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PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 12, p. e14254
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 140, Issue 7, pp. 2095 - 2106
Background & Aims Cholesterol gallstone disease (CGD) results from a biochemical imbalance of lipids and bile salts in the gallbladder bile. We investigated... 
Gastroenterology and Hepatology | Gene Regulation | Transgenic Mice | Nuclear Receptor | PCN | Fgf15 | ACTIVATION | GALLBLADDER-DISEASE | NUCLEAR RECEPTORS | SUSCEPTIBILITY LOCI | ACID SYNTHESIS | PREGNANE-X-RECEPTOR | BILIRUBIN DETOXIFICATION | LITH GENES | MICE | GASTROENTEROLOGY & HEPATOLOGY | DRUG-METABOLISM | Intestines - drug effects | Receptors, Steroid - metabolism | Gallstones - prevention & control | Cholesterol, Dietary - metabolism | Hypericum | Bile - metabolism | Male | Intestines - metabolism | Receptors, Steroid - agonists | Fibroblast Growth Factors - metabolism | Biological Transport | Liver - drug effects | Time Factors | Plant Preparations - pharmacology | Receptors, Steroid - deficiency | Phospholipids - metabolism | Disease Models, Animal | Gallstones - metabolism | Pregnenolone Carbonitrile - pharmacology | Liver - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Bile Acids and Salts - metabolism | Mice, Knockout | Animals | Gallstones - genetics | Receptors, Steroid - genetics | Cholesterol 7-alpha-Hydroxylase - metabolism | Mice | Receptors, Cytoplasmic and Nuclear - metabolism | Drug resistance in microorganisms | Pharmacogenetics | Cytochrome P-450 | Gallstones | Lipids | Biosynthesis | Cholesterol | Deoxycholic acid | Prevention | Quantitative genetics | Physiological aspects | Fibroblast growth factors | Progesterone | nuclear receptor | transgenic mice | gene regulation
Journal Article
Journal of Ethnopharmacology, ISSN 0378-8741, 2009, Volume 121, Issue 1, pp. 79 - 85
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 10/2011, Volume 301, Issue 4, pp. G634 - G643
Journal Article
Cellular Signalling, ISSN 0898-6568, 03/2014, Volume 26, Issue 3, pp. 512 - 520
Unconjugated bilirubin (UCB) is responsible for neonatal jaundice and high level of free bilirubin (Bf) can lead to kernicterus. Previous studies suggest that... 
Nrf2 pathway | Oxidative stress | Heme oxygenase 1 | Antioxidant Response Element | Bilirubin neurotoxicity | Stress response protein kinases | NEURODEGENERATIVE DISEASE | HUMAN-SKIN FIBROBLASTS | INDUCED APOPTOSIS | HEME OXYGENASE-1 GENE | ENDOPLASMIC-RETICULUM STRESS | CELL-SURVIVAL | CYTOCHROME-P450 2A5 | JAUNDICED GUNN RAT | CELL BIOLOGY | UNCONJUGATED BILIRUBIN | SIGNALING PATHWAY | Free Radical Scavengers - pharmacology | Transcription, Genetic - drug effects | Transcriptional Activation - genetics | Mitogen-Activated Protein Kinase 14 - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Humans | Transcriptional Activation - drug effects | Cell Survival - genetics | Bilirubin - pharmacology | RNA, Messenger - biosynthesis | RNA Interference | Antioxidant Response Elements - genetics | NF-E2-Related Factor 2 - genetics | Cell Survival - drug effects | MAP Kinase Kinase 1 - antagonists & inhibitors | Heme Oxygenase-1 - biosynthesis | Activating Transcription Factor 3 - biosynthesis | Protein Kinase C - antagonists & inhibitors | Antioxidants - pharmacology | Hep G2 Cells | Apoferritins - biosynthesis | NAD(P)H Dehydrogenase (Quinone) - biosynthesis | Signal Transduction - drug effects | MAP Kinase Kinase 2 - antagonists & inhibitors | NF-E2-Related Factor 2 - metabolism | Acetylcysteine - pharmacology | Cell Line, Tumor | RNA, Small Interfering | Oxidative Stress - drug effects | Transcription, Genetic - genetics | Antioxidants | Genetic research | Genetic transcription | Bilirubin | RNA | Index Medicus | Stresses | Cellular | Inhibitors | Pathways | Genes | Activation | Gene expression
Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 03/2004, Volume 172, Issue 6, pp. 3553 - 3563
Heme oxygenase-1 (HO-1) cleaves the porphyrin ring of heme into carbon monoxide, Fe2+, and biliverdin, which is then converted into bilirubin. Heme-derived... 
APOPTOSIS | TUMOR-NECROSIS-FACTOR | MAMMALIAN IRON | CARBON-MONOXIDE | ANTIOXIDANT | HUMAN VASCULAR ENDOTHELIUM | LUNG INJURY | ADENOVIRUS-MEDIATED EXPRESSION | IMMUNOLOGY | STRESS | NF-KAPPA-B | Endothelium, Vascular - cytology | Cell Adhesion Molecules | Humans | NF-kappa B - metabolism | Recombinant Proteins - biosynthesis | Endothelium, Vascular - enzymology | Bilirubin - pharmacology | MAP Kinase Signaling System - immunology | Heme - pharmacology | Carbon Monoxide - pharmacology | Heme Oxygenase (Decyclizing) - biosynthesis | Cattle | Swine | Intercellular Adhesion Molecule-1 - biosynthesis | E-Selectin - biosynthesis | Adenoviridae - genetics | Heme Oxygenase-1 | Heme Oxygenase (Decyclizing) - genetics | p38 Mitogen-Activated Protein Kinases | Endothelium, Vascular - immunology | Heme Oxygenase (Decyclizing) - physiology | NF-kappa B - antagonists & inhibitors | Transduction, Genetic | Cells, Cultured | Recombinant Proteins - pharmacology | Down-Regulation - drug effects | Membrane Proteins | Animals | Iron Chelating Agents - pharmacology | Mitogen-Activated Protein Kinases - physiology | Up-Regulation - immunology | Down-Regulation - immunology | Endothelium, Vascular - metabolism | Tumor Necrosis Factor-alpha - physiology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Journal Article
Journal Article
Molecular Immunology, ISSN 0161-5890, 2010, Volume 47, Issue 15, pp. 2443 - 2449