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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2016, Volume 113, Issue 6, pp. E782 - E790
Epstein–Barr virus (EBV) is an oncogenic herpesvirus that has been causally linked to the development of B-cell and epithelial malignancies. Early after... 
B cell | Epstein-Barr virus | Metabolism | Oncogene-induced senescence | Autophagy | TRANSFORMATION | SURVIVAL | autophagy | DNA-DAMAGE RESPONSE | MULTIDISCIPLINARY SCIENCES | CANCER | REPLICATION | SIGNALING PATHWAY | metabolism | TUMORIGENESIS | oncogene-induced senescence | LYMPHOCYTES | Metabolomics | Cell Transformation, Viral - drug effects | TOR Serine-Threonine Kinases - metabolism | DNA Replication - drug effects | Humans | Herpesvirus 4, Human - drug effects | Cellular Senescence - drug effects | Dimethylformamide - pharmacology | Autophagy - drug effects | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Deoxyglucose - pharmacology | Cell Respiration - drug effects | B-Lymphocytes - virology | Stress, Physiological - drug effects | B-Lymphocytes - pathology | B-Lymphocytes - ultrastructure | Oncogenes | DNA Damage - drug effects | Herpesvirus 4, Human - physiology | Tumor Suppressor Protein p53 - metabolism | Mitochondria - metabolism | Transcriptome - genetics | Mitochondria - drug effects | Transcription Factors - metabolism | B-Lymphocytes - drug effects | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Cell Proliferation - drug effects | Cell interaction | B cells | Host-virus relationships | Observations | Health aspects | Biological Sciences | PNAS Plus | Epstein–Barr virus
Journal Article
Seminars in Hematology, ISSN 0037-1963, 2010, Volume 47, Issue 2, pp. 115 - 123
Journal Article
Journal of clinical oncology, ISSN 1527-7755, 2015, Volume 33, Issue 25, pp. 2803 - 2811
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2011, Volume 108, Issue 50, pp. 20119 - 20124
Natural killer cell lymphoma (NKCL) constitutes a rare and aggressive form of non-Hodgkin lymphoma, and there is little insight into its pathogenesis. Here we... 
B lymphocytes | T cell lymphoma | B cell lymphoma | Cell lines | Cell cycle | Tumor suppressor genes | Natural killer cells | Methylation | Tumors | Apoptosis | Neoplastic transformation | NK-cell activation and homeostasis | Biotage pyrosequencing | CCNG2 | CCNG1 | PERIPHERAL T | LYMPHOMA/LEUKEMIA | biotage pyrosequencing | MULTIDISCIPLINARY SCIENCES | neoplastic transformation | TRANSCRIPTIONAL REPRESSOR BLIMP-1 | T-CELL | IDENTIFICATION | CONSISTENT PATTERNS | LYMPHOMA | DIFFERENTIATION | COMPARATIVE GENOMIC HYBRIDIZATION | LYMPHOCYTES | G2 Phase - genetics | Gene Silencing - drug effects | Apoptosis - drug effects | Humans | Killer Cells, Natural - pathology | Promoter Regions, Genetic - genetics | G2 Phase - drug effects | Lymphoma, Non-Hodgkin - pathology | Gene Knockdown Techniques | Time Factors | DNA Mutational Analysis | Tumor Suppressor Proteins - genetics | DNA Copy Number Variations - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Repressor Proteins - metabolism | Cell Division - genetics | Interleukin-2 - metabolism | Tumor Suppressor Proteins - metabolism | Transduction, Genetic | Repressor Proteins - genetics | DNA Copy Number Variations - genetics | DNA Methylation - genetics | Lymphoma, Non-Hodgkin - genetics | Cell Division - drug effects | Biopsy | Culture Media - pharmacology | Interleukin-2 - pharmacology | Killer Cells, Natural - drug effects | Killer Cells, Natural - metabolism | DNA Methylation - drug effects | Positive Regulatory Domain I-Binding Factor 1 | RNA, Small Interfering - metabolism | Development and progression | Killer cells | Genetic aspects | Non-Hodgkin's lymphomas | Health aspects | Life Sciences | Biochemistry, Molecular Biology | Biological Sciences
Journal Article
Blood, ISSN 1528-0020, 2017, Volume 129, Issue 6, pp. 729 - 739
Antibiotics cause multilineage alterations in murine hematopoiesis, with marked suppression of multipotent progenitors. Suppression of bone marrow populations... 
GRANULOPOIESIS | STEM-CELLS | HEMATOLOGY | NEUTROPENIA | T-CELLS | DISEASE | CD8-Positive T-Lymphocytes - pathology | STAT1 Transcription Factor - deficiency | Hematopoietic Stem Cells - pathology | Anemia - pathology | Gastrointestinal Microbiome - physiology | Leukopenia - therapy | Hematopoiesis - drug effects | Granulocytes - drug effects | Leukopenia - chemically induced | CD8-Positive T-Lymphocytes - metabolism | Thrombocytosis - microbiology | Bone Marrow Cells - drug effects | Granulocytes - pathology | Anti-Bacterial Agents - adverse effects | B-Lymphocytes - pathology | Granulocytes - metabolism | Thrombocytosis - pathology | B-Lymphocytes - metabolism | Hematopoietic Stem Cells - drug effects | Gene Expression | Leukopenia - microbiology | Macrophages - pathology | Signal Transduction | Germ-Free Life - genetics | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Anemia - chemically induced | Anemia - therapy | Hematopoiesis - genetics | Hematopoietic Stem Cells - metabolism | Germ-Free Life - drug effects | STAT1 Transcription Factor - genetics | Leukopenia - pathology | Fecal Microbiota Transplantation | Thrombocytosis - chemically induced | Mice, Knockout | B-Lymphocytes - drug effects | Macrophages - metabolism | Animals | Gastrointestinal Microbiome - drug effects | CD8-Positive T-Lymphocytes - drug effects | Macrophages - drug effects | Mice | Anemia - microbiology | Thrombocytosis - therapy | Bone Marrow Cells - metabolism | Hematopoiesis and Stem Cells
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 4, p. e61807
The Notch pathway can have both oncogenic and tumor suppressor roles, depending on cell context. For example, Notch signaling promotes T cell differentiation... 
PROMOTER DNA METHYLATION | LYMPHOMAS | TUMOR-SUPPRESSOR FUNCTION | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOCYTIC-LEUKEMIA | GROWTH | GENES | TRANSCRIPTION | MALIGNANCIES | CANCER | TUMORIGENESIS | Transcription, Genetic - drug effects | Apoptosis - drug effects | Receptors, Notch - metabolism | Humans | Apoptosis - genetics | Receptors, Notch - genetics | Gene Expression Profiling | Hematopoiesis - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | T-Lymphocytes - metabolism | Serrate-Jagged Proteins | T-Lymphocytes - drug effects | B-Lymphocytes - pathology | Precursor Cell Lymphoblastic Leukemia-Lymphoma - blood | B-Lymphocytes - metabolism | Repressor Proteins - metabolism | Bone Marrow - drug effects | Jagged-1 Protein | Basic Helix-Loop-Helix Transcription Factors - genetics | Membrane Proteins - genetics | Gene Expression Regulation, Leukemic - drug effects | Leukemia, B-Cell - pathology | Repressor Proteins - genetics | Hematopoiesis - genetics | Signal Transduction - genetics | Azacitidine - pharmacology | Leukemia, B-Cell - blood | Leukemia, B-Cell - genetics | Signal Transduction - drug effects | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - genetics | Cell Line, Tumor | Histones - metabolism | DNA Methylation - drug effects | Calcium-Binding Proteins - genetics | Gene Silencing - drug effects | Blood Cells - metabolism | Cell Lineage - drug effects | Intercellular Signaling Peptides and Proteins - metabolism | Epigenesis, Genetic - drug effects | Membrane Proteins - metabolism | T-Lymphocytes - pathology | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Blood Cells - drug effects | Hydroxamic Acids - pharmacology | Cell Lineage - genetics | Calcium-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Azacitidine - analogs & derivatives | DNA Methylation - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | B-Lymphocytes - drug effects | Bone Marrow - pathology | Receptor, Notch3 | Cell Proliferation - drug effects | Sulfites | Epigenetic inheritance | DNA microarrays | Genes | Genetic aspects | Genetic engineering | Acute lymphocytic leukemia | B cells | T cells | Methylation | Cell differentiation | Biotechnology | Transcription factors | Bisulfite | Pathogenesis | Leukemia | Differentiation (biology) | Lymphocytes T | Kinases | Inactivation | Signal transduction | Restoration | Lymphocytes | DNA methylation | Bone marrow | Tumorigenesis | Acetylation | Deoxyribonucleic acid--DNA | CpG islands | Acute lymphatic leukemia | Deactivation | Lymphatic leukemia | Tumor cell lines | Gene silencing | Signaling | Lymphocytes B | Regulatory mechanisms (biology) | Stem cells | Epigenetics | Ligands | Tumor suppressor genes | Leukemogenesis | Lymphomas | Notch protein | Aberration | Apoptosis | Cancer | Deoxyribonucleic acid | DNA
Journal Article
Nature (London), ISSN 1476-4687, 2017, Volume 542, Issue 7642, pp. 489 - 493
...). In addition, through off-target activity, AID has a much broader effect on genomic instability by initiating oncogenic chromosomal translocations and mutations involved in the development and progression of lymphoma(2... 
SUPER-ENHANCERS | INDUCED CYTIDINE DEAMINASE | DNA | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | IBRUTINIB | CLASS-SWITCH RECOMBINATION | MECHANISMS | TARGETING BTK | AID | SEQUENCING REVEALS | Isoquinolines - adverse effects | Class I Phosphatidylinositol 3-Kinases - metabolism | Humans | Lymphoma, Mantle-Cell - pathology | Phosphatidylinositol 3-Kinases - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Translocation, Genetic - drug effects | Isoquinolines - pharmacology | Antineoplastic Agents - adverse effects | Genomic Instability - drug effects | Female | Antineoplastic Agents - pharmacology | Purines - adverse effects | B-Lymphocytes - pathology | Immunoglobulin Class Switching - drug effects | Somatic Hypermutation, Immunoglobulin - drug effects | Lymphoma, Mantle-Cell - genetics | B-Lymphocytes - metabolism | Quinazolinones - pharmacology | Pyrazoles - adverse effects | Pyrazoles - pharmacology | Enzyme Inhibitors - adverse effects | B-Lymphocytes - enzymology | Purines - pharmacology | Enzyme Inhibitors - pharmacology | Pyrimidines - pharmacology | Class I Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | B-Lymphocytes - drug effects | Animals | Agammaglobulinaemia Tyrosine Kinase | Cytidine Deaminase - metabolism | Pyrimidines - adverse effects | Cell Line, Tumor | Mice | Immunoglobulin Heavy Chains - genetics | Quinazolinones - adverse effects | Recombination, Genetic - drug effects | Protein-Tyrosine Kinases - antagonists & inhibitors
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, p. e0172684
The massive meat production of broiler chickens make them continuously exposed to potential stressors that stimulate releasing of stress-related hormones like... 
GALLUS-GALLUS-DOMESTICUS | FAT ACCUMULATION | DEOXYRIBONUCLEIC-ACID DEGRADATION | MEAT QUALITY | BLOOD PARAMETERS | SHOWING LONG | MULTIDISCIPLINARY SCIENCES | PHYSIOLOGICAL STRESS | SHORT TONIC IMMOBILITY | ENDOGENOUS GLUCOCORTICOIDS | PROTEIN-SYNTHESIS | Aspartate Aminotransferases - genetics | Caspase 9 - genetics | Corticosterone - pharmacology | Spleen - immunology | Insulin-Like Growth Factor I - genetics | Spleen - drug effects | Meat | Insulin-Like Growth Factor I - immunology | Liver - immunology | Alanine Transaminase - genetics | Liver - drug effects | Triiodothyronine - genetics | T-Lymphocytes - drug effects | Weight Gain - drug effects | Cell Death - drug effects | Alanine Transaminase - immunology | Avian Proteins - immunology | Bursa of Fabricius - drug effects | B-Lymphocytes - cytology | Chickens - growth & development | Immunity, Innate - drug effects | Aspartate Aminotransferases - immunology | Injections, Intramuscular | Thymus Gland - drug effects | Chickens - immunology | Gene Expression Regulation - drug effects | B-Lymphocytes - drug effects | Caspase 9 - immunology | Eating - drug effects | Bursa of Fabricius - immunology | Animals | B-Lymphocytes - immunology | T-Lymphocytes - cytology | Animal Husbandry | T-Lymphocytes - immunology | Thymus Gland - immunology | Cell Proliferation - drug effects | Triiodothyronine - immunology | Avian Proteins - genetics | Usage | Immunology | Cell death | Corticosterone | Physiological aspects | Broilers (Poultry) | Genetic aspects | Research | Gene expression | Bursa of Fabricius | Plasma | Poultry | Insulin-like growth factor I | Liver | Body weight | Cytology | Biochemistry | Lymphocytes T | Insulin-like growth factors | Hormones | Juveniles | Thymus | Proteins | Lymphocytes | Rodents | Gangrene | Physiology | Spleen | Alanine | Immune response | Parameters | Ethanol | Hematology | Mortality | Caspase | Birds | Injection | Insulin | Stress | Beta cells | Immunosuppression | Hepatocytes | Protein synthesis | Chickens | Morphology | Caspase-9 | Chicks | Meat production | Apoptosis | Conversion ratio
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 2, pp. 219 - 234
Immune-mediated (IM) adverse drug reactions (ADRs) are an underrecognized source of preventable morbidity, mortality, and cost... 
Allergy and Immunology | Stevens-Johnson syndrome | human herpesvirus | hapten | human leukocyte antigen | drug reaction with eosinophilia and systemic symptoms | altered peptide | major histocompatibility complex | p-i | heterologous immunity | carbamazepine | toxic epidermal necrolysis | T-cell receptor | pharmacogenomics | adverse drug reaction | Abacavir | allopurinol | pharmacogenetics | IMMUNOLOGY | INDUCED LIVER-INJURY | CUTANEOUS ADVERSE-REACTIONS | ALLERGY | STEVENS-JOHNSON-SYNDROME | INDUCED HYPERSENSITIVITY SYNDROME | GENOME-WIDE ASSOCIATION | HLA CLASS-I | NEVIRAPINE HYPERSENSITIVITY | IMPLEMENTATION CONSORTIUM GUIDELINES | HLA-B-ASTERISK-1502 ALLELE | Stevens-Johnson Syndrome - immunology | Humans | Virus Diseases - virology | Genetic Loci | HLA Antigens - genetics | Drug Hypersensitivity - genetics | Dideoxynucleosides - adverse effects | B-Lymphocytes - pathology | T-Lymphocytes - pathology | Virus Diseases - genetics | Drug Hypersensitivity - etiology | Pharmacogenetics | Virus Diseases - immunology | Stevens-Johnson Syndrome - etiology | Gene Expression Regulation | HLA Antigens - immunology | Drug Hypersensitivity - pathology | Models, Immunological | Stevens-Johnson Syndrome - genetics | B-Lymphocytes - immunology | Carbamazepine - adverse effects | Drug Hypersensitivity - immunology | Virus Diseases - pathology | Allopurinol - adverse effects | T-Lymphocytes - immunology | Stevens-Johnson Syndrome - pathology | Drugs | Allergy | Medical colleges | Analysis | Mortality | Models | Drug therapy | T cells | Allergic reaction | Drug allergy | Copyright | Patents | Peptides | Royalties | Lymphocytes | T cell receptors | Accreditation | Research | Disclosure | Allergies | T cell receptor | DRESS | abacavir | TEN | SJS
Journal Article
The Journal of Immunology, ISSN 0022-1767, 08/2007, Volume 179, Issue 3, pp. 1634 - 1647
.... To address this, we examined the effects of 1,25(OH)(2)D(3) on B cell responses and found that it inhibited the ongoing proliferation of activated B cells and induced their apoptosis, whereas initial cell division was unimpeded... 
Immunologic Factors - deficiency | Apoptosis - drug effects | Humans | Middle Aged | Calcitriol - physiology | Apoptosis - genetics | Male | Receptors, Calcitriol - genetics | Plasma Cells - pathology | Receptors, Calcitriol - biosynthesis | Plasma Cells - drug effects | Cell Differentiation - genetics | Calcitriol - deficiency | Lupus Erythematosus, Systemic - drug therapy | Lupus Erythematosus, Systemic - immunology | Adult | Female | Growth Inhibitors - genetics | Steroid Hydroxylases - genetics | B-Lymphocytes - cytology | B-Lymphocytes - enzymology | Immunologic Factors - physiology | Cells, Cultured | Vitamin D3 24-Hydroxylase | Down-Regulation - drug effects | Down-Regulation - genetics | Vitamin D - blood | Gene Expression Regulation - drug effects | B-Lymphocytes - drug effects | Cell Differentiation - immunology | Apoptosis - immunology | B-Lymphocytes - immunology | Vitamin D - antagonists & inhibitors | Cell Differentiation - drug effects | Down-Regulation - immunology | Lymphocyte Activation - drug effects | ADP-ribosyl Cyclase 1 - biosynthesis | Calcitriol - pharmacology | Adolescent | Growth Inhibitors - physiology | Vitamin D - analogs & derivatives | Aged | Lupus Erythematosus, Systemic - pathology | Steroid Hydroxylases - biosynthesis | Immunologic Factors - pharmacology
Journal Article