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Journal of Clinical Investigation, ISSN 0021-9738, 06/2009, Volume 119, Issue 6, pp. 1537 - 1545
The forkhead box proteins A1 and A2 (Foxa1 and Foxa2) are transcription factors with critical roles in establishing the developmental competence of the foregut... 
TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATED PROTEIN-KINASE | LIVER MORPHOGENESIS | HEPATOCYTE GROWTH-FACTOR | GENE FAMILY | INTERLEUKIN-6-DEFICIENT MICE | BILIARY EPITHELIAL-CELLS | NF-KAPPA-B | EXPRESSION | CHOLANGIOCYTE PROLIFERATION | Bile Ducts - growth & development | Cell Proliferation | Hepatocyte Nuclear Factor 3-beta - genetics | Bile Ducts - metabolism | Receptors, Glucocorticoid - metabolism | Hepatocytes - metabolism | Fibrosis - metabolism | Bile Ducts - cytology | Hepatocytes - cytology | Hyperplasia - genetics | Bile Duct Diseases - metabolism | Cell Differentiation | Interleukin-6 - metabolism | Hyperplasia - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Microscopy, Electron, Transmission | Fibrosis - genetics | Interleukin-6 - genetics | Hepatocyte Nuclear Factor 3-alpha - deficiency | Liver - metabolism | Mice, Inbred C57BL | Bile Duct Diseases - genetics | Hepatocyte Nuclear Factor 3-alpha - genetics | Hepatocyte Nuclear Factor 3-alpha - metabolism | Mice, Knockout | Hyperplasia - pathology | Animals | Liver - cytology | Mice | Fibrosis - pathology | Bile Duct Diseases - pathology | Hepatocyte Nuclear Factor 3-beta - deficiency | Common bile duct | Growth | Bile ducts | Genes | Physiological aspects | Genetic aspects | Research | DNA binding proteins | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2007, Volume 132, Issue 3, pp. 1104 - 1116
Background & Aims: In polycystic liver diseases (PCLDs), increased cholangiocyte proliferation and fluid secretion are key features and cholangiocyte adenosine... 
Gastroenterology and Hepatology | KIDNEY-DISEASE | MOLECULAR CHARACTERIZATION | CYST FORMATION | CYCLIC ADENOSINE | TYROSINE KINASE INHIBITION | SOMATOSTATIN RECEPTORS | CELL-PROLIFERATION | HAN-SPRD RATS | ORTHOLOGOUS MODEL | GASTROENTEROLOGY & HEPATOLOGY | BILIARY EPITHELIUM | Cysts - etiology | Bile Ducts - metabolism | Bile Duct Diseases - etiology | Liver Diseases - pathology | Dose-Response Relationship, Drug | Polycystic Kidney, Autosomal Recessive - pathology | Cysts - metabolism | Liver - drug effects | Time Factors | Bile Duct Diseases - metabolism | Polycystic Kidney, Autosomal Recessive - complications | Liver Diseases - etiology | Liver Cirrhosis - metabolism | Polycystic Kidney, Autosomal Recessive - drug therapy | Receptors, Somatostatin - metabolism | Cyclic AMP - metabolism | Organ Culture Techniques | Disease Models, Animal | Liver Cirrhosis - etiology | Cysts - prevention & control | Kidney - drug effects | Liver Cirrhosis - prevention & control | Bile Ducts - drug effects | Liver - metabolism | Liver Diseases - prevention & control | Rats | Octreotide - therapeutic use | Cysts - pathology | Octreotide - pharmacology | Rats, Sprague-Dawley | Disease Progression | Gastrointestinal Agents - pharmacology | Animals | Bile Duct Diseases - prevention & control | Gastrointestinal Agents - therapeutic use | Receptors, Somatostatin - drug effects | Cell Proliferation - drug effects | Liver Diseases - metabolism | Polycystic Kidney, Autosomal Recessive - metabolism | Bile Duct Diseases - pathology | Adenosine | Analysis | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Pathology, ISSN 0021-9746, 07/2016, Volume 69, Issue 7, pp. 619 - 626
Journal Article
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2010, Volume 127, Issue 11, pp. 2576 - 2587
Peribiliary fibrosis caused by chronic infection with Opisthorchis viverrini (OV) is a risk factor of cholangiocarcinoma (CCA) in northeastern Thailand. Matrix... 
Rac1 | cholangiocarcinoma | MMP-9 | Opisthorchis viverrini | DNA damage | INTRAHEPATIC CHOLANGIOCARCINOMA | BILE-DUCT | NITRIC-OXIDE SYNTHASE | CELL INVASION | MECHANISMS | CANCER | MATRIX METALLOPROTEINASES | OPISTHORCHIS-VIVERRINI | CARCINOGENESIS | ONCOLOGY | INFLAMMATION | Nitric Oxide Synthase Type II - biosynthesis | Opisthorchiasis - genetics | Cholangiocarcinoma - parasitology | Male | Cholangiocarcinoma - metabolism | RNA, Messenger - biosynthesis | Matrix Metalloproteinase 9 - metabolism | Bile Duct Diseases - metabolism | Cell Transformation, Neoplastic - genetics | Mesocricetus | Collagen - biosynthesis | Collagen - genetics | Disease Models, Animal | Cricetinae | Matrix Metalloproteinase 9 - biosynthesis | Actins - biosynthesis | Matrix Metalloproteinase 2 - metabolism | Liver - metabolism | Bile Duct Diseases - genetics | Cell Transformation, Neoplastic - metabolism | Opisthorchiasis - metabolism | Collagen - metabolism | Cholangiocarcinoma - pathology | Animals | Urokinase-Type Plasminogen Activator - metabolism | Opisthorchiasis - pathology | Fibrosis | Cholangiocarcinoma - genetics | rac1 GTP-Binding Protein - biosynthesis | DNA Damage | Cell Transformation, Neoplastic - pathology | Opisthorchis | Bile Duct Diseases - pathology | Bile Ducts - pathology | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics | Index Medicus
Journal Article
Toxicologic Pathology, ISSN 0192-6233, 8/2011, Volume 39, Issue 5, pp. 795 - 808
Journal Article
Journal Article
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