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bimel (6) 6
index medicus (5) 5
apoptosis (4) 4
apoptosis regulatory proteins - metabolism (4) 4
bcl-2-like protein 11 (4) 4
bim (4) 4
membrane proteins - metabolism (4) 4
proto-oncogene proteins - metabolism (4) 4
animals (3) 3
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extracellular signal-regulated map kinases - metabolism (3) 3
humans (3) 3
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phosphorylation (3) 3
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activation (2) 2
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apoptosis - physiology (2) 2
bax-dependent apoptosis (2) 2
bh3-only proteins (2) 2
cell adhesion - drug effects (2) 2
cell biology (2) 2
death (2) 2
degradation (2) 2
female (2) 2
mice (2) 2
mitochondria (2) 2
pathway (2) 2
phosphorylation - drug effects (2) 2
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20s proteasome (1) 1
acute promyelocytic leukemia (1) 1
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analysis (1) 1
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anoikis - drug effects (1) 1
anoikis - genetics (1) 1
antineoplastic agents - pharmacology (1) 1
antineoplastic combined chemotherapy protocols - pharmacology (1) 1
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bcl-2-like protein 11 - metabolism (1) 1
beta-trcp (1) 1
bfgf, basic fibroblast growth factor (1) 1
bh, bcl-2 homology (1) 1
bh3-only protein bim (1) 1
bim controls apoptosis (1) 1
bim, bcl-2-interacting mediator of cell death (1) 1
bimel, extra-long bim (1) 1
biml, long bim (1) 1
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biochemistry (1) 1
biology (1) 1
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casp8 and fadd-like apoptosis regulating protein - genetics (1) 1
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caspases - genetics (1) 1
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cdc2 protein kinase - metabolism (1) 1
cdk, cyclin-dependent kinase (1) 1
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cip, calf intestine phosphatase (1) 1
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Journal Article
Biochemical Journal, ISSN 0264-6021, 05/2005, Volume 388, Issue 1, pp. 185 - 194
The regulation of survival and cell death is a key determinant of cell fate. Recent evidence shows that survival and death machineries are regulated along the... 
Signal transduction | Bim (Bcl-2-interacting mediator of cell death) | Fibroblast growth factor (FGF) | Mitosis | Cell cycle | Apoptosis | BCL-2 RELATIVE BIM | SURVIVAL | PROAPOPTOTIC ACTIVITY | cell cycle | signal transduction | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | DEATH | mitosis | FAMILY-MEMBER BIM | MICROTUBULE DYNAMICS | PATHWAY | fibroblast growth factor (FGF) | CELL-CYCLE | BH3-ONLY PROTEINS | Proto-Oncogene Proteins - metabolism | NIH 3T3 Cells | Phosphorylation | Signal Transduction | MAP Kinase Kinase Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Apoptosis Regulatory Proteins - metabolism | CDC2 Protein Kinase - metabolism | Animals | Bcl-2-Like Protein 11 | Mitosis - physiology | Membrane Proteins - metabolism | Mice | Protein Processing, Post-Translational | Apoptosis - physiology | Fibroblast Growth Factors - physiology | Index Medicus | TUNEL, terminal deoxynucleotidyl transferase-mediated dUTP nick-end labelling | PI3K, phosphoinositide 3-kinase | CIP, calf intestine phosphatase | HEK, human embryonic kidney | TBST, Tris-buffered saline with Tween 20 | BH, Bcl-2 homology | ERK kinase | DTT, dithiothreitol | ERK, extracellular-signal-regulated kinase | DMEM, Dulbecco's modified Eagle's medium | P-Bim, phosphorylated Bim | Bim, Bcl-2-interacting mediator of cell death | BimEL, extra-long Bim | Cdk, cyclin-dependent kinase | FGF, fibroblast growth factor | MEK, mitogen-activated protein kinase | HA, haemagglutinin | IP, immunoprecipitation | BimS, short Bim | MT, microtubule | bFGF, basic fibroblast growth factor | BimL, long BIM | FBS, foetal bovine serum
Journal Article
Journal of Cell Science, ISSN 0021-9533, 03/2011, Volume 124, Issue 6, pp. 969 - 977
BIM-extra long (BIMEL), a pro-apoptotic BH3-only protein and part of the BCL-2 family, is degraded by the proteasome following activation of the ERK1/2... 
Ubiquitin | ERK1/2 | 20S proteasome | Intrinsically disordered protein | BIM | CANCER-CELLS | BETA-TRCP | MCL-1 | BIMEL | KAPPA-B-ALPHA | CELL BIOLOGY | UBA DOMAIN | UBIQUITIN CHAINS | PATHWAY | DEGRADATION | REG-GAMMA-PROTEASOME | BH3-ONLY PROTEINS
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 2010, Volume 31, Issue 11, pp. 1915 - 1925
Huntington's disease (HD) is a devastating neurodegenerative disorder caused by an expanded polyglutamine repeat within the N-terminus of the huntingtin... 
Phosphorylation | Protein degradation | Mice | Huntington's disease | Ubiquitin-proteasome system | BimEL | NEURONAL APOPTOSIS | BCL-2 FAMILY-MEMBER | BAX-DEPENDENT APOPTOSIS | SYMPATHETIC NEURONS | ENDOPLASMIC-RETICULUM STRESS | POLYGLUTAMINE EXPANSION | mice | NEUROSCIENCES | ubiquitin-proteasome system | BH3-ONLY PROTEIN BIM | AGGREGATE FORMATION | CYTOCHROME-C RELEASE | phosphorylation | protein degradation | PARKINSONS-DISEASE | RNA, Small Interfering - genetics | Humans | Endoplasmic Reticulum - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Bcl-2-Like Protein 11 | Apoptosis Regulatory Proteins - genetics | Mice, Inbred CBA | Membrane Proteins - metabolism | Nuclear Proteins - genetics | Huntington Disease - physiopathology | Disease Models, Animal | Proto-Oncogene Proteins - metabolism | Cell Line | Membrane Proteins - genetics | Mice, Inbred C57BL | bcl-2-Associated X Protein - metabolism | Mice, Transgenic | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Nerve Tissue Proteins - genetics | Proteasome Endopeptidase Complex - physiology | Apoptosis Regulatory Proteins - metabolism | Nerve Tissue Proteins - metabolism | Huntingtin Protein | Animals | Cell Death - physiology | Enzyme Activation | RNA, Small Interfering - metabolism | Cell research | Nervous system diseases | RNA | Huntington's chorea | Cell death | Proteolysis | Analysis | Biochemistry | Index Medicus | Huntington’s disease
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2008, Volume 12, Issue 6a, pp. 2482 - 2496
Journal Article
Biochimie, ISSN 0300-9084, 02/2012, Volume 94, Issue 2, pp. 287 - 299
SAHA, an inhibitor of histone deacetylase activity, has been shown to sensitize tumor cells to apoptosis induced by TRAIL, a member of TNF-family. In this... 
SAHA | FAK | Anoikis | TRAIL | EGFR | BimEL | HEPATOCELLULAR-CARCINOMA CELLS | HISTONE DEACETYLASE INHIBITOR | SUBEROYLANILIDE HYDROXAMIC ACID | INTEGRIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | ENDOCRINE THERAPY | PROTEIN BIM | RESISTANCE | TRAIL-INDUCED APOPTOSIS | FOCAL ADHESION KINASE | Focal Adhesion Kinase 1 - genetics | Receptor, Epidermal Growth Factor - genetics | Recombinant Fusion Proteins - pharmacology | Humans | Recombinant Fusion Proteins - metabolism | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Receptor, Epidermal Growth Factor - metabolism | Caspases - metabolism | TNF-Related Apoptosis-Inducing Ligand - pharmacology | Bcl-2-Like Protein 11 | Apoptosis Regulatory Proteins - genetics | Female | Antineoplastic Agents - pharmacology | Membrane Proteins - metabolism | Gene Expression Regulation, Neoplastic - drug effects | CASP8 and FADD-Like Apoptosis Regulating Protein - metabolism | Focal Adhesion Kinase 1 - metabolism | Hydroxamic Acids - pharmacology | Proto-Oncogene Proteins - metabolism | Anoikis - genetics | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Caspases - genetics | Membrane Proteins - genetics | Proto-Oncogene Proteins - genetics | Cell Adhesion - drug effects | TNF-Related Apoptosis-Inducing Ligand - metabolism | Breast Neoplasms - drug therapy | Apoptosis Regulatory Proteins - metabolism | Breast Neoplasms - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Cell Line, Tumor | Anoikis - drug effects | Care and treatment | Breast cancer | Epidermal growth factor | Estrogen | Cancer | Index Medicus
Journal Article
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