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Cancer Cell, ISSN 1535-6108, 2007, Volume 11, Issue 1, pp. 83 - 95
Journal Article
Stroke, ISSN 0039-2499, 02/2010, Volume 41, Issue 2, pp. 307 - 312
Background and Purpose-The Intensive Blood Pressure Reduction In Acute Cerebral Haemorrhage Trial (INTERACT) study suggests that early intensive blood pressure... 
Hypertension | Clinical trial | Blood pressure | Treatment | Intracerebral hemorrhage | clinical trial | MORTALITY | blood pressure | treatment | MANAGEMENT | ENLARGEMENT | CLINICAL NEUROLOGY | ACTIVATED FACTOR-VII | intracerebral hemorrhage | ACUTE STROKE | PERIPHERAL VASCULAR DISEASE | SCALE | hypertension | Brain - diagnostic imaging | Outcome Assessment (Health Care) | Humans | Middle Aged | Antihypertensive Agents - administration & dosage | Hypertension - drug therapy | Male | Tomography, X-Ray Computed | Intracranial Hemorrhage, Hypertensive - pathology | Cerebral Arteries - drug effects | Brain - blood supply | Time Factors | China | Adult | Female | Blood Pressure - drug effects | Blood Pressure - physiology | Intracranial Hemorrhage, Hypertensive - complications | Intracranial Hemorrhage, Hypertensive - drug therapy | Republic of Korea | Brain Edema - etiology | Drug Administration Schedule | Cerebral Arteries - diagnostic imaging | Brain Edema - pathology | Treatment Outcome | Acute Disease - therapy | Disease Progression | Antihypertensive Agents - adverse effects | Emergency Medical Services - statistics & numerical data | Cerebral Arteries - pathology | Brain - pathology | Hypertension - complications | Aged | Brain Edema - drug therapy | Emergency Medical Services - methods | Australia | Early Diagnosis | Practice Guidelines as Topic | Index Medicus
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 04/2010, Volume 207, Issue 4, pp. 881 - 896
Endothelial cell-cell junctions regulate vascular permeability, vasculogenesis, and angiogenesis. Familial cerebral cavernous malformations (CCMs) in humans... 
CELL PERMEABILITY | PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNE-RESPONSE | P38 MAPK ACTIVATION | ACUTE LUNG INJURY | VE-CADHERIN | CCM2 | IMMUNOLOGY | 2-HIT MECHANISM | BRAIN | ENDOTHELIAL BARRIER FUNCTION | RNA, Small Interfering - genetics | Humans | Capillary Permeability - drug effects | Endothelium, Vascular - drug effects | rhoA GTP-Binding Protein - metabolism | Animal Structures - blood supply | Mutation - physiology | Phosphorylation - genetics | rho-Associated Kinases - metabolism | Edema, Cardiac - pathology | Phosphorylation - drug effects | Endothelial Cells - metabolism | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - therapeutic use | Hemangioma, Cavernous, Central Nervous System - drug therapy | Edema, Cardiac - genetics | Hemangioma, Cavernous, Central Nervous System - pathology | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Mice | Myosin Light Chains - metabolism | Animal Structures - metabolism | Protein Binding - physiology | Capillary Permeability - physiology | KRIT1 Protein | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Hemangioma, Cavernous, Central Nervous System - metabolism | rho-Associated Kinases - antagonists & inhibitors | Brain Edema - genetics | Edema, Cardiac - drug therapy | Mice, Mutant Strains | Pulmonary Edema - pathology | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Proto-Oncogene Proteins - metabolism | Pulmonary Edema - genetics | Brain Edema - pathology | Mice, Inbred C57BL | Proto-Oncogene Proteins - genetics | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Protein Kinase Inhibitors - therapeutic use | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Signal Transduction - physiology | Brain Edema - drug therapy | Protein Kinase Inhibitors - pharmacology | Endothelial Cells - drug effects | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e84767 - e84767
Ischemic stroke results in disruption of the blood-brain barrier (BBB), edema formation and neuronal cell loss. Some neuroprotective factors such as vascular... 
MOUSE-BRAIN | NERVOUS-SYSTEM | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | ALZHEIMERS-DISEASE | VEGF | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | PROLYL HYDROXYLASES | NEUROPROTECTION | ENDOTHELIAL GROWTH-FACTOR | VASCULAR LEAKAGE | Neurons - pathology | Brain Injuries - drug therapy | Brain Infarction - metabolism | Brain Infarction - pathology | Male | Hypoxia-Inducible Factor-Proline Dioxygenases - antagonists & inhibitors | Brain Injuries - metabolism | Stroke - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neurons - metabolism | Neurons - drug effects | Brain Infarction - drug therapy | Tight Junction Proteins - metabolism | Astrocytes - drug effects | Endothelial Cells - metabolism | Edema - drug therapy | Stroke - drug therapy | Protein Transport | Edema - metabolism | Stroke - metabolism | Animals | Signal Transduction - drug effects | Intracellular Space - metabolism | Mice | Brain Injuries - pathology | Astrocytes - metabolism | Endothelial Cells - drug effects | Brain | Dropsy | Edema | Ischemia | Neurons | Glycoproteins | DNA binding proteins | Hydroxylases | Vascular endothelial growth factor | Endothelium | Neuroprotection | Diabetic retinopathy | Transcription factors | Phosphorylation | Erythropoietin | Damage prevention | Proteins | Prevention | Angiogenesis | Signal transduction | Blood-brain barrier | Neurodegeneration | Rodents | Physiology | Prolyl hydroxylase | Inhibition | Leakage | Deprivation | Stroke | Cell survival | Astrocytes | Hydroxylase | Permeability | Zonula occludens-1 protein | Endothelial cells | Signaling | Injury prevention | Hypoxia | Brain injury | Veins & arteries | Index Medicus
Journal Article
Journal Article
Medicine (United States), ISSN 0025-7974, 11/2017, Volume 96, Issue 46, pp. e7358 - e7358
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1282, pp. 133 - 141
Abstract Background: Oxidative and cytotoxic damage plays an important role in cerebral ischemic pathogenesis and may represent a target for treatment.... 
Neurology | Nuclear factor erythroid 2-related factor 2(Nrf2) | Neuroprotection | Cerebral ischemia | Curcumin | OXIDATIVE STRESS | ACTIVATION | MACROPHAGES | NEUROSCIENCES | ANTIOXIDANT DEFENSE | CEREBRAL-ARTERY OCCLUSION | IN-VIVO | NITRIC-OXIDE | NEURONS | KEAP1/NRF2 PATHWAY | HEME OXYGENASE-1 | Infarction, Middle Cerebral Artery - physiopathology | Heme Oxygenase-1 - metabolism | Brain Ischemia - metabolism | RNA, Messenger - analysis | Brain Infarction - pathology | Heme Oxygenase-1 - drug effects | Male | Cytoprotection - physiology | Infarction, Middle Cerebral Artery - metabolism | RNA, Messenger - metabolism | Brain - metabolism | Brain Edema - prevention & control | Brain Infarction - prevention & control | Heme Oxygenase-1 - genetics | Neuroprotective Agents - pharmacology | Time Factors | Up-Regulation - physiology | Cytoprotection - drug effects | NF-E2-Related Factor 2 - genetics | Infarction, Middle Cerebral Artery - drug therapy | Brain Infarction - drug therapy | Disease Models, Animal | Curcumin - therapeutic use | Brain - physiopathology | Brain Edema - pathology | NF-E2-Related Factor 2 - drug effects | Curcumin - pharmacology | Enzyme Inhibitors - pharmacology | Gene Expression Regulation - physiology | Rats | Brain Ischemia - physiopathology | Rats, Sprague-Dawley | Brain - drug effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Brain Ischemia - drug therapy | NF-E2-Related Factor 2 - metabolism | Brain Edema - drug therapy | Index Medicus
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 02/2009, Volume 29, Issue 2, pp. 317 - 330
Subarachnoid hemorrhage (SAH) causes secondary brain injury due to vasospasm and inflammation. Here, we studied a rat model of mild-to-moderate SAH intended to... 
Sulfonylurea receptor 1 | Vasogenic edema | Inflammation | Subarachnoid hemorrhage | Caspase-3 | Zona occludens 1 | APOPTOSIS | CYTOKINES | zona occludens 1 | sulfonylurea receptor 1 | PERMEABILITY | CYTOSKELETON | ISCHEMIA | vasogenic edema | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | PATHOPHYSIOLOGY | inflammation | subarachnoid hemorrhage | JUNCTIONS | ENDOCRINOLOGY & METABOLISM | CEREBRAL VASOSPASM | HEMATOLOGY | CELL | caspase-3 | Inflammation - pathology | Rats, Wistar | Receptors, Drug - genetics | Carotid Artery Injuries | Caspase 3 - metabolism | Male | Sulfonylurea Receptors | Brain Edema - enzymology | ATP-Binding Cassette Transporters - genetics | Inflammation - drug therapy | ATP-Binding Cassette Transporters - metabolism | Glyburide - therapeutic use | Subarachnoid Hemorrhage - enzymology | Subarachnoid Hemorrhage - drug therapy | Brain Edema - pathology | Rats | Potassium Channels, Inwardly Rectifying - genetics | Up-Regulation - genetics | Carotid Artery, Internal | Enzyme Activation - drug effects | Subarachnoid Hemorrhage - pathology | Blood-Brain Barrier - drug effects | Receptors, Drug - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Animals | Hypoxia - pathology | Brain Edema - drug therapy | Inflammation - enzymology | Potassium Channels, Inwardly Rectifying - metabolism | Index Medicus
Journal Article
Brain Research, ISSN 0006-8993, 2013, Volume 1548, pp. 56 - 62
Abstract Lavender belongs to the family Labiatae and has a variety of cosmetic uses as well as therapeutic purposes in herbal medicine. The present study was... 
Neurology | Oxidative stress | Bcl-2 and Bax | Rat | Lavender oil | VEGF | Focal cerebral ischemia | Brain edema | Antioxidant | Apoptosis | AQUEOUS EXTRACT | ARTERY OCCLUSION | ANGIOGENESIS | PERFORMANCE | ISCHEMIA |