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PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e78935 - e78935
Background: An important phenomenon observed in glioma metabolism is increased aerobic glycolysis in tumor cells, which is generally referred to as the Warburg... 
GLIOBLASTOMA | MALIGNANCY | MULTIDISCIPLINARY SCIENCES | THROMBOSPONDIN-1 | MATRIX METALLOPROTEINASE-2 | LATENT TGF-BETA | PROTEINS | EXPRESSION | BINDING | CANCER STEM-CELLS | MOTILITY | L-Lactate Dehydrogenase - metabolism | Apoptosis - drug effects | Transforming Growth Factor beta2 - metabolism | Humans | Transcriptional Activation - drug effects | Apoptosis - genetics | Cell Movement - genetics | Glioma - metabolism | Glioma - genetics | RNA Interference | Isoenzymes - metabolism | Glioma - pathology | Thrombospondin 1 - genetics | Tumor Cells, Cultured | Lactic Acid - pharmacology | Enzyme-Linked Immunosorbent Assay | Isoenzymes - genetics | Lactic Acid - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Cell Movement - drug effects | L-Lactate Dehydrogenase - genetics | Cell Line, Tumor | Cell Proliferation - drug effects | Thrombospondin 1 - metabolism | Transforming Growth Factor beta2 - genetics | Gliomas | Physiological aspects | Development and progression | Genetic aspects | Research | Transforming growth factors | Cell migration | Boyden chamber | Brain cancer | Glioblastoma | Smooth muscle | Neurosurgery | Proteins | Angiogenesis | Proteolysis | Glioma cells | Extracellular matrix | Inhibition | Growth factors | Enzyme-linked immunosorbent assay | Thrombospondin | Transforming growth factor-b1 | Cytokines | Tumor cells | siRNA | Metabolism | Gene expression | Beta2 protein | Latency | Neurology | Immunosuppression | Sodium | Ribonucleic acids | Glycolysis | Hypoxia | Lactic acid | Index Medicus
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 06/2017, Volume 56, Issue 6, pp. 796 - 803
Airway remodeling, a characteristic feature of asthma, begins in early life. Recurrent human rhinovirus (HRV) infections are a potential inciting stimulus for... 
Airway epithelial cell | Airway smooth muscle | Chemotaxis | Ccl5 | Rhinovirus | BIOCHEMISTRY & MOLECULAR BIOLOGY | EOSINOPHILIC INFLAMMATION | airway smooth muscle | airway epithelial cell | INDUCTION | HYPERPLASIA | rhinovirus | ILLNESSES | CHILDREN | CELL BIOLOGY | HYPERTROPHY | PRESCHOOL WHEEZERS | RESPIRATORY SYSTEM | CCL5 | GROWTH | ASTHMA | EXPRESSION | chemotaxis | Epithelial Cells - metabolism | Molecular Weight | Epithelial Cells - drug effects | Humans | Middle Aged | Myocytes, Smooth Muscle - pathology | Culture Media, Conditioned - pharmacology | Male | Rhinovirus - physiology | Picornaviridae Infections - virology | Young Adult | Chemotactic Factors - pharmacology | Flow Cytometry | Adult | Female | Chemokine CCL5 - metabolism | Myocytes, Smooth Muscle - drug effects | Bronchi - pathology | Cyclic AMP - metabolism | Myocytes, Smooth Muscle - metabolism | Picornaviridae Infections - pathology | Rhinovirus - drug effects | Virus Replication - drug effects | Lung - pathology | Epithelial Cells - pathology | Cell Movement - drug effects | Epithelial Cells - virology | Intracellular Space - metabolism | Adolescent | Pertussis Toxin - toxicity | Flow cytometry | Boyden chamber | Leukocyte migration | Epithelial cells | Antibodies | Smooth muscle | Viruses | Infections | Experiments | Pertussis toxin | Respiratory tract | Pertussis | Angiogenesis | Formoterol | Growth factors | Chronic illnesses | Medical research | Cyclic AMP | Epithelium | Asthma | Medicine | CXCL10 protein | Forskolin | Chemokines | Cell migration | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2018, Volume 13, Issue 5, pp. e0197751 - e0197751
Background The endocannabinoid (eCB) 2-arachidonoylglycerol (2-AG) is a known modulator of inflammation and ligand to both, pro-inflammatory cannabinoid... 
CELLS | INHIBITION | METABOLISM | ENDOCANNABINOIDS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | INVOLVEMENT | ATHEROSCLEROSIS | CANNABINOID CB2 RECEPTOR | KNOCKOUT MICE | BRAIN | Endocannabinoids - metabolism | Cell Line | Atherosclerosis - immunology | Atherosclerosis - chemically induced | Atherosclerosis - genetics | Diet, High-Fat - adverse effects | Male | Glycerides - metabolism | Antigens, CD - metabolism | Cell Movement - drug effects | Arachidonic Acids - metabolism | Animals | Piperidines - pharmacology | Antigens, Differentiation, Myelomonocytic - metabolism | Apolipoproteins E - genetics | Piperidines - adverse effects | Benzodioxoles - adverse effects | Mice | Benzodioxoles - pharmacology | Macrophages - immunology | Heart | CC chemokine receptors | Atherogenesis | Boyden chamber | Transcription | High cholesterol diet | Leukocyte migration | Migration | Adhesion tests | Cardiovascular disease | Macrophages | Lipase | CCR1 protein | Receptors | 2-Arachidonylglycerol | Apolipoprotein E | Atherosclerosis | Aorta | Physiology | Cardiology | Heart diseases | CCR5 protein | Internal medicine | Data processing | Inflammation | Metabolism | Coronary artery disease | Chemokine receptors | Cholesterol | Molecular chains | Medicine | Hospitals | Inhibitors | Arteriosclerosis | Vascular tissue | Ligands | Fats and oils | Cardiovascular diseases | Immunofluorescence | Chemokines | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e44039 - e44039
Background: Withaferin A, which is a naturally derived steroidal lactone, has been found to prevent angiogenesis and metastasis in diverse tumor models. It has... 
MATRIX METALLOPROTEINASES | APOPTOSIS | ACTIVATION | METASTASIS | GELATINASE-A | MULTIDISCIPLINARY SCIENCES | COMPOUND WITHAFERIN-A | TRAFFICKING | WITHANOLIDES | EXPRESSION | TUMOR-CELL | Phosphorylation | Apoptosis - drug effects | Antineoplastic Agents - chemical synthesis | Humans | Neoplasm Invasiveness - prevention & control | Gene Expression Regulation, Neoplastic | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Proto-Oncogene Proteins c-akt - genetics | Receptors, Thrombin - antagonists & inhibitors | Laminin | Matrix Metalloproteinase Inhibitors - chemical synthesis | Biological Assay | Neovascularization, Pathologic - prevention & control | Antineoplastic Agents - pharmacology | Azides - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Azides - chemical synthesis | Proteoglycans | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Withanolides - chemical synthesis | Cell Movement - drug effects | Matrix Metalloproteinase 2 - genetics | Matrix Metalloproteinase Inhibitors - pharmacology | Animals | Receptors, Thrombin - genetics | Withanolides - pharmacology | Collagen | Cell Line, Tumor | Receptors, Thrombin - metabolism | Mice | Drug Combinations | Animal experimentation | Chemotherapy | Metastasis | Neovascularization | Analysis | Cancer | Cell culture | Animal models | Boyden chamber | Motility | Laboratories | Clinical trials | AKT protein | Kinases | Machinery | Metastases | Angiogenesis | Signal transduction | Carcinogens | Gelatin | Penicillin | Biocompatibility | Inhibition | Conditioning | Wound healing | Secretion | Extracellular signal-regulated kinase | Pharmacology | Gelatinase | Cervix | Gelatinase A | Medicine | Signaling | Natural products | Prostate apoptosis response 4 protein | Tumor suppressor genes | Prostate cancer | Prostate | Integrative medicine | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2017, Volume 12, Issue 12, pp. e0189017 - e0189017
Complement activation correlates to rheumatoid arthritis disease activity, and increased amounts of the complement split product C5a is observed in synovial... 
COLLAGEN-INDUCED ARTHRITIS | POLYMORPHONUCLEAR LEUKOCYTES | CARTILAGE DESTRUCTION | COLONY-STIMULATING FACTOR | T-CELL IMMUNITY | MULTIDISCIPLINARY SCIENCES | FELTYS-SYNDROME | AUTOIMMUNE ARTHRITIS | RECEPTOR EXPRESSION | COMPLEMENT PATHWAY ACTIVATION | MONOCLONAL-ANTIBODY | Chemotaxis, Leukocyte | Immunohistochemistry | Arthritis, Rheumatoid - metabolism | Leukocytes - pathology | Flow Cytometry | Enzyme-Linked Immunosorbent Assay | Humans | Synovial Fluid - metabolism | Complement C5a - metabolism | Arthritis, Psoriatic - metabolism | Receptor, Anaphylatoxin C5a - metabolism | Development and progression | Care and treatment | Genetic aspects | Complement (Immunology) | Health aspects | Rheumatoid arthritis | Index Medicus | Flow cytometry | Boyden chamber | Leukocyte migration | Pathogenesis | Complement component C5a | Lymphocytes T | Statistical methods | Arthritis | Tissue analysis | Macrophages | Blood | Synovial fluid | Cell activation | Biomedical materials | Lymphocytes | Rodents | Classification | Biocompatibility | Rheumatism | Bioindicators | Enzyme-linked immunosorbent assay | Statistical analysis | Cytokines | Computational fluid dynamics | Medical treatment | Neutrophils | Leukocytes (neutrophilic) | Inflammation | Statistical tests | Joint diseases | Patients | White blood cells | Cytometry | Complement activation | Monocytes | Hospitals | Rheumatoid synovitis | Collagen | Monoclonal antibodies | Biomarkers | Infiltration | Osteoarthritis
Journal Article
Acta Ophthalmologica, ISSN 1755-375X, 09/2017, Volume 95, Issue S259, p. n/a
Journal Article
International Surgery, ISSN 0020-8868, 05/2016, Volume 101, Issue 5-6, pp. 282 - 290
The objective of this study was to evaluate the effects of mistletoe alkali on human osteosarcoma cells (U2OS) in vitro. Osteosarcoma is the most common... 
Anticancer drug | Mistletoe alkali | U2OS | Boyden chamber transwell migration assay | Apoptosis | SURGERY | RECEPTOR | CELL-SURVIVAL | SUPPRESSION | CANCER | CHEMOTHERAPY | THERAPY | GROWTH | LIVER | EXPRESSION | EXTRACT
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 5, pp. e20348 - e20348
Background: Glioma cells are exposed to elevated interstitial fluid flow during the onset of angiogenesis, at the tumor periphery while invading normal... 
VASCULAR NORMALIZATION | ANTIANGIOGENIC THERAPY | GLIOBLASTOMA | TUMOR BLOOD-FLOW | MECHANISM | BIOLOGY | COLLAGEN I | INTERSTITIAL FLOW | GROWTH-FACTOR | UP-REGULATION | BRAIN-TUMORS | Cell Survival - drug effects | Permeability - drug effects | Reproducibility of Results | Glioma - enzymology | Gels | Apoptosis - drug effects | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Humans | Fluorescence | Rats | Stress, Mechanical | Hydrodynamics | Chemotaxis - drug effects | Down-Regulation - drug effects | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Chemotactic Factors - pharmacology | Animals | Glioma - pathology | Cell Line, Tumor | Matrix Metalloproteinase 1 - metabolism | Rheology - drug effects | Gliomas | Development and progression | Biotechnology | Cell culture | Surgical implants | Boyden chamber | Motility | Leukocyte migration | Brain tumors | Brain cancer | Fluid flow | Central nervous system | Parenchyma | Metastasis | Compaction | Matrix metalloproteinase | Shear stresses | Angiogenesis | Modulation | Glioma cells | Extracellular matrix | Physiology | Metalloproteinase | Interstitial collagenase | Biomedical engineering | Medical research | Computational fluid dynamics | Gene expression | Substantia alba | Chemotaxis | Gelatinase A | Polymerase chain reaction | Brain research | Three dimensional flow | Collagen | Cell lines | Shear stress | Mechanotransduction | Mechanical stimuli | Three dimensional models | Cell migration | Tumors | Index Medicus
Journal Article
Tissue engineering. Part C, Methods, ISSN 1937-3384, 03/2017, Volume 23, Issue 3, pp. 180 - 187
We present a novel mechanobiology-based invasiveness assay to rapidly and quantitatively evaluate the mechanical invasiveness of metastatic cancer cells and... 
Breast Neoplasms - secondary | Neoplasm Invasiveness | Humans | Gels - metabolism | Cell Communication | Female | Gels - chemistry | Tumor Cells, Cultured | Cell Movement | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, pp. e28134 - e28134
Cardiac fibroblasts (CFs) are the primary cell type responsible for cardiac fibrosis during pathological myocardial remodeling. Several studies have... 
LEFT-VENTRICULAR ENLARGEMENT | COLLAGEN PRODUCTION | MATRIX-METALLOPROTEINASE | PHENOTYPIC MODULATION | HEART-FAILURE | BIOLOGY | TISSUE INHIBITORS | NECROSIS-FACTOR-ALPHA | CELL-PROLIFERATION | PULMONARY-FIBROSIS | EXPERIMENTAL MYOCARDIAL-INFARCTION | Actins - metabolism | Cytokines - secretion | Myofibroblasts - enzymology | Rats | Myofibroblasts - secretion | Myofibroblasts - drug effects | Rats, Sprague-Dawley | Myocardium - cytology | Myofibroblasts - cytology | Cell Movement - drug effects | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Matrix Metalloproteinase 9 - metabolism | Animals | Cell Differentiation - drug effects | Pyridones - toxicity | Cell Proliferation - drug effects | Collagen - pharmacology | Pyridones - pharmacology | Bone morphogenetic proteins | B cells | Cytokines | Cardiology | Analysis | Collagen | Heart | Neonates | Animal models | Boyden chamber | Heart attacks | Transplants & implants | Leukocyte migration | Liver | Interleukin | Smooth muscle | Cardiovascular disease | Matrix metalloproteinase | Remodeling | Tissue inhibitor of metalloproteinase 1 | Proteins | Actin | Rodents | Fibroblasts | Bone marrow | Attenuation | Tumor necrosis factor-TNF | Metalloproteinase | Growth factors | Heart diseases | Heart failure | Secretion | Bioassays | Contractility | Inflammation | Gene expression | Muscle contraction | Adenoviruses | Fibrosis | Interleukin 10 | Stem cells | TNF inhibitors | Hypoxia | Kidney diseases | Laboratory animals | Differentiation | Cell migration | Chemokines | Index Medicus
Journal Article