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Journal of Clinical Investigation, ISSN 0021-9738, 08/2012, Volume 122, Issue 8, pp. 2911 - 2915
Journal Article
Hepatology, ISSN 0270-9139, 04/2014, Volume 59, Issue 4, pp. 1427 - 1434
Journal Article
Journal of Immunology Research, ISSN 2314-8861, 2014, Volume 2014, pp. 149185 - 19
Acute inflammation is a response to an alteration induced by a pathogen or a physical or chemical insult, which functions to eliminate the source of the damage... 
EPITHELIAL-MESENCHYMAL TRANSITION | ACTIVATED PROTEIN-KINASE | CARCINOMA-CELL-LINES | TGF-BETA | NITRIC-OXIDE SYNTHASE | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | TNF-ALPHA | IMMUNOLOGY | ENDOTHELIAL GROWTH-FACTOR | PRIMARY SCLEROSING CHOLANGITIS | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Oxidative Stress | Colitis - genetics | Colorectal Neoplasms - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | Colitis - complications | Colitis - pathology | Cholangiocarcinoma - metabolism | Inflammation - complications | Colorectal Neoplasms - etiology | Inflammation - metabolism | Bile Ducts, Intrahepatic - metabolism | Tumor Microenvironment - genetics | Interleukin-10 - metabolism | Bile Duct Neoplasms - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Bile Ducts, Intrahepatic - pathology | Gene Expression | Bile Duct Neoplasms - metabolism | Interleukin-6 - genetics | Carcinogenesis - genetics | Cholangiocarcinoma - pathology | Transforming Growth Factor beta - genetics | Interleukin-10 - genetics | Colitis - metabolism | Cholangiocarcinoma - genetics | Inflammation - genetics | Bile Duct Neoplasms - pathology | Colorectal Neoplasms - pathology | Chronic Disease | Transforming Growth Factor beta - metabolism | Free radicals | Cytokines | Disease | Mortality | Clinical trials | Metastasis | Cancer therapies | Proteins | Angiogenesis | Medical prognosis | Tumorigenesis | Nonsteroidal anti-inflammatory drugs | Chemokines | Immune system | Tumors | Cancer | Apoptosis | Review
Journal Article
Oncogene, ISSN 0950-9232, 06/2013, Volume 32, Issue 25, pp. 3091 - 3100
Journal Article
Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, p. 2166
Isocitrate dehydrogenase (IDH) genes 1 and 2 are frequently mutated in acute myeloid leukaemia (AML), low-grade glioma, cholangiocarcinoma (CC) and... 
DNA METHYLATION | MAMMALIAN DNA | 5-METHYLCYTOSINE | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | PHENOTYPE | RETINOIC ACID RECEPTORS | ACUTE MYELOID-LEUKEMIA | MUTATIONS | 5-CARBOXYLCYTOSINE | DISCOVERY | Neoplasms - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Gene Expression Regulation, Neoplastic | Cholangiocarcinoma - metabolism | Bone Neoplasms - pathology | Receptors, Retinoic Acid - genetics | Bone Neoplasms - metabolism | Glioma - metabolism | DNA-Binding Proteins - metabolism | Glioma - genetics | DNA Methylation | Neoplasms - genetics | Glioma - pathology | Trans-Activators - genetics | Chondrosarcoma - genetics | Chondrosarcoma - pathology | Bone Neoplasms - genetics | Bile Duct Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Central Nervous System Neoplasms - metabolism | Bile Duct Neoplasms - metabolism | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Central Nervous System Neoplasms - genetics | Isocitrate Dehydrogenase - genetics | Proto-Oncogene Proteins - genetics | Receptors, Retinoic Acid - metabolism | DNA-Binding Proteins - genetics | Central Nervous System Neoplasms - pathology | Cholangiocarcinoma - pathology | Chondrosarcoma - metabolism | Cholangiocarcinoma - genetics | Isocitrate Dehydrogenase - metabolism | Trans-Activators - metabolism | Glutarates - metabolism | Bile Duct Neoplasms - pathology | Mutation | Neoplasms - pathology | Leukemia, Myeloid, Acute - genetics
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 04/2013, Volume 37, Issue 4, pp. 496 - 505
Journal Article
Molecular Cell, ISSN 1097-2765, 10/2015, Volume 60, Issue 2, pp. 328 - 337
The Hippo/YAP signaling pathway is a crucial regulator of tissue growth, stem cell activity, and tumorigenesis. However, the mechanism by which YAP controls... 
REGULATOR | COACTIVATOR | ACTIVATION | COMPLEX | CHROMATIN | YORKIE | MEDIATOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | SIZE | CELL | REQUIREMENT | CELL BIOLOGY | Chromatin - metabolism | Mediator Complex - metabolism | Humans | Gene Expression Regulation, Neoplastic | Intracellular Signaling Peptides and Proteins - metabolism | Cholangiocarcinoma - metabolism | DNA Polymerase II - metabolism | Phosphoproteins - metabolism | Carcinogenesis - metabolism | Piperidines - pharmacology | Transcription, Genetic | Antineoplastic Agents - pharmacology | Flavonoids - pharmacology | Bile Duct Neoplasms - genetics | DNA Polymerase II - genetics | Intracellular Signaling Peptides and Proteins - genetics | Bile Duct Neoplasms - drug therapy | Chromatin - chemistry | Bile Duct Neoplasms - metabolism | Signal Transduction | Cyclin-Dependent Kinase 9 - genetics | Carcinogenesis - genetics | Mediator Complex - genetics | Mice, Transgenic | Phosphoproteins - genetics | Carcinogenesis - drug effects | Carcinogenesis - pathology | Xenograft Model Antitumor Assays | Cholangiocarcinoma - pathology | Animals | Cholangiocarcinoma - drug therapy | Enhancer Elements, Genetic | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Cholangiocarcinoma - genetics | Protein Binding | Cyclin-Dependent Kinase 9 - metabolism | Mice | Bile Duct Neoplasms - pathology | Adaptor Proteins, Signal Transducing - metabolism | Genetic aspects | Genetic transcription | Growth | Analysis | Genomics | Stem cells | Yuan (China) | Chromatin
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 147, Issue 3, pp. 690 - 701
Background & Aims Aberrant activation of β-catenin and Yes-associated protein 1 (Yap1) signaling pathways have been associated with the development of multiple... 
Gastroenterology and Hepatology | Gene Regulation | Liver Cancer | Oncogene | Tumor Formation | YES-ASSOCIATED PROTEIN | MUTANT | NOTCH PATHWAY | TRANSCRIPTION | CANCER | HEPATOCELLULAR-CARCINOMA | GENE | LIVER | GASTROENTEROLOGY & HEPATOLOGY | TUMORIGENESIS | EXPRESSION | Hepatoblastoma - genetics | Cell Proliferation | United States | Humans | Gene Expression Regulation, Neoplastic | Cholangiocarcinoma - metabolism | Phosphoproteins - metabolism | Bile Ducts, Intrahepatic - metabolism | Transfection | RNA Interference | Time Factors | Cell Transformation, Neoplastic - genetics | Cell Death | Liver Neoplasms - pathology | Transcription, Genetic | Hepatoblastoma - metabolism | Hepatoblastoma - pathology | Bile Ducts, Intrahepatic - pathology | Bile Duct Neoplasms - metabolism | Liver Neoplasms - genetics | Signal Transduction | Europe | Phosphoproteins - genetics | Cell Transformation, Neoplastic - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Hep G2 Cells | Cholangiocarcinoma - pathology | Animals | Adaptor Proteins, Signal Transducing - genetics | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Protein Binding | Mice | Bile Duct Neoplasms - pathology | Adaptor Proteins, Signal Transducing - metabolism | Cell Transformation, Neoplastic - pathology | Carcinoma, Hepatocellular - metabolism | liver cancer | tumor formation | oncogene | gene regulation
Journal Article
Hepatology, ISSN 0270-9139, 11/2006, Volume 44, Issue 5, pp. 1333 - 1343
Journal Article