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Nature Medicine, ISSN 1078-8956, 10/2012, Volume 18, Issue 10, pp. 1560 - 1569
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 01/2018, Volume 233, Issue 1, pp. 688 - 698
Understanding the role and underlying regulation mechanism of autophagy in lipopolysaccharide‐induced lung injury (LPS‐LI) may provide potentially new... 
autophagy | lipopolysaccharide | acute lung injury | microvascular barrier | APOPTOSIS | ACTIVATION | PHYSIOLOGY | PERMEABILITY | MODEL | RESPIRATORY-DISTRESS-SYNDROME | PROTECTS | CELL BIOLOGY | LIVER | Capillary Permeability | Autophagy-Related Protein 7 - metabolism | Humans | Acute Lung Injury - genetics | Autophagy-Related Protein 5 - genetics | Autophagy | Lipopolysaccharides | Endothelial Cells - ultrastructure | Transfection | RNA Interference | Lung - ultrastructure | Blood-Air Barrier - ultrastructure | Pulmonary Edema - pathology | Acute Lung Injury - metabolism | Lung - metabolism | Zonula Occludens-1 Protein - metabolism | Pulmonary Edema - metabolism | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Acute Lung Injury - pathology | Autophagy-Related Protein 7 - genetics | Pulmonary Edema - chemically induced | Stress Fibers - metabolism | Animals | Blood-Air Barrier - metabolism | Acute Lung Injury - chemically induced | Autophagy-Related Protein 5 - metabolism | Lung - blood supply | Acute respiratory distress syndrome | Permeability | Mitogens | Endothelium | Interleukin | Hemorrhage | Metastases | Bronchoalveolar lavage | Rodents | Interleukin 1 | Tumor necrosis factor-TNF | Inhibition | Alveoli | Injuries | Edema | Cytokines | Bronchus | Lactate dehydrogenase | Pharmacology | L-Lactate dehydrogenase | Zonula occludens-1 protein | Endothelial cells | Lungs | Tumor necrosis factor | Infiltration | In vivo methods and tests | Microvasculature | Lactic acid | Viability | Phagocytosis | Integrity | Index Medicus
Journal Article
EXPERIMENTAL CELL RESEARCH, ISSN 0014-4827, 07/2007, Volume 313, Issue 11, pp. 2504 - 2520
Pyostaglandin E-2 (PGE(2)) and prostacyclin are lipid mediators produced by cyclooxygenase and implicated in the regulation of vascular function, wound repair,... 
cytoskeleton | PROTEIN-KINASE | permeability | small GTPases | PKA | EXCHANGE FACTOR TIAM1 | lung endothelium | GEFs | SPHINGOSINE 1-PHOSPHATE | CELL BIOLOGY | SIGNAL-TRANSDUCTION | CYCLIC-AMP | PROSTANOID RECEPTORS | CELL JUNCTIONS | ONCOLOGY | IN-VIVO | RHO GTPASES | CYTOSKELETAL REARRANGEMENT | Proto-Oncogene Proteins c-vav - antagonists & inhibitors | Proto-Oncogene Proteins c-vav - genetics | Humans | rac GTP-Binding Proteins - metabolism | Male | Lung - cytology | Respiratory Distress Syndrome, Adult - metabolism | Adherens Junctions - metabolism | rac GTP-Binding Proteins - antagonists & inhibitors | Blood-Air Barrier - drug effects | Cyclic AMP-Dependent Protein Kinases - genetics | Guanine Nucleotide Exchange Factors - metabolism | rac GTP-Binding Proteins - genetics | Epoprostenol - pharmacology | Lung - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic AMP - metabolism | T-Lymphoma Invasion and Metastasis-inducing Protein 1 | Cyclic AMP-Dependent Protein Kinases - metabolism | Dinoprostone - pharmacology | Guanine Nucleotide Exchange Factors - genetics | Cytoskeleton - pathology | Respiratory Distress Syndrome, Adult - prevention & control | Endothelial Cells - metabolism | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Cells, Cultured | Blood-Air Barrier - cytology | Permeability | Adherens Junctions - pathology | Epoprostenol - analogs & derivatives | Animals | Blood-Air Barrier - metabolism | Guanine Nucleotide Exchange Factors - antagonists & inhibitors | Lung - drug effects | Adherens Junctions - drug effects | rac1 GTP-Binding Protein - antagonists & inhibitors | Cytoskeleton - metabolism | Mice | Cytoskeleton - drug effects | Proto-Oncogene Proteins c-vav - metabolism | rac1 GTP-Binding Protein - metabolism | Endothelial Cells - drug effects | rac1 GTP-Binding Protein - genetics
Journal Article
Toxicology Letters, ISSN 0378-4274, 09/2017, Volume 279, pp. 22 - 32
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 06/2008, Volume 215, Issue 3, pp. 715 - 724
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e110286
A disintegrin and metalloproteinase15 (ADAM15) has been shown to be upregulated and mediate endothelial hyperpermeability during inflammation and sepsis. This... 
METALLOPROTEINASE ADAM15 | DISINTEGRIN | LET-7 MICRORNA | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | LUNG INJURY | GROWTH-FACTOR | SEPSIS | CATALYTIC-ACTIVITY | UP-REGULATION | EPIDEMIOLOGY | Human Umbilical Vein Endothelial Cells | ADAM Proteins - chemistry | Endothelial Cells - metabolism | Membrane Proteins - genetics | Down-Regulation | Humans | Gene Expression Regulation | Immunophenotyping | Permeability | ADAM Proteins - metabolism | Blood-Air Barrier - metabolism | Membrane Proteins - chemistry | RNA Interference | Endothelium, Vascular - metabolism | Base Sequence | Cell Membrane - metabolism | Membrane Proteins - metabolism | MicroRNAs - genetics | 3' Untranslated Regions | ADAM Proteins - genetics | Binding Sites | MicroRNAs - chemistry | Infection | MicroRNA | Luciferase | Inflammation | Gene therapy | Health aspects | Membrane proteins | Endothelium | Metastasis | Kinases | Cell surface | Cell adhesion & migration | Lipopolysaccharides | Proteins | Angiogenesis | Transfection | Rodents | Surgery | Cell adhesion | Cell cycle | Extracellular matrix | Physiology | Biodegradation | Therapeutic applications | Neutrophils | MiRNA | Pharmacology | Gene expression | Ribonucleic acid--RNA | Endothelial cells | Inflammatory bowel disease | Medicine | 3' Untranslated regions | MicroRNAs | Sepsis | Cancer | RNA | Ribonucleic acid
Journal Article
PLoS Pathogens, ISSN 1553-7366, 11/2013, Volume 9, Issue 11, p. e1003727
Journal Article
Particle and Fibre Toxicology, ISSN 1743-8977, 08/2013, Volume 10, Issue 1, pp. 35 - 35
Background: Nanotechnology, particularly the use of multi-walled carbon nanotubes (MWCNT), is a rapidly growing discipline with implications for advancement in... 
Pulmonary inflammation | MWCNT | Pulmonary fibrosis | Airway epithelium | Co-culture | Endothelium | LUNG | PROTEIN | PARTICLES | TOXICITY | IN-VITRO MODELS | NANOPARTICLES | REACTIVE OXYGEN | INFLAMMATION | TOXICOLOGY | NF-KAPPA-B | EXPRESSION | Phosphorylation | Reactive Oxygen Species - metabolism | Respiratory Mucosa - drug effects | Cadherins - metabolism | Capillaries - pathology | Coculture Techniques | Humans | Actins - metabolism | Neovascularization, Pathologic | Capillary Permeability - drug effects | Capillaries - drug effects | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Antigens, CD - metabolism | Respiratory Mucosa - pathology | Blood-Air Barrier - drug effects | Nanotubes, Carbon - toxicity | Alveolar Epithelial Cells - pathology | Time Factors | Blood-Air Barrier - pathology | Inflammation Mediators - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Capillaries - metabolism | STAT3 Transcription Factor - metabolism | Endothelial Cells - metabolism | Alveolar Epithelial Cells - metabolism | Alveolar Epithelial Cells - drug effects | Intercellular Adhesion Molecule-1 - metabolism | Blood-Air Barrier - metabolism | Respiratory Mucosa - metabolism | Endothelial Cells - pathology | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects | Muscle proteins | Epithelial cells | Analysis | Nanotubes | Nanoparticles | Angiogenesis | Occupational safety | Transmission electron microscopy | Lungs | Rodents | Nanomaterials | Carbon | Cell adhesion & migration | Index Medicus
Journal Article