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Circulation Research, ISSN 0009-7330, 04/2012, Volume 110, Issue 9, pp. 1217 - 1225
RATIONALE:The function of Nox4, a source of vascular H2O2, is unknown. Other Nox proteins were identified as mediators of endothelial dysfunction. OBJECTIVE:We... 
Hypertension | Angiogenesis | Carbon monoxide | Nitric oxide | Redox regulation | INDUCED ACTIVATION | nitric oxide | CARDIAC & CARDIOVASCULAR SYSTEMS | redox regulation | angiogenesis | carbon monoxide | ANGIOTENSIN-II | BLOOD-PRESSURE | NAD(P)H OXIDASE | ENDOTHELIAL-CELLS | IN-VIVO | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HYDROGEN-PEROXIDE | HEMATOLOGY | hypertension | UP-REGULATION | Catalase - pharmacology | Heme Oxygenase-1 - metabolism | Humans | NADPH Oxidases - metabolism | Male | Ischemia - genetics | Carbonates - pharmacology | RNA, Messenger - metabolism | NADPH Oxidases - deficiency | Time Factors | NADPH Oxidases - genetics | Carbonates - metabolism | Hemin - pharmacology | Hypertension - enzymology | Hypertension - genetics | Ischemia - pathology | Cytoprotection | Disease Models, Animal | Muscle, Skeletal - blood supply | NADH, NADPH Oxidoreductases - genetics | Hypertension - pathology | Hydrogen Peroxide - metabolism | Mice, Knockout | Human Umbilical Vein Endothelial Cells - enzymology | Organometallic Compounds - metabolism | Mice | Oxidative Stress - drug effects | Endothelial Cells - pathology | Endothelial Cells - enzymology | Hypertrophy | Ischemia - enzymology | Boranes - metabolism | Transfection | NADH, NADPH Oxidoreductases - deficiency | Hypertension - chemically induced | NF-E2-Related Factor 2 - genetics | Membrane Proteins - metabolism | Nitric Oxide Synthase Type III - metabolism | Angiotensin II | Polyethylene Glycols - pharmacology | Carbon Dioxide - metabolism | Mice, Inbred C57BL | Cells, Cultured | Boranes - pharmacology | Antioxidants - pharmacology | Ischemia - physiopathology | NADPH Oxidase 4 | Hypertension - physiopathology | NADPH Oxidase 1 | Animals | Lung - blood supply | Neovascularization, Physiologic | Organometallic Compounds - pharmacology | Apoptosis | Endothelial Cells - drug effects
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 02/2011, Volume 300, Issue 2, pp. C256 - C265
Basuroy S, Tcheranova D, Bhattacharya S, Leffler CW, Parfenova H. Nox4 NADPH oxidase-derived reactive oxygen species, via endogenous carbon monoxide, promote... 
Cerebral vascular endothelial cells | CORM-A1 | CO-releasing molecule | Inflammatory cytokines | Heme oxygenase | HO-2 | NOX1 | NOX2 | heme oxygenase | OXIDATIVE STRESS | KINASE PATHWAY | PHYSIOLOGY | PHYSIOLOGICAL ROLES | PHOSPHATIDYLINOSITOL 3-KINASE | MAP KINASE | inflammatory cytokines | CELL BIOLOGY | INHIBITS APOPTOSIS | NAD(P)H OXIDASE | cerebral vascular endothelial cells | SMOOTH-MUSCLE-CELLS | HEME OXYGENASE-1 | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Reactive Oxygen Species - metabolism | Humans | Microvessels - metabolism | NADPH Oxidases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Carbon Monoxide - metabolism | Brain - metabolism | Carbonates - administration & dosage | Isoenzymes - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Heme Oxygenase (Decyclizing) - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Animals, Newborn | Cell Survival - drug effects | Swine - metabolism | Endothelial Cells - metabolism | Cells, Cultured | Microvessels - drug effects | Brain - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | Signal Transduction - drug effects | Boranes - administration & dosage | Oxidative Stress - drug effects | Apoptosis | Endothelial Cells - drug effects | RNA, Small Interfering - metabolism | Vascular Biology
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2014, Volume 61, Issue 5, pp. 1029 - 1037
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 12/2009, Volume 41, Issue 6, pp. 639 - 650
Carbon monoxide (CO) is currently being evaluated as a therapeutic modality in the treatment of patients with acute lung injury and acute respiratory distress... 
Acute respiratory distress syndrome | channel | Carbon monoxide | Sodium/potassium-exchanging ATPase | Carbon monoxide-releasing molecule | Epithelial Na | CELLS | sodium/potassium-exchanging ATPase | BIOCHEMISTRY & MOLECULAR BIOLOGY | carbon monoxide | HYPEROXIC LUNG INJURY | MONOLAYERS | RESPIRATORY-DISTRESS-SYNDROME | CELL BIOLOGY | SMOOTH-MUSCLE | RESPIRATORY SYSTEM | DISEASE | acute respiratory distress syndrome | HEME OXYGENASE-1 | PROVIDES PROTECTION | ABSORPTION | carbon monoxide-releasing molecule | epithelial Na+ channel | NA+ CONDUCTANCE | Heme Oxygenase-1 - metabolism | Humans | Carbonates - pharmacology | Epithelial Sodium Channels - metabolism | Body Fluids - drug effects | Carbon Monoxide - metabolism | Respiratory Distress Syndrome, Adult - metabolism | Amiloride - metabolism | Carbon Monoxide - pharmacology | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - drug effects | Ion Transport - drug effects | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Cell Line | Rabbits | Signal Transduction | Carbon Monoxide - toxicity | Rats | Guanylate Cyclase - metabolism | Boranes - pharmacology | Respiratory Distress Syndrome, Adult - drug therapy | Epithelial Sodium Channels - chemistry | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Cyclic GMP - metabolism | Body Fluids - metabolism | Amiloride - pharmacology | Epithelial Sodium Channel Blockers | Histidine - chemistry | In Vitro Techniques | Energy Metabolism - drug effects | Organometallic Compounds - pharmacology | Index Medicus
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2012, Volume 32, Issue 9, pp. 2149 - 2157
OBJECTIVE—We compared the antithrombotic effects in vivo of 2 chemically different carbon monoxide–releasing molecules (CORM-A1 and CORM-3) on arterial and... 
carbon monoxide-releasing molecules | intravital microscopy | plasminogen activator inhibitor-1 | thrombosis | rat | fibrin generation | carbon monoxide | green fluorescent protein mice | platelet aggregation | PLATELET-AGGREGATION | RATS | ARTERIAL THROMBOSIS | ASPIRIN | INHIBITION | CO-RMS | IN-VIVO | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | MICE | HEME OXYGENASE-1 | HEMATOLOGY | Fibrinolysis - drug effects | Boranes - chemistry | Male | Green Fluorescent Proteins - genetics | Carbonates - pharmacology | Carbonates - administration & dosage | Time Factors | Carbonates - chemistry | Carbonates - metabolism | Disease Models, Animal | Thrombosis - physiopathology | Solubility | Rats | Mice, Transgenic | Plasminogen Activator Inhibitor 1 - blood | Microscopy, Confocal | Organometallic Compounds - metabolism | Blood Platelets - metabolism | Green Fluorescent Proteins - biosynthesis | Boranes - administration & dosage | Mice | Arterial Occlusive Diseases - prevention & control | Fibrin - metabolism | Organometallic Compounds - administration & dosage | Arterial Occlusive Diseases - blood | Fibrinolytic Agents - chemistry | Injections, Intravenous | Rats, Wistar | Prothrombin Time | Venous Thrombosis - blood | Fibrinolytic Agents - metabolism | Carbon Monoxide - metabolism | Boranes - metabolism | Dose-Response Relationship, Drug | Water - chemistry | Venous Thrombosis - prevention & control | Blood Pressure - drug effects | Platelet Aggregation - drug effects | Thrombosis - blood | Thrombosis - prevention & control | Arterial Occlusive Diseases - physiopathology | Blood Platelets - drug effects | Mice, Inbred C57BL | Blood Gas Analysis | Venous Thrombosis - physiopathology | Boranes - pharmacology | Venous Thrombosis - etiology | Arterial Occlusive Diseases - etiology | Fibrinolytic Agents - pharmacology | Animals | Blood Coagulation - drug effects | Thrombosis - etiology | Fibrinogen - metabolism | Organometallic Compounds - chemistry | Fibrinolytic Agents - administration & dosage | Organometallic Compounds - pharmacology
Journal Article
Journal Article
by Li, WL and Li, X and Zhang, B and Gao, CM and Chen, YZ and Jiang, YY
CURRENT MEDICINAL CHEMISTRY, ISSN 0929-8673, 2016, Volume 23, Issue 23, pp. 2507 - 2554
Journal Article
Angewandte Chemie International Edition, ISSN 1433-7851, 04/2018, Volume 57, Issue 16, pp. 4143 - 4148
Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 501, Issue 7465, pp. 84 - 87
The reduction of nitrogen (N-2) to ammonia (NH3) is a requisite transformation for life(1). Although it is widely appreciated that the iron-rich cofactors of... 
TRANSFORMATION | N-2 | HYDROGENATION | CARBON | REDUCTION | MECHANISM | MOLECULAR NITROGEN | MOLYBDENUM | MULTIDISCIPLINARY SCIENCES | DINITROGEN | CLEAVAGE | Molybdenum - chemistry | Molybdoferredoxin - metabolism | Nitrogenase - metabolism | Oxidation-Reduction | Molybdenum - metabolism | Iron - chemistry | Nitrogenase - chemistry | Boranes - chemistry | Phosphines - chemistry |