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Journal of immunology research, ISSN 2314-7156, 2014, Volume 2014, pp. 1 - 19
Acute inflammation is a response to an alteration induced by a pathogen or a physical or chemical insult, which functions to eliminate the source of the damage... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Oxidative Stress | Colitis - genetics | Colorectal Neoplasms - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | Colitis - complications | Colitis - pathology | Cholangiocarcinoma - metabolism | Inflammation - complications | Colorectal Neoplasms - etiology | Inflammation - metabolism | Bile Ducts, Intrahepatic - metabolism | Tumor Microenvironment - genetics | Interleukin-10 - metabolism | Bile Duct Neoplasms - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Bile Ducts, Intrahepatic - pathology | Gene Expression | Bile Duct Neoplasms - metabolism | Interleukin-6 - genetics | Carcinogenesis - genetics | Cholangiocarcinoma - pathology | Transforming Growth Factor beta - genetics | Interleukin-10 - genetics | Colitis - metabolism | Cholangiocarcinoma - genetics | Inflammation - genetics | Bile Duct Neoplasms - pathology | Colorectal Neoplasms - pathology | Chronic Disease | Transforming Growth Factor beta - metabolism | Free radicals | Cytokines | Disease | Mortality | Clinical trials | Metastasis | Cancer therapies | Proteins | Angiogenesis | Medical prognosis | Tumorigenesis | Nonsteroidal anti-inflammatory drugs | Chemokines | Immune system | Tumors | Cancer | Apoptosis | Index Medicus | Review
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 08/2018, Volume 155, Issue 2, pp. 557 - 571.e14
MAF bZIP transcription factor G (MAFG) is activated by the farnesoid X receptor to repress bile acid synthesis. However, expression of MAFG increases during... 
FXR | Obeticholic Acid | S-Adenosylmethionine | Ursodeoxycholic Acid | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Diethylnitrosamine - toxicity | Up-Regulation | Carcinoma, Hepatocellular - mortality | Liver - pathology | Humans | Male | Cholangiocarcinoma - etiology | Liver Neoplasms - mortality | Gene Knockdown Techniques | Cholestasis - etiology | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Liver Neoplasms, Experimental - genetics | Bile Duct Neoplasms - genetics | Gene Expression Regulation, Neoplastic - drug effects | Liver Neoplasms, Experimental - pathology | MafG Transcription Factor - metabolism | MafG Transcription Factor - genetics | Repressor Proteins - metabolism | Liver Neoplasms - virology | Liver Neoplasms - genetics | Mice, Inbred C57BL | Repressor Proteins - genetics | Carcinoma, Hepatocellular - virology | Receptors, Cytoplasmic and Nuclear - agonists | S-Adenosylmethionine - pharmacology | Liver Neoplasms, Experimental - etiology | Bile Duct Neoplasms - etiology | Xenograft Model Antitumor Assays | Cholestasis - pathology | Cholic Acids - pharmacology | Cholangiocarcinoma - pathology | Animals | Mice, Nude | Bile Duct Neoplasms - mortality | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Cholangiocarcinoma - genetics | Mice | Mice, Inbred BALB C | Bile Duct Neoplasms - pathology | RNA, Small Interfering - metabolism | Liver cancer | Analysis | Index Medicus | Abridged Index Medicus | obeticholic acid | ursodeoxycholic acid | S-adenosylmethionine
Journal Article
Oncogene, ISSN 1476-5594, 07/2012, Volume 32, Issue 25, pp. 3091 - 3100
Journal Article
International journal of cancer, ISSN 0020-7136, 02/2015, Volume 136, Issue 4, pp. 844 - 853
Journal Article
Nature (London), ISSN 1476-4687, 07/2014, Volume 513, Issue 7516, pp. 110 - 114
Mutations in isocitrate dehydrogenase 1 (IDH1) and IDH2 are among the most common genetic alterations in intrahepatic cholangiocarcinoma (IHCC), a deadly liver... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Hepatocyte Nuclear Factor 4 - antagonists & inhibitors | Humans | ras Proteins - metabolism | Hepatocytes - pathology | Male | Hepatocytes - metabolism | Bile Duct Neoplasms - enzymology | Cell Differentiation - genetics | Neoplasm Metastasis | Hepatocyte Nuclear Factor 4 - biosynthesis | Female | Bile Duct Neoplasms - genetics | Cell Lineage - genetics | Cholangiocarcinoma - enzymology | Disease Models, Animal | Cell Division - genetics | Proto-Oncogene Proteins - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Bile Ducts, Intrahepatic - pathology | Mutant Proteins - genetics | Isocitrate Dehydrogenase - genetics | Mice, Transgenic | Mutant Proteins - metabolism | Proto-Oncogene Proteins - genetics | Hepatocyte Nuclear Factor 4 - genetics | Mutation - genetics | Bile Ducts, Intrahepatic - enzymology | Cholangiocarcinoma - pathology | Animals | Cholangiocarcinoma - genetics | Stem Cells - pathology | Isocitrate Dehydrogenase - metabolism | Glutarates - metabolism | Mice | Bile Duct Neoplasms - pathology | Hepatocytes - enzymology | Index Medicus | Càncer | Diferenciació cel·lular | Gallbladder diseases | Metabolisme | Cell diferentiation | Metabolism | Malalties de la vesícula biliar | Cancer
Journal Article
Journal Article