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1. Full Text Epigenetic dysregulation of ACE2 and interferon-regulated genes might suggest increased COVID-19 susceptibility and severity in lupus patients
by Sawalha, Amr H and Zhao, Ming and Coit, Patrick and Lu, Qianjin
Clinical immunology (Orlando, Fla.), ISSN 1521-6616, 06/2020, Volume 215, pp. 108410 - 108410
Lupus | COVID-19 | ACE2 | SARS-CoV-2 | Epigenetics | Interferon | Methylation | Life Sciences & Biomedicine | Immunology | Science & Technology | Betacoronavirus - pathogenicity | Lupus Erythematosus, Systemic - complications | Pandemics | Epigenesis, Genetic | Humans | NF-kappa B - immunology | Oxidative Stress - immunology | Host-Pathogen Interactions - immunology | Betacoronavirus - immunology | Peptidyl-Dipeptidase A - genetics | DNA Methylation | Pneumonia, Viral - genetics | CD11a Antigen - genetics | Coronavirus Infections - epidemiology | Pneumonia, Viral - epidemiology | Pneumonia, Viral - complications | Lupus Erythematosus, Systemic - immunology | Coronavirus Infections - complications | Spike Glycoprotein, Coronavirus - immunology | Cytokines - genetics | Cytokines - immunology | Genetic Predisposition to Disease | Spike Glycoprotein, Coronavirus - genetics | Receptors, KIR - genetics | Signal Transduction | Lupus Erythematosus, Systemic - epidemiology | Oxidative Stress - genetics | Coronavirus Infections - genetics | Interferon Regulatory Factors - genetics | Viremia - genetics | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Disease Progression | Angiotensin-Converting Enzyme 2 | Viremia - complications | Interferon Regulatory Factors - immunology | Host-Pathogen Interactions - genetics | NF-kappa B - genetics | Lupus Erythematosus, Systemic - genetics | Protein Binding | Viremia - epidemiology | Viremia - immunology | CD11a Antigen - immunology | Peptidyl-Dipeptidase A - immunology | Receptors, KIR - immunology | Virus diseases | Epigenetic inheritance | Genes | Genetic research | Disease susceptibility | Biological response modifiers | T cells | Health aspects | Risk factors | Index Medicus
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2. Full Text Irisin modulates genes associated with severe coronavirus disease (COVID-19) outcome in human subcutaneous adipocytes cell culture
by de Oliveira, Miriane and De Sibio, Maria Teresa and Mathias, Lucas Solla and Rodrigues, Bruna Moretto and Sakalem, Marna Eliana and Nogueira, Célia Regina
Molecular and cellular endocrinology, ISSN 0303-7207, 09/2020, Volume 515, pp. 110917 - 110917
Irisin | ADAM10 | SARS-CoV-2 | TRIB3 | FURIN | Adipose tissue | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Amyloid Precursor Protein Secretases - genetics | Humans | ADAM10 Protein - metabolism | Adipocytes - drug effects | COVID-19 | Histone Acetyltransferases - metabolism | Peptidyl-Dipeptidase A - metabolism | Gene Ontology | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | Histone Deacetylase 2 - genetics | Fibronectins - pharmacology | Signal Transduction | rab1 GTP-Binding Proteins - metabolism | Membrane Proteins - genetics | Cell Cycle Proteins - metabolism | Repressor Proteins - genetics | Betacoronavirus - metabolism | Toll-Like Receptor 3 - metabolism | Angiotensin-Converting Enzyme 2 | Amyloid Precursor Protein Secretases - metabolism | Coronavirus Infections - virology | Models, Biological | Betacoronavirus - genetics | Jumonji Domain-Containing Histone Demethylases - metabolism | Sirtuin 1 - metabolism | Pandemics | ADAM10 Protein - genetics | Histone Acetyltransferases - genetics | Adipocytes - cytology | Sirtuin 1 - genetics | Peptidyl-Dipeptidase A - genetics | Furin - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Toll-Like Receptor 3 - genetics | Nuclear Proteins - genetics | Furin - metabolism | Obesity - virology | Molecular Sequence Annotation | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Jumonji Domain-Containing Histone Demethylases - genetics | Fibronectins - metabolism | rab1 GTP-Binding Proteins - genetics | Gene Expression Regulation - drug effects | Adipocytes - metabolism | Fibronectins - genetics | Histone Deacetylase 2 - metabolism | Pneumonia, Viral - virology | Virus diseases | Enzymes | Anopheles | Genes | Genetic aspects | Coronaviruses | Health aspects | Cells | Risk factors | Index Medicus
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3. Full Text Genomewide Association Study of Severe Covid-19 with Respiratory Failure
by Ellinghaus, David and Degenhardt, Frauke and Buti, Maria and Albillos, Agustin and Prati, Daniele and Asselta, Rosanna and Milani, Chiara and Kassens, Jan and May, Sandra and Wienbrandt, Lars and Uellendahl-Werth, Florian and Zheng, Tenghao and Yi, Xiaoli and Rodriguez-Frias, Francisco and Zanella, Alberto and Bandera, Alessandra and Protti, Alessandro and Aghemo, Alessio and Lleo, Ana and Biondi, Andrea and Caballero-Garralda, Andrea and Latiano, Anna and Fracanzani, Anna Ludovica and Peschuck, Anna and Pesenti, Antonio and Mateos, Beatriz and Jimenez, Beatriz Nafria and Quereda, Carmen and Paccapelo, Cinzia and Angelini, Claudio and Cea, Cristina and Solier, Aurora and Sandoval, Elena and Sanchez, Felix Garcia and Ceriotti, Ferruccio and Martinelli-Boneschi, Filippo and Blasi, Francesco and Tellez, Luis and Blanco-Grau, Albert and Hemmrich-Stanisak, Georg and Grasselli, Giacomo and Cardamone, Giulia and Aneli, Serena and Kurihara, Hayato and ElAbd, Hesham and Galvan-Femenia, Ivan and Erdmann, Jeanette and Ferrusquia-Acosta, Jose and Garcia-Etxebarria, Koldo and Santoro, Luigi and Roade, Luisa and Ruhlemann, Malte C and Carrabba, Maria and Riveiro-Barciela, Mar and Valsecchi, Maria G and Hernandez-Tejero, Maria and Acosta-Herrera, Marialbert and D'Angio, Mariella and Baldini, Marina and Cazzaniga, Marina and Schulzky, Martin and Cecconi, Maurizio and Rodriguez-Gandia, Miguel and Bocciolone, Monica and Miozzo, Monica and Montano, Nicola and Braun, Nicole and Sacchi, Nicoletta and Faverio, Paola and Preatoni, Paoletta and Bonfanti, Paolo and Omodei, Paolo and Tentorio, Paolo and Castro, Pedro and Rodrigues, Pedro M and Blandino, Aaron and de Cid, Rafael and Ferrer, Ricard and Nieto, Rosa and Goerg, Siegfried and Badalamenti, Salvatore and Marsal, Sara and Matullo, Giuseppe and Juzenas, Simonas and Aliberti, Stefano and Monzani, Valter and Moreno, Victor and Lenz, Tobias L and Pumarola, Tomas and Rimoldi, Valeria and Bosari, Silvano and Albrecht, Wolfgang and Peter, Wolfgang and D'Amato, Mauro and Duga, Stefano and Banales, Jesus M and Hov, Johannes R and Folseraas, Trine and Franke, Andre and Karlsen, Tom H and The Severe Covid-19 GWAS Group and Severe Covid-19 GWAS Grp and Severe Covid-19 GWAS Group
The New England journal of medicine, ISSN 0028-4793, 10/2020, Volume 383, Issue 16, pp. 1522 - 1534
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Multigene Family | Pandemics | Humans | Middle Aged | Respiratory Insufficiency - etiology | Male | Genetic Loci | Case-Control Studies | COVID-19 | Chromosomes, Human, Pair 9 - genetics | SARS-CoV-2 | Pneumonia, Viral - genetics | Pneumonia, Viral - complications | ABO Blood-Group System - genetics | Betacoronavirus | Coronavirus Infections - complications | Female | Genetic Predisposition to Disease | Genome-Wide Association Study | Coronavirus Infections - genetics | Spain | Respiratory Insufficiency - genetics | Italy | Aged | Polymorphism, Single Nucleotide | Chromosomes, Human, Pair 3 - genetics | Disease susceptibility | Genetic aspects | Respiratory insufficiency | Epidemics | Blood group A | Disease | Pathogenesis | Association analysis | Single-nucleotide polymorphism | Ethics | Genetic factors | Blood groups | Drug dosages | Age | Ventilators | Medical research | Blood & organ donations | Susceptibility | Mortality | Phlebotomy | ABO system | RNA polymerase | Coronaviruses | Chromosome 3 | Severe acute respiratory syndrome coronavirus 2 | Blood group O | Hospitals | Chromosome 9 | Quality control | Informed consent | CCR9 protein | Index Medicus | Abridged Index Medicus | Medicin och hälsovetenskap
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4. Full Text Genetic gateways to COVID‐19 infection: Implications for risk, severity, and outcomes
by Debnath, Monojit and Banerjee, Moinak and Berk, Michael
The FASEB journal, ISSN 0892-6638, 07/2020, Volume 34, Issue 7, pp. 8787 - 8795
ACE | cytokine storm | genetics | COVID‐19 | SARS‐CoV‐2 | immunity | HLA | COVID-19 | SARS-CoV-2 | Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Metagenomics | Pandemics | Prognosis | Humans | Peptidyl-Dipeptidase A - physiology | Risk | Disease Resistance - immunology | HLA Antigens - genetics | Mutation, Missense | Receptors, Virus - genetics | Host-Pathogen Interactions - immunology | Receptors, Virus - physiology | Coronavirus Infections - transmission | Genetic Variation | Pneumonia, Viral - transmission | Peptidyl-Dipeptidase A - genetics | Pneumonia, Viral - genetics | Betacoronavirus - physiology | Pneumonia, Viral - complications | Coronavirus Infections - complications | Genetic Predisposition to Disease | Toll-Like Receptors - immunology | Cytokine Release Syndrome - genetics | Coronavirus Infections - genetics | HLA Antigens - immunology | Treatment Outcome | Inflammation | Continental Population Groups - genetics | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Angiotensin-Converting Enzyme 2 | Complement Activation - genetics | Cytokine Release Syndrome - etiology | Host-Pathogen Interactions - genetics | Toll-Like Receptors - genetics | Quantitative Trait Loci | Index Medicus
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5. Full Text Development of epitope‐based peptide vaccine against novel coronavirus 2019 (SARS‐COV‐2): Immunoinformatics approach
by Bhattacharya, Manojit and Sharma, Ashish R and Patra, Prasanta and Ghosh, Pratik and Sharma, Garima and Patra, Bidhan C and Lee, Sang‐Soo and Chakraborty, Chiranjib
Journal of medical virology, ISSN 0146-6615, 06/2020, Volume 92, Issue 6, pp. 618 - 631
epitopes | vaccine | immunoinformatics | SARS‐COV‐2 | Life Sciences & Biomedicine | Science & Technology | Virology | Betacoronavirus - pathogenicity | Toll-Like Receptor 5 - genetics | Epitopes, B-Lymphocyte - chemistry | Humans | Pandemics - prevention & control | Epitopes, T-Lymphocyte - genetics | Epitopes, B-Lymphocyte - immunology | Middle East Respiratory Syndrome Coronavirus - immunology | Betacoronavirus - immunology | COVID-19 | Epitopes - immunology | SARS-CoV-2 | Epitopes, B-Lymphocyte - genetics | Binding Sites | Amino Acid Sequence | Computational Biology - methods | Spike Glycoprotein, Coronavirus - genetics | Coronavirus Infections - immunology | SARS Virus - pathogenicity | Toll-Like Receptor 5 - immunology | Epitopes, T-Lymphocyte - chemistry | Histocompatibility Antigens Class II - immunology | Coronavirus Infections - virology | Middle East Respiratory Syndrome Coronavirus - genetics | COVID-19 Vaccines | Epitopes - chemistry | Molecular Docking Simulation | Betacoronavirus - genetics | Spike Glycoprotein, Coronavirus - chemistry | Coronavirus Infections - prevention & control | SARS Virus - immunology | SARS Virus - genetics | Spike Glycoprotein, Coronavirus - immunology | Protein Interaction Domains and Motifs | Epitopes, T-Lymphocyte - immunology | Protein Structure, Tertiary | Vaccines, Subunit | Histocompatibility Antigens Class I - immunology | Histocompatibility Antigens Class I - genetics | Histocompatibility Antigens Class II - chemistry | Epitopes - genetics | Histocompatibility Antigens Class I - chemistry | Toll-Like Receptor 5 - chemistry | Pneumonia, Viral - immunology | Middle East Respiratory Syndrome Coronavirus - pathogenicity | Pneumonia, Viral - prevention & control | Viral Vaccines - chemistry | Protein Binding | Histocompatibility Antigens Class II - genetics | Pneumonia, Viral - virology | Viral Vaccines - immunology | Antigens | TLR5 protein | Peptides | Severe acute respiratory syndrome | Viral diseases | Glycoproteins | Coronaviruses | Vaccines | Epitopes | Major histocompatibility complex | Immunogenicity | Toll-like receptors | Spike glycoprotein | Index Medicus
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6. Full Text COVID-19 infections are also affected by human ACE1 D/I polymorphism
by Delanghe, Joris R and Speeckaert, Marijn M and De Buyzere, Marc L
Clinical chemistry and laboratory medicine, ISSN 1434-6621, 06/2020, Volume 58, Issue 7, pp. 1125 - 1126
COVID-19 | polymorphism | plasma proteins | angiotensin-converting enzyme | Life Sciences & Biomedicine | Medical Laboratory Technology | Science & Technology | Betacoronavirus - pathogenicity | Pandemics | Prevalence | Humans | Coronavirus Infections - genetics | Haptoglobins - genetics | INDEL Mutation - genetics | Coronavirus Infections - mortality | Peptidyl-Dipeptidase A - genetics | SARS-CoV-2 | Pneumonia, Viral - genetics | Vitamin D-Binding Protein - genetics | Hemochromatosis Protein - genetics | Peptidyl-Dipeptidase A - metabolism | Polymorphism, Genetic - genetics | Pneumonia, Viral - mortality | Betacoronavirus - genetics | Complement C3 - genetics | Polymorphism | Index Medicus
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7. Full Text SARS-CoV-2 Reverse Genetics Reveals a Variable Infection Gradient in the Respiratory Tract
by Hou, Yixuan J and Okuda, Kenichi and Edwards, Caitlin E and Martinez, David R and Asakura, Takanori and Dinnon, Kenneth H and Kato, Takafumi and Lee, Rhianna E and Yount, Boyd L and Mascenik, Teresa M and Chen, Gang and Olivier, Kenneth N and Ghio, Andrew and Tse, Longping V and Leist, Sarah R and Gralinski, Lisa E and Schäfer, Alexandra and Dang, Hong and Gilmore, Rodney and Nakano, Satoko and Sun, Ling and Fulcher, M. Leslie and Livraghi-Butrico, Alessandra and Nicely, Nathan I and Cameron, Mark and Cameron, Cheryl and Kelvin, David J and de Silva, Aravinda and Margolis, David M and Markmann, Alena and Bartelt, Luther and Zumwalt, Ross and Martinez, Fernando J and Salvatore, Steven P and Borczuk, Alain and Tata, Purushothama R and Sontake, Vishwaraj and Kimple, Adam and Jaspers, Ilona and O’Neal, Wanda K and Randell, Scott H and Boucher, Richard C and Baric, Ralph S
Cell (Cambridge), ISSN 0092-8674, 07/2020, Volume 182, Issue 2, pp. 429 - 446.e14
COVID-19 | ACE2 | SARS-CoV-2 | reverse genetics | respiratory tropism | infectious clone | primary cells | reporter virus | nasal infection | neutralization assay | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Betacoronavirus - pathogenicity | Respiratory System - pathology | Pandemics | Humans | Middle Aged | Reverse Genetics - methods | Virulence | Cystic Fibrosis - pathology | Male | Nasal Mucosa - pathology | Betacoronavirus - immunology | Respiratory System - virology | Antibodies, Neutralizing - immunology | Lung - virology | Peptidyl-Dipeptidase A - metabolism | Female | Lung - metabolism | Nasal Mucosa - metabolism | Vero Cells | Antibodies, Monoclonal - immunology | Furin - metabolism | Nasal Mucosa - virology | Cell Line | Lung - pathology | Chlorocebus aethiops | Pneumonia, Viral - pathology | Cells, Cultured | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Angiotensin-Converting Enzyme 2 | Animals | Coronavirus Infections - virology | Virus Replication | Coronavirus Infections - therapy | Coronavirus Infections - pathology | DNA, Recombinant | Immunization, Passive | Aged | Serine Endopeptidases - metabolism | Betacoronavirus - genetics | Pneumonia, Viral - virology | Health aspects | Severe acute respiratory syndrome | Index Medicus
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8. Full Text Genetic diversity and evolution of SARS-CoV-2
by Phan, Tung
Infection, genetics and evolution, ISSN 1567-1348, 07/2020, Volume 81, pp. 104260 - 104260
Mutations | SARS-CoV-2 | Coronavirus | Genomic diversity | Life Sciences & Biomedicine | Infectious Diseases | Science & Technology | Genetic Variation | Genome, Viral - genetics | Mutation | Betacoronavirus - genetics | Betacoronavirus - classification | Evolution, Molecular | Sequence Deletion - genetics | Index Medicus
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9. Full Text Host Polymorphisms May Impact SARS-CoV-2 Infectivity
by Brest, Patrick and Refae, Sadal and Mograbi, Baharia and Hofman, Paul and Milano, Gerard
Trends in genetics, ISSN 0168-9525, 11/2020, Volume 36, Issue 11, pp. 813 - 815
genetic variation | coronavirus | infection | SNP | pharmacogenetics | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Disease Susceptibility | Pandemics | Humans | Coronavirus Infections - genetics | Virus Shedding | Promoter Regions, Genetic - genetics | Virus Internalization | ADAM17 Protein - metabolism | COVID-19 | Angiotensin-Converting Enzyme 2 | Peptidyl-Dipeptidase A - genetics | ADAM17 Protein - genetics | SARS-CoV-2 | Pneumonia, Viral - genetics | Coronavirus Infections - virology | Betacoronavirus - physiology | Peptidyl-Dipeptidase A - metabolism | Serine Endopeptidases - genetics | Polymorphism, Single Nucleotide - genetics | Polymorphism, Genetic - genetics | Serine Endopeptidases - metabolism | Pneumonia, Viral - virology | Index Medicus
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10. Full Text Functional prediction and comparative population analysis of variants in genes for proteases and innate immunity related to SARS-CoV-2 infection
by Klaassen, Kristel and Stankovic, Biljana and Zukic, Branka and Kotur, Nikola and Gasic, Vladimir and Pavlovic, Sonja and Stojiljkovic, Maja
Infection, genetics and evolution, ISSN 1567-1348, 10/2020, Volume 84, pp. 104498 - 104498
COVID-19 | SARS-CoV-2 | Host genomics | Gene variants | Functional prediction | Population genomics | Allele frequencies | Susceptibility and resistance | Life Sciences & Biomedicine | Infectious Diseases | Science & Technology | Betacoronavirus - pathogenicity | Eye Proteins - immunology | Metagenomics | Pandemics | Mannose-Binding Lectin - immunology | Humans | Plasminogen - genetics | Mannose-Binding Lectin - genetics | Host-Pathogen Interactions - immunology |