X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (18205) 18205
Newspaper Article (278) 278
Book Chapter (134) 134
Publication (30) 30
Book / eBook (28) 28
Conference Proceeding (24) 24
Dissertation (21) 21
Trade Publication Article (11) 11
Web Resource (7) 7
Newsletter (6) 6
Magazine Article (5) 5
Streaming Video (3) 3
Reference (2) 2
Book Review (1) 1
Government Document (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (11360) 11360
blood-brain barrier (9342) 9342
humans (8798) 8798
blood-brain-barrier (6868) 6868
neurosciences (6328) 6328
male (5847) 5847
mice (4247) 4247
rats (4048) 4048
female (3576) 3576
brain (3456) 3456
disease models, animal (2792) 2792
brain - pathology (2748) 2748
permeability (2658) 2658
blood-brain barrier - metabolism (2640) 2640
clinical neurology (2593) 2593
central-nervous-system (2592) 2592
inflammation (2498) 2498
blood-brain barrier - drug effects (2249) 2249
neurology (2158) 2158
brain - metabolism (1980) 1980
proteins (1971) 1971
pathology (1967) 1967
blood-brain barrier - pathology (1893) 1893
alzheimer's disease (1840) 1840
expression (1806) 1806
blood–brain barrier (1731) 1731
cell biology (1662) 1662
analysis (1618) 1618
research (1593) 1593
biochemistry & molecular biology (1569) 1569
rats, sprague-dawley (1569) 1569
stroke (1558) 1558
immunology (1534) 1534
pharmacology & pharmacy (1495) 1495
rodents (1476) 1476
mice, inbred c57bl (1447) 1447
oxidative stress (1428) 1428
alzheimers-disease (1382) 1382
central nervous system (1368) 1368
nervous system (1333) 1333
middle aged (1322) 1322
magnetic resonance imaging (1316) 1316
adult (1299) 1299
neurons (1255) 1255
immunohistochemistry (1228) 1228
medicine (1214) 1214
health aspects (1192) 1192
blood-brain barrier - physiology (1188) 1188
ischemia (1176) 1176
multidisciplinary sciences (1149) 1149
apoptosis (1148) 1148
physiological aspects (1120) 1120
multiple sclerosis (1118) 1118
research article (1089) 1089
aged (1085) 1085
brain research (1072) 1072
time factors (1070) 1070
cells, cultured (1045) 1045
review (1045) 1045
astrocytes (1039) 1039
disease (1025) 1025
endothelium (1006) 1006
cytokines (997) 997
neurodegeneration (991) 991
in-vivo (975) 975
rats, wistar (974) 974
in-vitro (964) 964
medicine, research & experimental (953) 953
cerebrospinal-fluid (936) 936
brain - drug effects (934) 934
cells (924) 924
brain - blood supply (923) 923
biomedicine (917) 917
gene expression (917) 917
blood-brain barrier - physiopathology (914) 914
endothelial-cells (901) 901
microglia (893) 893
neurodegenerative diseases (887) 887
endothelial cells (858) 858
pathogenesis (847) 847
oncology (839) 839
science (831) 831
biology (820) 820
multiple-sclerosis (806) 806
activation (794) 794
neuroprotection (790) 790
risk factors (789) 789
brain neoplasms - pathology (780) 780
care and treatment (774) 774
edema (759) 759
experimental autoimmune encephalomyelitis (755) 755
dementia (742) 742
mouse model (733) 733
animal models (731) 731
endothelial cells - metabolism (721) 721
neurons - pathology (712) 712
alzheimer disease - pathology (704) 704
blood brain barrier (701) 701
studies (701) 701
endocrinology & metabolism (696) 696
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (18434) 18434
Japanese (115) 115
German (80) 80
French (67) 67
Russian (67) 67
Chinese (45) 45
Polish (19) 19
Spanish (18) 18
Italian (8) 8
Dutch (4) 4
Portuguese (4) 4
Czech (2) 2
Danish (2) 2
Serbian (2) 2
Hungarian (1) 1
Korean (1) 1
Lithuanian (1) 1
Romanian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Molecular Neurobiology, ISSN 0893-7648, 5/2016, Volume 53, Issue 4, pp. 2451 - 2467
.... However, the role of hydrogen sulfide, as well as N-methyl-d-aspartate receptor (NMDAR) activation, in hyperhomocysteinemia (HHcy) induced blood–brain barrier (BBB... 
Cerebrovascular pathology | Neurology | Neurosciences | Biomedicine | Blood–brain barrier dysfunction | Alzheimer’s disease | Neurobiology | Homocysteine | Cell Biology | Dementia | COGNITIVE PERFORMANCE | NEUROSCIENCES | MATRIX METALLOPROTEINASES | INDUCED MEMORY IMPAIRMENT | RISK-FACTOR | Blood-brain barrier dysfunction | RECEPTOR TRAFFICKING | MATRIX-METALLOPROTEINASE-9 | RAT-BRAIN | Alzheimer's disease | PLASMA HOMOCYSTEINE | EXPRESSION | Memory - drug effects | Glial Fibrillary Acidic Protein - genetics | Cadherins - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Blood-Brain Barrier - physiopathology | Claudin-5 - metabolism | Microvessels - pathology | Male | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Antigens, CD - genetics | Alzheimer Disease - pathology | Antigens, CD - metabolism | Matrix Metalloproteinase 9 - metabolism | Cadherins - genetics | Synapses - drug effects | Alzheimer Disease - physiopathology | Hydrogen Sulfide - pharmacology | Cerebrovascular Circulation - drug effects | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Alzheimer Disease - drug therapy | Microvessels - drug effects | Claudin-5 - genetics | Cystathionine beta-Synthase - metabolism | Permeability | Blood-Brain Barrier - drug effects | Nerve Tissue Proteins - genetics | Blood-Brain Barrier - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Blood-Brain Barrier - pathology | Hydrogen Sulfide - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Intercellular Adhesion Molecule-1 - genetics | Avoidance Learning - drug effects | Alzheimer Disease - genetics | Dizocilpine Maleate - pharmacology | Hydrogen sulfide | Methyl aspartate | Brain | RNA | Neurons | Intermediate filament proteins | cerebrovascular pathology | dementia | blood brain barrier dysfunction
Journal Article
Brain Research, ISSN 0006-8993, 2007, Volume 1157, Issue 1, pp. 126 - 137
...–brain barrier (BBB) and blood–spinal cord barrier (BSCB) in G93A SOD1 mice modeling ALS at different stages of disease... 
Neurology | Spinal cord | Mitochondria | Ultrastructure | Blood–spinal cord barrier | Brainstem | Motor neuron | Blood–brain barrier | G93A SOD1 mice | Endothelium | Basement membrane | Blood-brain barrier | Blood-spinal cord barrier | blood-brain barrier | MOTOR-NEURONS | endothelium | IMMUNOGLOBULIN SUPERFAMILY | blood-spinal cord barrier | basement membrane | WHITE-MATTER | mitochondria | IGG | AMYOTROPHIC-LATERAL-SCLEROSIS | ultrastructure | CEREBROSPINAL-FLUID | NEUROSCIENCES | motor neuron | MOUSE MODEL | DISEASE | OCCLUDIN | AQUAPORIN-4 | brainstem | spinal cord | Basement Membrane - ultrastructure | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Capillaries - pathology | Humans | Astrocytes - pathology | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Mitochondria - ultrastructure | Spinal Cord - ultrastructure | Blood-Brain Barrier - ultrastructure | Motor Neurons - pathology | Motor Neurons - ultrastructure | Brain - blood supply | Endothelial Cells - ultrastructure | Spinal Cord - pathology | Brain - ultrastructure | Disease Models, Animal | Extracellular Space - physiology | Microscopy, Electron, Transmission | Capillaries - physiopathology | Capillaries - ultrastructure | Brain Edema - etiology | Brain Edema - pathology | Mice, Inbred C57BL | Mice, Transgenic | Mitochondria - pathology | Spinal Cord - blood supply | Mutation - genetics | Astrocytes - ultrastructure | Tight Junctions - ultrastructure | Blood-Brain Barrier - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Brain Edema - physiopathology | Brain - pathology | Mice | Superoxide Dismutase-1 | Endothelial Cells - pathology | Tight Junctions - pathology
Journal Article
Acta neuropathologica, ISSN 1432-0533, 2009, Volume 118, Issue 1, pp. 103 - 113
.... Cerebral hypoperfusion and impaired amyloid β-peptide (Aβ) clearance across the blood–brain barrier (BBB) may contribute to the onset and progression of dementia AD type... 
Pathology | Neurosciences | Medicine & Public Health | Alzheimer’s disease | Neurovascular | Clearance | Blood–brain barrier | Blood-brain barrier | Alzheimer's disease | CEREBRAL AMYLOID ANGIOPATHY | MICROVASCULAR PATHOLOGY | TOPOGRAPHICAL DISTRIBUTION | PATHOLOGY | MILD COGNITIVE IMPAIRMENT | A-BETA PEPTIDES | NEUROSCIENCES | CLINICAL NEUROLOGY | APOLIPOPROTEIN-E | A beta | SERUM RESPONSE FACTOR | CENTRAL-NERVOUS-SYSTEM | SMOOTH-MUSCLE-CELLS | RECEPTOR-RELATED PROTEIN-1 | Alzheimer Disease - complications | Reactive Oxygen Species - metabolism | Humans | Blood-Brain Barrier - physiopathology | Apolipoproteins E - metabolism | Alzheimer Disease - pathology | Brain - blood supply | Cerebral Amyloid Angiopathy - complications | Microglia - physiology | Cerebrovascular Disorders - complications | Amyloid beta-Peptides - metabolism | Macrophages - physiology | Cerebrovascular Disorders - physiopathology | Alzheimer Disease - physiopathology | Cerebral Amyloid Angiopathy - physiopathology | Serum Response Factor - metabolism | Brain - physiopathology | Nuclear Proteins - metabolism | Cerebrovascular Circulation | Blood-Brain Barrier - pathology | Cerebral Amyloid Angiopathy - pathology | Animals | Brain - pathology | Trans-Activators - metabolism | Matrix Metalloproteinases - metabolism | Cerebrovascular Disorders - pathology
Journal Article
Fluids and barriers of the CNS, ISSN 2045-8118, 2018, Volume 15, Issue 1, pp. 24 - 17
Breakdown of the blood-brain barrier (BBB) or inner blood-retinal barrier (BRB), induced by pathologically elevated levels of vascular endothelial growth factor (VEGF... 
Plasmalemma vesicle-associated protein | Blood-brain barrier | Blood-retinal barrier | Cerebral edema | Diabetic macular edema | INDUCED RETINOPATHY | VASCULAR-PERMEABILITY | TRANSCELLULAR TRANSPORT | NEUROSCIENCES | DIFFERENTIATED MICRODOMAINS | MEDIATED TRANSCYTOSIS | FENESTRATED ENDOTHELIA | ANTIGEN PAL-E | LUMINAL SURFACE | CAPILLARY ENDOTHELIUM | ENDOTHELIAL GROWTH-FACTOR | Eye - metabolism | Brain Edema - metabolism | Capillary Permeability | Brain Edema - pathology | Diabetes Complications - metabolism | Humans | Macular Edema - pathology | Blood-Retinal Barrier - pathology | Macular Edema - metabolism | Blood-Brain Barrier - metabolism | Blood-Retinal Barrier - metabolism | Brain - metabolism | Blood-Brain Barrier - pathology | Animals | Carrier Proteins - metabolism | Macular Edema - complications | Membrane Proteins - metabolism | Diabetes Complications - pathology | Diabetic retinopathy | Brain | Nervous system diseases | Neurons | Mortality | Cell membranes | Permeability | Endothelium | Stroke (Disease) | Proteins | Health aspects | Vascular endothelial growth factor | Corticoids | Neuroprotection | Retinopathy | Brain cancer | Central nervous system | Retina | Angiogenesis | Ischemia | Protein transport | Edema | Stroke | Wound healing | Caveolae | Therapeutic applications | Diabetes mellitus | Morbidity | Endothelial cells | Plasma membranes | Diabetes | Cancer | Structure-function relationships | Blood–retinal barrier | Blood–brain barrier
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 9/2011, Volume 122, Issue 3, pp. 293 - 311
...), leading to disturbed cerebral blood flow and cognitive dysfunction, posing the question how cerebrovascular pathology contributes to the pathology of AD... 
Pathology | Neurosciences | Congophilic amyloid angiopathy | Medicine & Public Health | Alzheimer’s disease | Astrocytes | Cerebral glucose metabolism | Alzheimer's disease | MICROVASCULAR PATHOLOGY | ALZHEIMERS-DISEASE | NEUROVASCULAR MECHANISMS | PATHOLOGY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | GLUCOSE | MOUSE MODEL | LACTATE | A-BETA | SMOOTH-MUSCLE-CELLS | ANGIOPATHY | Microdialysis - methods | Humans | Astrocytes - pathology | Dystroglycans - metabolism | Glucose Transporter Type 1 - metabolism | Muscle, Smooth - ultrastructure | Glial Fibrillary Acidic Protein - metabolism | Microscopy, Electron, Scanning - methods | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | Cell Culture Techniques | Disease Models, Animal | Endothelium - pathology | Mice, Transgenic | Cerebral Arteries - ultrastructure | Basement Membrane - metabolism | Disease Progression | Symporters - metabolism | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Cerebral Arteries - metabolism | Brain - pathology | Glucose - metabolism | Plaque, Amyloid - metabolism | Mice | Astrocytes - metabolism | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Cerebral Amyloid Angiopathy - complications | Monocarboxylic Acid Transporters - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Hemorrhage - etiology | Amyloid beta-Protein Precursor - metabolism | Lactase - metabolism | Cerebral Amyloid Angiopathy - genetics | Astrocytes - drug effects | Plaque, Amyloid - pathology | Gene Expression Regulation - genetics | Hemorrhage - metabolism | Muscle, Smooth - metabolism | Cerebrovascular Disorders - etiology | Cerebral Amyloid Angiopathy - pathology | Animals | Endothelium - metabolism | Glucose Transporter Type 1 - genetics | Symporters - genetics | Cerebral Arteries - pathology | Laminin - metabolism | Monocarboxylic Acid Transporters - genetics | Cerebrovascular Disorders - pathology | Hemorrhage - pathology | Muscle, Smooth - pathology | Glucose transporter | Leakage | Brain | Neurodegenerative diseases | Cognitive ability | Transgenic mice | Blood vessels | Data processing | Smooth muscle | beta -Amyloid | Glucose transport | Metabolism | Amyloid precursor protein | Blood-brain barrier | Cerebral blood flow | Lactic acid | Mutation | Original Paper
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2017, Volume 51, pp. 104 - 112
Abstract Blood-brain barrier (BBB) dysfunction might be an important component of many neurodegenerative disorders... 
Neurology | Internal Medicine | APOE ε4 | Amyloid | Diabetes | Blood-brain barrier | Vascular pathology | Dementia | ALZHEIMERS-DISEASE | COGNITIVE IMPAIRMENT | APOE epsilon 4 | FRONTOTEMPORAL LOBAR DEGENERATION | CEREBROSPINAL-FLUID | DEPENDENT MANNER | NEUROSCIENCES | GERIATRICS & GERONTOLOGY | TIGHT JUNCTIONS | VASCULAR DEMENTIA | DYSFUNCTION | DIAGNOSTIC-CRITERIA | LEWY BODIES | Diabetes Mellitus - blood | Capillary Permeability | Humans | Middle Aged | Blood-Brain Barrier - physiopathology | Immunoglobulins - cerebrospinal fluid | Vascular Endothelial Growth Factor A - cerebrospinal fluid | Cell Adhesion Molecules - cerebrospinal fluid | Genotype | Male | Diabetes Mellitus - cerebrospinal fluid | Dementia - etiology | Serum Albumin | Cell Adhesion Molecule-1 | Apolipoproteins E - genetics | Amyloid beta-Peptides - metabolism | Aged, 80 and over | Albumins - cerebrospinal fluid | Diabetes Mellitus - etiology | Female | Aged | Cohort Studies | Nervous system diseases | Neurosciences | PET imaging | Endothelial growth factors | Physiological aspects | Brain damage | Genetic aspects | Permeability | Apolipoproteins | Alzheimer's disease | Endothelium | Medical research | Medicine, Experimental | lewy bodies | vascular dementia | Geriatrics & Gerontology | Neurosciences & Neurology | dependent manner | dysfunction | cerebrospinal-fluid | Endokrinologi och diabetes | diagnostic-criteria | cognitive impairment | tight junctions | Geriatrik | frontotemporal lobar degeneration | Neurovetenskaper | Endocrinology and Diabetes | Geriatrics | alzheimers-disease
Journal Article