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Cell Stem Cell, ISSN 1934-5909, 01/2015, Volume 16, Issue 1, pp. 51 - 66
Mesenchymal stem cells (MSCs) reside in the perivascular niche of many organs, including kidney, lung, liver, and heart, although their roles in these tissues... 
REGULATOR | ORIGIN | SONIC HEDGEHOG | PATHWAY | BONE-MARROW NICHE | MYOFIBROBLASTS | NG2 PROTEOGLYCAN | EXPRESSION | MESENCHYMAL STEM-CELLS | GROWTH FACTOR-AA | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Organ Specificity - drug effects | Diphtheria Toxin - pharmacology | Neovascularization, Physiologic - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Fibrosis - metabolism | Cell Lineage - drug effects | Myofibroblasts - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Kruppel-Like Transcription Factors - metabolism | Pericytes - pathology | Bone Marrow Cells - drug effects | Colony-Forming Units Assay | Aorta - physiopathology | Homeostasis - drug effects | Heart Ventricles - pathology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Endothelial Cells - metabolism | Aorta - drug effects | Pericytes - metabolism | Cells, Cultured | Proteoglycans - metabolism | Antigens - metabolism | Aorta - pathology | Myofibroblasts - cytology | Animals | Heart Ventricles - physiopathology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Blood Vessels - drug effects | Mice | Stem Cell Niche - drug effects | Zinc Finger Protein GLI1 | Fibrosis - pathology | Bone Marrow Cells - metabolism | Endothelial Cells - drug effects | Heart Ventricles - drug effects
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 06/2010, Volume 207, Issue 6, pp. 1247 - 1260
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 2014, Volume 20, Issue 11, pp. 1270 - 1278
Osteogenesis during bone modeling and remodeling is coupled with angiogenesis. A recent study showed that a specific vessel subtype, strongly positive for CD31... 
MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | CORTICAL BONE | OSTEOCLASTS | RESORPTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | BONE-FORMATION | CATHEPSIN-K | MICE | DIFFERENTIATION | Neovascularization, Physiologic - drug effects | Cell Count | Tartrate-Resistant Acid Phosphatase | Osteoclasts - secretion | Culture Media, Conditioned - pharmacology | Femur - diagnostic imaging | X-Ray Microtomography | Protease Inhibitors - pharmacology | Acid Phosphatase - metabolism | Mesenchymal Stromal Cells - cytology | Isoenzymes - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Female | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Cathepsin K - antagonists & inhibitors | Mesenchymal Stromal Cells - drug effects | Ovariectomy | Endothelial Cells - metabolism | Femur - metabolism | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Mice, Inbred C57BL | Osteogenesis - drug effects | Mesenchymal Stromal Cells - metabolism | Femur - drug effects | Cell Movement - drug effects | Animals | Cathepsin K - metabolism | Endothelial Cells - cytology | Proto-Oncogene Proteins c-sis - secretion | Osteoclasts - enzymology | Osteoclasts - drug effects | Endothelial Cells - drug effects | Platelet-derived growth factor | Growth | Analysis | Physiological aspects | Bones | Research | Osteoclasts (Biology) | Homeostasis | Angiogenesis | Cell growth | Blood platelets
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, p. e40677
Background: Myeloid derived suppressor cells (MDSC) are important regulators of immune responses. We evaluated the mechanistic role of MDSC depletion on... 
MHC CLASS-I | DENDRITIC CELLS | NK CELLS | MECHANISM | TUMOR-ANTIGENS | TOLERANCE | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | ANERGY | INDUCTION | CYTOTOXIC T-LYMPHOCYTES | Spleen - immunology | Ovalbumin - immunology | Apoptosis - drug effects | Vaccination | Carcinoma, Lewis Lung - immunology | Antineoplastic Agents - therapeutic use | Spleen - drug effects | Carcinoma, Lewis Lung - blood supply | Neoplasm Metastasis | Myeloid Cells - immunology | T-Lymphocytes - drug effects | Angiogenesis Inhibitors - therapeutic use | Biomarkers, Tumor - metabolism | Killer Cells, Natural - immunology | Bone Marrow Cells - drug effects | Myeloid Cells - drug effects | Antineoplastic Agents - pharmacology | Cytotoxicity, Immunologic - drug effects | Disease Models, Animal | Antigen-Presenting Cells - drug effects | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Angiogenesis Inhibitors - pharmacology | Treatment Outcome | Tumor Burden | Cell Adhesion - drug effects | Antigen-Presenting Cells - immunology | Carcinoma, Lewis Lung - drug therapy | Animals | Carcinoma, Lewis Lung - pathology | T-Lymphocytes - immunology | Cell Proliferation - drug effects | Killer Cells, Natural - drug effects | Mice | Myeloid Cells - pathology | Antigens | Lymphocytes | Lung cancer | Comparative analysis | Health aspects | Vascular endothelial growth factor | Tumors | Regulators | Animal models | Leukocyte migration | Laboratories | Veterans | CD8 antigen | Immunocytochemistry | Cytotoxicity | Lymphocytes T | Suppressor cells | Anticancer properties | Angiogenesis | Cell activation | Immunology | Immunotherapy | Bone marrow | Perforin | Medical research | Immunological memory | Immune response | Inflammation | CD3 antigen | T cell receptors | CXCR3 protein | Adenomatous polyposis coli | Medicine | Depletion | Antigen-presenting cells | γ-Interferon | CXCL10 protein | Interleukin 10 | Antitumor activity | Lymphomas | Cell migration | Apoptosis | Cancer
Journal Article
Cell Metabolism, ISSN 1550-4131, 12/2016, Volume 24, Issue 6, pp. 795 - 806
NAD availability decreases with age and in certain disease conditions. Nicotinamide mononucleotide (NMN), a key NAD intermediate, has been shown to enhance NAD... 
NAD | NAD+ precursor | insulin sensitivity | eye function | anti-aging | mitochondria | aging | nicotinamide mononucleotide | glucose metabolism | NMN | energy metabolism | precursor | LIFE-SPAN | STEM-CELLS | ACTIVATION | MITOCHONDRIAL | NAD BIOSYNTHESIS | INSULIN-SECRETION | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | RIBOSIDE | SIRT1 | ADIPOSE-TISSUE | CELL BIOLOGY | Darkness | Aging - drug effects | Male | Muscle, Skeletal - metabolism | Nicotinamide Mononucleotide - administration & dosage | Aging - genetics | Time Factors | Lipids - blood | Muscle, Skeletal - drug effects | Cell Respiration - drug effects | Myeloid Cells - drug effects | Weight Gain - drug effects | NAD - metabolism | Physical Conditioning, Animal | Food | Lymphocytes - metabolism | Insulin - pharmacology | Administration, Oral | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Gene Expression Regulation - drug effects | Eating - drug effects | Bone Density - drug effects | Animals | Aging - physiology | Lymphocytes - drug effects | Myeloid Cells - metabolism | Nicotinamide Mononucleotide - pharmacology | Drinking - drug effects | Nicotinamide Mononucleotide - blood | Energy Metabolism - drug effects | Niacinamide | Medical colleges | Exercise | Pharmacy | Genes | Body weight | Physiological aspects | Muscles | Mice | Ophthalmology | Gene expression
Journal Article
Cell Metabolism, ISSN 1550-4131, 01/2014, Volume 19, Issue 1, pp. 96 - 108
The transcriptional coactivator peroxisome proliferator-activated receptor-gamma coactivator-1α (PGC-1α) regulates metabolic genes in skeletal muscle and... 
SKELETAL-MUSCLE | PPAR-ALPHA | ACTIVATION | MITOCHONDRIAL UNCOUPLING PROTEIN | PGC-1-ALPHA | IN-VIVO | ENDOCRINOLOGY & METABOLISM | MICE | EXPRESSION | EXERCISE | GENOME-WIDE ASSOCIATION | CELL BIOLOGY | Organ Specificity - drug effects | Transcription, Genetic - drug effects | Metabolic Diseases - pathology | Adipocytes, Brown - metabolism | Humans | Adipose Tissue, White - metabolism | Cardiovascular Diseases - pathology | Organ Specificity - genetics | Adipocytes, White - drug effects | Adipose Tissue, White - cytology | Exercise | Liver - drug effects | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Oxidation-Reduction - drug effects | Weight Gain - drug effects | Physical Conditioning, Animal | Aminoisobutyric Acids - pharmacology | Induced Pluripotent Stem Cells - metabolism | Adipocytes, Brown - pathology | Glucose Tolerance Test | Cardiovascular Diseases - metabolism | Induced Pluripotent Stem Cells - drug effects | Adipocytes, Brown - drug effects | Liver - metabolism | Risk Factors | Aminoisobutyric Acids - blood | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Adipose Tissue, Brown - cytology | Phenotype | Adipocytes, White - pathology | Animals | Metabolic Diseases - metabolism | Cell Differentiation - drug effects | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Mice | PPAR alpha - metabolism | Adipocytes, White - metabolism | Adipose Tissue, White - drug effects
Journal Article
Science, ISSN 0036-8075, 2014, Volume 344, Issue 6184, pp. 630 - 634
Journal Article
Nature Reviews Cancer, ISSN 1474-175X, 10/2013, Volume 13, Issue 10, pp. 739 - 752
Journal Article