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Gastrointestinal Endoscopy, ISSN 0016-5107, 2016, Volume 83, Issue 6, pp. 1228 - 1235
Journal Article
Journal of immunology research, ISSN 2314-7156, 2014, Volume 2014, pp. 1 - 19
Acute inflammation is a response to an alteration induced by a pathogen or a physical or chemical insult, which functions to eliminate the source of the damage... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Oxidative Stress | Colitis - genetics | Colorectal Neoplasms - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | Colitis - complications | Colitis - pathology | Cholangiocarcinoma - metabolism | Inflammation - complications | Colorectal Neoplasms - etiology | Inflammation - metabolism | Bile Ducts, Intrahepatic - metabolism | Tumor Microenvironment - genetics | Interleukin-10 - metabolism | Bile Duct Neoplasms - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Bile Ducts, Intrahepatic - pathology | Gene Expression | Bile Duct Neoplasms - metabolism | Interleukin-6 - genetics | Carcinogenesis - genetics | Cholangiocarcinoma - pathology | Transforming Growth Factor beta - genetics | Interleukin-10 - genetics | Colitis - metabolism | Cholangiocarcinoma - genetics | Inflammation - genetics | Bile Duct Neoplasms - pathology | Colorectal Neoplasms - pathology | Chronic Disease | Transforming Growth Factor beta - metabolism | Free radicals | Cytokines | Disease | Mortality | Clinical trials | Metastasis | Cancer therapies | Proteins | Angiogenesis | Medical prognosis | Tumorigenesis | Nonsteroidal anti-inflammatory drugs | Chemokines | Immune system | Tumors | Cancer | Apoptosis | Index Medicus | Review
Journal Article
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 08/2018, Volume 155, Issue 2, pp. 557 - 571.e14
MAF bZIP transcription factor G (MAFG) is activated by the farnesoid X receptor to repress bile acid synthesis. However, expression of MAFG increases during... 
FXR | Obeticholic Acid | S-Adenosylmethionine | Ursodeoxycholic Acid | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Diethylnitrosamine - toxicity | Up-Regulation | Carcinoma, Hepatocellular - mortality | Liver - pathology | Humans | Male | Cholangiocarcinoma - etiology | Liver Neoplasms - mortality | Gene Knockdown Techniques | Cholestasis - etiology | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Liver Neoplasms, Experimental - genetics | Bile Duct Neoplasms - genetics | Gene Expression Regulation, Neoplastic - drug effects | Liver Neoplasms, Experimental - pathology | MafG Transcription Factor - metabolism | MafG Transcription Factor - genetics | Repressor Proteins - metabolism | Liver Neoplasms - virology | Liver Neoplasms - genetics | Mice, Inbred C57BL | Repressor Proteins - genetics | Carcinoma, Hepatocellular - virology | Receptors, Cytoplasmic and Nuclear - agonists | S-Adenosylmethionine - pharmacology | Liver Neoplasms, Experimental - etiology | Bile Duct Neoplasms - etiology | Xenograft Model Antitumor Assays | Cholestasis - pathology | Cholic Acids - pharmacology | Cholangiocarcinoma - pathology | Animals | Mice, Nude | Bile Duct Neoplasms - mortality | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Cholangiocarcinoma - genetics | Mice | Mice, Inbred BALB C | Bile Duct Neoplasms - pathology | RNA, Small Interfering - metabolism | Liver cancer | Analysis | Index Medicus | Abridged Index Medicus | obeticholic acid | ursodeoxycholic acid | S-adenosylmethionine
Journal Article