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The Journal of Clinical Investigation, ISSN 0021-9738, 06/2008, Volume 118, Issue 6, pp. 2260 - 2268
Brugada syndrome is a genetic disease associated with sudden cardiac death that is characterized by ventricular fibrillation and right precordial ST segment... 
SKELETAL-MUSCLE | SCN5A MUTATION | BETA-1 SUBUNIT | FUNCTIONAL EXPRESSION | EXTRACELLULAR DOMAIN | XENOPUS OOCYTES | LONG-QT SYNDROME | FEBRILE SEIZURES | ST-SEGMENT ELEVATION | UMCG Approved | BETA SUBUNIT | MEDICINE, RESEARCH & EXPERIMENTAL
Journal Article
Current Opinion in Neurobiology, ISSN 0959-4388, 2003, Volume 13, Issue 3, pp. 284 - 290
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 10683 - 13
Journal Article
Channels, ISSN 1933-6950, 03/2012, Volume 6, Issue 2, pp. 75 - 85
Perturbations to body temperature affect almost all cellular processes and, within certain limits, results in minimal effects on overall physiology. Genetic... 
paramyotonia congenita | Brugada syndrome | sodium channel | Long QT syndrome | erythromelalgia | GEFS | febrile seizures | severe myoclonic epilepsy | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Febrile seizures | Sodium channel | Severe myoclonic epilepsy | Paramyotonia congenita | Erythromelalgia | GEFS(+) | NA+ CHANNEL MUTATION | long QT syndrome | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXPERIMENTAL FEBRILE SEIZURES | SKELETAL-MUSCLE | BRUGADA-SYNDROME | PARAMYOTONIA-CONGENITA | brugada syndrome | BETA-1 SUBUNIT MUTATION | SLOW INACTIVATION | LONG-QT-SYNDROME | IDIOPATHIC VENTRICULAR-FIBRILLATION | Erythromelalgia - physiopathology | Channelopathies - physiopathology | Brugada Syndrome - physiopathology | Electrophysiological Phenomena - genetics | Humans | Electrophysiological Phenomena - physiology | Epilepsies, Myoclonic - metabolism | Epilepsies, Myoclonic - physiopathology | Channelopathies - metabolism | Body Temperature - physiology | Myotonic Disorders - metabolism | Long QT Syndrome - physiopathology | Long QT Syndrome - metabolism | Channelopathies - genetics | Seizures, Febrile - physiopathology | Brugada Syndrome - metabolism | Myotonic Disorders - physiopathology | Homeostasis - physiology | Erythromelalgia - metabolism | Sodium Channels - genetics | Mutation | Seizures, Febrile - metabolism
Journal Article
Journal of Immunology, ISSN 0022-1767, 05/2005, Volume 174, Issue 10, pp. 6054 - 6061
We show that the pertussis toxin B oligomer (PTX-B), and the PTX mutant PT9K/129G, which is safely administered in vivo, inhibit both transcription and... 
RESPONSE ELEMENT | GROWTH-FACTOR-BETA | HIV-I TAT | DENDRITIC CELLS | TRANSFORMING GROWTH-FACTOR-BETA-1 | DEPENDENT PATHWAY | IMMUNOLOGY | HUMAN-IMMUNODEFICIENCY-VIRUS | NF-KAPPA-B | T-CELLS | TERMINAL KINASE | Protein Subunits - immunology | Vaccines, Subunit - genetics | Humans | Apoptosis - genetics | RNA, Messenger - metabolism | Transforming Growth Factor beta - biosynthesis | HIV Infections - immunology | Protein Subunits - toxicity | Protein-Serine-Threonine Kinases - metabolism | Protein Subunits - genetics | Glycine - genetics | bcl-X Protein | Adjuvants, Immunologic - physiology | HIV Infections - virology | Pertussis Toxin - immunology | Down-Regulation - genetics | Killer Cells, Natural - cytology | Lysine - genetics | Pertussis Toxin - genetics | Killer Cells, Natural - metabolism | Pertussis Toxin - toxicity | tat Gene Products, Human Immunodeficiency Virus | Phosphorylation | Gene Products, tat - antagonists & inhibitors | Transcription Factor AP-1 - metabolism | Enzyme Activation - immunology | Proto-Oncogene Proteins c-bcl-2 - metabolism | HIV Infections - pathology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Killer Cells, Natural - immunology | Transforming Growth Factor beta - antagonists & inhibitors | Proto-Oncogene Proteins - metabolism | Adjuvants, Immunologic - antagonists & inhibitors | Proto-Oncogene Proteins - antagonists & inhibitors | Cells, Cultured | Gene Products, tat - physiology | Transforming Growth Factor beta - physiology | Vaccines, Subunit - immunology | Proto-Oncogene Proteins c-akt | HIV-1 - immunology | Transcription Factor AP-1 - antagonists & inhibitors | Apoptosis - immunology | Down-Regulation - immunology | Vaccines, Subunit - toxicity | Transcription, Genetic - immunology | Amino Acid Substitution - genetics | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Proto-Oncogene Proteins c-bcl-2 - genetics | Transforming Growth Factor beta - metabolism
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2004, Volume 24, Issue 44, pp. 10022 - 10034
Journal Article
Epilepsy research, ISSN 0920-1211, 05/2005, Volume 64, Issue 3, p. 77
Journal Article
AMERICAN JOURNAL OF NEPHROLOGY, ISSN 0250-8095, 2010, Volume 32, Issue 5, pp. 414 - 424
Background: Glomerular filtration rate (GFR) is a heritable trait, and hyperfiltration (GFR increment in remnant nephrons) may accelerate renal functional... 
KIDNEY-DISEASE | POPULATION | BETA-1 SUBUNIT | African-American Study of Kidney Disease and Hypertension | BLOOD-PRESSURE | AFRICAN-AMERICAN | Glomerular filtration rate | SMOOTH-MUSCLE | KCNMB1 | Hypertensive nephrosclerosis | MESANGIAL CELLS | UROLOGY & NEPHROLOGY | HYPERTENSION | LINKAGE | CREATININE CLEARANCE
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 10/2009, Volume 297, Issue 4, pp. 758 - 766
Large conductance, calcium- and voltage-activated potassium (BKCa) channels are important modulators of pulmonary vascular smooth muscle membrane potential,... 
Channel subunits | LARGE-CONDUCTANCE | PHYSIOLOGY | MECHANISM | BETA-1 SUBUNIT | PKC | ACTIVATED POTASSIUM CHANNELS | CA2+-ACTIVATED K+ CHANNEL | MESENTERIC MICROVESSELS | RESPIRATORY SYSTEM | channel subunits | ELEVATED BLOOD-PRESSURE | CGMP | SMOOTH-MUSCLE-CELLS
Journal Article
HUMAN MUTATION, ISSN 1059-7794, 10/2018, Volume 39, Issue 10, pp. 1402 - 1415
Voltage-dependent sodium channels are responsible of the rising phase of the action potential in excitable cells. These integral membrane proteins are composed... 
epilepsy | AUXILIARY SUBUNIT | BETA-1 SUBUNIT | EXTRACELLULAR DOMAIN | GEFS | SIALIC-ACID | MAMMALIAN-CELLS | INACTIVATION | GENETICS & HEREDITY | GENERALIZED EPILEPSY | EXPRESSION | protein maturation voltage-gated sodium channel | MODULATION | BRAIN
Journal Article
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