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Oncogene, ISSN 0950-9232, 07/2001, Volume 20, Issue 33, pp. 4457 - 4465
Defects in PTEN a tumor suppressor, have been found in cancers arising in a variety of human tissues. To elucidate the tumor-suppressive function of this gene,... 
PTEN | EGR2 | PINK1 | BPOZ | Tumor suppressor gene | ZINC FINGER PROTEIN | NERVOUS-SYSTEM | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | CELL BIOLOGY | ONCOLOGY | PROSTATE-CANCER | BTB/POZ DOMAIN | GENETICS & HEREDITY | MOLECULAR-CLONING | MUTATIONS | DIFFERENTIATION | tumor suppressor gene | HUMAN OVARIAN-CANCER | Protein Kinases - genetics | Oligodeoxyribonucleotides, Antisense - pharmacology | Humans | Repressor Proteins | Transcriptional Activation | Ovarian Neoplasms - pathology | Male | Neoplasm Proteins - pharmacology | Gene Expression Profiling | Ovarian Neoplasms - genetics | Protein Tyrosine Phosphatases - genetics | Blood Proteins - genetics | Neoplasm Proteins - genetics | Genes, Tumor Suppressor | Protein Phosphatase 1 | Expressed Sequence Tags | Phosphoprotein Phosphatases | Amino Acid Sequence | Tumor Stem Cell Assay | DNA-Binding Proteins - physiology | Tumor Cells, Cultured - metabolism | Signal Transduction - genetics | Immediate-Early Proteins - genetics | Basic-Leucine Zipper Transcription Factors | Cell Cycle Proteins - physiology | Protein Tyrosine Phosphatases - physiology | Cell Cycle - genetics | Dual Specificity Phosphatase 1 | Neoplasm Proteins - physiology | Recombinant Fusion Proteins - physiology | Molecular Sequence Data | Adaptor Proteins, Vesicular Transport | Tumor Cells, Cultured - pathology | RNA Splicing | Transfection | Early Growth Response Protein 2 | PTEN Phosphohydrolase | Cell Cycle Proteins - genetics | Adult | Female | Cell Division - genetics | Protein Structure, Tertiary | Proteins - physiology | Nerve Tissue Proteins - physiology | Phosphoric Monoester Hydrolases - genetics | Transcription Factors - physiology | Immediate-Early Proteins - physiology | Neoplasm Proteins - biosynthesis | Intracellular Signaling Peptides and Proteins | Ankyrin Repeat | Phosphoric Monoester Hydrolases - physiology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Subcellular Fractions - metabolism | Proteins - genetics | Sequence Alignment | Blood Proteins - physiology | Protein Kinases - physiology | Tumor Suppressor Proteins | G-Box Binding Factors
Journal Article
2008, 1. Aufl., ISBN 9780470016749, x, 526
Human Blood Plasma Proteinsgives an overview of the proteins found in human blood plasma, with special emphasis on their structure and function and... 
Enzymes | Peptides | Blood Coagulation | physiology | Hormones | blood | Blood proteins
Book
Cellular and Molecular Life Sciences, ISSN 1420-682X, 3/2011, Volume 68, Issue 5, pp. 765 - 783
Journal Article
Neuron, ISSN 0896-6273, 08/2017, Volume 95, Issue 5, pp. 1056 - 1073.e5
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1497 - 1508
Synaptic plasticity is the ability of synapses to modulate the strength of neuronal connections; however, the molecular factors that regulate this feature are... 
DENDRITIC SPINES | MEDICINE, RESEARCH & EXPERIMENTAL | EMERGING ROLES | MEMORY | EXCITATORY SYNAPSES | APOPTOTIC CELLS | RECEPTOR | BRAIN ANGIOGENESIS INHIBITOR-1 | LONG-TERM POTENTIATION | PSD-95 | POSTSYNAPTIC DENSITY | Disks Large Homolog 4 Protein | Guanylate Kinases - genetics | Angiogenic Proteins - genetics | Memory Disorders - physiopathology | Synaptic Transmission - physiology | Memory Disorders - genetics | Humans | Nerve Tissue Proteins - deficiency | Angiogenic Proteins - physiology | Learning Disorders - physiopathology | Proto-Oncogene Proteins c-mdm2 - physiology | Recombinant Fusion Proteins - metabolism | Spatial Learning - physiology | Brain - blood supply | Membrane Proteins - deficiency | Neuronal Plasticity - genetics | Ubiquitination | Neuronal Plasticity - physiology | Neurons - ultrastructure | HEK293 Cells | Long-Term Potentiation - physiology | Membrane Proteins - metabolism | Learning Curve | Protein Structure, Tertiary | Nerve Tissue Proteins - physiology | Angiogenic Proteins - deficiency | Maze Learning - physiology | Membrane Proteins - genetics | Mice, Inbred C57BL | Hippocampus - pathology | Nerve Tissue Proteins - genetics | Mice, Knockout | Protein Interaction Mapping | Guanylate Kinases - deficiency | Animals | Guanylate Kinases - metabolism | Learning Disorders - genetics | Mice | Protein Processing, Post-Translational | Hippocampus - physiopathology | Cell receptors | Nervous system diseases | Neural transmission | Development and progression | Genetic aspects | Hippocampus (Brain) | Health aspects | Proteins | Angiogenesis | Rodents | Memory | Cloning | Software | Behavior | Gene expression
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1208 - 1216
The ataxia telangiectasia mutated (ATM) kinase, a master regulator of the DNA damage response (DDR), acts as a barrier to cellular senescence and... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | OXYGEN-INDUCED RETINOPATHY | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | HISTONE H2AX | MECHANISMS | TUMOR ANGIOGENESIS | CELL BIOLOGY | MOUSE MODEL | NEOVASCULARIZATION | ATAXIA-TELANGIECTASIA | Protein-Serine-Threonine Kinases - deficiency | Reactive Oxygen Species - metabolism | Humans | Male | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Retinal Vessels - pathology | Tumor Suppressor Proteins - deficiency | Lipid Peroxidation - drug effects | Neovascularization, Pathologic - physiopathology | Ischemia - pathology | Hydrogen Peroxide - toxicity | Cells, Cultured - drug effects | DNA-Binding Proteins - physiology | p38 Mitogen-Activated Protein Kinases - physiology | Retinal Vessels - drug effects | Ataxia Telangiectasia Mutated Proteins | Mice, Knockout | Tumor Suppressor Proteins - physiology | Signal Transduction - drug effects | Cells, Cultured - metabolism | Eye Proteins - physiology | Mice | Enzyme Activation | Retinal Neovascularization - physiopathology | Cell Cycle Proteins - physiology | Hydrogen Peroxide - antagonists & inhibitors | Vascular Endothelial Growth Factor A - physiology | Retinal Neovascularization - pathology | Oxidative Stress - physiology | Pyrones - pharmacology | DNA-Binding Proteins - deficiency | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Retinal Neovascularization - prevention & control | Female | Neovascularization, Pathologic - prevention & control | Acetylcysteine - therapeutic use | Melanoma, Experimental - blood supply | Protein-Serine-Threonine Kinases - physiology | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Protein-Serine-Threonine Kinases - genetics | Morpholines - pharmacology | Cell Cycle Proteins - deficiency | Antioxidants - pharmacology | Ischemia - physiopathology | DNA-Binding Proteins - genetics | Antioxidants - therapeutic use | Animals | Ischemia - drug therapy | DNA Repair | Acetylcysteine - pharmacology | Apoptosis | Physiological aspects | Oxidative stress | Research | Neovascularization | Biological control systems | Endothelium | Angiogenesis | Signal transduction | Cellular biology | Kinases | DNA damage
Journal Article
Journal of Virology, ISSN 0022-538X, 09/2012, Volume 86, Issue 18, pp. 9899 - 9910
Journal Article
Oncogene, ISSN 0950-9232, 12/2001, Volume 20, Issue 58, pp. 8342 - 8357
The basic-helix-loop-helix (bHLH) proteins are a superfamily of DNA-binding transcription factors that regulate numerous biological processes in both... 
Basic-helic-loop-helix | Hairy | Transcription | Corepressor | Notch | PAN-NEURAL GENE | transcription | BIOCHEMISTRY & MOLECULAR BIOLOGY | corepressor | SOMITE BOUNDARY FORMATION | NOTCH PATHWAY | FATE SPECIFICATION | LOOP-HELIX REPRESSOR | LUNATIC-FRINGE | CELL BIOLOGY | hairy | XENOPUS-EMBRYOS | ONCOLOGY | PROMOTES NEURONAL DIFFERENTIATION | basic-helix-loop-helix | GENETICS & HEREDITY | CENTRAL-NERVOUS-SYSTEM | PRESOMITIC MESODERM | Vertebrates - genetics | Humans | Mesoderm - cytology | Phylogeny | Blood Vessels - embryology | Drosophila Proteins - physiology | Cell Transformation, Neoplastic - genetics | Membrane Proteins - physiology | Transcription, Genetic | Nervous System - embryology | Dimerization | Basic Helix-Loop-Helix Transcription Factors | Gene Expression Regulation, Developmental - physiology | Amino Acid Sequence | DNA-Binding Proteins - physiology | Morphogenesis - genetics | Membrane Proteins - genetics | Muscles - embryology | Repressor Proteins - genetics | Terminology as Topic | Xenopus Proteins | Embryonic and Fetal Development - physiology | Mice, Knockout | Cell Lineage | Mice | Transcription Factors | Morphogenesis - physiology | Muscles - cytology | Drosophila melanogaster - embryology | Multigene Family | Drosophila melanogaster - physiology | Embryonic and Fetal Development - genetics | Vertebrates - embryology | Vertebrates - physiology | Gene Expression Regulation, Developmental - genetics | Molecular Sequence Data | Neurons - cytology | Helix-Loop-Helix Motifs | Drosophila melanogaster - genetics | Repressor Proteins - physiology | Cell Differentiation - genetics | Receptors, Notch | Cell Differentiation - physiology | Protein Structure, Tertiary | Proteins - physiology | Nerve Tissue Proteins - physiology | Leukemia-Lymphoma, Adult T-Cell - genetics | Neovascularization, Physiologic - genetics | Leukemia-Lymphoma, Adult T-Cell - pathology | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Blood Vessels - cytology | Proteins - genetics | Sequence Homology, Amino Acid | Neovascularization, Physiologic - physiology | Sequence Alignment | Animals | Drosophila Proteins - genetics | Evolution, Molecular
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 04/2007, Volume 34, Issue 4, pp. 255 - 262
SUMMARY • In general, cardiac hypertrophy (an increase in heart mass) is a poor prognostic sign. Cardiac enlargement is a characteristic of most forms of heart... 
heart failure | pathological | cardioprotection | exercise | cardiac function | cardiac hypertrophy | athlete's heart | physiological | Athlete's heart | Heart failure | Cardioprotection | Exercise | Cardiac function | Pathological | Cardiac hypertrophy | Physiological | PRESSURE-OVERLOAD | PHYSIOLOGY | MYOCARDIAL-INFARCTION | DILATED CARDIOMYOPATHY | BLOOD-PRESSURE | PHARMACOLOGY & PHARMACY | PHOSPHOINOSITIDE-3-OH KINASE | PHYSICAL-ACTIVITY | CARDIOVASCULAR RISK | GROWTH-FACTOR-I | TRANSGENIC MICE | Gene Expression - drug effects | Humans | Cardiomegaly - drug therapy | Heart Failure - physiopathology | Cardiomegaly - physiopathology | Cardiomegaly - pathology | Heart Failure - pathology | Heart Failure - drug therapy | Animals | Signal Transduction - drug effects | Models, Biological | Myocardium - metabolism | Signal Transduction - physiology | Exercise - physiology | Sarcoplasmic Reticulum Calcium-Transporting ATPases, physiology | Actins, physiology | Ribosomal Protein S6 Kinases, physiology | Endothelin-1, physiology | Myosin Heavy Chains, physiology | Nerve Growth Factor, physiology | Calcineurin, physiology | Insulin-Like Growth Factor I, physiology | Cardiomegaly, pathology | Sports, physiology | Cardiomegaly, physiopathology | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating), physiology | Angiotensin II, physiology | Proto-Oncogene Proteins c-akt, physiology | Cardiac Myosins, physiology | Mice, Transgenic | Mitogen-Activated Protein Kinase Kinases, physiology | 1-Phosphatidylinositol 3-Kinase, physiology | Cardiomegaly, etiology | Mice, Knockout | Natriuretic Peptide, Brain, physiology | Signal Transduction, physiology | Mice | Atrial Natriuretic Factor, physiology | GTP-Binding Proteins, physiology | Physiological aspects | Heart enlargement | Health aspects
Journal Article
Stroke, ISSN 0039-2499, 05/2015, Volume 46, Issue 5, pp. 1337 - 1343
BACKGROUND AND PURPOSE—Cerebral cavernous malformation (CCM) is a neurovascular dysplasia characterized by conglomerates of enlarged endothelial channels in... 
DLL4 protein | Notch3 protein | angiogenesis effect | pericytes | NOTCH | BLOOD-BRAIN-BARRIER | CLINICAL NEUROLOGY | FEATURES | INHIBITS SPROUTING ANGIOGENESIS | MUSCLE-CELL FORMATION | VASCULAR INTEGRITY | IN-VIVO | PERIPHERAL VASCULAR DISEASE | LIGAND |