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Journal Article
Nutrition and Metabolism, ISSN 1743-7075, 04/2016, Volume 13, Issue 1, p. 30
Journal Article
Molecular metabolism (Germany), ISSN 2212-8778, 2020, Volume 32, pp. 85 - 96
Ureagenesis predominantly occurs in the liver and functions to remove ammonia, and the dysregulation of ureagenesis leads to the development of hyperammonemia.... 
Urea cycle | BAF60a | YB-1 | Nutrient signals | Hyperammonemia | UREA-CYCLE | TRANSCRIPTION | PHOSPHATE SYNTHETASE-I | DEFICIENCY | CIRCADIAN CLOCK | BOX BINDING PROTEIN-1 | GENE | METABOLISM | DNA | ENDOCRINOLOGY & METABOLISM | EXPRESSION
Journal Article
Journal of cellular physiology, ISSN 0021-9541, 7/2019, Volume 234, Issue 7, pp. 11780 - 11791
SWI/SNF chromatin remodeling enzymes are multi-subunit complexes that contain one of two catalytic subunits, BRG1 or BRM, and 9–11 additional subunits called... 
Chromatin Remodeling | BAF60A | MITF | Melanocyte differentiation
Journal Article
STEM CELLS, ISSN 1066-5099, 05/2008, Volume 26, Issue 5, pp. 1155 - 1165
Journal Article
Hepatology (Baltimore, Md.), ISSN 1527-3350, 2020, Volume 71, Issue 4, pp. 1228 - 1246
Background and Aims Nonalcoholic steatohepatitis (NASH) is a progressive liver disease that is characterized by liver injury, inflammation, and fibrosis. NASH... 
TRUNCATED TRKB | PROTEIN | INHIBITION | GENE | BAF60A | NEUROTROPHIC FACTOR | MOUSE MODEL | DISEASE | RECEPTORS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Chromatin | Immunoprecipitation | Liver diseases | Pathogenesis | Liver | Inflammation | Ribonucleic acid--RNA | Gene expression | Membrane proteins | Hepatocytes | Diet | Cell death | Fibrosis | TrkB receptors | Matrix protein
Journal Article
Journal of cellular physiology, ISSN 1097-4652, 2018, Volume 234, Issue 7, pp. 11780 - 11791
SWI/SNF chromatin remodeling enzymes are multisubunit complexes that contain one of two catalytic subunits, BRG1 or BRM and 9–11 additional subunits called... 
microphthalmia‐associated transcription factor (MITF) | chromatin remodeling | melanocyte differentiation | BAF60A | microphthalmia-associated transcription factor (MITF) | ACTIVATION | PHYSIOLOGY | BRG1 | DOPACHROME-TAUTOMERASE | MUSCLE | MITF | SOX10 | CELL BIOLOGY | REGULATOR | CHROMATIN-REMODELING COMPLEXES | EXPRESSION | LINEAGE | Microphthalmia-Associated Transcription Factor - metabolism | Chromosomal Proteins, Non-Histone - metabolism | Membrane Glycoproteins - metabolism | Microphthalmia-Associated Transcription Factor - chemistry | Oxidoreductases - metabolism | Oxidoreductases - genetics | Humans | Gene Expression Regulation | Melanocytes - metabolism | Nuclear Proteins - metabolism | Promoter Regions, Genetic - genetics | Protein Subunits - metabolism | Melanins - biosynthesis | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Cell Differentiation - genetics | DNA Helicases - metabolism | Animals | Cell Cycle | Melanocytes - cytology | Models, Biological | HEK293 Cells | Protein Binding | Mice | Chromatin | DNA binding proteins | Microphthalmia-associated transcription factor | Regulators | Transcription factors | BRG1 protein | Gene expression | Remodeling | Recruitment | Chromatin remodeling | Depletion | SWI/SNF complex | Isoforms | Catalysis | Differentiation | Melanin | Recombinant | Catalytic subunits
Journal Article
The Journal of pathology, ISSN 0022-3417, 2014, Volume 233, Issue 2, pp. 159 - 169
Many mammalian physiological processes show diurnal oscillation and are controlled by a circadian clock. Disruption of the circadian clock has been implicated... 
physiology | smarcd1 | hyperlipidaemia | vascular smooth muscle cells | circadian clock | LIPID-METABOLISM | MAMMALIAN SWI/SNF COMPLEXES | MOUSE | TRANSCRIPTION | PATHOLOGY | SUBUNIT BAF60A | RHYTHMS | ONCOLOGY | LIVER | GENE-EXPRESSION | MICE | ENERGY-METABOLISM | Hyperlipidemias - genetics | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Homeostasis | Male | CLOCK Proteins - genetics | Circadian Clocks - genetics | DNA-Binding Proteins - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 1 - metabolism | CLOCK Proteins - metabolism | Transfection | RNA Interference | Time Factors | Diet, High-Fat | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Transcription, Genetic | Fatty Acids - metabolism | Myocytes, Smooth Muscle - metabolism | Aorta, Thoracic - pathology | Disease Models, Animal | ARNTL Transcription Factors - genetics | Hyperlipidemias - metabolism | Signal Transduction | Down-Regulation | Cells, Cultured | ARNTL Transcription Factors - metabolism | Aorta, Thoracic - metabolism | Rats | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Rats, Sprague-Dawley | Transcription Factors - metabolism | Muscle, Smooth, Vascular - pathology | Animals | Hyperlipidemias - etiology | Hyperlipidemias - pathology | Enzyme Activation | Nuclear Receptor Subfamily 1, Group F, Member 1 - genetics | Cell Movement | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Smooth muscle | Hyperlipidemia | Analysis
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2001, Volume 276, Issue 4, pp. 2852 - 2857
Fos family proteins form stable heterodimers with Jun family proteins, and each heterodimer shows distinctive transactivating potential for regulating cellular... 
BAF60a protein | SWI/SNF complex | chromatin remodeling
Journal Article
Journal Article