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Circulation research, ISSN 1524-4571, 2015, Volume 116, Issue 6, pp. 1074 - 1095
Hypertension is the most common modifiable risk factor for cardiovascular disease and death, and lowering blood pressure with antihypertensive drugs reduces target organ damage and prevents cardio... 
blood pressure | treatment | hypertension | interventional | drug | CARDIAC & CARDIOVASCULAR SYSTEMS | CAROTID-BODY CHEMORECEPTORS | PRESERVED EJECTION FRACTION | BAROREFLEX ACTIVATION THERAPY | ANGIOTENSIN-CONVERTING ENZYME | AMINOPEPTIDASE-A INHIBITORS | RENAL SYMPATHETIC DENERVATION | SOLUBLE EPOXIDE HYDROLASE | ALDOSTERONE-SYNTHASE INHIBITION | RECEPTOR-NEPRILYSIN INHIBITOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | AMBULATORY BLOOD-PRESSURE | Kidney Tubules - physiopathology | Antihypertensive Agents - pharmacology | Drugs, Investigational - pharmacology | Sympathetic Nervous System - drug effects | Humans | Drugs, Investigational - therapeutic use | Hypertension - drug therapy | Ion Channels - drug effects | Sympathetic Nervous System - physiopathology | Molecular Targeted Therapy | Receptors, Cell Surface - antagonists & inhibitors | High-Intensity Focused Ultrasound Ablation | Decompression, Surgical | Pre-Eclampsia - prevention & control | Multicenter Studies as Topic | Sympathectomy - methods | Drug Design | Mineralocorticoid Receptor Antagonists - pharmacology | Female | Models, Animal | Hypertension - enzymology | Hypertension - therapy | Stents | Kidney Tubules - drug effects | Arteriovenous Shunt, Surgical | Carotid Body - surgery | Comorbidity | Enzyme Inhibitors - pharmacology | Pressoreceptors - physiology | Therapies, Investigational | Renin-Angiotensin System - physiology | Clinical Trials as Topic | Antihypertensive Agents - therapeutic use | Electric Stimulation Therapy | Enzyme Inhibitors - therapeutic use | Hypertension - pathology | Mineralocorticoid Receptor Antagonists - therapeutic use | Randomized Controlled Trials as Topic | Pregnancy | Animals | Pre-Eclampsia - drug therapy | Kidney - innervation | Renin-Angiotensin System - drug effects | Baroreflex - physiology | Oxidative Stress - drug effects | Renal Artery - surgery
Journal Article
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 04/2016, Volume 310, Issue 8, pp. R697 - R706
We tested the hypothesis that an increase in the anti-inflammatory cholinergic pathway, when induced by pyridostigmine (PY), may modulate subtypes of... 
Myocardial infarction | T lymphocytes | Anticholinesterase drugs | Macrophage activation | Cholinergic anti-inflammatory reflex | IN-VITRO | PHYSIOLOGY | STIMULATION | BAROREFLEX SENSITIVITY | PATHWAY | HEART-RATE-VARIABILITY | AUTONOMIC FUNCTION | REGULATORY T-CELLS | NICOTINIC ACETYLCHOLINE-RECEPTOR | INFLAMMATORY RESPONSE | FAILURE | T-Lymphocytes, Regulatory - metabolism | Myocardium - immunology | Spleen - immunology | Rats, Inbred WKY | Male | Spleen - drug effects | Myocardial Infarction - immunology | T-Lymphocytes, Regulatory - immunology | Heart Rate - drug effects | Chemotaxis, Leukocyte - drug effects | Time Factors | Forkhead Transcription Factors - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | CD8-Positive T-Lymphocytes - metabolism | Myocardial Infarction - physiopathology | Blood Pressure - drug effects | Macrophages - immunology | Disease Models, Animal | Biomarkers - metabolism | Pyridostigmine Bromide - pharmacology | Anti-Inflammatory Agents - pharmacology | Cholinergic Neurons - metabolism | Ventricular Function, Left - drug effects | Myocardium - pathology | Cholinergic Neurons - drug effects | Myocardial Infarction - metabolism | Macrophages - metabolism | Phenotype | T-Lymphocytes, Regulatory - drug effects | Animals | Myocardial Infarction - drug therapy | Cholinesterase Inhibitors - pharmacology | Interleukin-2 Receptor alpha Subunit - metabolism | Spleen - metabolism | CD8-Positive T-Lymphocytes - drug effects | Macrophages - drug effects | CD8-Positive T-Lymphocytes - immunology | Macrophage Activation - drug effects | Cholinergic Neurons - pathology | Anti-inflammatory drugs | Heart attack | Euthanasia | Physiological aspects | Dosage and administration | Lymphocytes | Cholinesterase inhibitors | Health aspects | cholinergic anti-inflammatory reflex | anticholinesterase drugs | Translational Physiology | macrophage activation | myocardial infarction
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 09/2009, Volume 54, Issue 10, pp. 919 - 927
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 309, Issue 9, pp. H1479 - H1489
...) were continuously determined. With no hemodynamic effect at rest, fentanyl blockade during exercise attenuated both cardiac output and QL similar to 17... 
Dynamic exercise | Exercise pressor reflex | Afferent feedback | CARDIAC-OUTPUT | dynamic exercise | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | CAROTID BAROREFLEX CONTROL | exercise pressor reflex | afferent feedback | AFFERENTS CONTRIBUTE | METABOLIC-RESPONSE | SKELETAL-MUSCLE | ADVANCING AGE | ARTERIAL-BLOOD PRESSURE | FIBER-TYPE | PERIPHERAL VASCULAR DISEASE | Cardiac Output - physiology | Reflex - physiology | Humans | Analgesics, Opioid - pharmacology | Cardiovascular System - innervation | Reflex - drug effects | Male | Fentanyl - pharmacology | Young Adult | Regional Blood Flow - drug effects | Arterial Pressure - drug effects | Quadriceps Muscle - drug effects | Adult | Cardiovascular Physiological Phenomena - drug effects | Vasodilation - physiology | Autonomic Nervous System - drug effects | Cardiac Output - drug effects | Autonomic Nervous System - physiology | Cardiovascular System - drug effects | Arterial Pressure - physiology | Muscle, Skeletal - physiology | Femoral Artery - drug effects | Aging - physiology | Leg - blood supply | Muscle Contraction - physiology | Quadriceps Muscle - physiology | Aged | Femoral Artery - physiology | Vasodilation - drug effects | Regional Blood Flow - physiology | Complications and side effects | Care and treatment | Exercise | Aging | Influence | Research | Cardiovascular diseases | Health aspects | Call for Papers
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, p. e100536
Cannabinoid type 1 (CB1) receptors are expressed in the nervous and cardiovascular systems. In mice, CB1 receptor deficiency protects from metabolic... 
HEART | SLEEP | ACTIVATION | BAROREFLEX SENSITIVITY | MULTIDISCIPLINARY SCIENCES | CARDIOVASCULAR FUNCTION | ENDOCANNABINOID SYSTEM | ANGIOTENSIN-II | DIET-INDUCED OBESITY | BLOOD-PRESSURE | AUTONOMIC NERVOUS-SYSTEM | Wakefulness - drug effects | Diet, High-Fat - adverse effects | Male | Heart Rate - drug effects | Arterial Pressure - genetics | Respiration - drug effects | Arterial Pressure - drug effects | Behavior, Animal - drug effects | Cardiovascular Physiological Phenomena - drug effects | Respiration - genetics | Sleep - physiology | Wakefulness - genetics | Heart Rate - genetics | Circadian Rhythm - genetics | Behavior, Animal - physiology | Respiratory Physiological Phenomena - drug effects | Circadian Rhythm - physiology | Signal Transduction - genetics | Sleep - genetics | Sleep - drug effects | Adrenergic Antagonists - pharmacology | Mice, Knockout | Phenotype | Animals | Receptor, Cannabinoid, CB1 - genetics | Signal Transduction - drug effects | Wakefulness - physiology | Mice | Receptor, Cannabinoid, CB1 - deficiency | Circadian Rhythm - drug effects | Heart | Baroreceptors | Biomedical research | High fat diet | Receptors | Autonomic nervous system | Rodents | Surgery | Blood pressure | Vagus nerve | Heart diseases | Sleep and wakefulness | Reflexes | Cardiovascular system | Hypertension | Carbohydrates | Medical research | Obesity | Cannabinoid CB1 receptors | Metabolism | Heart rate | Signaling | Sleep | Hospitals | Diet | Breathing | Respiration | Laboratory animals | Endocrinology | Anomalies
Journal Article
Experimental physiology, ISSN 0958-0670, 2012, Volume 97, Issue 6, pp. 699 - 709
In this study, we evaluated whether the activation of endogenous angiotensin‐converting enzyme 2 (ACE2) would improve the cardiovascular autonomic dysfunction... 
VAGUS NERVE | SYSTEM | PHYSIOLOGY | NEUROPATHY | HEART-RATE CONTROL | REPERFUSION ARRHYTHMIAS | AMBIGUUS NA | SYMPATHETIC OUTFLOW | NUCLEUS-TRACTUS-SOLITARII | MYOCARDIAL DYSFUNCTION | BAROREFLEX CONTROL | Diabetes Mellitus, Experimental - drug therapy | Chemoreceptor Cells - drug effects | Rats, Wistar | Sympathetic Nervous System - drug effects | Cardiovascular Diseases - drug therapy | Male | Sympathetic Nervous System - physiopathology | Hyperglycemia - drug therapy | Heart Rate - drug effects | Peptidyl-Dipeptidase A - metabolism | Blood Pressure - drug effects | Blood Pressure - physiology | Parasympathetic Nervous System - physiopathology | Diabetes Mellitus, Experimental - metabolism | Hyperglycemia - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Heart - physiopathology | Tachycardia - drug therapy | Vasoconstrictor Agents - pharmacology | Cardiovascular Diseases - physiopathology | Autonomic Nervous System Diseases - metabolism | Cardiovascular Diseases - metabolism | Bradycardia - physiopathology | Rats | Chemoreceptor Cells - physiology | Tachycardia - physiopathology | Vasoconstriction - drug effects | Baroreflex - drug effects | Animals | Autonomic Nervous System Diseases - physiopathology | Autonomic Nervous System Diseases - drug therapy | Parasympathetic Nervous System - drug effects | Heart - drug effects | Baroreflex - physiology | Bradycardia - drug therapy | Chemoreflex | ACE2 activation | Baroreflex
Journal Article
Journal of Cardiovascular Pharmacology, ISSN 0160-2446, 03/2019, Volume 73, Issue 3, pp. 143 - 148
...–infusion hypertension. To examine effects of a putative preferential mitochondrial ROS scavenger in the brain of (mRen2... 
NADPH OXIDASE | SUPEROXIDE | CARDIAC & CARDIOVASCULAR SYSTEMS | RECEPTOR BLOCKADE | angiotensin peptides | mitochondria | spontaneous baroreflex function | TRANSGENIC RATS | ANGIOTENSIN-II | MECHANISMS | reactive oxygen species | intracerebroventricular | PHARMACOLOGY & PHARMACY | MEDULLA | hypertension | BRAIN | BAROREFLEX | Free Radical Scavengers - pharmacology | Antihypertensive Agents - pharmacology | Reactive Oxygen Species - metabolism | Tetrazoles - pharmacology | Sympathetic Nervous System - drug effects | Vagus Nerve - drug effects | Hypertension - drug therapy | Male | Peptide Fragments - pharmacology | Sympathetic Nervous System - physiopathology | Angiotensin II Type 1 Receptor Blockers - pharmacology | Brain - metabolism | Heart Rate - drug effects | Renin - genetics | Piperidines - pharmacology | Arterial Pressure - drug effects | Organophosphorus Compounds - pharmacology | Angiotensin I - pharmacology | Hypertension - genetics | Disease Models, Animal | Heart - innervation | Rats, Transgenic | Brain - physiopathology | Mitochondria - metabolism | Mitochondria - drug effects | Hypertension - physiopathology | Hypertension - metabolism | Vagus Nerve - physiopathology | Baroreflex - drug effects | Brain - drug effects | Animals | Benzimidazoles - pharmacology | Drug Combinations | Blood pressure | Dosage and administration | Research | Drug therapy | Candesartan
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 309, Issue 9, pp. H1579 - H1590
Vagal nerve stimulation (VNS) has been shown to have antiarrhythmic effects, but many of these benefits were demonstrated in the setting of vagal nerve decentralization... 
Electrophysiology | Parasympathetic | Afferent cardiac neurotransmission | Vagotomy | VAGUS NERVE | ARRHYTHMIAS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | afferent cardiac neurotransmission | BAROREFLEX SENSITIVITY | vagotomy | VENTRICULAR-FIBRILLATION | RECOVERY INTERVALS | REPERFUSION INJURY | parasympathetic | electrophysiology | HEART-RATE-VARIABILITY | ACUTE MYOCARDIAL-INFARCTION | PERIPHERAL VASCULAR DISEASE | MODULATION | RABBIT HEART | Ventricular Function - physiology | Efferent Pathways - physiology | Heart - physiology | Male | Hemodynamics - physiology | Vagus Nerve Stimulation | Heart Rate - drug effects | Swine | Heart Rate - physiology | Female | Parasympathetic Nervous System - physiology | Action Potentials - drug effects | Heart - innervation | Ventricular Function - drug effects | Atropine - pharmacology | Vagus Nerve - surgery | Parasympatholytics - pharmacology | Action Potentials - physiology | Vagus Nerve - physiology | Animals | Afferent Pathways - physiology | Parasympathetic Nervous System - drug effects | Heart - drug effects | Heart Ventricles - innervation | Hemodynamics - drug effects | Sus scrofa | Heart Ventricles - drug effects | Usage | Analysis | Anti-arrhythmia drugs | Influence | Vagus nerve stimulation | Neural transmission | Research | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
Journal Article