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Expert review of anti-infective therapy, ISSN 1744-8336, 2014, Volume 9, Issue 12, pp. 1149 - 1156
Propionibacterium acnes is a Gram-positive bacterium that forms part of the normal flora of the skin, oral cavity, large intestine, the conjunctiva and the... 
infection | Propionibacterium acnes | inflammation | opportunistic pathogen | device related | skin commensal | INFECTIOUS DISEASES | PROSTHETIC VALVE ENDOCARDITIS | LYMPH-NODES | MICROBIOLOGY | SAPHO SYNDROME | QUANTITATIVE-ANALYSIS | GRANULOMA | COMPLETE GENOME SEQUENCE | DNA | LUMBAR SPONDYLODISCITIS | PHARMACOLOGY & PHARMACY | SARCOIDOSIS | BIOFILM FORMATION | Bone and Bones - pathology | Inflammation - pathology | Brain - microbiology | Skin - microbiology | Mouth - drug effects | Humans | Male | Bone and Bones - drug effects | Anti-Bacterial Agents - therapeutic use | Inflammation - complications | Prostate - pathology | Acquired Hyperostosis Syndrome - drug therapy | Inflammation - drug therapy | Acquired Hyperostosis Syndrome - pathology | Gram-Positive Bacterial Infections - microbiology | Prostate - drug effects | Gram-Positive Bacterial Infections - complications | Skin - pathology | Inflammation - microbiology | Acquired Hyperostosis Syndrome - complications | Propionibacterium acnes - physiology | Prostate - microbiology | Sarcoidosis - drug therapy | Bone and Bones - microbiology | Brain - drug effects | Gram-Positive Bacterial Infections - pathology | Sarcoidosis - complications | Mouth - microbiology | Prostatic Neoplasms - microbiology | Brain - pathology | Mouth - pathology | Sarcoidosis - microbiology | Acquired Hyperostosis Syndrome - microbiology | Gram-Positive Bacterial Infections - drug therapy | Propionibacterium acnes - drug effects | Anti-Bacterial Agents - administration & dosage | Skin - drug effects | Sarcoidosis - pathology | Infection | Implants, Artificial | Prosthesis | Genomics | Skin | Inflammation | Conjunctivitis | Toy industry | Heart | Brain | Synovitis | Virulence | Opportunist infection | Genomes | Eye | Mouth | Osteitis | Ear canal | Prosthetics | Shunts | Pathogens | Sarcoidosis | pustulosis | Acne | Conjunctiva | Joint diseases | Oral cavity | Reviews | Large intestine | Prostate | Prostate cancer | Cancer
Journal Article
Journal of clinical neuroscience, ISSN 0967-5868, 2017, Volume 38, pp. 8 - 12
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2009, Volume 325, Issue 5936, pp. 83 - 87
Type IV pili mediate the initial interaction of many bacterial pathogens with their host cells. In Neisseria meningitidis, the causative agent of cerebrospinal... 
Intercellular junctions | Cell lines | Actins | Small interfering RNA | Bacteria | Cadherins | Reports | Bacterial adhesion | Infections | Blood brain barrier | Endothelial cells | CDC42 | ADHESION | HUMAN EPITHELIAL-CELLS | PATHOGENIC NEISSERIA | TWITCHING MOTILITY | MULTIDISCIPLINARY SCIENCES | NEISSERIA-MENINGITIDIS | BARRIER | PAR3 | RECEPTOR | ADHERENS | Cell Polarity | Brain - microbiology | Cadherins - metabolism | Humans | cdc42 GTP-Binding Protein - metabolism | Intercellular Junctions - microbiology | Phosphoproteins - metabolism | Blood-Brain Barrier - microbiology | Antigens, CD - metabolism | Catenins | Endothelial Cells - microbiology | Endothelium, Vascular - ultrastructure | Brain - blood supply | Protein Kinase C - metabolism | Membrane Proteins - metabolism | Neisseria meningitidis - physiology | Neisseria meningitidis - pathogenicity | Cell Line | Brain - cytology | Endothelial Cells - metabolism | Cell Cycle Proteins - metabolism | Intercellular Junctions - metabolism | Cell Adhesion Molecules - metabolism | Blood-Brain Barrier - metabolism | Endothelium, Vascular - microbiology | Bacterial Adhesion | Endothelium, Vascular - metabolism | Fimbriae, Bacterial - physiology | Adaptor Proteins, Signal Transducing - metabolism | Intercellular Junctions - ultrastructure | Measurement | Polarity (Biology) | Neisseria meningitidis | Properties | Health aspects | Endothelium | Brain | Meningitis | Eukaryotes | Cell adhesion & migration | Fimbriae, Bacterial | Endothelial Cells | Cell Adhesion Molecules | Intercellular Junctions | Membrane Proteins | Life Sciences | Microbiology and Parasitology | Adaptor Proteins, Signal Transducing | Phosphoproteins | cdc42 GTP-Binding Protein | Endothelium, Vascular | Cell Cycle Proteins | Antigens, CD | Protein Kinase C | Blood-Brain Barrier | cytology | meningitis | microbiology | physiology | ultrastructure | blood brain barrier | pathogenicity | blood supply | metabolism
Journal Article
Nature medicine, ISSN 1546-170X, 2016, Volume 22, Issue 5, pp. 516 - 523
Journal Article
Nature communications, ISSN 2041-1723, 2017, Volume 8, Issue 1, p. 24
Neuroinflammation caused by local deposits of A beta(42) in the brain is key for the pathogenesis and progression of Alzheimer's disease. However, inflammation... 
STROKE | SYSTEM | IMMUNE-RESPONSE | ENERGY HOMEOSTASIS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | NEURODEGENERATION | MECHANISMS | MODEL | BRAIN | COMMUNICATION | Antimicrobial Cationic Peptides - immunology | Alzheimer Disease - complications | Brain - microbiology | Drosophila melanogaster - immunology | Hemocytes - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | MAP Kinase Kinase 4 - immunology | Drosophila Proteins - immunology | Dysbiosis - microbiology | Enterobacteriaceae Infections - complications | Alzheimer Disease - pathology | Cell Movement - immunology | Enterobacteriaceae Infections - immunology | Hemocytes - pathology | Dysbiosis - immunology | Gastrointestinal Tract - immunology | Enterobacteriaceae - immunology | Leukocyte Reduction Procedures | Amyloid beta-Peptides - genetics | Peptide Fragments - immunology | Tumor Necrosis Factor-alpha - immunology | Hemocytes - microbiology | Alzheimer Disease - immunology | Antimicrobial Cationic Peptides - genetics | Peptide Fragments - genetics | Enterobacteriaceae - growth & development | Disease Models, Animal | Alzheimer Disease - microbiology | Microbiota - immunology | Signal Transduction | Enterobacteriaceae Infections - microbiology | Gene Expression Regulation | Gastrointestinal Tract - microbiology | Enterobacteriaceae Infections - pathology | Dysbiosis - complications | Disease Progression | Drosophila melanogaster - microbiology | Animals | Brain - pathology | MAP Kinase Kinase 4 - genetics | Amyloid beta-Peptides - immunology | Drosophila Proteins - genetics | Dysbiosis - pathology | Brain - immunology
Journal Article
PloS one, ISSN 1932-6203, 02/2007, Volume 2, Issue 2, p. e241
Neisseria meningitidis is a human pathogen that causes septicemia and meningitis with high mortality. The disease progression is rapid and much remains unknown... 
BIOLOGY | Thyroid Gland - microbiology | Recombinant Fusion Proteins - physiology | Male | Respiratory System - microbiology | Luminescent Measurements | Sepsis - pathology | Meningococcal Infections - pathology | Meningitis, Meningococcal - microbiology | Female | Central Nervous System - microbiology | Mice, Transgenic | Bacterial Proteins - physiology | Membrane Cofactor Protein - physiology | Bacterial Translocation | Adhesins, Bacterial - physiology | Disease Progression | Animals | Meningitis, Meningococcal - cerebrospinal fluid | Nasal Cavity - microbiology | Luminescent Proteins - genetics | Membrane Cofactor Protein - genetics | Mice | Bacterial Outer Membrane Proteins - physiology | Bacteremia - pathology | Meningococcal Infections - blood | Sepsis - microbiology | Blood-Brain Barrier | Medical research | Medicine, Experimental | Meningitis | Bacteria | Thyroid gland | Diagnostic imaging | Vaccines | Health aspects | Neuroimaging | Pathogenesis | Mucosa | Biochemistry | Streptococcus infections | Respiratory tract | Proteins | Signal transduction | Blood-brain barrier | Transgenic animals | Rodents | Combined vaccines | Thyroxine | Trends | Deoxyribonucleic acid--DNA | Thyroid | Septicemia | Gene expression | Dynamic tests | Sepsis | In vivo methods and tests | Meningococcal disease | Deoxyribonucleic acid | DNA
Journal Article
PloS one, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e63244
Journal Article
Current Opinion in Microbiology, ISSN 1369-5274, 2011, Volume 15, Issue 1, pp. 44 - 49
Journal Article
Journal of NeuroVirology, ISSN 1355-0284, 2/2016, Volume 22, Issue 1, pp. 22 - 32
We seek an aetiopathogenic model for the spectrum of Parkinson’s disease (PD), functional bowel disease, depression and cognitive impairment. The adopted... 
Autoimmunity | Neurosciences | Intestinal dysbiosis | Pathogenesis | Helicobacter | Aetiology | Virology | Parkinson’s and overlap diseases | Neurology | Bystander damage | Biomedicine | Immunology | Infectious Diseases | CHRONIC INFECTION | INTESTINAL MICROBIOTA | ALZHEIMERS-DISEASE | IDIOPATHIC PARKINSONISM | Parkinson's and overlap diseases | DOPAMINERGIC-NEURONS | BRAIN PATHOLOGY | SUBSTANTIA-NIGRA | NEUROSCIENCES | HUMAN GUT MICROBIOME | VIROLOGY | HELICOBACTER-PYLORI ERADICATION | GROWTH-FACTOR-ALPHA | Depression - pathology | Humans | Helicobacter pylori - pathogenicity | Killer Cells, Natural - pathology | Peptic Ulcer - epidemiology | CD4-Positive T-Lymphocytes - pathology | Dysbiosis - microbiology | Helicobacter Infections - pathology | Inflammatory Bowel Diseases - epidemiology | Cognition Disorders - microbiology | Peptic Ulcer - microbiology | Inflammatory Bowel Diseases - pathology | Parkinson Disease - epidemiology | Neutrophils - microbiology | Killer Cells, Natural - microbiology | Neutrophils - pathology | Parkinson Disease - pathology | CD4-Positive T-Lymphocytes - microbiology | Depression - epidemiology | Comorbidity | Helicobacter pylori - growth & development | Cognition Disorders - pathology | Gastrointestinal Microbiome | Helicobacter Infections - epidemiology | Depression - microbiology | Inflammation | Peptic Ulcer - pathology | Animals | Parkinson Disease - microbiology | Dysbiosis - epidemiology | Inflammatory Bowel Diseases - microbiology | Dysbiosis - pathology | Cognition Disorders - epidemiology | Helicobacter Infections - microbiology | Mini-Review
Journal Article