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Neuroscience, ISSN 0306-4522, 2009, Volume 158, Issue 3, pp. 983 - 994
Journal Article
Stroke, ISSN 0039-2499, 09/2008, Volume 39, Issue 9, pp. 2538 - 2543
Background and Purpose-The concept of the neurovascular unit suggests that effects on brain vasculature must be considered if neuroprotection is to be achieved... 
Baicalein | Edema | Endothelial cell | Blood-brain barrier | Lipoxygenase | blood-brain barrier | edema | RAT | endothelial cell | baicalein | 15-LIPOXYGENASE | MODEL | BLOOD-BRAIN-BARRIER | CLINICAL NEUROLOGY | STROKE | lipoxygenase | INHIBITION | DISRUPTION | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | CEREBRAL-ISCHEMIA | ORGANELLE DEGRADATION | Arachidonate 12-Lipoxygenase - metabolism | Humans | Oxidative Stress - physiology | Ischemic Attack, Transient - enzymology | Brain Edema - enzymology | Brain Edema - prevention & control | Brain Ischemia - enzymology | Cytoprotection - drug effects | Membrane Proteins - metabolism | Brain Infarction - drug therapy | Arachidonate 12-Lipoxygenase - genetics | Ischemic Attack, Transient - physiopathology | Tight Junctions - drug effects | Brain Infarction - enzymology | Tight Junctions - metabolism | Ischemic Attack, Transient - drug therapy | Flavanones - therapeutic use | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Hydrogen Peroxide - pharmacology | Arachidonate 15-Lipoxygenase - drug effects | Claudin-5 | Brain Ischemia - physiopathology | Brain Infarction - physiopathology | Arachidonate 15-Lipoxygenase - metabolism | Blood-Brain Barrier - drug effects | Enzyme Inhibitors - therapeutic use | Blood-Brain Barrier - metabolism | Mice, Knockout | Animals | Arachidonate 15-Lipoxygenase - genetics | Brain Edema - physiopathology | Brain Ischemia - drug therapy | Arachidonate 12-Lipoxygenase - drug effects | Mice | Oxidative Stress - drug effects | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Immunoglobulin G - metabolism | blood–brain barrier
Journal Article
Stroke, ISSN 0039-2499, 2007, Volume 38, Issue 11, pp. 3000 - 3006
Background and Purpose - Cerebral ischemia/reperfusion is associated with reactive oxygen species (ROS) generation, and NADPH oxidases are important sources of... 
Oxidative stress | Ischemia/reperfusion | RhoA | Statins | Endothelium | REDUCTASE INHIBITORS | endothelium | FOCAL CEREBRAL-ISCHEMIA | NEURONAL DAMAGE | RAT MODEL | CLINICAL NEUROLOGY | FREE-RADICALS | IN-VITRO | statins | ENDOTHELIAL-CELLS | ischemia/ reperfusion | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | SUPEROXIDE-DISMUTASE | oxidative stress | Infarction, Middle Cerebral Artery - physiopathology | Cell Hypoxia - physiology | Reactive Oxygen Species - metabolism | Blood-Brain Barrier - physiopathology | Male | Protein Transport - physiology | Protein Transport - drug effects | Stroke - physiopathology | rho-Associated Kinases - antagonists & inhibitors | Brain Edema - enzymology | MAP Kinase Signaling System - genetics | rho-Associated Kinases - metabolism | NADH, NADPH Oxidoreductases - metabolism | Disease Models, Animal | NADH, NADPH Oxidoreductases - genetics | Brain Edema - etiology | Endothelial Cells - metabolism | Reperfusion Injury - enzymology | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Enzyme Activation - drug effects | NADPH Oxidase 1 | Mice, Knockout | Stroke - enzymology | Animals | Blood-Brain Barrier - enzymology | MAP Kinase Signaling System - drug effects | Brain Edema - physiopathology | Reperfusion Injury - physiopathology | rac1 GTP-Binding Protein - antagonists & inhibitors | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Infarction, Middle Cerebral Artery - enzymology | Enzyme Activation - physiology | Sus scrofa | rac1 GTP-Binding Protein - metabolism
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 02/2009, Volume 29, Issue 2, pp. 317 - 330
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 3/2016, Volume 53, Issue 2, pp. 1310 - 1321
The development and/or progression of perihematomal edema (PHE) in patients with acute spontaneous intracerebral hemorrhage (ICH) vary substantially with... 
Neurology | Perihematomal edema | Neurosciences | Biomedicine | MicroRNA-130a | Neurobiology | Biomarkers | Clinical outcome prediction | Intracerebral hemorrhage | Cell Biology | MANAGEMENT | ANGIOGENESIS | PERIHEMORRHAGIC EDEMA | GUIDELINES | MICRORNAS | HEALTH-CARE PROFESSIONALS | NEUROSCIENCES | INVASION | EXPRESSION | Demography | Prognosis | Humans | Middle Aged | Microvessels - pathology | Male | Cerebral Hemorrhage - genetics | Hematoma - complications | RNA, Messenger - metabolism | Brain Edema - enzymology | Behavior, Animal | Brain Edema - genetics | Cerebral Hemorrhage - blood | Matrix Metalloproteinase 9 - metabolism | Brain Edema - complications | Thrombin - pharmacology | Hematoma - genetics | Matrix Metalloproteinase 9 - genetics | Female | Disease Models, Animal | Acute Disease | Endothelial Cells - metabolism | Matrix Metalloproteinase 2 - metabolism | RNA, Messenger - genetics | Treatment Outcome | Up-Regulation - genetics | Rats, Sprague-Dawley | Blood-Brain Barrier - metabolism | Caveolin 1 - metabolism | Blood-Brain Barrier - pathology | Matrix Metalloproteinase 2 - genetics | Animals | Brain - pathology | Cerebral Hemorrhage - enzymology | MicroRNAs - blood | Endothelial Cells - pathology | Cerebral Hemorrhage - complications | Brain Edema - blood | Endothelial Cells - drug effects | Hematoma - blood | Dropsy | Edema | Thrombin | Permeability | Analysis | Brain | Hemorrhage | MicroRNAs
Journal Article
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 5/2012, Volume 32, Issue 5, pp. 919 - 932
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e34504
Background: Oxidative stress is known to play an important role in the pathology of traumatic brain injury. Mitochondria are thought to be the major source of... 
SYNAPTIC PROTEINS | OXYGEN | CONTROLLED CORTICAL IMPACT | SUPEROXIDE-PRODUCTION | ALZHEIMERS-DISEASE | EDEMA | MULTIDISCIPLINARY SCIENCES | RATS | ISCHEMIA | MICE | AMYLOID PRECURSOR PROTEIN | Microglia - metabolism | Membrane Glycoproteins - metabolism | Oxidative Stress | Cerebral Cortex - pathology | NADPH Oxidases - metabolism | Male | Hippocampus - drug effects | Cerebral Cortex - metabolism | Brain Edema - enzymology | Membrane Glycoproteins - antagonists & inhibitors | Microglia - physiology | Neuroprotective Agents - pharmacology | Isoenzymes - metabolism | Acetophenones - pharmacology | Amyloid beta-Peptides - metabolism | Superoxides - metabolism | Neurons - metabolism | Cerebral Cortex - drug effects | Neurons - drug effects | Microglia - drug effects | Oxidation-Reduction | Brain Edema - pathology | NADPH Oxidases - antagonists & inhibitors | Brain Injuries - enzymology | Hippocampus - pathology | NADPH Oxidase 2 | Hippocampus - metabolism | Animals | Neurons - enzymology | Mice | Enzyme Activation | Brain Injuries - pathology | Isoenzymes - antagonists & inhibitors | Oxidases | Brain | Neurons | Amyloid beta-protein | Brain damage | Superoxide | Mediation | Alzheimer's disease | Injuries | Health sciences | Oxidative stress | Neuroprotection | Reactive oxygen species | Cerebral cortex | Traumatic brain injury | Peptides | Laboratories | Cognitive ability | Oxidase | Activation | Neurosurgery | NAD(P)H oxidase | Proteins | Mitochondria | Head injuries | Ischemia | CYBB protein | Localization | Growth factors | Enzymes | Neurodegenerative diseases | Cortex | Amyloid precursor protein | Microglia | Medicine | Pathology | Neurology | Brain research | Inhibitors | β-Amyloid | Alzheimers disease | Hippocampus | Dementia | Animal cognition
Journal Article