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Journal of Neurochemistry, ISSN 0022-3042, 04/2006, Volume 97, Issue 2, pp. 475 - 487
Dysfunction of cholinergic basal forebrain (CBF) neurons of the nucleus basalis (NB) is a cardinal feature of Alzheimer's disease (AD) and correlates with... 
microarray | trkA | mild cognitive impairment | RNA amplification | neurotrophin | nucleus basalis | Neurotrophin | Nucleus basalis | Microarray | Mild cognitive impairment | Humans | Cognition Disorders - metabolism | Male | Receptors, Nerve Growth Factor - metabolism | RNA, Messenger - metabolism | Choline O-Acetyltransferase - metabolism | Mental Status Schedule - statistics & numerical data | Alzheimer Disease - pathology | Reverse Transcriptase Polymerase Chain Reaction - methods | Aged, 80 and over | Female | Neurons - metabolism | In Situ Hybridization - methods | Prosencephalon - pathology | Severity of Illness Index | Oligonucleotide Array Sequence Analysis - methods | Cognition Disorders - pathology | Gene Expression Regulation - physiology | Nerve Tissue Proteins - genetics | Disease Progression | Receptors, Nerve Growth Factor - genetics | Immunohistochemistry - methods | Nerve Tissue Proteins - metabolism | Receptor, trkA - metabolism | Alzheimer Disease - metabolism | Receptor, trkA - genetics | Aged | Alzheimer Disease - genetics | Postmortem Changes | Cohort Studies | Neurosciences | Alzheimers disease | Gene expression | Neurons | Cell survival | Neurodegenerative diseases | Cognitive ability | Nerve growth factor | Forebrain (basal) | Neurotrophin receptors | Polymerase chain reaction | DNA microarrays | Cerebral blood flow | Degeneration | TrkA receptors | TrkB receptors | Alzheimer's disease
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 7, p. e0134421
Journal Article