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1. Full Text Calcium Metabolism in Health and Disease
by Munro Peacock
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 01/2010, Volume 5, Issue Supplement 1, pp. S23 - S30
This brief review focuses on calcium balance and homeostasis and their relationship to dietary calcium intake and calcium supplementation in healthy subjects... 
SUPPLEMENTATION | MORTALITY | HOMEOSTASIS | BONE-MINERAL DENSITY | UROLOGY & NEPHROLOGY | PARATHYROID-HORMONE | CALCIFICATION | PHOSPHATE BINDERS | CHRONIC KIDNEY-DISEASE | ADOLESCENT | CHILDREN | Calcium - metabolism | Hypocalcemia - metabolism | Humans | Homeostasis | Hyperphosphatemia - metabolism | Kidney Diseases - drug therapy | Hypercalcemia - metabolism | Metabolic Diseases - drug therapy | Hypophosphatemia - metabolism | Intestines - metabolism | Nutrition Policy | Kidney - metabolism | Phosphates - metabolism | Animals | Metabolic Diseases - metabolism | Phosphorus - metabolism | Bone and Bones - metabolism | Calcium, Dietary - metabolism | Vitamin D - therapeutic use | Bone Remodeling | Chronic Disease | Dietary Supplements | Kidney Diseases - metabolism
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2. Full Text Insulin‐like growth factor‐II: its role in metabolic and endocrine disease
by Livingstone, Callum and Borai, Anwar
Clinical Endocrinology, ISSN 0300-0664, 06/2014, Volume 80, Issue 6, pp. 773 - 781
Insulin‐like growth factor‐ II ( IGF ‐ II ) is a widely expressed 7·5 kDa mitogenic peptide hormone. Although it is abundant in serum, understanding of its... 
POLYCYSTIC-OVARY-SYNDROME | BREAST-CANCER | BECKWITH-WIEDEMANN-SYNDROME | RECEPTOR ISOFORM | PROLIFERATIVE DIABETIC-RETINOPATHY | FACTOR BINDING PROTEIN-2 | ENDOCRINOLOGY & METABOLISM | INDUCED WEIGHT-LOSS | C-ASSOCIATED OSTEOSCLEROSIS | IGF-II | TRANSGENIC MICE | Endocrine System Diseases - metabolism | Polycystic Ovary Syndrome - metabolism | Humans | Gene Expression Regulation | Diabetes Mellitus - metabolism | Male | Endocrine System | Insulin-Like Growth Factor II - metabolism | Obesity - metabolism | Bone Diseases, Metabolic - metabolism | Animals | Growth Disorders - metabolism | Metabolic Diseases - metabolism | Female | Mice | Insulin-Like Growth Factor I - metabolism
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3. Full Text Klinefelter syndrome: Integrating genetics, neuropsychology, and endocrinology
by Gravholt, Claus H and Chang, Simon and Wallentin, Mikkel and Fedder, Jens and Moore, Philip and Skakkebæk, Anne
Endocrine Reviews, ISSN 0163-769X, 2018, Volume 39, Issue 4, pp. 389 - 423
Although first identified over 70 years ago, Klinefelter syndrome (KS) continues to pose substantial diagnostic challenges, as many patients are still... 
TESTICULAR SPERM EXTRACTION | METABOLIC SYNDROME | X-CHROMOSOME | BONE-MINERAL DENSITY | ENDOCRINOLOGY & METABOLISM | IDIOPATHIC HYPOGONADOTROPIC HYPOGONADISM | MITRAL-VALVE-PROLAPSE | OF-THE-LITERATURE | TURNER-SYNDROME | SEX-CHROMOSOME ABNORMALITIES | TESTOSTERONE REPLACEMENT THERAPY | Humans | Infertility, Male - etiology | Musculoskeletal Diseases - physiopathology | Male | Cardiovascular Diseases - genetics | Musculoskeletal Diseases - genetics | Klinefelter Syndrome - genetics | Infertility, Male - genetics | Metabolic Diseases - physiopathology | Cognitive Dysfunction - genetics | Cardiovascular Diseases - etiology | Klinefelter Syndrome - metabolism | Cardiovascular Diseases - physiopathology | Musculoskeletal Diseases - etiology | Cardiovascular Diseases - metabolism | Musculoskeletal Diseases - metabolism | Klinefelter Syndrome - complications | Cognitive Dysfunction - metabolism | Cognitive Dysfunction - etiology | Cognitive Dysfunction - physiopathology | Infertility, Male - physiopathology | Animals | Klinefelter Syndrome - physiopathology | Metabolic Diseases - metabolism | Metabolic Diseases - etiology | Metabolic Diseases - genetics | Infertility, Male - metabolism | Neonates | Therapy | Animal models | Senescence | Cognitive ability | Medical services | Cognition | Socioeconomics | Genetic effects | Genetics | Children | Diabetes mellitus (non-insulin dependent) | Phenotypes | Mortality | Medical treatment | Diabetes mellitus | Socio-economic aspects | Health risks | Breast cancer | Ribonucleic acid--RNA | Metabolism | Gene expression | Patients | Morbidity | Hypogonadism | Testosterone | Infertility | Diagnostic systems | Cardiovascular diseases | Endocrinology | Metabolic disorders | Tumors
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4. Adiponectin in metabolic bone disease
by Kanazawa, I
Current Medicinal Chemistry, ISSN 0929-8673, 11/2012, Volume 19, Issue 32, pp. 5481 - 5492
Adiponectin has attracted widespread attention because of its pivotal role in glucose metabolism and energy homeostasis. Adiponectin and its receptor are shown... 
Osteocalcin | Osteoblast | Bone mineral density | Osteoclast | Bone turnover | Type 2 diabetes mellitus | Fracture risk | Adiponectin | CHEMISTRY, MEDICINAL | BIOCHEMICAL MARKERS | HUMAN OSTEOBLASTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | bone turnover | osteoblast | TURNOVER MARKERS | fracture risk | DIABETES-MELLITUS | bone mineral density | MINERAL DENSITY | VERTEBRAL FRACTURES | INSULIN-RESISTANCE | type 2 diabetes mellitus | osteocalcin | PHARMACOLOGY & PHARMACY | osteoclast | POSTMENOPAUSAL WOMEN | SERUM ADIPONECTIN | Osteocalcin - metabolism | Humans | Receptors, Adiponectin - metabolism | Diabetes Mellitus, Type 2 - metabolism | Osteoclasts - cytology | Osteoclasts - metabolism | Fractures, Bone - metabolism | Bone Diseases, Metabolic - metabolism | Adipose Tissue - metabolism | Animals | Bone and Bones - metabolism | Adiponectin - metabolism | Osteoblasts - cytology | Osteoblasts - metabolism
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5. Full Text Estrogen Prevents Bone Loss via Estrogen Receptor α and Induction of Fas Ligand in Osteoclasts
by Nakamura, Takashi and Imai, Yuuki and Matsumoto, Takahiro and Sato, Shingo and Takeuchi, Kazusane and Igarashi, Katsuhide and Harada, Yoshifumi and Azuma, Yoshiaki and Krust, Andree and Yamamoto, Yoko and Nishina, Hiroshi and Takeda, Shu and Takayanagi, Hiroshi and Metzger, Daniel and Kanno, Jun and Takaoka, Kunio and Martin, T. John and Chambon, Pierre and Kato, Shigeaki
Cell, ISSN 0092-8674, 2007, Volume 130, Issue 5, pp. 811 - 823
Estrogen prevents osteoporotic bone loss by attenuating bone resorption; however, the molecular basis for this is unknown. Here, we report a critical role for... 
HUMDISEASE | DEVBIO | SIGNALING | ANDROGEN RECEPTOR | APOPTOSIS | RESORPTION | DISEASES | BIOCHEMISTRY & MOLECULAR BIOLOGY | MASS | MECHANISMS | DEFICIENT MICE | OSTEOPETROSIS | EXPRESSION | OSTEOPOROSIS | CELL BIOLOGY | Fas Ligand Protein - metabolism | Male | fas Receptor - metabolism | Bone Remodeling - drug effects | Selective Estrogen Receptor Modulators - therapeutic use | Estrogen Receptor alpha - metabolism | Female | Integrases - metabolism | Mice, Inbred DBA | Cell Differentiation | Estradiol - metabolism | Osteoclasts - pathology | Bone Density | Cathepsin K | Ovariectomy | Signal Transduction | Bone Diseases, Metabolic - prevention & control | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Osteoclasts - metabolism | Estrogen Receptor alpha - deficiency | Mice, Knockout | Bone Diseases, Metabolic - metabolism | Estrogen Receptor alpha - drug effects | Phenotype | Animals | Estrogen Receptor alpha - genetics | Cathepsins - genetics | Cathepsins - metabolism | Tamoxifen - therapeutic use | Tamoxifen - pharmacology | Mice | Bone Diseases, Metabolic - physiopathology | Fas Ligand Protein - genetics | Integrases - genetics | Selective Estrogen Receptor Modulators - pharmacology | Osteoclasts - drug effects | Apoptosis | Cathepsins | Antigens, CD95 | Estradiol | Life Sciences | Estrogen Receptor alpha | Biochemistry, Molecular Biology | Tamoxifen | Bone Diseases, Metabolic | Integrases | Fas Ligand Protein | Selective Estrogen Receptor Modulators | Osteoclasts | Molecular biology | Bone Remodeling
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6. Full Text Taxonomy of rare genetic metabolic bone disorders
by Masi, L and Agnusdei, D and Bilezikian, J and Chappard, D and Chapurlat, R and Cianferotti, L and Devolgelaer, J.-P and El Maghraoui, A and Ferrari, S and Javaid, M K and Kaufman, J.-M and Liberman, U A and Lyritis, G and Miller, P and Napoli, N and Roldan, E and Papapoulos, S and Watts, N B and Brandi, M L
Osteoporosis International, ISSN 0937-941X, 10/2015, Volume 26, Issue 10, pp. 2529 - 2558
This article reports a taxonomic classification of rare skeletal diseases based on metabolic phenotypes. It was prepared by The Skeletal Rare Diseases Working... 
Metabolic bone diseases | Medicine & Public Health | Rare bone diseases | Taxonomy | Orthopedics | Rheumatology | Bone metabolism | Endocrinology | Genetic bone diseases | OSTEOCYTES | OSTEOBLAST | RESORPTION | BIOCHEMICAL MARKERS | MEMBRANE | VACUOLAR PROTON PUMP | MECHANISMS | OSTEOPOROSIS | DISEASES | ENDOCRINOLOGY & METABOLISM | OSTEOPETROSIS | Rare Diseases - genetics | Humans | Osteoblasts - physiology | Bone Diseases, Metabolic - diagnosis | Rare Diseases - diagnosis | Proteoglycans - metabolism | Bone Diseases, Developmental - genetics | Osteocytes - physiology | Bone Diseases, Metabolic - metabolism | Bone Diseases, Metabolic - classification | Phenotype | Bone Diseases, Developmental - metabolism | Rare Diseases - classification | Bone Diseases, Developmental - classification | Bone Diseases, Metabolic - genetics | Osteoclasts - physiology | Bone Diseases, Developmental - diagnosis | Rare Diseases - metabolism | Genetic research | Genetic aspects | Bone diseases | Drug therapy | Metabolism | Genetic disorders
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7. Full Text Bone turnover markers in the diagnosis and monitoring of metabolic bone disease
by Greenblatt, Matthew B and Tsai, Joy N and Wein, Marc N
Clinical Chemistry, ISSN 0009-9147, 02/2017, Volume 63, Issue 2, pp. 464 - 474
BACKGROUND: Disorders of bone metabolism, most notably osteoporosis, are highly prevalent and predispose to fractures, causing high patient morbidity and... 
RHEUMATOID-ARTHRITIS | ZOLEDRONIC ACID | MINERAL DENSITY | COLLAGEN TYPE-I | POSTMENOPAUSAL OSTEOPOROSIS | VERTEBRAL FRACTURE RISK | BIOCHEMICAL MARKERS | MEDICAL LABORATORY TECHNOLOGY | BIOLOGICAL VARIABILITY | PYRIDINOLINE CROSS-LINKS | C-TERMINAL TELOPEPTIDE | Biomarkers - metabolism | Bone Diseases, Metabolic - metabolism | Bone and Bones - metabolism | Humans | Biomarkers - analysis | Bone Diseases, Metabolic - diagnosis | Bone Remodeling | Osteoporosis | Mortality | Physiological aspects | Hostages | Bones | Diagnosis | Health aspects | Density | Development and progression | Care and treatment | Genetic aspects | Biological markers | Urine | Laboratories | Collagen | Arthritis | Metastasis | Metabolism | Tumors
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8. Full Text CKD-induced wingless/integration1 inhibitors and phosphorus cause the CKD-mineral and bone disorder
by Fang, Yifu and Fang, Yifu and Ginsberg, Charles and Seifert, Michael and Agapova, Olga and Sugatani, Toshifumi and Register, Thomas C and Freedman, Barry I and Monier-Faugere, Marie-Claude and Malluche, Hartmut and Hruska, Keith A
Journal of the American Society of Nephrology : JASN, ISSN 1046-6673, 08/2014, Volume 25, Issue 8, pp. 1760 - 1773
In chronic kidney disease, vascular calcification, renal osteodystrophy, and phosphate contribute substantially to cardiovascular risk and are components of... 
MURINE MODEL | STEM-CELLS | VASCULAR CALCIFICATION | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | PHOSPHATE BINDERS | PREVALENCE | CHRONIC KIDNEY-DISEASE | FGF23 | CARDIOVASCULAR RISK | PROGRESSION | Double-Blind Method | Renal Insufficiency, Chronic - etiology | Humans | Mice, Inbred C57BL | Bone Diseases, Metabolic - etiology | Glucuronidase - metabolism | Glycoproteins - metabolism | Male | Wnt1 Protein - physiology | Renal Insufficiency, Chronic - drug therapy | Bone Diseases, Metabolic - drug therapy | Bone Diseases, Metabolic - metabolism | Renal Insufficiency, Chronic - metabolism | Fibroblast Growth Factors - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Phosphorus, Dietary | Bone Morphogenetic Proteins - metabolism | Phosphates - metabolism | Animals | Adult | Female | Lanthanum - therapeutic use | Disease Models, Animal | Basic Research
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