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Journal of Bone and Mineral Research, ISSN 0884-0431, 01/2009, Volume 24, Issue 1, pp. 12 - 21
Osteoarthritis (OA) is a degenerative joint disease, and the mechanism of its pathogenesis is poorly understood. Recent human genetic association studies... 
Cre‐recombination | articular chondrocyte | β‐catenin | osteoarthritis | chondrocyte differentiation | β-catenin | Cre-recombination | Articular chondrocyte | Chondrocyte differentiation | Osteoarthritis
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2012, Volume 325, Issue 1, pp. 42 - 53
Abstract Here we show that activation of the canonical WNT/β-catenin pathway increases the expression of stem cell genes and promotes the migratory and... 
Hematology, Oncology and Palliative Medicine | β-Catenin | ZEB1 | Epithelial-to-mesenchymal transition (EMT) | Invasion | Glioblastoma | STEM-CELLS | LENTIVIRAL VECTORS | NEGATIVE REGULATOR | BREAST-CANCER | GLIOMA TUMORIGENESIS | ONCOLOGY | WNT PATHWAY | COLORECTAL-CANCER | GROWTH | beta-Catenin | EXPRESSION | Immunohistochemistry | Homeodomain Proteins - metabolism | Humans | Glycoproteins - metabolism | Peptides - genetics | Wnt Proteins - metabolism | Antigens, CD - genetics | Cell Movement - genetics | Cell Movement - physiology | Antigens, CD - metabolism | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Peptides - metabolism | Wnt Proteins - genetics | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Cell Growth Processes - genetics | Wnt Signaling Pathway | Glycoproteins - genetics | Signal Transduction | Neoplasm Invasiveness | Intercellular Signaling Peptides and Proteins - genetics | Transcription Factors - genetics | AC133 Antigen | beta Catenin - metabolism | Homeodomain Proteins - genetics | beta Catenin - genetics | Transcription Factors - metabolism | Glioblastoma - pathology | Cell Line, Tumor | Zinc Finger E-box-Binding Homeobox 1
Journal Article
Nature: international weekly journal of science, ISSN 0028-0836, 01/2014, Volume 506, Issue 7487, pp. 240 - 244
textabstractCells of the osteoblast lineage affect the homing and the number of long-term repopulating haematopoietic stem cells, haematopoietic stem cell... 
PROGENITOR CELLS | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | BONE-MARROW NICHE | NOTCH ACTIVATION | STEM-CELL NICHE | MYELOID-LEUKEMIA | MICE | DIFFERENTIATION | EXPRESSION | MYELODYSPLASTIC SYNDROMES | Osteoblasts - secretion | Receptors, Notch - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Hematopoietic Stem Cells - pathology | Anemia - pathology | Male | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Membrane Proteins - deficiency | Anemia - genetics | Cell Nucleus - metabolism | Tumor Microenvironment - genetics | Cell Transformation, Neoplastic - genetics | Serrate-Jagged Proteins | Base Sequence | Female | Membrane Proteins - metabolism | Intercellular Signaling Peptides and Proteins - deficiency | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Myelodysplastic Syndromes - metabolism | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Calcium-Binding Proteins - deficiency | Hematopoietic Stem Cells - metabolism | Mutation - genetics | beta Catenin - metabolism | beta Catenin - genetics | Anemia - metabolism | Cell Lineage | Osteoblasts - pathology | Animals | Chromosome Aberrations | Myeloid Cells - metabolism | Ligands | Mice | Myelodysplastic Syndromes - genetics | Myeloid Cells - pathology | Cell Transformation, Neoplastic - pathology | Myelodysplastic Syndromes - pathology | Osteoblasts - metabolism | Calcium-Binding Proteins - genetics | Leukemia, Myeloid, Acute - genetics
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 9/2016, Volume 53, Issue 7, pp. 4286 - 4301
Oxidative stress and neuroinflammation are known causative factors in progressive degeneration of dopaminergic (DAergic) neurons in Parkinson’s disease (PD).... 
Neurology | Oxidative stress | Neuroprotection | Neurosciences | Biomedicine | Neuroblasts | Neurobiology | Acetyl- l -carnitine (ALCAR) | Neural stem cells | Wnt/ β -catenin pathway | Cell Biology | Acetyl-l-carnitine (ALCAR) | Wnt/β-catenin pathway | ACETYL-L-CARNITINE | Wnt/beta-catenin pathway | ALPHA-LIPOIC ACID | DOPAMINERGIC-NEURONS | SUBVENTRICULAR ZONE | SUBSTANTIA-NIGRA | NEUROSCIENCES | MGLU2 RECEPTORS | NEURAL STEM-CELLS | MOUSE MODEL | Acetyl-L-carnitine (ALCAR) | HIPPOCAMPAL NEUROGENESIS | CORPUS STRIATUM | Neuroprotective Agents - therapeutic use | Reactive Oxygen Species - metabolism | Acetylcarnitine - pharmacology | Dopaminergic Neurons - pathology | Neuroglia - pathology | Antioxidants - metabolism | Nitrites - metabolism | Male | Parkinson Disease - drug therapy | Oxidopamine | Neuroglia - drug effects | Behavior, Animal | Brain - metabolism | Cell Nucleus - metabolism | Neuroprotective Agents - pharmacology | Dopaminergic Neurons - metabolism | Lipid Peroxidation - drug effects | Ataxia - drug therapy | Dopaminergic Neurons - drug effects | Neurogenesis - drug effects | Parkinson Disease - metabolism | Phosphorylation - drug effects | Disease Models, Animal | Parkinson Disease - complications | Parkinson Disease - pathology | Neural Stem Cells - drug effects | Axons - drug effects | Axons - metabolism | Glycogen Synthase Kinase 3 beta - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Axons - pathology | Brain - pathology | Neuroglia - metabolism | Ataxia - complications | Acetylcarnitine - therapeutic use | Oxidative Stress - drug effects | Cell Nucleus - drug effects | Neural Stem Cells - metabolism
Journal Article
by Feng, Y and Ren, JF and Gui, Y and Wei, W and Shu, BY and Lu, QM and Xue, X and Sun, XL and He, WC and Yang, JW and Dai, CS
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY, ISSN 1046-6673, 01/2018, Volume 29, Issue 1, pp. 182 - 193
The Wnt/beta-catenin pathway is crucial in normal development and throughout life, but aberrant activation of this pathway has been linked to kidney fibrosis,... 
RENAL FIBROSIS | FIBROBLAST ACTIVATION | REPAIR | DENDRITIC CELLS | DISEASE | INJURY | UROLOGY & NEPHROLOGY | GROWTH-FACTOR | BETA-CATENIN | WNT | POLARIZATION | β-catenin | macrophages | Wnt | kidney fibrosis | Basic Research
Journal Article
Journal Article