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1. Full Text Expression of Follicle-Stimulating Hormone Receptor in Tumor Blood Vessels
by Radu, Aurelian and Pichon, Christophe and Camparo, Philippe and Antoine, Martine and Allory, Yves and Couvelard, Anne and Fromont, Gaëlle and Hai, Mai Thu Vu and Ghinea, Nicolae
The New England journal of medicine, ISSN 0028-4793, 10/2010, Volume 363, Issue 17, pp. 1621 - 1630
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Biological and medical sciences | General aspects | Medical sciences | Endothelial Cells - chemistry | Prostatic Neoplasms - blood supply | Neoplasm Transplantation | Prostatic Neoplasms - chemistry | Blood Vessels - chemistry | Immunologic Techniques | Receptors, FSH - immunology | Electrophoresis, Polyacrylamide Gel | Humans | Male | Neoplasms - blood supply | Transplantation, Heterologous | Microscopy, Immunoelectron | Neoplasms - chemistry | Animals | In Situ Hybridization | Antibodies, Monoclonal - analysis | Neoplasms, Experimental - chemistry | Mice | Disease Models, Animal | Neoplasms, Experimental - blood supply | Receptors, FSH - analysis | Care and treatment | Follicle-stimulating hormone | Blood-vessels | Analysis | Physiological aspects | Genetic aspects | Gene expression | Health aspects | Cancer | Tumors | Proteins | Microscopy | Rodents | Surgery | Index Medicus | Abridged Index Medicus
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2. Full Text Monitoring of Blood Vessels and Tissues by a Population of Monocytes with Patrolling Behavior
by Cedric Auffray and Darin Fogg and Meriem Garfa and Gaelle Elain and Olivier Join-Lambert and Samer Kayal and Sabine Sarnacki and Ana Cumano and Gregoire Lauvau and Frederic Geissmann
Science (American Association for the Advancement of Science), ISSN 0036-8075, 8/2007, Volume 317, Issue 5838, pp. 666 - 670
Monocytes | Microscopy | Blood vessels | Reports | Infections | Macrophages | Blood | Chemokine receptors | Endothelial cells | Endothelium | Integrins | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Myeloid cells: ontogeny, maturation, markers, receptors | Fundamental immunology | Biological and medical sciences | Immunobiology | Monocytes, macrophages | Monocytes - cytology | Receptors, Chemokine - analysis | Mesenteric Arteries - immunology | Monocytes - immunology | Mesenteric Veins - immunology | Blood Vessels - immunology | Listeriosis - immunology | Cell Differentiation | Monocytes - physiology | Macrophages - immunology | Endothelium, Vascular - immunology | Lymphocyte Function-Associated Antigen-1 - physiology | Gene Expression Regulation | Listeria monocytogenes | Inflammation - immunology | Macrophages - cytology | Immunity, Innate | Cell Adhesion | Dermis - immunology | Microscopy, Confocal | Animals | Receptors, Chemokine - physiology | Mice | CX3C Chemokine Receptor 1 | Cell Movement | Immune response | Causes of | Inflammation | Research | Leukocytes | Leucocytes | Immunology | Cellular biology | Molecular biology | Tissues | Index Medicus | Blood Vessels | Mesenteric Veins | Dermis | Listeria Infections | Immunity, Natural | Life Sciences | Lymphocyte Function-Associated Antigen-1 | Mesenteric Arteries | Receptors, Chemokine | Endothelium, Vascular
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3. Full Text Angiotensin inhibition enhances drug delivery and potentiates chemotherapy by decompressing tumour blood vessels
by Chauhan, Vikash P and Martin, John D and Liu, Hao and Lacorre, Delphine A and Jain, Saloni R and Kozin, Sergey V and Stylianopoulos, Triantafyllos and Mousa, Ahmed S and Han, Xiaoxing and Adstamongkonkul, Pichet and Popović, Zoran and Huang, Peigen and Bawendi, Moungi G and Boucher, Yves and Jain, Rakesh K
Nature communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 2516 - 2516
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Stromal Cells - pathology | Humans | Pancreatic Neoplasms - blood supply | Transforming Growth Factor beta1 - metabolism | Gene Expression Regulation, Neoplastic | Stress, Mechanical | Angiotensin II Type 1 Receptor Blockers - pharmacology | Cell Hypoxia | Pancreatic Neoplasms - drug therapy | Receptor, Angiotensin, Type 1 - genetics | Mammary Neoplasms, Experimental - pathology | Stromal Cells - drug effects | Angiotensins - metabolism | Female | Mammary Neoplasms, Experimental - blood supply | Antineoplastic Agents - pharmacology | Mammary Neoplasms, Experimental - drug therapy | Angiotensins - antagonists & inhibitors | Endothelin-1 - genetics | Endothelin-1 - metabolism | Pancreatic Neoplasms - pathology | Stromal Cells - metabolism | Losartan - pharmacology | Transforming Growth Factor beta1 - genetics | Drug Repositioning | Drug Synergism | Collagen - metabolism | Mechanotransduction, Cellular | Animals | Hyaluronic Acid - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Connective Tissue Growth Factor - genetics | Fluorouracil - pharmacology | Mice | Connective Tissue Growth Factor - metabolism | Index Medicus
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4. Full Text Local Overexpression of Toll-Like Receptors at the Vessel Wall Induces Atherosclerotic Lesion Formation: Synergism of TLR2 and TLR4
by Shinohara, Masakazu and Hirata, Ken-ichi and Yamashita, Tomoya and Takaya, Tomofumi and Sasaki, Naoto and Shiraki, Rio and Ueyama, Tomomi and Emoto, Noriaki and Inoue, Nobutaka and Yokoyama, Mitsuhiro and Kawashima, Seinosuke
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 11/2007, Volume 27, Issue 11, pp. 2384 - 2391
Toll-like receptors | Adhesion molecules | Inflammation | Gene transfer | Atherosclerosis | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Diseases of the cardiovascular system | Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous | Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy | Atherosclerosis (general aspects, experimental research) | Biological and medical sciences | Anesthesia | Medical sciences | Anesthesia depending on type of surgery | Thoracic and cardiovascular surgery. Cardiopulmonary bypass | Blood and lymphatic vessels | Radiotherapy. Instrumental treatment. Physiotherapy. Reeducation. Rehabilitation, orthophony, crenotherapy. Diet therapy and various other treatments (general aspects) | Carotid Artery Diseases - immunology | Rabbits | Animals, Genetically Modified | Humans | Carotid Artery Diseases - physiopathology | Inflammation - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Diet, Atherogenic | Animals | Carotid Arteries - physiopathology | Carotid Arteries - immunology | Disease Models, Animal | Index Medicus
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5. Full Text Excess vascular endothelial growth factor-A disrupts pericyte recruitment during blood vessel formation
by Darden, Jordan and Darden, Jordan and Payne, Laura Beth and Payne, Laura Beth and Zhao, Huaning and Zhao, Huaning and Chappell, John C and Chappell, John C
Angiogenesis (London), ISSN 0969-6970, 2/2019, Volume 22, Issue 1, pp. 167 - 183
Biomedicine, general | Pericyte | Angiogenesis | Biomedicine | Development | Cancer Research | Oncology | Ophthalmology | Cardiology | VEGF-A | Mouse embryonic stem cells | Cell Biology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Signal Transduction | Endothelial Cells - metabolism | Models, Cardiovascular | Receptors, Notch - metabolism | Becaplermin - metabolism | Pericytes - cytology | Pericytes - metabolism | Receptors, Notch - genetics | Vascular Endothelial Growth Factor A - metabolism | Becaplermin - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Vascular Endothelial Growth Factor A - genetics | Mice, Knockout | Animals | Receptors, Vascular Endothelial Growth Factor - metabolism | Mice | Receptors, Vascular Endothelial Growth Factor - genetics | Neovascularization, Physiologic | Receptor, Platelet-Derived Growth Factor beta - metabolism | Blood vessels | Genetic aspects | Cellular signal transduction | Endothelial growth factors | Health aspects | Animal models | Platelet-derived growth factor | Transcription | Splicing | Migration | Crosstalk | Embryos | Blood | Endothelial cells | Proteins | Signal transduction | Signaling | Receptors | Pathways | Developmental stages | Proteolysis | Pericytes | Skin | Vascular endothelial growth factor | Growth factors | Investment | Index Medicus | development | mouse embryonic stem cells | pericyte | angiogenesis
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6. Full Text VEGFR-3 controls tip to stalk conversion at vessel fusion sites by reinforcing Notch signalling
by Tammela, Tuomas and Zarkada, Georgia and Nurmi, Harri and Jakobsson, Lars and Heinolainen, Krista and Tvorogov, Denis and Zheng, Wei and Franco, Claudio A and Murtomäki, Aino and Aranda, Evelyn and Miura, Naoyuki and Ylä-Herttuala, Seppo and Fruttiger, Marcus and Mäkinen, Taija and Eichmann, Anne and Pollard, Jeffrey W and Gerhardt, Holger and Alitalo, Kari
Nature cell biology, ISSN 1465-7392, 10/2011, Volume 13, Issue 10, pp. 1202 - 1213
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptors, Notch - antagonists & inhibitors | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Vascular Endothelial Growth Factor D - genetics | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Transduction, Genetic | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rhombencephalon - blood supply | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Forkhead Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | Antibodies - pharmacology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Vascular Endothelial Growth Factor B - genetics | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Physiological aspects | Blood vessels | Cellular signal transduction | Growth factor receptors | Research | Vascular endothelial growth factor | Index Medicus | Medicin och hälsovetenskap
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7. Full Text Development and plasticity of meningeal lymphatic vessels
by Antila, Salli and Karaman, Sinem and Nurmi, Harri and Airavaara, Mikko and Voutilainen, Merja H and Mathivet, Thomas and Chilov, Dmitri and Li, Zhilin and Koppinen, Tapani and Park, Jun-Hee and Fang, Shentong and Aspelund, Aleksanteri and Saarma, Mart and Eichmann, Anne and Thomas, Jean-Léon and Alitalo, Kari
The Journal of experimental medicine, ISSN 0022-1007, 12/2017, Volume 214, Issue 12, pp. 3645 - 3667
Immunology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Spinal Cord - drug effects | Humans | Injections, Intraventricular | Male | Lymphatic Vessels - drug effects | Microspheres | Sunitinib | Gene Deletion | Indoles - pharmacology | Lymph Nodes - drug effects | Biological Transport - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Animals, Newborn | Dependovirus - metabolism | Lymphatic Vessels - physiology | Signal Transduction | Mice, Inbred C57BL | Lymph Nodes - metabolism | Meninges - drug effects | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Lymphangiogenesis - drug effects | Pyrroles - pharmacology | Vascular Endothelial Growth Factor D - metabolism | Animals | Cerebrospinal Fluid - metabolism | Vascular Endothelial Growth Factor C - metabolism | Spinal Cord - physiology | Protein Kinase Inhibitors - pharmacology | Meninges - physiology | Tyrosine | Spinal nerves | Lymphatic system | Neuropathology | Developmental plasticity | Central nervous system | Muscles | Blood vessels | Smooth muscle | Cerebrospinal fluid | Wound drainage | Endothelial cells | Vascular endothelial growth factor receptors | Signal transduction | Inhibitors | Clonal deletion | Rodents | Nerves | Plasticity | Plastic properties | Mice | Meninges | Vascular endothelial growth factor | Protein-tyrosine kinase | Index Medicus | 301 | 320
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