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1. Full Text Mechanisms involved in the nociception produced by peripheral protein kinase c activation in mice
by Ferreira, Juliano and Trichês, Karen M and Medeiros, Rodrigo and Calixto, João B
Pain (Amsterdam), ISSN 0304-3959, 2005, Volume 117, Issue 1, pp. 171 - 181
Nociception | Protein kinase C | Substance P | Phorbol ester | MAP kinase | Mice | Glutamate | Neurosciences | Clinical Neurology | Anesthesiology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Fundamental and applied biological sciences. Psychology | Vertebrates: nervous system and sense organs | Behavioral psychophysiology | Psychology. Psychoanalysis. Psychiatry | Psychology. Psychophysiology | Biological and medical sciences | Somesthesis and somesthetic pathways (proprioception, exteroception, nociception); interoception; electrolocation. Sensory receptors | Neurotransmission and behavior | Bradykinin - analogs & derivatives | Analgesics - pharmacology | Tetradecanoylphorbol Acetate - pharmacology | Capsaicin - pharmacology | Nociceptors - physiology | Tetradecanoylphorbol Acetate - analogs & derivatives | Male | Peptide Fragments - pharmacology | Extracellular Signal-Regulated MAP Kinases - metabolism | Pain - chemically induced | Dose-Response Relationship, Drug | Salicylates - pharmacology | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Drug Interactions | Pain - enzymology | Time Factors | Pain - drug therapy | Protein Kinase C - metabolism | Sympatholytics - pharmacology | Behavior, Animal - drug effects | Indoles - pharmacology | Bradykinin - pharmacology | Blotting, Western - methods | Protein Kinase C - physiology | Calcitonin Gene-Related Peptide - pharmacology | Dipeptides - pharmacology | Enzyme Inhibitors - pharmacology | Capsaicin - analogs & derivatives | Chelating Agents - pharmacology | Enzyme Activation - drug effects | Antibodies - pharmacology | Pain Measurement - methods | Animals | Egtazic Acid - pharmacology | Ruthenium Red - pharmacology | Dizocilpine Maleate - pharmacology | Excitatory Amino Acid Antagonists | Guanethidine - pharmacology | Nociceptors - drug effects | Methyl aspartate | Nerve growth factor | Cell research | Protein kinases | Index Medicus
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2. Full Text A role for the sodium pump in H2O2-induced vasorelaxation in porcine isolated coronary arteries
by Wong, P.S and Garle, M.J and Alexander, S.P.H and Randall, M.D and Roberts, R.E
Pharmacological research, ISSN 1043-6618, 12/2014, Volume 90, pp. 25 - 35
Porcine coronary artery | Ca2+-reintroduction | Ouabain-sensitive sodium–potassium pump | KCl relaxation | Rubidium uptake | Hydrogen peroxide (H2O2) relaxation | reintroduction | relaxation | Hydrogen peroxide (H | Ouabain-sensitive sodium-potassium pump | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Catalase - pharmacology | Sodium-Potassium-Exchanging ATPase - physiology | Male | Nitroprusside - pharmacology | Quinoxalines - pharmacology | Oxadiazoles - pharmacology | Swine | Female | Vasodilation - physiology | Bradykinin - pharmacology | Ouabain - pharmacology | Polyethylene Glycols - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Coronary Vessels - drug effects | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Colforsin - pharmacology | Coronary Vessels - physiology | Hydrogen Peroxide - pharmacology | Verapamil - pharmacology | Indomethacin - pharmacology | Peptides - pharmacology | Carbenoxolone - pharmacology | Animals | Vasodilation - drug effects | In Vitro Techniques | Index Medicus
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3. Full Text Evidence for the role of neurogenic inflammation components in trypsin‐elicited scratching behaviour in mice
by Costa, R and Marotta, D M and Manjavachi, M N and Fernandes, E S and Lima‐Garcia, J F and Paszcuk, A F and Quintão, N L M and Juliano, L and Brain, S D and Calixto, J B
British journal of pharmacology, ISSN 0007-1188, 07/2008, Volume 154, Issue 5, pp. 1094 - 1103
neuropeptides | Trypsin | PAR‐2 | TRPV1 | mast cells | scratching behaviour | PAR-2 | Neuropeptides | Mast cells | Scratching behaviour | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Biological and medical sciences | Pharmacology. Drug treatments | Medical sciences | Receptor, PAR-2 - metabolism | Male | Peptide Fragments - pharmacology | Antipruritics - pharmacology | Receptors, Calcitonin Gene-Related Peptide - metabolism | Quinolines - pharmacology | TRPV Cation Channels - metabolism | Receptor, PAR-2 - antagonists & inhibitors | Cromolyn Sodium - pharmacology | TRPV Cation Channels - antagonists & inhibitors | Neurogenic Inflammation - chemically induced | Bradykinin Receptor Antagonists | Dioxoles - pharmacology | Behavior, Animal - drug effects | Pruritus - chemically induced | Disease Models, Animal | Injections, Intradermal | Pyrazoles - pharmacology | Reproducibility of Results | Plant Proteins - pharmacology | Cyclooxygenase 2 Inhibitors - pharmacology | Calcitonin Gene-Related Peptide - pharmacology | Receptors, Calcitonin Gene-Related Peptide - antagonists & inhibitors | p-Methoxy-N-methylphenethylamine - pharmacology | Celecoxib | Sulfonamides - pharmacology | Cinnamates - pharmacology | Mast Cells - drug effects | Trypsin - administration & dosage | TRPV Cation Channels - genetics | Mice, Knockout | Neurogenic Inflammation - prevention & control | Pruritus - metabolism | Animals | Receptors, Bradykinin - metabolism | Signal Transduction - drug effects | Nerve Fibers, Unmyelinated - metabolism | Anilides - pharmacology | Cyclooxygenase 2 - metabolism | Neurogenic Inflammation - metabolism | Pruritus - prevention & control | Mice | Oligopeptides - pharmacology | Cell Degranulation - drug effects | Index Medicus | Research Papers
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4. Full Text Sex differences in endothelial function in porcine coronary arteries: a role for H2O2 and gap junctions?
by Wong, P S and Roberts, R E and Randall, M D
British journal of pharmacology, ISSN 0007-1188, 06/2014, Volume 171, Issue 11, pp. 2751 - 2766
porcine coronary artery (PCA) | gap junctions | endothelium‐derived hyperpolarization (EDH) | nitric oxide (NO) | intermediate‐conductance calcium‐activated K+ channel (IKca) | connexin (Cx) | bradykinin | hydrogen peroxide (H2O2) | small‐conductance calcium‐activated K+ channel (SKca) | sex differences | endothelium-derived hyperpolarization (EDH) | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Catalase - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Vasodilator Agents - pharmacology | Coronary Vessels - physiology | Glycyrrhetinic Acid - analogs & derivatives | Hydrogen Peroxide - pharmacology | Male | Sex Characteristics | Apamin - pharmacology | Indomethacin - pharmacology | Vasodilation | Connexins - metabolism | Endothelium, Vascular - physiology | Carbenoxolone - pharmacology | Glycyrrhetinic Acid - pharmacology | Animals | Gap Junctions - physiology | Swine | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Female | Bradykinin - pharmacology | Polyethylene Glycols - pharmacology | Pyrazoles - pharmacology | Postmenopausal women | Hydrogen peroxide | Nitric oxide | Arteries | Endothelium | Index Medicus | Research Papers | intermediate-conductance calcium-activated K+ channel (IKca) | small-conductance calcium-activated K+ channel (SKca)
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5. Full Text Angiotensin II Type 2 Receptor–Mediated and Nitric Oxide–Dependent Renal Vasodilator Response to Compound 21 Unmasked by Angiotensin-Converting Enzyme Inhibition in Spontaneously Hypertensive Rats In Vivo
by Brouwers, Sofie and Smolders, Ilse and Massie, Ann and Dupont, Alain G
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 11/2013, Volume 62, Issue 5, pp. 920 - 926
renal circulation | receptor angiotensin type 2 | blood pressure | compound 21 | angiotensin II | receptor, angiotensin type 1 | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Arterial hypertension. Arterial hypotension | Medical sciences | Blood and lymphatic vessels | Bradykinin - analogs & derivatives | Kidney - blood supply | Captopril - pharmacology | Rats, Inbred WKY | omega-N-Methylarginine - pharmacology | Dose-Response Relationship, Drug | Fenoldopam - pharmacology | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Blood Pressure - drug effects | Blood Pressure - physiology | Vasodilation - physiology | Bradykinin - pharmacology | Rats, Inbred SHR | Dopamine Agonists - pharmacology | Receptor, Angiotensin, Type 2 - agonists | Kidney - drug effects | Vasodilator Agents - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Thiophenes - pharmacology | Imidazoles - pharmacology | Sulfonamides - pharmacology | Indomethacin - pharmacology | Animals | Pyridines - pharmacology | Vasodilation - drug effects | Nitric Oxide - metabolism | Index Medicus
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6. Full Text The Binding of Oxidized Low Density Lipoprotein (ox-LDL) to ox-LDL Receptor-1 Reduces the Intracellular Concentration of Nitric Oxide in Endothelial Cells through an Increased Production of Superoxide
by Luciano Cominacini and Anna Rigoni and Anna Fratta Pasini and Ulisse Garbin and Anna Davoli and Mario Campagnola and Antonio M. Pastorino and Vincenzo Lo Cascio and Tatsuya Sawamura
The Journal of biological chemistry, ISSN 0021-9258, 04/2001, Volume 276, Issue 17, pp. 13750 - 13755
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Free Radical Scavengers - pharmacology | Reactive Oxygen Species - metabolism | Humans | Scavenger Receptors, Class E | Hemostatics - pharmacology | omega-N-Methylarginine - pharmacology | Aorta - metabolism | Oxygen - metabolism | Dose-Response Relationship, Drug | Probucol - pharmacology | Flow Cytometry | Thrombin - pharmacology | Chromans - pharmacology | Time Factors | Cattle | Superoxides - metabolism | Lipoproteins, LDL - metabolism | Aspirin - pharmacology | Bradykinin - pharmacology | CHO Cells | Cricetinae | Receptors, Oxidized LDL | Antibodies, Monoclonal - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Receptors, LDL - metabolism | Antioxidants - pharmacology | Imidazolines | Animals | Endothelium, Vascular - metabolism | Ascorbic Acid - pharmacology | Protein Binding | Allopurinol - pharmacology | Mice | Nitric Oxide - metabolism | Catecholamines - pharmacology | Index Medicus
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7. Full Text Involvement of bradykinin, cytokines, sympathetic amines and prostaglandins in formalin‐induced orofacial nociception in rats
by Chichorro, Juliana G and Lorenzetti, Berenice B and Zampronio, Aleksander R
British journal of pharmacology, ISSN 0007-1188, 04/2004, Volume 141, Issue 7, pp. 1175 - 1184
Formalin | bradykinin | prostaglandins | cytokines | noradrenaline | Prostaglandins | Cytokines | Noradrenaline | Bradykinin | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Biological and medical sciences | Pharmacology. Drug treatments | Medical sciences | Bradykinin - analogs & derivatives | Bradykinin B1 Receptor Antagonists | Captopril - pharmacology | Rats, Wistar | Nociceptors - physiology | Male | Cytokines - physiology | Facial Pain - physiopathology | Cytokines - chemistry | Dose-Response Relationship, Drug | Facial Pain - chemically induced | Tyramine - chemistry | Arachidonic Acids - metabolism | Injections, Subcutaneous | Formaldehyde - administration & dosage | Thiazines - pharmacology | Behavior, Animal - drug effects | Interleukin-8 - pharmacology | Biogenic Amines - physiology | Bradykinin - pharmacology | Atenolol - pharmacology | Pyrazoles - pharmacology | Lip - drug effects | Tyramine - physiology | Rats | Behavior, Animal - physiology | Hindlimb - innervation | Receptor, Bradykinin B2 - physiology | Bradykinin B2 Receptor Antagonists | Celecoxib | Sulfonamides - pharmacology | Arachidonic Acids - pharmacology | Indomethacin - pharmacology | Drug Synergism | Bradykinin - physiology | Tumor Necrosis Factor-alpha - pharmacology | Prostaglandins - physiology | Animals | Interleukin-6 - pharmacology | Lip - innervation | Receptor, Bradykinin B1 - physiology | Hindlimb - drug effects | Thiazoles - pharmacology | Guanethidine - pharmacology | Nociceptors - drug effects | Facial Pain - drug therapy | Index Medicus | Papers
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8. Full Text Bestrophin-1 Encodes for the Ca2+-Activated Anion Channel in Hippocampal Astrocytes
by Park, Hyungju and Oh, Soo-Jin and Han, Kyung-Seok and Woo, Dong Ho and Park, Hyekyung and Mannaioni, Guido and Traynelis, Stephen F and Lee, C. Justin
The Journal of neuroscience, ISSN 0270-6474, 10/2009, Volume 29, Issue 41, pp. 13063 - 13073
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | RNA, Small Interfering - genetics | Membrane Potentials - genetics | Ion Channels | Calcium - metabolism | Humans | Chloride Channels - genetics | Cerebral Cortex - cytology | Adenosine Triphosphate - pharmacology | Transfection - methods | Bestrophins | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Egtazic Acid - analogs & derivatives | Lysophospholipids - pharmacology | Electric Stimulation - methods | Bradykinin - pharmacology | Animals, Newborn | Membrane Potentials - drug effects | Dinoprostone - pharmacology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Chelating Agents - pharmacology | Thionucleotides - pharmacology | Hippocampus - cytology | Adenosine Triphosphate - analogs & derivatives | Astrocytes - physiology | Patch-Clamp Techniques | Animals | Egtazic Acid - pharmacology | Analysis of Variance | Eye Proteins - metabolism | Thapsigargin - pharmacology | Mice | In Vitro Techniques | Oligopeptides - pharmacology | Chloride Channels - physiology | Index Medicus
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