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blood-spinal cord barrier (195) 195
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cell injury (14) 14
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tight junction (14) 14
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Biochemical and Biophysical Research Communications, ISSN 0006-291X, 01/2018, Volume 495, Issue 4, pp. 2525 - 2531
Journal Article
Journal of Neural Transmission, ISSN 0300-9564, 1/2011, Volume 118, Issue 1, pp. 155 - 176
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 3/2018, Volume 55, Issue 3, pp. 2577 - 2590
After spinal cord injury (SCI), tight junction (TJ) protein degradation increases permeability and disrupts the blood–spinal cord barrier (BSCB). The BSCB is... 
Neurology | Neurosciences | Biomedicine | Tight junctions | Blood–spinal cord barrier | Neurobiology | Neuroinflammation | MMP-8 | Spinal cord injury | Cell Biology | Occludin | FUNCTIONAL RECOVERY | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | NEUTROPHIL INFILTRATION | VASCULAR EVENTS | NEUROSCIENCES | BRAIN-BARRIER | CELL JUNCTIONS | MESSENGER-RNA | Blood-spinal cord barrier | NITRIC-OXIDE | HEME OXYGENASE-1 | CEREBRAL-ISCHEMIA | Inflammation - pathology | Spinal Cord - drug effects | Spinal Cord - enzymology | Rats | Tight Junctions - enzymology | Blood-Brain Barrier - drug effects | Rats, Sprague-Dawley | Matrix Metalloproteinase Inhibitors - therapeutic use | Spinal Cord Injuries - pathology | Dose-Response Relationship, Drug | Spinal Cord Injuries - prevention & control | Blood-Brain Barrier - pathology | Matrix Metalloproteinase Inhibitors - pharmacology | Animals | Blood-Brain Barrier - enzymology | Spinal Cord Injuries - enzymology | Inflammation - drug therapy | Matrix Metalloproteinase 8 - metabolism | Spinal Cord - pathology | Female | Inflammation - enzymology | Tight Junctions - pathology | Disease Models, Animal | Tight Junctions - drug effects | Prevention | Immunohistochemistry | Cysteine | Proteolysis | Surgery | Nervous system | Spinal cord injuries | Inflammation | Permeability | Recombinant proteins | Endothelium | Compression | Matrix metalloproteinase | Blood | Western blotting | Degradation | Interleukin 6 | Proteins | Antioxidants | Cell activation | Extravasation | Rodents | Metalloproteinase | Neutrophils | Leukocytes (neutrophilic) | Tumor necrosis factor-α | Zonula occludens-1 protein | Nitric-oxide synthase | Endothelial cells | Cell injury | Sodium | Neutrophil collagenase | Protein expression | Infiltration | N-Acetyl-L-cysteine
Journal Article
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 1/2013, Volume 125, Issue 1, pp. 111 - 120
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 06/2012, Volume 121, Issue 5, pp. 818 - 829
J. Neurochem. (2012) 121, 818–829. The disruption of blood–spinal cord barrier (BSCB) after spinal cord injury (SCI) elicits an intensive local inflammation by... 
matrix metalloprotease | spinal cord injury | blood–spinal cord barrier | inflammation | histone deacetylase inhibitor | tight junction | blood-spinal cord barrier | ACTIVATION | PROTEIN | HISTONE DEACETYLASE INHIBITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | POTENTIAL ROLES | NEUROSCIENCES | CELL-DEATH | BRAIN-BARRIER | INTRACEREBRAL HEMORRHAGE | RAT-BRAIN | EXPRESSION | Immunohistochemistry | In Situ Nick-End Labeling | Enzyme-Linked Immunosorbent Assay | Recovery of Function - drug effects | Capillary Permeability - drug effects | Rats | Male | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Valproic Acid - pharmacology | Blotting, Western | Spinal Cord Injuries - pathology | Matrix Metalloproteinase 9 - metabolism | Animals | Neuroprotective Agents - pharmacology | Spinal Cord Injuries - enzymology | Histone Deacetylase Inhibitors - pharmacology | Neurochemistry | Leucocytes | Spinal cord injuries | Pharmacology | Protease inhibitors | Cellular biology | Histone deacetylase | Tight junctions | Therapeutic applications | Blood cells | Heat shock proteins | Recovery of function | Inflammation | Permeability | Macrophages | Spinal cord injury | Valproic acid | Caspase-3 | Gelatinase B | Zonula occludens-1 protein | Metastases | Cell death | Chromium | Tumor necrosis factor- alpha | Apoptosis
Journal Article
Ultrasound in Medicine & Biology, ISSN 0301-5629, 09/2019, Volume 45, Issue 9, pp. 2417 - 2426
The blood–spinal cord barrier (BSCB) considerably limits the delivery and efficacy of treatments for spinal cord diseases. The blood–brain barrier can be... 
Spinal cord | Drug delivery | Ultrasound | Blood–spinal cord barrier | Blood–brain barrier | BRAIN-BARRIER | ACOUSTICS | FOCUSED ULTRASOUND | DISRUPTION | Blood-brain barrier | Blood-spinal cord barrier | VALIDATION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | DELIVERY | Drugs | Sects | Drug delivery systems | Analysis | Surgery | Nervous system | Vehicles | Index Medicus
Journal Article
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2016, Volume 20, Issue 6, pp. 1062 - 1075
After spinal cord injury (SCI), disruption of blood–spinal cord barrier (BSCB) elicits blood cell infiltration such as neutrophils and macrophages,... 
spinal cord injury | Rac1 | blood–spinal cord barrier | epidermal growth factor | PI3K/Akt | Blood-spinal cord barrier | Epidermal growth factor | Spinal cord injury | MEDICINE, RESEARCH & EXPERIMENTAL | FUNCTIONAL RECOVERY | blood-spinal cord barrier | ACTIVATION | MODEL | RHOA | CELL BIOLOGY | BRAIN-BARRIER | ENDOTHELIAL-CELLS | MEK/ERK | ISCHEMIA-REPERFUSION | EXPRESSION | Epidermal Growth Factor - administration & dosage | Spinal Cord - drug effects | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Epidermal Growth Factor - therapeutic use | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Adherens Junctions - metabolism | Proteolysis - drug effects | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Female | Spinal Cord Injuries - blood | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Tight Junctions - drug effects | Tight Junctions - metabolism | Oxygen | Endothelial Cells - metabolism | Morpholines - pharmacology | Rats, Sprague-Dawley | Glucose - deficiency | Animals | Signal Transduction - drug effects | Adherens Junctions - drug effects | Epidermal Growth Factor - pharmacology | rac1 GTP-Binding Protein - metabolism | Endothelial Cells - drug effects | Spinal cord injuries | Permeability | Analysis | PI3K | Akt | Original
Journal Article
Brain Research, ISSN 0006-8993, 2007, Volume 1157, Issue 1, pp. 126 - 137
Abstract The purpose of this study was to determine the ultrastructure of the blood–brain barrier (BBB) and blood–spinal cord barrier (BSCB) in G93A SOD1 mice... 
Neurology | Spinal cord | Mitochondria | Ultrastructure | Blood–spinal cord barrier | Brainstem | Motor neuron | Blood–brain barrier | G93A SOD1 mice | Endothelium | Basement membrane | Blood-brain barrier | Blood-spinal cord barrier | blood-brain barrier | MOTOR-NEURONS | endothelium | IMMUNOGLOBULIN SUPERFAMILY | blood-spinal cord barrier | basement membrane | WHITE-MATTER | mitochondria | IGG | AMYOTROPHIC-LATERAL-SCLEROSIS | ultrastructure | CEREBROSPINAL-FLUID | NEUROSCIENCES | motor neuron | MOUSE MODEL | DISEASE | OCCLUDIN | AQUAPORIN-4 | brainstem | spinal cord | Basement Membrane - ultrastructure | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Capillaries - pathology | Humans | Astrocytes - pathology | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Mitochondria - ultrastructure | Spinal Cord - ultrastructure | Blood-Brain Barrier - ultrastructure | Motor Neurons - pathology | Motor Neurons - ultrastructure | Brain - blood supply | Endothelial Cells - ultrastructure | Spinal Cord - pathology | Brain - ultrastructure | Disease Models, Animal | Extracellular Space - physiology | Microscopy, Electron, Transmission | Capillaries - physiopathology | Capillaries - ultrastructure | Brain Edema - etiology | Brain Edema - pathology | Mice, Inbred C57BL | Mice, Transgenic | Mitochondria - pathology | Spinal Cord - blood supply | Mutation - genetics | Astrocytes - ultrastructure | Tight Junctions - ultrastructure | Blood-Brain Barrier - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Brain Edema - physiopathology | Brain - pathology | Mice | Superoxide Dismutase-1 | Endothelial Cells - pathology | Tight Junctions - pathology
Journal Article