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Journal of neuroimmunology, ISSN 0165-5728, 2010, Volume 225, Issue 1, pp. 22 - 33
... antigen-specific cytotoxicity a... 
Neurology | Allergy and Immunology | IDO | Immunomodulation | Immunotherapy | Glioblastoma | TGF-β | PDL-1 | T cells | GLIOMA-CELLS | FAS LIGAND | HLA-G EXPRESSION | RECURRENT GLIOMA | IMMUNE PRIVILEGE | lmmunotherapy | IMMUNOLOGY | POTENTIAL MECHANISM | NEUROSCIENCES | ADJUVANT TEMOZOLOMIDE | TGF-beta | GROWTH-FACTOR-BETA | TRANSFORMING GROWTH-FACTOR-BETA-1 | Indoleamine-Pyrrole 2,3,-Dioxygenase - metabolism | Fas Ligand Protein - metabolism | Flow Cytometry - methods | Transforming Growth Factor beta2 - metabolism | Humans | HLA Antigens - genetics | Interferon-gamma - metabolism | Indoleamine-Pyrrole 2,3,-Dioxygenase - pharmacology | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Antigens, CD - metabolism | Histocompatibility Antigens Class I - metabolism | T-Lymphocytes - drug effects | Antigens, Neoplasm - metabolism | Cytotoxicity, Immunologic - drug effects | B7-H1 Antigen | Oligonucleotide Array Sequence Analysis - methods | Gene Expression Profiling - methods | Lectins - pharmacology | Histocompatibility Antigens Class I - genetics | HLA-G Antigens | HLA Antigens - metabolism | Gene Expression Regulation - drug effects | Glioblastoma - immunology | Glioblastoma - pathology | Cell Line, Tumor | Antigens, CD - pharmacology | MART-1 Antigen | T-Lymphocytes - immunology | Cell Proliferation - drug effects | HLA-A2 Antigen - metabolism | Cytotoxicity, Immunologic - immunology | Fas Ligand Protein - genetics | Interferon-gamma - pharmacology | Cell research | Histocompatibility antigens | Messenger RNA | HLA histocompatibility antigens | Lectins | Biochemistry | Transforming growth factors | Glioblastoma multiforme | Apoptosis | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Histocompatibility Antigens Class I | Gene Expression Profiling | Life Sciences | Flow Cytometry | Immunology | Neoplasm Proteins | Transforming Growth Factor beta2 | Interferon-gamma | Antigens, Neoplasm | T-Lymphocytes | Gene Expression Regulation | Fas Ligand Protein | HLA-A2 Antigen | Indoleamine-Pyrrole 2,3,-Dioxygenase | HLA Antigens | RNA, Messenger | Antigens, CD | Cytotoxicity, Immunologic | Cancer
Journal Article
Gene therapy, ISSN 0007-0610, 03/2014, Volume 217, Issue 1, pp. 262 - 271
Journal Article
Gut, ISSN 0017-5749, 02/2018, Volume 67, Issue 2, pp. 320 - 332
ObjectiveLimited efficacy of immune checkpoint inhibitors in pancreatic ductal adenocarcinoma (PDAC) has prompted investigation into combination therapy. We... 
INTERLEUKINS | IMMUNOTHERAPY | PANCREATIC CANCER | MULTIPLE-MYELOMA | INTERFERON-ALPHA | ACTIVATION | ADENOCARCINOMA | SILTUXIMAB | MONOCLONAL-ANTIBODY | MICE | SUPPRESSOR-CELLS | INTERLEUKIN-6 | GASTROENTEROLOGY & HEPATOLOGY | T-CELLS | Antineoplastic Agents, Immunological - administration & dosage | Interleukin-6 - antagonists & inhibitors | Pancreatic Neoplasms - metabolism | Humans | Actins - metabolism | Carcinoma, Pancreatic Ductal - metabolism | STAT Transcription Factors - metabolism | Janus Kinases - metabolism | L-Selectin - metabolism | Pancreatic Neoplasms - drug therapy | Th1 Cells - metabolism | Pancreatic Stellate Cells - immunology | Hyaluronan Receptors - metabolism | Female | Lymphocytes, Tumor-Infiltrating - metabolism | Carcinoma, Pancreatic Ductal - immunology | Interleukin-6 - metabolism | Disease Models, Animal | Mice, Inbred C57BL | Survival Rate | B7-H1 Antigen - immunology | Disease Progression | Tumor Microenvironment - immunology | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | B7-H1 Antigen - metabolism | Animals | B7-H1 Antigen - antagonists & inhibitors | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Pancreatic Neoplasms - immunology | Interleukin-6 - immunology | Mice | Pancreatic Stellate Cells - metabolism | Adenocarcinoma | Immunohistochemistry | Flow cytometry | Animal models | Transcription | CD8 antigen | Multiple myeloma | Smooth muscle | Lymphocytes T | Kinases | Metastases | Interleukin 6 | Lymphocytes | Actin | CD44 antigen | Immunotherapy | Fibroblasts | BRCA2 protein | Stellate cells | Cytokines | Stroma | CD62L protein | CD4 antigen | Immune checkpoint | Antibiotics | Pancreatic cancer | Medical prognosis | PD-L1 protein | Ligands | Genetic engineering | Apoptosis | PD-L1 | pancreatic cancer | IL-6
Journal Article
Blood, ISSN 1528-0020, 2012, Volume 120, Issue 24, pp. 4772 - 4782
..., demonstrating that LECs are significant, albeit suboptimal, antigen-presenting cells. Because LECs express numerous peripheral tissue antigens, lack of costimulation coupled to rapid high-level up-regulation of inhibitory receptors may be generally important in systemic peripheral tolerance. 
DENDRITIC CELLS | PROGRAMMED DEATH-1 | PERIPHERAL TOLERANCE | IN-VIVO | EFFECTOR FUNCTION | STEADY-STATE | CHRONIC VIRAL-INFECTION | HEMATOLOGY | CD28 COSTIMULATION | SELF-TOLERANCE | CROSS-PRESENTATION | Adoptive Transfer | Vitiligo - metabolism | Receptors, OX40 - immunology | Monophenol Monooxygenase - metabolism | Autoimmune Diseases - genetics | Signal Transduction - immunology | Receptors, OX40 - metabolism | Immune Tolerance - immunology | CD8-Positive T-Lymphocytes - metabolism | Receptors, Antigen, T-Cell - immunology | Monophenol Monooxygenase - genetics | Autoimmune Diseases - metabolism | Antigen-Presenting Cells - metabolism | Lymphatic Vessels - cytology | Receptors, Antigen, T-Cell - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | Tumor Necrosis Factor Receptor Superfamily, Member 9 - immunology | Vitiligo - immunology | Autoimmune Diseases - immunology | Lymph Nodes - metabolism | Programmed Cell Death 1 Receptor - metabolism | Mice, Transgenic | Tumor Necrosis Factor Receptor Superfamily, Member 9 - metabolism | Lymph Nodes - immunology | Antigen-Presenting Cells - immunology | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Mice, Knockout | Endothelial Cells - immunology | B7-H1 Antigen - metabolism | Animals | Monophenol Monooxygenase - immunology | Mice | Receptors, Antigen, T-Cell - genetics | Programmed Cell Death 1 Receptor - immunology | CD8-Positive T-Lymphocytes - immunology | Vitiligo - genetics | Programmed Cell Death 1 Receptor - genetics | Microscopy, Fluorescence | Immunobiology
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2013, Volume 288, Issue 17, pp. 11771 - 11785
PD-1, a receptor expressed by T cells, B cells, and monocytes, is a potent regulator of immune responses and a promising therapeutic target. The structure and... 
SECONDARY STRUCTURE DETERMINATION | NMR ASSIGNMENT | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL DOMAIN | TISSUE INHIBITOR | QUANTITATIVE-ANALYSIS | MOLECULAR-BASIS | HCV INFECTION | T-CELLS | PD-1 EXPRESSION | Surface Plasmon Resonance | Cell Communication - immunology | Programmed Cell Death 1 Receptor - chemistry | B7-1 Antigen - genetics | Humans | Programmed Cell Death 1 Ligand 2 Protein - metabolism | Structure-Activity Relationship | B7-H1 Antigen - chemistry | Programmed Cell Death 1 Ligand 2 Protein - chemistry | T-Lymphocytes - metabolism | B7-1 Antigen - chemistry | Nuclear Magnetic Resonance, Biomolecular | Antigen-Presenting Cells - metabolism | Protein Structure, Secondary | B7-1 Antigen - immunology | Programmed Cell Death 1 Ligand 2 Protein - genetics | Programmed Cell Death 1 Receptor - metabolism | B7-1 Antigen - metabolism | Antigen-Presenting Cells - immunology | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Programmed Cell Death 1 Ligand 2 Protein - immunology | Antigen-Presenting Cells - chemistry | B7-H1 Antigen - metabolism | Models, Immunological | Animals | T-Lymphocytes - chemistry | Protein Binding | T-Lymphocytes - immunology | Mice | Programmed Cell Death 1 Receptor - immunology | Programmed Cell Death 1 Receptor - genetics | Nuclear Magnetic Resonance | Signaling | Thermodynamics | Receptors | Immunology | Surface Plasmon Resonance (SPR) | Cell Surface Protein | Complex Formation | Affinity
Journal Article
Nature immunology, ISSN 1529-2916, 2009, Volume 10, Issue 11, pp. 1185 - 1192
Journal Article
Journal Article
Blood, ISSN 0006-4971, 09/2010, Volume 116, Issue 13, pp. 2266 - 2276
Oral tolerance is a key feature of intestinal immunity, generating systemic tolerance to fed antigens... 
RESPONSES | HOMEOSTASIS | RETINOIC-ACID | DISEASE | DIFFERENTIATION | HEMATOLOGY | FOXP3 | PD-1 | T-Lymphocytes, Regulatory - metabolism | B7-1 Antigen - genetics | Membrane Glycoproteins - metabolism | Ovalbumin - immunology | Dendritic Cells - immunology | Peptides - genetics | Peptides - deficiency | T-Lymphocytes, Regulatory - immunology | Antigens - administration & dosage | Peptides - metabolism | Antigen Presentation | Immunoglobulin G - biosynthesis | Forkhead Transcription Factors - metabolism | Mesentery - immunology | Dendritic Cells - metabolism | Recombinant Proteins - metabolism | Receptors, Antigen, T-Cell - metabolism | Administration, Oral | B7-H1 Antigen | Mice, Inbred C57BL | Programmed Cell Death 1 Ligand 2 Protein | Immune Tolerance | Mice, Transgenic | B7-1 Antigen - metabolism | Recombinant Proteins - genetics | Lymph Nodes - immunology | Lymph Nodes - cytology | Mesentery - cytology | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Ovalbumin - administration & dosage | Mice | Mice, Inbred BALB C | Receptors, Antigen, T-Cell - genetics | In Vitro Techniques | Membrane Glycoproteins - deficiency | Ovalbumin | Antigens | Dendritic Cells | Peptides | Immunoglobulin G | Antigens, CD80 | Mesentery | Receptors, Antigen, T-Cell | Recombinant Proteins | Lymph Nodes | Life Sciences | Immunology | T-Lymphocytes, Regulatory | Membrane Glycoproteins | Forkhead Transcription Factors | Immunobiology
Journal Article
European journal of cancer (1990), ISSN 0959-8049, 2016, Volume 54, pp. 139 - 148
Journal Article
European journal of immunology, ISSN 0014-2980, 2011, Volume 41, Issue 2, pp. 413 - 424
During infection, TLR agonists are released and trigger mature as well as differentiating innate immune cells. Early encounter with TLR agonists (R848; LPS)... 
Tolerance | STAT‐3 | TLR | PD‐L1 | PD-L1 | STAT-3 | MYCOBACTERIUM-TUBERCULOSIS | ALLOGRAFT-REJECTION | HUMAN DENDRITIC CELLS | IFN-GAMMA | TRANSPLANT TOLERANCE | IMMUNOLOGY | CUTTING EDGE | IMMUNE-RESPONSES | REGULATORY T-CELLS | B7 FAMILY | DIFFERENTIATION | T-Lymphocyte Subsets - immunology | Antigen-Presenting Cells - cytology | Immune Tolerance - physiology | Monocytes - cytology | Dendritic Cells - immunology | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | STAT Transcription Factors - metabolism | Monocytes - immunology | Antigens, CD - metabolism | Lipopolysaccharide Receptors - metabolism | Lymphocyte Culture Test, Mixed | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Interleukin-4 - pharmacology | Interleukin-10 - metabolism | Histocompatibility Antigens Class II - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Antigen-Presenting Cells - metabolism | B7-H1 Antigen | Gene Expression Regulation - immunology | Antigen-Presenting Cells - drug effects | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Imidazoles - pharmacology | Antigen-Presenting Cells - immunology | Toll-Like Receptors - agonists | Monocytes - drug effects | Cell Differentiation - immunology | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Differentiation - drug effects | Antigens, CD1 - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Dendritic Cells - cytology | Protein Kinase Inhibitors - pharmacology | STAT Transcription Factors - antagonists & inhibitors | STAT3 Transcription Factor - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - metabolism | Medical research | Rodents
Journal Article