteins | Receptors | Cell activation | Toll-like receptors | Bacteria | Immune system | NF-κB protein | Pathogens | Integration | Cytokines | Neutrophils | Crosslinking | Leukocytes (neutrophilic) | Inflammation | T cell receptors | Pattern recognition | TLR4 protein | Epithelium | Rac2 protein | Signaling | Lymphocytes B | Effectors | Bcl-10 protein | Human behavior
Journal Article
Immunity, ISSN 1074-7613, 05/2014, Volume 40, Issue 5, pp. 692 - 705
Glutamine has been implicated as an immunomodulatory nutrient, but how glutamine uptake is mediated during T cell activation is poorly understood. We have... 
MAMMALIAN TARGET | RAPAMYCIN | EFFECTOR | METABOLISM | PATHWAY | C-MYC | DIFFERENTIATION | IMMUNOLOGY | GLUCOSE-UPTAKE | NF-KAPPA-B | TRANSLATIONAL CONTROL | TOR Serine-Threonine Kinases - metabolism | Minor Histocompatibility Antigens | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Glutamine - metabolism | Adoptive Transfer | Neoplasm Proteins - metabolism | Th1 Cells - immunology | Enzyme Activation - immunology | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Signal Transduction - immunology | CARD Signaling Adaptor Proteins - metabolism | Lymphocyte Activation - immunology | Caspases - metabolism | Biological Transport | Receptors, Antigen, T-Cell - immunology | Amino Acid Transport System ASC - genetics | Leucine - metabolism | Encephalomyelitis, Autoimmune, Experimental - pathology | Amino Acid Transport System ASC - immunology | Jurkat Cells | Mice, Inbred C57BL | Amino Acid Transport System ASC - metabolism | CD28 Antigens - immunology | Inflammation - immunology | Mice, Knockout | Cell Differentiation - immunology | Animals | B-Cell CLL-Lymphoma 10 Protein | Mice | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein | Adaptor Proteins, Signal Transducing - metabolism | Interleukin-2 - biosynthesis | Autoimmunity | T cells | Analysis | Glutamine | Cytokines | Sucrose | Colleges & universities | Homeostasis | Amino acids | T cell receptors | Glucose | Kinases | Proteins | Cell growth | Lymphocytes | Immune system | Cancer | ASCT2 | CARMA1 | TCR signaling | EAE | T-cell differentiation | mTORC1 | Glutamine uptake
Journal Article
Nature, ISSN 0028-0836, 08/2006, Volume 442, Issue 7103, pp. 651 - 656
Journal Article
Immunity, ISSN 1074-7613, 06/2012, Volume 36, Issue 6, pp. 947 - 958
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 05/2014, Volume 5, Issue 1, p. 3755
A variety of reactive organic compounds, called haptens, can cause allergic contact dermatitis. However, the innate immune mechanisms by which haptens... 
DAMAGED CELLS | CANDIDA-ALBICANS | MULTIDISCIPLINARY SCIENCES | NLRP3 INFLAMMASOME ACTIVATION | IN-VIVO | IL-17-PRODUCING T-CELLS | INNATE IMMUNE-RESPONSE | TOLL-LIKE RECEPTORS | HOST-DEFENSE | NALP3 INFLAMMASOME | LANGERHANS CELLS | Receptors, Interleukin-1 Type I - immunology | NLR Family, Pyrin Domain-Containing 3 Protein | Dendritic Cells - immunology | Caspase 1 - metabolism | Intracellular Signaling Peptides and Proteins - immunology | Enzyme Activation - immunology | Protein-Tyrosine Kinases - immunology | CARD Signaling Adaptor Proteins - genetics | Interleukin-1 - biosynthesis | Signal Transduction - immunology | Dermatitis, Contact - immunology | Protein-Tyrosine Kinases - genetics | Receptors, Interleukin-1 Type I - antagonists & inhibitors | Reactive Oxygen Species - immunology | Myeloid Differentiation Factor 88 - immunology | Adaptor Proteins, Signal Transducing - immunology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | CARD Signaling Adaptor Proteins - immunology | Mice, Inbred C57BL | Myeloid Differentiation Factor 88 - genetics | Signal Transduction - genetics | Mice, Knockout | Carrier Proteins - genetics | Animals | B-Cell CLL-Lymphoma 10 Protein | Inflammasomes - immunology | Adaptor Proteins, Signal Transducing - genetics | Immunoreceptor Tyrosine-Based Activation Motif - immunology | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | Interleukin-1 - secretion
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 134, Issue 2, pp. 276 - 284
Next-generation DNA sequencing has accelerated the genetic characterization of many human primary immunodeficiency diseases (PIDs). These discoveries can be... 
Allergy and Immunology | nuclear factor κB | next-generation sequencing | CARMA1 | primary immunodeficiency diseases | CARD11-BCL10-MALT1 signalosome complex | paracaspase | congenital B-cell lymphocytosis | combined immunodeficiency | nuclear factor kappa B | IMMUNE-DEFICIENCY | T-CELL | BCL10 | IMMUNOLOGY | CLEAVAGE | MALT LYMPHOMA | JNK ACTIVATION | ALLERGY | ECTODERMAL DYSPLASIA | CARD11 MUTATIONS | NF-KAPPA-B | Immunologic Deficiency Syndromes - pathology | Caspases - immunology | Humans | NF-kappa B - immunology | Neoplasm Proteins - immunology | CARD Signaling Adaptor Proteins - genetics | Adaptor Proteins, Signal Transducing - immunology | Germ-Line Mutation | B-Lymphocytes - pathology | Immunologic Deficiency Syndromes - diagnosis | Immunologic Deficiency Syndromes - immunology | Neoplasm Proteins - genetics | CARD Signaling Adaptor Proteins - immunology | Guanylate Cyclase - immunology | Signal Transduction | Caspases - genetics | Gene Expression Regulation | Receptors, Antigen, B-Cell - immunology | B-Lymphocytes - immunology | NF-kappa B - genetics | B-Cell CLL-Lymphoma 10 Protein | Adaptor Proteins, Signal Transducing - genetics | Immunologic Deficiency Syndromes - genetics | Guanylate Cyclase - genetics | Receptors, Antigen, B-Cell - genetics | High-Throughput Nucleotide Sequencing | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein | Lymphomas | Immunodeficiency | Primary immunodeficiency diseases | Congenital B cell lymphocytosis | NF-κB | Combined immunodeficiency | CARD11–BCL10–MALT1 (CBM) signalosome complex | Paracaspase | Next generation sequencing
Journal Article
Journal Article