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Circulation: Heart Failure, ISSN 1941-3289, 01/2016, Volume 9, Issue 1, pp. e002424 - e002424
Background Diabetic heart disease is associated with tetrahydrobiopterin oxidation and high arginase activity, leading to endothelial nitric oxide synthase... 
Diabetic cardiomyopathy | type 2 diabetes mellitus | tetrahydrobiopterin | ischemia reperfusion injury | nitric oxide synthase | A ADENOSINE RECEPTOR | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | NITRIC-OXIDE SYNTHASE | REPERFUSION INJURY | diabetic cardiomyopathy | ENDOTHELIAL FUNCTION | INFARCT SIZE | OVEREXPRESSION | IN-VIVO | ARGINASE ACTIVITY | Myocardial Reperfusion Injury - etiology | Phosphorylation | Diabetic Cardiomyopathies - metabolism | Age Factors | Diabetic Cardiomyopathies - etiology | Coronary Vessels - physiopathology | Protein Multimerization | Diabetes Mellitus, Type 2 - metabolism | Biopterin - analogs & derivatives | Diabetic Cardiomyopathies - physiopathology | Biopterin - metabolism | Coronary Vessels - metabolism | Myocardial Reperfusion Injury - pathology | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Diabetic Cardiomyopathies - prevention & control | Myocardial Infarction - physiopathology | Pterins - administration & dosage | Nitric Oxide Synthase Type III - metabolism | Drug Therapy, Combination | Myocardial Infarction - etiology | Diabetes Mellitus, Type 2 - complications | Disease Models, Animal | Coronary Vessels - drug effects | Drug Administration Schedule | Endothelial Cells - metabolism | Isolated Heart Preparation | Obesity - complications | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Cells, Cultured | Myocardium - pathology | Myocardial Infarction - metabolism | Myocardial Reperfusion Injury - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Cardiotonic Agents - administration & dosage | Diabetic Cardiomyopathies - pathology | Mice, Obese | Citrulline - administration & dosage | Myocardial Infarction - prevention & control | Coronary Circulation - drug effects | Vasodilation - drug effects | Diabetes Mellitus, Type 2 - drug therapy | Nitric Oxide - metabolism | Myocardial Reperfusion Injury - prevention & control | Endothelial Cells - drug effects
Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 10/2011, Volume 589, Issue 19, pp. 4787 - 4797
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2003, Volume 285, Issue 2, pp. 875 - 882
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 09/2000, Volume 87, Issue 7, pp. 566 - 573
We have reported that a deficiency of tetrahydrobiopterin (BH4), an active cofactor of endothelial NO synthase (eNOS), contributes to the endothelial... 
Free radicals | Tetrahydrobiopterin | Insulin | Endothelium | endothelium | tetrahydrobiopterin | free radicals | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | insulin | TRANSCRIPTION | COMPENSATORY HYPERINSULINEMIA | BLOOD-PRESSURE | SYNTHASE | GLUCOSE | DISEASE | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | RADICALS | HYPERTENSION | HEMATOLOGY
Journal Article
Journal Article