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Circulation, ISSN 0009-7322, 07/2007, Volume 116, Issue 1, pp. 77 - 84
NERVOUS-SYSTEM | CARDIAC & CARDIOVASCULAR SYSTEMS | antioxidants | SUDDEN-DEATH | nervous system, autonomic | INJURY | apoptosis | death, sudden | nervous system, sympathetic | INTRACRANIAL HEMORRHAGE | DAMAGE | MYOCARDIAL LESIONS | cerebral infarction | INSULAR CORTEX | SUBARACHNOID HEMORRHAGE | PERIPHERAL VASCULAR DISEASE | cardiomyopathy | STRESS | Thyroxine - toxicity | Calcium - metabolism | Humans | Death, Sudden, Cardiac - etiology | Sympathetic Nervous System - physiopathology | Fludrocortisone - analogs & derivatives | Arrhythmias, Cardiac - physiopathology | Necrosis | Cardiotonic Agents - therapeutic use | Stress, Physiological - complications | Fludrocortisone - toxicity | Electrocardiography | Myocardial Stunning - etiology | Parasympathetic Nervous System - physiopathology | Heart Diseases - chemically induced | Culture | Helplessness, Learned | Ergocalciferols - toxicity | Cats | Psychophysiologic Disorders - physiopathology | Myocardial Stunning - physiopathology | Heart Diseases - physiopathology | Myocardial Stunning - psychology | Rats | Catecholamines - physiology | Myocardium - pathology | Stress, Physiological - physiopathology | Arrhythmias, Cardiac - etiology | Catecholamines - toxicity | Immobilization - adverse effects | Subarachnoid Hemorrhage - complications | Hypothalamus - physiopathology | Emotions | Myocardial Reperfusion Injury - physiopathology | Vagus Nerve - physiopathology | Catecholamines - antagonists & inhibitors | Heart Diseases - etiology | Heart Diseases - psychology | Subarachnoid Hemorrhage - physiopathology | Animals | Psychophysiologic Disorders - psychology | Heart Conduction System - physiopathology | Dogs | Mice | Stress, Physiological - psychology | Heart | Physiological aspects | Brain | Research
Journal Article
Journal Article
European Heart Journal, ISSN 0195-668X, 05/2015, Volume 36, Issue 17, pp. 1049 - 1058
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, pp. 1798 - 1808
Journal Article
Neuroscience, ISSN 0306-4522, 2014, Volume 273, pp. 39 - 51
Highlights • We used brain slices from transgenic mice with features of human essential hypertension. • Brain slices were exposed to oxygen–glucose deprivation... 
Neurology | cerebral cortex | brain edema | hippocampus | stroke | hypertension | NADPH oxidase | Hypertension | Cerebral cortex | Stroke | Brain edema | Hippocampus | CONVERTING ENZYME 2 | HIPPOCAMPAL SLICES | OXIDATIVE STRESS | PROTECTS DOPAMINERGIC-NEURONS | NEUROSCIENCES | INDUCED MICROGLIAL ACTIVATION | INCREASED SUPEROXIDE | HUMAN RENIN | LONG-TERM POTENTIATION | INTRINSIC OPTICAL SIGNALS | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Receptors, G-Protein-Coupled - metabolism | NADPH Oxidases - metabolism | Stroke - complications | Stroke - physiopathology | Cerebral Cortex - physiopathology | Peptidyl-Dipeptidase A - genetics | Neurons - physiology | Peptidyl-Dipeptidase A - metabolism | Brain Ischemia - complications | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Peptide Fragments - metabolism | Angiotensin II - metabolism | Brain Edema - etiology | Tissue Culture Techniques | Brain - physiopathology | Mice, Transgenic | Brain Ischemia - physiopathology | Hypoxia - complications | Angiotensin II - analogs & derivatives | NADPH Oxidase 4 | Angiotensin I - metabolism | NADPH Oxidase 2 | Glucose - deficiency | Animals | Brain Edema - physiopathology | Cell Death - physiology | Receptor, Angiotensin, Type 1 - metabolism | Receptors, G-Protein-Coupled - antagonists & inhibitors | Hypoxia - physiopathology | Signal Transduction - physiology | CA1 Region, Hippocampal - physiopathology | Oxidases | Glucose metabolism | Brain | Cell death | Analysis | Angiotensin | Biochemistry | Genetic engineering | Glucose | Dextrose | Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 138, Issue 1, pp. 310 - 312.e3
Journal Article
Neurology, ISSN 0028-3878, 07/2007, Volume 69, Issue 5, pp. 434 - 441
Objective: Ictal asystole (IA) is a rare event mostly seen in patients with temporal lobe epilepsy (TLE) and a potential contributor to sudden unexplained... 
CARDIAC ASYSTOLE | ARRHYTHMIAS | TEMPORAL-LOBE EPILEPSY | BRADYCARDIA SYNDROME | SUDDEN-DEATH | SYNCOPE | DROP ATTACKS | PARTIAL SEIZURES | INSULAR CORTEX STIMULATION | ECG-CHANGES | CLINICAL NEUROLOGY | Predictive Value of Tests | Electroencephalography - standards | Brain - anatomy & histology | Monitoring, Physiologic - standards | Syncope - diagnosis | Bradycardia - diagnosis | Humans | Middle Aged | Child, Preschool | Death, Sudden, Cardiac - prevention & control | Epilepsy, Temporal Lobe - physiopathology | Male | Death, Sudden, Cardiac - etiology | Epilepsy - physiopathology | Epilepsy, Temporal Lobe - complications | Epilepsy, Temporal Lobe - diagnosis | Adult | Female | Electroencephalography - trends | Epilepsy, Generalized - etiology | Autonomic Pathways - physiopathology | Video Recording - standards | Brain - physiopathology | Bradycardia - physiopathology | Brain Ischemia - physiopathology | Brain Ischemia - etiology | Heart Arrest - physiopathology | Electrodiagnosis - methods | Electroencephalography - methods | Heart Arrest - diagnosis | Epilepsy - complications | Epilepsy - diagnosis | Epilepsy, Generalized - physiopathology | Syncope - physiopathology | Electrodiagnosis - standards | Video Recording - trends | Bradycardia - etiology | Video Recording - methods | Adolescent | Electrodiagnosis - trends | Syncope - etiology | Brain Ischemia - diagnosis | Heart Arrest - etiology | Monitoring, Physiologic - methods | Monitoring, Physiologic - instrumentation | Aged | Early Diagnosis
Journal Article
2013, 1. Aufl., ISBN 9781461443032, Volume 9781461443049, 368
Addressing all aspects of brain death and thoroughly detailing how a potential organ donor should be maintained to ensure maximum use of the organs and cells,... 
Other branches of medicine | Brain death | Transplantation of organs, tissues, etc | Donation of organs, tissues, etc
eBook
Journal of Neurotrauma, ISSN 0897-7151, 04/2010, Volume 27, Issue 4, pp. 753 - 762
In this study we tested the hypothesis that long-term neuropathological outcome is worsened by hyperoxic compared to normoxic reperfusion in a rat global... 
Morris water maze | Astrocyte | Hippocampus | Resuscitation | Cardiac arrest | Microglia | hippocampus | BRAIN-INJURY | 100-PERCENT OXYGEN | CARDIOPULMONARY | NORMOBARIC OXYGEN | RATS | FOREBRAIN ISCHEMIA | cardiac arrest | NEUROSCIENCES | CLINICAL NEUROLOGY | STROKE | CARDIAC-ARREST | resuscitation | astrocyte | microglia | CRITICAL CARE MEDICINE | Neurons - pathology | Hyperoxia - physiopathology | Rats, Inbred F344 | Nerve Degeneration - physiopathology | Learning Disorders - physiopathology | Male | Gliosis - physiopathology | Time | Learning Disorders - etiology | Oxygen Consumption - physiology | Oxygen - metabolism | Gliosis - pathology | Learning Disorders - pathology | Encephalitis - physiopathology | Cognition Disorders - etiology | Hyperoxia - complications | Neurons - physiology | Reperfusion Injury - complications | Cell Survival - physiology | Gliosis - etiology | Resuscitation - adverse effects | Disease Models, Animal | Brain Ischemia - complications | Cognition Disorders - physiopathology | Oxygen - toxicity | Reperfusion Injury - pathology | Cognition Disorders - pathology | Encephalitis - pathology | Rats | Brain Ischemia - physiopathology | Hippocampus - pathology | Nerve Degeneration - pathology | Hyperoxia - pathology | Animals | Brain Ischemia - pathology | Reperfusion Injury - physiopathology | Nerve Degeneration - etiology | Hippocampus - physiopathology | Encephalitis - etiology | Cerebral ischemia | Care and treatment | Neurons | Patient outcomes | Physiological aspects | Reperfusion (Physiology) | Research | Hippocampus (Brain) | Stroke | Ischemia | Hyperoxia | Rodents | Apoptosis | Original
Journal Article