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Journal Article
The Plant Cell, ISSN 1040-4651, 8/2013, Volume 25, Issue 8, pp. 3117 - 3132
Arabidopsis thaliana plants fend off insect attack by constitutive and inducible production of toxic metabolites, such as glucosinolates (GSs). A triple mutant... 
Transcriptional regulatory elements | Insect larvae | RESEARCH ARTICLES | Genes | Glucosinolates | Biosynthesis | Gene expression regulation | Herbivores | Phytophagous insects | Genotypes | Plant cells | HERBIVORES | PLUTELLA-XYLOSTELLA | BIOCHEMISTRY & MOLECULAR BIOLOGY | PLANT SCIENCES | CELL BIOLOGY | INDUCED RESISTANCE | THALIANA | DEFENSE SIGNALING PATHWAYS | MYB | GENE-EXPRESSION | ACCUMULATION | PROTEINS | JASMONATE RESPONSES | Herbivory - physiology | Trans-Activators - chemistry | Promoter Regions, Genetic - genetics | Arabidopsis Proteins - metabolism | Biosynthetic Pathways - genetics | Gene Expression Regulation, Plant | Trans-Activators - genetics | Insecta - physiology | Protein Structure, Tertiary | Arabidopsis Proteins - genetics | Plant Leaves - parasitology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - chemistry | Mutation - genetics | Feeding Behavior - physiology | Arabidopsis - metabolism | Arabidopsis - genetics | Glucosinolates - biosynthesis | Animals | Arabidopsis Proteins - chemistry | Plant Leaves - metabolism | Models, Biological | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Protein Binding | Arabidopsis - parasitology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Trans-Activators - metabolism | Arabidopsis thaliana | Glucose metabolism | Transcription factors | Physiological aspects | Genetic aspects | Research
Journal Article
European Urology, ISSN 0302-2838, 2015, Volume 69, Issue 4, pp. 646 - 657
Abstract Context Renal cancer is a common urologic malignancy, and therapeutic options for metastatic disease are limited. Most clear cell renal cell... 
Urology | Hypoxia | Hypoxia-inducible transcription factors | Clear cell renal cell carcinoma | von Hippel-Lindau | HIPPEL-LINDAU DISEASE | Clear cell renal cell carcinoma von Hippel-Lindau | CELL CARCINOMA | OXYGEN-SENSING PATHWAY | GENE-EXPRESSION | UROLOGY & NEPHROLOGY | TUMOR-SUPPRESSOR PROTEIN | SUSCEPTIBILITY LOCUS | FACTOR (HIF)-1-ALPHA | HIF-ALPHA | ENDOTHELIAL GROWTH-FACTOR | GENOME-WIDE ASSOCIATION | Up-Regulation | Kidney Neoplasms - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Prognosis | Signal Transduction | Carcinoma, Renal Cell - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Renal Cell - genetics | Tumor Microenvironment | Kidney Neoplasms - metabolism | Oxygen - metabolism | Cell Hypoxia | Carcinoma, Renal Cell - metabolism | Animals | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | Carcinoma, Renal Cell - therapy | Kidney Neoplasms - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Kidney Neoplasms - therapy | Medical colleges | DNA binding proteins | Metastasis | Health aspects | Medical genetics | Cancer | Analysis | Laws, regulations, etc | Genetic research | Imports | Genetic polymorphisms
Journal Article
Journal Article
International Journal of Cancer, ISSN 0020-7136, 05/2018, Volume 142, Issue 9, pp. 1817 - 1828
The factors involved in cell differentiation have recently garnered interest for their role in inhibition of pathogenesis in various tumors. However, their... 
hypoxia | glioblastoma | NeuroD2 | miR‐210 | p53 | miR-210 | STEM-CELLS | CONVERSION | PATTERNS | FIBROBLASTS | BREAST-CANCER | ONCOLOGY | AGGRESSIVENESS | CENTRAL-NERVOUS-SYSTEM | PROTEINS | EXPRESSION | Cell Hypoxia - physiology | Humans | Middle Aged | Brain Neoplasms - pathology | Tumor Microenvironment | Male | MicroRNAs - metabolism | Brain Neoplasms - metabolism | Tumor Suppressor Protein p53 - genetics | Young Adult | Basic Helix-Loop-Helix Transcription Factors - metabolism | Glioblastoma - genetics | Neuropeptides - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | Adult | Female | Glioblastoma - metabolism | Neuropeptides - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Down-Regulation | Tumor Suppressor Protein p53 - metabolism | Brain Neoplasms - genetics | Neuropeptides - metabolism | Glioblastoma - pathology | Aged | MicroRNAs - genetics | Tumor proteins | Cell differentiation | Neurons | Glioblastoma multiforme | Analysis | Genomics | Cell proliferation | Pathogenesis | p53 Protein | Glioblastoma | Differentiation (biology) | AKT protein | Genomes | Gelatin | Extracellular matrix | Inhibition | Phenotypes | Cytokines | MiRNA | Patients | Spheroids | 1-Phosphatidylinositol 3-kinase | Signaling | Tumor necrosis factor | Biomarkers | Tumor suppressor genes | Hypoxia | Cell migration | Tumors | Cancer | Apoptosis
Journal Article
Neuron, ISSN 0896-6273, 04/2008, Volume 58, Issue 1, pp. 52 - 64
Expression of the Notch effector gene is required for maintenance of neural progenitors in the embryonic brain, but persistent and high levels of expression... 
STEMCELL | DEVBIO | MOLNEURO | STEM-CELLS | MAMMALIAN HAIRY | DELTA-HOMOLOG | LOOP-HELIX FACTORS | NEGATIVE AUTOREGULATION | HES1 EXPRESSION | CENTRAL-NERVOUS-SYSTEM | SOMITE SEGMENTATION | PRESOMITIC MESODERM | NEUROSCIENCES | FEEDBACK LOOP | Transcription Factor HES-1 | Intercellular Signaling Peptides and Proteins - biosynthesis | Neurons - cytology | Receptors, Notch - genetics | Stem Cells - cytology | Intercellular Signaling Peptides and Proteins - physiology | Receptors, Notch - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | Nerve Tissue Proteins - biosynthesis | Neurons - physiology | Female | Cell Differentiation - physiology | Basic Helix-Loop-Helix Transcription Factors - physiology | Nerve Tissue Proteins - physiology | Basic Helix-Loop-Helix Transcription Factors - genetics | Homeodomain Proteins - biosynthesis | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Receptors, Notch - physiology | Mice, Transgenic | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Pregnancy | Animals | Biological Clocks - physiology | Signal Transduction - physiology | Stem Cells - physiology | Mice | Homeodomain Proteins - physiology | Heparan sulfate | Proteins | Signal transduction | Rodents | Cell division | Ligands | Protein expression | Genomes | Gene expression
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2014, Volume 289, Issue 3, pp. 1866 - 1875
Journal Article
Industrial Crops & Products, ISSN 0926-6690, 02/2019, Volume 128, pp. 115 - 125
Artemisinin based combination therapies are still an extensively used remedy for the cure of Malaria. Although artemisinin biosynthetic pathway has been fully... 
Transcription factors | Artemisia annua | Malaria | AaMYC2-Like | Basic helix loop helix | Artemisinin | ANTIMALARIAL-DRUG | AGRONOMY | ACID | INITIATION | TRICHOME DEVELOPMENT | AGRICULTURAL ENGINEERING | GENE | METABOLISM | AAWRKY1 | PROTEINS | EXPRESSION | JASMONATE
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2014, Volume 111, Issue 28, pp. 10359 - 10364
Journal Article
Cell Stem Cell, ISSN 1934-5909, 2010, Volume 6, Issue 6, pp. 578 - 590
After central nervous system (CNS) demyelination—such as occurs during multiple sclerosis—there is often spontaneous regeneration of myelin sheaths, mainly by... 
STEMCELL | PDGF ALPHA-RECEPTOR | MULTIPLE-SCLEROSIS LESIONS | NEURAL PROGENITOR | REMYELINATION OCCURS | EMBRYONIC SPINAL-CORD | CENTRAL-NERVOUS-SYSTEM | INTRASPINAL INJECTION | RAT-BRAIN | NG2 PROTEOGLYCAN | PRECURSOR CELLS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Oligodendroglia - metabolism | Demyelinating Diseases - genetics | Astrocytes - pathology | Central Nervous System - pathology | Demyelinating Diseases - metabolism | Central Nervous System - surgery | Lysophosphatidylcholines - administration & dosage | Multiple Sclerosis - physiopathology | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | Nerve Tissue Proteins - biosynthesis | Female | Demyelinating Diseases - chemically induced | Adult Stem Cells - pathology | Demyelinating Diseases - pathology | Receptor, Fibroblast Growth Factor, Type 3 - biosynthesis | Disease Models, Animal | Multiple Sclerosis - metabolism | Receptor, Platelet-Derived Growth Factor alpha - biosynthesis | Demyelinating Diseases - physiopathology | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Cells, Cultured | Mice, Transgenic | Multiple Sclerosis - genetics | Schwann Cells - pathology | Oligodendrocyte Transcription Factor 2 | Schwann Cells - metabolism | Nerve Regeneration | Nerve Tissue Proteins - genetics | Oligodendroglia - pathology | Adult Stem Cells - metabolism | Animals | Microscopy | Receptor, Platelet-Derived Growth Factor alpha - genetics | Central Nervous System - drug effects | Multiple Sclerosis - pathology | Mice | Integrases - genetics | Astrocytes - metabolism
Journal Article