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Journal of molecular medicine (Berlin, Germany), ISSN 1432-1440, 2017, Volume 95, Issue 9, pp. 915 - 925
Inflammatory bowel disease (IBD) is a devastating disease that is associated with excessive inflammation in the intestinal tract in genetically susceptible... 
Human Genetics | Adenosine | CD73 | Internal Medicine | ENPP | Innate immunity | Adaptive immunity | Inflammation | AHR | Macrophages | ENTPD | Inflammatory bowel disease | Extracellular nucleotides | Biomedicine | P1 receptors | Lymphocytes | P2 receptors | CD39 | Molecular Medicine | ATP | MEDICINE, RESEARCH & EXPERIMENTAL | GLIAL-CELLS | BOWEL-DISEASE | ADENOSINE RECEPTORS | IMMUNE-SYSTEM | ARYL-HYDROCARBON RECEPTOR | GENETICS & HEREDITY | REGULATORY T-CELLS | ECTO-ATPASE | NERVOUS-SYSTEM | CROHNS-DISEASE | EPITHELIAL-CELLS | Exosomes - metabolism | Lymphocytes - metabolism | Signal Transduction | Purines - metabolism | Humans | Apyrase - metabolism | Gastrointestinal Microbiome | Dysbiosis | Inflammatory Bowel Diseases - metabolism | Fecal Microbiota Transplantation | Hypoxia-Inducible Factor 1, alpha Subunit | Antigens, CD - metabolism | Macrophages - metabolism | Animals | Lymphocytes - immunology | Inflammatory Bowel Diseases - pathology | Adenosine - metabolism | Biomarkers | Receptors, Aryl Hydrocarbon - metabolism | Inflammatory Bowel Diseases - etiology | Inflammatory Bowel Diseases - therapy | Macrophages - immunology | Inflammatory bowel diseases | Care and treatment | Dendritic cells | Physiological aspects | B cells | Research | Purine nucleotides | Risk factors | Disease | Pathogenesis | Liver | Indole | Nucleotides | Bilirubin | Immunity | Machinery | Microparticles | Machinery and equipment | Microorganisms | Hemostasis | Aromatic compounds | Intestine | Biocompatibility | CD73 antigen | Hemostatics | Colon | Thromboembolism | Carbonyls | Immune system | Integration | Myeloid cells | Liver diseases | Cytokines | Scavenging | Health risks | Heredity | Carboxylic acids | Patients | Thrombosis | Immunological tolerance | Biological activity | Thrombophilia | Immunosuppression | Lymphocytes B | Ligands | Ectonucleotidase | Colitis | Aberration | Autoimmune diseases | Cancer | Integrity | lymphocytes | extracellular nucleotides | adaptive immunity | inflammatory bowel disease | adenosine | macrophages | innate immunity | inflammation
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2017, Volume 214, Issue 6, pp. 1737 - 1752
MicroRNA (miRNA)-mediated RNA interference regulates many immune processes, but how miRNA circuits orchestrate aberrant intestinal inflammation during... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | IL-1 | ULCERATIVE-COLITIS | IN-VIVO | MICE | IL-1-BETA | COLON | IMMUNOLOGY | MICRORNA EXPRESSION | BOWEL-DISEASE | Dextran Sulfate | Inflammation - pathology | Inflammasomes - metabolism | Antibodies - metabolism | Nanoparticles - chemistry | Colitis - genetics | Humans | Middle Aged | Colitis - pathology | MicroRNAs - metabolism | Monocytes - metabolism | Hematopoiesis | Interleukin-1beta - metabolism | Base Sequence | Colitis - chemically induced | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Adult | Neutrophils - metabolism | Disease Susceptibility | Intestines - pathology | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Mice, Inbred C57BL | Mice, Knockout | Receptors, CCR2 - metabolism | Animals | Inflammation - genetics | Myeloid Cells - metabolism | MicroRNAs - genetics | CC chemokine receptors | Animal models | Inflammatory bowel diseases | Nanoparticles | Cell activation | CCR2 protein | Intestine | Rodents | miRNA | Colon | Immune system | Immune response | RNA-mediated interference | Pharmacology | Inflammation | IL-1β | Ribonucleic acid--RNA | Disease control | Constraining | Inflammatory bowel disease | Monocytes | MicroRNAs | Mice | Aberration | Colitis | Monocyte chemoattractant protein 1 | Binding sites | 311 | 319
Journal Article
The FASEB Journal, ISSN 0892-6638, 11/2017, Volume 31, Issue 11, pp. 4707 - 4719
Endogenous circadian clocks regulate 24‐h rhythms of physiology and behavior. Circadian rhythm disruption (CRD) is suggested as a risk factor for inflammatory... 
epithelial cells | inflammatory bowel disease | necroptosis | Inflammatory bowel disease | Epithelial cells | Necroptosis | MPER2 | CROHNS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RISK | SLEEP DISTURBANCES | PROLIFERATION | BOWEL-DISEASE | CELL BIOLOGY | GENES | BIOLOGY | MICE | CELL-DIFFERENTIATION | CLOCK | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Protein-Tyrosine Kinases - metabolism | Homeostasis | Inflammatory Bowel Diseases - metabolism | Necrosis | Protein-Tyrosine Kinases - genetics | Caspases - metabolism | Period Circadian Proteins - genetics | Mice, Mutant Strains | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Cell Cycle Proteins - genetics | Cell Cycle Checkpoints - genetics | Indoles - pharmacology | Inflammatory Bowel Diseases - chemically induced | Nuclear Proteins - genetics | Cell Line | Caspases - genetics | Cell Cycle Proteins - metabolism | Nuclear Proteins - metabolism | Imidazoles - pharmacology | Circadian Rhythm | Tumor Necrosis Factor-alpha - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Period Circadian Proteins - metabolism | Tumor Necrosis Factor-alpha - adverse effects | Cell Cycle Checkpoints - drug effects | Mice | Mutation | Immunohistochemistry | Inflammatory bowel diseases | Mucosa | Bromodeoxyuridine | Kinases | Small intestine | Risk factors | Period 1 protein | Intestine | Rodents | Sodium sulfate | Cell cycle | Biological clocks | Circadian rhythms | Oral administration | Health risks | Caspase | Rhythm | Inflammation | siRNA | Risk taking | Rhythms | Circadian rhythm | Electron microscopy | Epithelium | Ribonucleic acid--RNA | Ablation | Dextran | Molecular modelling | Sodium | Protein kinase | Cell death | Colitis | Disruption | Sulfate | Apoptosis | Research
Journal Article
Journal of cellular physiology, ISSN 0021-9541, 2013, Volume 228, Issue 8, pp. 1793 - 1798
Journal Article
Oncogene, ISSN 1476-5594, 2018, Volume 38, Issue 7, pp. 1067 - 1079
Solute carrier family 7 member 2 (SLC7A2, also known as CAT2) is an inducible transporter of the semi-essential amino acid L-arginine (L-Arg), which has been... 
PLASMA ARGININE | ARGINASE | CHEMOKINES | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMINO-ACID TRANSPORTER-2 | BOWEL-DISEASE | CELL BIOLOGY | NITRIC-OXIDE PRODUCTION | COLITIS | ONCOLOGY | GENETICS & HEREDITY | L-ARGININE TRANSPORT | M1 MACROPHAGE ACTIVATION | HELICOBACTER-PYLORI | Inflammation - chemically induced | Inflammation - pathology | Colonic Neoplasms - genetics | Amino Acid Transport Systems, Basic - genetics | Colonic Neoplasms - chemically induced | Cell Transformation, Neoplastic - chemically induced | Neoplasm Proteins - metabolism | Azoxymethane - toxicity | Inflammatory Bowel Diseases - metabolism | Colonic Neoplasms - metabolism | Inflammation - metabolism | Cell Transformation, Neoplastic - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Inflammatory Bowel Diseases - chemically induced | Neoplasm Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Mice, Knockout | Animals | Colonic Neoplasms - pathology | Cell Line, Tumor | Inflammation - genetics | Mice | Amino Acid Transport Systems, Basic - metabolism | Cell Transformation, Neoplastic - pathology | Dextran | Gastrointestinal diseases | Colorectal cancer | Development and progression | Colitis | Comparative analysis | Diseases | Inflammatory bowel diseases | Colorectal carcinoma | Amino acids | Carcinogenesis | Interleukin 4 | Cell activation | Azoxymethane | Arginine | Interleukin 3 | Intestine | Rodents | Dextran sulfate | Bone marrow | Tumorigenesis | Chimeras | Colon | Genotypes | Phenotypes | Dysplasia | CD11b antigen | Wound healing | CCL3 protein | CCL4 protein | Inflammatory bowel disease | CXCL10 protein | Chemokines | Tumors | M2 macrophages | tumorigenesis | inflammation | colon cancer | SLC7A2
Journal Article
Pediatrics (Evanston), ISSN 1098-4275, 2012, Volume 130, Issue 4, pp. e794 - e803
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 5, p. e0125375
Aminosalicylates, corticosteroids and immunosuppressants are currently the therapeutic choices in inflammatory bowel diseases (IBD), however, with limited... 
IN-VITRO | PERMEABILITY | MODELS | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | MECHANISMS | INDUCTION | NF-KAPPA-B | EXPRESSION | BOWEL-DISEASE | Caco-2 Cells | Hydrogen Peroxide - toxicity | Inflammatory Bowel Diseases - drug therapy | Cell Survival - drug effects | Plant Extracts - chemistry | Intestinal Mucosa - metabolism | Antioxidants - chemistry | Epithelial Cells - drug effects | Humans | Epithelial Cells - pathology | NF-kappa B - metabolism | Inflammatory Bowel Diseases - metabolism | Intestinal Mucosa - drug effects | Plant Extracts - administration & dosage | Boswellia - chemistry | Inflammatory Bowel Diseases - pathology | Antioxidants - administration & dosage | Triterpenes - administration & dosage | Tight Junction Proteins - metabolism | Intestinal Mucosa - pathology | Anti-inflammatory drugs | Tumor necrosis factor | Analysis | Gastrointestinal diseases | Alternative medicine | Inflammation | Permeability | Health aspects | Immunosuppressive agents | Organic acids | Monomolecular films | Corticoids | Reactive oxygen species | Phosphorylation | Inflammatory bowel diseases | Hydrogen peroxide | Epithelial cells | Small intestine | Antioxidants | Proteins | Ultrasonic imaging | Intestine | Rodents | Remission | Tumor necrosis factor-TNF | Pretreatment | Sensors | NF-κB protein | Oxygen | Corticosteroids | Cytokines | Pharmacology | Tumor necrosis factor-α | Gene expression | Epithelium | Anti-inflammatory agents | Chromatography | Zonula occludens-1 protein | Medicine | Inflammatory bowel disease | Herbal medicine | Side effects | Acids | Monolayers | Biomarkers | Scientific imaging | Immunofluorescence | Mass spectrometry | Dismantling | Pharmaceuticals | Integrity
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2018, Volume 128, Issue 9, pp. 4044 - 4056
Neutrophil influx into the intestinal lumen is a critical response to infectious agents, but is also associated with severe intestinal damage observed in... 
MEDICINE, RESEARCH & EXPERIMENTAL | HEPOXILIN A | STEM-CELLS | CANNABINOID RECEPTOR | COLITIS | NEUTROPHIL TRANSEPITHELIAL MIGRATION | CB2 RECEPTOR | IN-VITRO EXPANSION | MODEL | MULTIDRUG-RESISTANCE | BOWEL-DISEASE | Endocannabinoids - metabolism | Intestinal Mucosa - metabolism | Humans | Homeostasis | Receptor, Cannabinoid, CB2 - genetics | Inflammatory Bowel Diseases - metabolism | Neutrophil Infiltration - physiology | Intestinal Mucosa - cytology | Biological Transport, Active | Inflammatory Bowel Diseases - pathology | Female | ATP Binding Cassette Transporter, Subfamily B - genetics | Disease Models, Animal | Cell Line | Inflammatory Bowel Diseases - prevention & control | Signal Transduction | Mice, Inbred C57BL | ATP Binding Cassette Transporter, Subfamily B - deficiency | ATP Binding Cassette Transporter, Subfamily B, Member 1 - deficiency | ATP Binding Cassette Transporter, Subfamily B - metabolism | ATP Binding Cassette Transporter, Subfamily B, Member 1 - genetics | Mice, Knockout | ATP Binding Cassette Transporter, Subfamily B, Member 1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Models, Biological | Mice | Multidrug Resistance-Associated Proteins - metabolism | Receptor, Cannabinoid, CB2 - deficiency | Prevention | Immune response | Gastrointestinal system | Neutrophils | Physiological aspects | Development and progression | Inflammation | Glycoproteins | Health aspects | Endocannabinoids | Ethanolamine | Inflammatory bowel diseases | Epithelial cells | Secretion | Multidrug resistance | Lipids | Infections | Recruitment | Inflammatory bowel disease | Proteins | Pathology | Intestine | Rodents | Stem cells | Cannabinoid CB2 receptors | Colon | P-Glycoprotein | Cell migration | Immune system
Journal Article