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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2011, Volume 108, Issue 4, pp. 1609 - 1614
.... There is no prevention or treatment for irradiation-induced bone injury. We irradiated the distal half of the mouse left femur to study the mechanism of irradiation-induced bone injury and found that no mesenchymal stem cells (MSCs... 
Femur | Free radicals | Stem cells | Irradiation | Bone marrow | Bones | Physical trauma | Bone marrow cells | Radiation damage | Mesenchymal stem cells | CFU-fibroblast | Self-renewal | Angiography | Repopulation | Differentiation | MIGRATION | CAPACITY | differentiation | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | OSTEOGENESIS | self-renewal | angiography | CHEMOTHERAPY | TRANSPLANTATION | ORIGIN | ENGRAFTMENT | THERAPY | repopulation | Femur - pathology | Leukocyte Common Antigens - metabolism | Mesenchymal Stromal Cells - radiation effects | Blood Vessels - pathology | Bone Marrow - radiation effects | Blood Vessels - radiation effects | Radiation Injuries, Experimental - etiology | Free Radicals - metabolism | Antigens, Ly - metabolism | Colony-Forming Units Assay | Membrane Proteins - metabolism | Radiation Injuries, Experimental - metabolism | Osteoblasts - radiation effects | Femur - metabolism | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Mesenchymal Stromal Cells - metabolism | Thiobarbituric Acid Reactive Substances - metabolism | Bone Marrow Cells - radiation effects | Radiation Injuries, Experimental - pathology | Fibroblasts - pathology | Femur - radiation effects | Integrin beta1 - metabolism | Osteoblasts - pathology | Animals | Fibroblasts - radiation effects | Bone Marrow - pathology | Mice | Mesenchymal Stromal Cells - pathology | Bone Marrow Cells - metabolism | Physiological aspects | Complications and side effects | Stem cell research | Care and treatment | Radiotherapy | Biological Sciences
Journal Article
Circulation Research, ISSN 0009-7330, 02/2013, Volume 112, Issue 3, pp. 510 - 522
RATIONALE:The impact of diabetes mellitus on bone marrow (BM) structure is incompletely understood... 
microangiopathy | stem cells | macroangiopathy | diabetes mellitus type 2 | bone marrow | CARDIAC & CARDIOVASCULAR SYSTEMS | TRANSCRIPTION | MICRORNAS | TRANSPLANTATION | HEART | ENDOTHELIAL PROGENITOR CELLS | DISEASE | PERIPHERAL VASCULAR DISEASE | COMPLICATIONS | NEOVASCULARIZATION | DIFFERENTIATION | HEMATOLOGY | EXPRESSION | Immunohistochemistry | Humans | Middle Aged | Microvessels - metabolism | Hematopoietic Stem Cells - pathology | Microvessels - pathology | Male | MicroRNAs - metabolism | Ischemia - genetics | Adipose Tissue - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Forkhead Transcription Factors - metabolism | Bone Marrow Cells - immunology | Aged, 80 and over | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Ischemia - pathology | Biomarkers - metabolism | Peripheral Arterial Disease - metabolism | Stem Cell Niche | Signal Transduction | Endothelial Cells - metabolism | Adipose Tissue - pathology | Bone Marrow Cells - pathology | Bone Marrow Examination | Cell Lineage | MicroRNAs - genetics | Diabetes Mellitus, Type 2 - pathology | Endothelial Cells - pathology | Forkhead Box Protein O3 | Antigens, CD34 - metabolism | Peripheral Arterial Disease - pathology | Diabetes Mellitus, Type 2 - genetics | Diabetes Mellitus, Type 2 - metabolism | Microvessels - immunology | Case-Control Studies | Hematopoietic Stem Cells - immunology | Diabetic Angiopathies - pathology | Flow Cytometry | Transfection | Peripheral Arterial Disease - genetics | Adult | Female | Diabetic Angiopathies - genetics | Diabetic Angiopathies - metabolism | Cells, Cultured | Gene Expression Regulation | Ischemia - metabolism | Hematopoietic Stem Cells - metabolism | Forkhead Transcription Factors - genetics | Aged | Bone Marrow Cells - metabolism | Apoptosis
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 1945-7197, 2013, Volume 98, Issue 3, pp. 935 - 945
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 2, p. e87893
.... In this study, we found that spontaneously-formed tumorigenic hybrids between bone marrow-derived mesenchymal stem cells (MSCs... 
BREAST-CANCER | TRANSITION | MSCS | METASTASIS | FUSION | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | FOCUS | IDENTIFICATION | TUMORS | PROGRESSION | Cell Proliferation | Epithelial Cells - metabolism | Humans | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | Lung Neoplasms - pathology | Gene Expression Profiling | Epithelial-Mesenchymal Transition - genetics | Cell Fusion | Neoplastic Stem Cells - metabolism | Cell Shape | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Female | Cell Differentiation | Fibroblasts - metabolism | Hybrid Cells - metabolism | Lung Neoplasms - genetics | Neoplasm Invasiveness | Spheroids, Cellular - metabolism | Bone Marrow Cells - pathology | Carcinogenesis - genetics | Mesenchymal Stromal Cells - metabolism | Epithelial Cells - pathology | Fibroblasts - pathology | Mice, SCID | Carcinogenesis - pathology | Phenotype | Animals | Hybrid Cells - pathology | Cell Line, Tumor | Mesenchymal Stromal Cells - pathology | Bone Marrow Cells - metabolism | Cell Movement | Lung cancer, Non-small cell | Development and progression | Metastasis | Vimentin | Hybrids | Transcription factors | Motility | Mesenchyme | Oct-4 protein | Brain cancer | Lung cancer | Cytology | Stem cell transplantation | Smooth muscle | Cell fusion | E-cadherin | Fibronectin | Metastases | Genotype & phenotype | Actin | Rodents | Bone marrow | Fibroblasts | Population | Subpopulations | Myeloid cells | Invasiveness | Muscles | Non-small cell lung carcinoma | Forming | Inflammation | Tumor cell lines | Gene expression | Medicine | Properties (attributes) | Bone cancer | Hospitals | Overexpression | Morphology | Stem cells | Aberration | Cancer
Journal Article
Journal Article
Nature cell biology, ISSN 1476-4679, 2017, Volume 19, Issue 6, pp. 677 - 688
.... Using genetic fate mapping we found that bone marrow leptin receptor (Lepr)-expressing mesenchymal stromal lineage cells expanded extensively and were the fibrogenic cells in PMF... 
FIBROSIS | HEPATIC STELLATE CELLS | HEMATOPOIETIC STEM-CELLS | PDGFR-ALPHA | MYELOPROLIFERATIVE NEOPLASMS | CALRETICULIN MUTANTS | LARGE GENE LISTS | MYELOID METAPLASIA | IMATINIB MESYLATE | THROMBOPOIETIN RECEPTOR | CELL BIOLOGY | Cell Proliferation | Receptors, Leptin - genetics | Primary Myelofibrosis - pathology | Myofibroblasts - metabolism | Time Factors | Bone Marrow Cells - drug effects | Cell Differentiation | Primary Myelofibrosis - metabolism | Disease Models, Animal | Mesenchymal Stromal Cells - drug effects | Myofibroblasts - pathology | Primary Myelofibrosis - prevention & control | Thrombopoietin - metabolism | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Stem Cell Niche | Signal Transduction | Imatinib Mesylate - pharmacology | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Mesenchymal Stromal Cells - metabolism | Genotype | Mice, Transgenic | Myofibroblasts - drug effects | Cell Lineage | Thrombopoietin - genetics | Phenotype | Primary Myelofibrosis - genetics | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Hematopoiesis, Extramedullary | Protein Kinase Inhibitors - pharmacology | Mesenchymal Stromal Cells - pathology | Receptors, Leptin - metabolism | Bone Marrow Cells - metabolism | Osteogenesis | Cell Movement | Imatinib | Myelofibrosis | Platelet-derived growth factor | Mesenchyme | Mapping | Gene expression | Signal transduction | Signaling | Biomedical materials | Clonal deletion | Cell fate | Stromal cells | Stem cells | Fibrosis | Bone marrow | Biocompatibility | Gene mapping
Journal Article
Blood, ISSN 1528-0020, 2014, Volume 124, Issue 4, pp. 555 - 566
Journal Article
Cancer Research, ISSN 0008-5472, 12/2007, Volume 67, Issue 23, pp. 11438 - 11446
Journal Article
Nature medicine, ISSN 1546-170X, 2018, Volume 24, Issue 9, pp. 1459 - 1468
Journal Article