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Scientific Reports, ISSN 2045-2322, 01/2016, Volume 6, Issue 1, pp. 18778 - 18778
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 10/2004, Volume 167, Issue 2, pp. 257 - 267
The Pit1-Oct1-Unc86 domain (POU domain) transcription factor Brn3a controls sensory neuron survival by regulating the expression of Trk receptors and members... 
Sensory ganglia | Messenger RNA | Neurons | Cell death | Exons | Genes | Sensory neurons | Gene expression regulation | Trigeminal ganglion | Apoptosis | SIGNAL-TRANSDUCTION | NERVOUS-SYSTEM | TRIGEMINAL GANGLION | PROTEIN KINASE-2 | IN-VIVO | RECEPTOR EXPRESSION | TRK RECEPTORS | DEFICIENT MICE | NT-3 ACTIVATION | CELL-DEATH | CELL BIOLOGY | Immunohistochemistry | Up-Regulation | Immunoprecipitation | Luciferases - metabolism | RNA, Messenger - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | In Situ Hybridization | Time Factors | Gene Deletion | Transcription Factor Brn-3A | Transcription, Genetic | Neurons - metabolism | Carrier Proteins - physiology | Protein Structure, Tertiary | Gene Targeting | Green Fluorescent Proteins - metabolism | bcl-X Protein | DNA-Binding Proteins - physiology | Transcription Factors - physiology | Cell Survival | Protein-Serine-Threonine Kinases - physiology | Down-Regulation | Gene Expression Regulation | DNA - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Two-Hybrid System Techniques | Animals | Receptor, trkA - metabolism | Transcription Factor Brn-3 | Models, Biological | Protein Binding | Mice | Models, Genetic | Mutation | DNA, Complementary - metabolism | Cytology | Genetic transcription | Research | Proteins | Cellular biology | Deoxyribonucleic acid | DNA | Index Medicus
Journal Article
03/2002, ISBN 9780471350545, 19
This chapter reviews findings on BCL‐6 translocations and mutations in various types of NHL, mechanisms of its deregulation in lymphomas, and the current... 
non‐Hodgkin's lymphoma | B cells | BCL‐6 expression | target genes | T cells | histogenic marker | interacting proteins | upstream signals | BCL‐6 function | nonlymphoid cell types | regulation | BCL‐6 mutations | transcriptional repressor | BCL‐6 translocations | BCL‐6 protein
Book Chapter
The EMBO Journal, ISSN 0261-4189, 07/1999, Volume 18, Issue 14, pp. 3990 - 4003
The Wilms‘ tumor suppressor gene, WT1 , encodes a zinc finger transcription factor that has been demonstrated to negatively regulate several growth factor and... 
apoptosis | Bcl‐2 | Wilms' tumor | WT1 | transcriptional regulation | Bcl-2 | Transcriptional regulation | Apoptosis | DNA-BINDING | SUPPRESSOR GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | WILMS-TUMOR GENE | KIDNEY DEVELOPMENT | RENAL TUMORS | CANCER-THERAPY | I RECEPTOR GENE | CELL BIOLOGY | POINT MUTATIONS | GROWTH-FACTOR | CELLULAR PROLIFERATION | Vincristine - pharmacology | Kidney Neoplasms - genetics | Transcription Factors - chemistry | Wilms Tumor - genetics | Apoptosis - drug effects | Humans | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Kidney Neoplasms - metabolism | RNA, Messenger - metabolism | Promoter Regions, Genetic - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Transfection | Protein Isoforms - chemistry | Tumor Cells, Cultured | Wilms Tumor - metabolism | DNA-Binding Proteins - physiology | Transcription Factors - physiology | RNA, Messenger - genetics | DNA - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | DNA-Binding Proteins - chemistry | Genes, bcl-2 - genetics | Protein Isoforms - physiology | DNA - genetics | Animals | WT1 Proteins | Consensus Sequence - genetics | Mutation | Proto-Oncogene Proteins c-bcl-2 - genetics | Doxorubicin - pharmacology | Staurosporine - pharmacology | Protein Isoforms - genetics | Index Medicus
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 09/2003, Volume 285, Issue 3, pp. 740 - 754
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 06/2014, Volume 89, Issue 4, pp. 526 - 535
Glucocorticoids (GCs) are steroid hormones widely used as coadjuvants in the treatment of solid tumors due to their anti-inflammatory effects. However,... 
Mitogen activated protein kinase phosphatase-1 | Cyclooxygenase-2 | 21OH-6,19-epoxyprogesterone | Glucocorticoid receptor | Paclitaxel | Mitogen activated protein kinase | APOPTOSIS | DEXAMETHASONE | BCL-X GENE | MAP KINASE PHOSPHATASE-1 | FACTOR-KAPPA-B | TUMOR-CELLS | OXIDE SYNTHASE INOS | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | phosphatase-1 | EXPRESSION | MAMMARY EPITHELIAL-CELLS | Lung Neoplasms - drug therapy | Neoplasm Transplantation | Receptors, Glucocorticoid - antagonists & inhibitors | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Progesterone - administration & dosage | Humans | Lung Neoplasms - metabolism | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Neoplasm Proteins - antagonists & inhibitors | Receptors, Glucocorticoid - metabolism | Neoplasm Proteins - metabolism | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Breast Neoplasms - metabolism | Progesterone - therapeutic use | Antineoplastic Agents, Hormonal - adverse effects | Breast Neoplasms - enzymology | Anti-Inflammatory Agents - adverse effects | Antineoplastic Agents, Hormonal - therapeutic use | Anti-Inflammatory Agents - therapeutic use | Anti-Inflammatory Agents - administration & dosage | Female | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Macrophages, Peritoneal - drug effects | Progesterone - pharmacology | Cell Survival - drug effects | Lung Neoplasms - enzymology | Specific Pathogen-Free Organisms | Antineoplastic Agents, Hormonal - pharmacology | Anti-Inflammatory Agents - pharmacology | Antineoplastic Agents, Hormonal - administration & dosage | Cells, Cultured | Macrophages, Peritoneal - enzymology | Progesterone - analogs & derivatives | Random Allocation | Progesterone - adverse effects | Breast Neoplasms - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Cell Line, Tumor | Mice | Mice, Inbred BALB C | Macrophages, Peritoneal - metabolism | Cell Line, Transformed | Antimitotic agents | Chemotherapy | Corticosteroids | Breast cancer | Antineoplastic agents | Cancer | Steroids | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 05/2007, Volume 26, Issue 26, pp. 3797 - 3810
Journal Article
Expert Opinion on Therapeutic Targets, ISSN 1472-8222, 06/2012, Volume 16, Issue 6, pp. 573 - 585
Journal Article