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Journal of Neurotrauma, ISSN 0897-7151, 04/2010, Volume 27, Issue 4, pp. 739 - 751
Spinal cord injury results in disruption of the cord microstructure, which is followed by inflammation leading to additional deterioration. Perivascular... 
Glia cell response to injury | Extracellular matrix | Inflammation | Spinal cord injury | spinal cord injury | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | COLLAGEN-IV | GLIAL SCAR | COMPRESSION INJURY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | REPAIR | inflammation | EXTRAVASATION | glia cell response to injury | REACTIVE ASTROCYTES | CENTRAL-NERVOUS-SYSTEM | EXPRESSION | extracellular matrix | CRITICAL CARE MEDICINE | Immunohistochemistry | Monocytes - cytology | Spinal Cord Injuries - complications | Blood Vessels - pathology | Astrocytes - pathology | Blood-Brain Barrier - physiopathology | Extracellular Matrix - metabolism | Myelitis - etiology | Gliosis - physiopathology | Basement Membrane - pathology | Chemotaxis, Leukocyte - physiology | Spinal Cord Injuries - pathology | Blood Vessels - physiopathology | Gliosis - pathology | Spinal Cord - pathology | Female | Monocytes - physiology | Gliosis - etiology | Macrophages - physiology | Endothelial Cells - metabolism | Myelitis - physiopathology | Mice, Inbred C57BL | Myelitis - pathology | Macrophages - cytology | Spinal Cord - blood supply | Basement Membrane - physiopathology | Blood-Brain Barrier - pathology | Animals | Mice | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Endothelial Cells - pathology | Extracellular Matrix - pathology | Astrocytes - metabolism | Physiological aspects | Care and treatment | Spinal cord injuries | Models | Patient outcomes | Neuroglia | Membranes | Cellular biology | Rodents | Inflammatory diseases
Journal Article
The Journal of Comparative Neurology, ISSN 0021-9967, 04/2002, Volume 445, Issue 4, pp. 308 - 324
After spinal cord injury (SCI), the absence of an adequate blood supply to injured tissues has been hypothesized to contribute to the lack of regeneration. In... 
blood vessels | macrophages | laminin | nerve regeneration | astrocytes | neovascularization | Blood vessels | Laminin | Neovascularization | Macrophages | Astrocytes | Nerve regeneration | REGENERATION | SECONDARY INJURY | MONOCLONAL-ANTIBODY | NEUROSCIENCES | IN-VITRO | BRAIN ANGIOGENESIS | ZOOLOGY | NEURITE OUTGROWTH | ENDOTHELIAL-CELLS | REACTIVE ASTROCYTES | CENTRAL-NERVOUS-SYSTEM | EXPRESSION | Blood Vessels - metabolism | Blood Vessels - pathology | Nerve Regeneration - physiology | Basement Membrane - pathology | Neovascularization, Pathologic - pathology | Spinal Cord Injuries - pathology | Blood Vessels - physiopathology | Spinal Cord - pathology | Rats, Inbred F344 - metabolism | Antigens, Surface - metabolism | Recovery of Function - physiology | Female | Neovascularization, Pathologic - physiopathology | Rats, Inbred F344 - anatomy & histology | Collagen Type IV - metabolism | Spinal Cord Injuries - metabolism | GAP-43 Protein - metabolism | Endothelium, Vascular - physiopathology | Rats | Antibodies, Monoclonal | Spinal Cord - blood supply | Basement Membrane - metabolism | Basement Membrane - physiopathology | Animals | Rats, Inbred F344 - injuries | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Neovascularization, Pathologic - metabolism | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Wound Healing - physiology | Laminin - metabolism | Neurofilament Proteins - metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e70288
Cerebral palsy (CP) is a static encephalopathy occurring when a lesion to the developing brain results in disordered movement and posture. Patients present... 
MEDIATED GENE-THERAPY | MULTIDISCIPLINARY SCIENCES | EXTRAJUNCTIONAL ACETYLCHOLINE-RECEPTORS | ACTIVE ZONES | MUSCULAR-DYSTROPHY | MAMMALIAN MUSCLE | UTROPHIN-DEFICIENT MICE | EXTRACELLULAR-MATRIX | POSTSYNAPTIC ABNORMALITIES | HUMAN SKELETAL-MUSCLE | VERTEBRATE NEUROMUSCULAR-JUNCTION | Basement Membrane - ultrastructure | Prospective Studies | Membrane Glycoproteins - metabolism | Neuromuscular Junction - metabolism | Cerebral Palsy - metabolism | Humans | Cerebral Palsy - physiopathology | Male | Muscle, Skeletal - metabolism | Case-Control Studies | Acetylcholinesterase - genetics | Synapses - metabolism | Female | Child | Gene Expression | Cerebral Palsy - pathology | Receptors, Cholinergic - metabolism | Muscle, Skeletal - ultrastructure | Synapses - ultrastructure | Microscopy, Electron | Neuromuscular Junction - physiopathology | Basement Membrane - metabolism | Nerve Tissue Proteins - genetics | Neuromuscular Junction - ultrastructure | Membrane Glycoproteins - genetics | Laminin - genetics | Nerve Tissue Proteins - metabolism | Basement Membrane - physiopathology | Muscle, Skeletal - physiopathology | Adolescent | Laminin - metabolism | Acetylcholinesterase - metabolism | Cerebral Palsy - genetics | Receptors, Cholinergic - genetics | Cerebral palsy | Medical research | Telomeres | Laminin | Analysis | Myosin | Medicine, Experimental | Muscle proteins | Drugs | Brain | Early intervention | Biomedical research | Motor task performance | Bone surgery | Posture | Mitochondria | Esterase | Scoliosis | Encephalopathy | Basal lamina | Children | Paralysis | Localization | Dietitians | Myogenin | Maturation | Muscles | Electron microscopy | Gene expression | Patients | Studies | Brain research | Transmission electron microscopy | Biopsy | Orthopedics | Acetylcholine receptors | Neuromuscular junctions | Disruption | Position (location) | Synapses | Acetylcholine
Journal Article