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Journal Article
Cardiovascular research, ISSN 0008-6363, 2009, Volume 84, Issue 2, pp. 273 - 282
Our objective was to study the effect of the genetic background on the wound healing process after myocardial infarction (MI) in mice. MI was induced in five... 
Heart failure | Myocardial infarction | Genetic background | Infarct rupture | Inflammatory cells | CARDIAC & CARDIOVASCULAR SYSTEMS | PHENOTYPE | ISCHEMIA | MODEL | FAILURE | RANDOMIZED TRIAL | INHIBITION | GRANULATION-TISSUE | PREVENTS CARDIAC RUPTURE | MICE | TARGETED DELETION | Inflammation - pathology | Myocardial Infarction - genetics | Antihypertensive Agents - pharmacology | Matrix Metalloproteinases - genetics | Species Specificity | Blood Pressure - genetics | Ventricular Function, Left - genetics | Male | RNA, Messenger - metabolism | Time Factors | Atrial Natriuretic Factor - genetics | Myocardial Infarction - pathology | Polymerase Chain Reaction | Myocardial Infarction - physiopathology | Heart Rupture, Post-Infarction - prevention & control | Wound Healing - genetics | Disease Models, Animal | Natriuretic Peptide, Brain - genetics | Echocardiography | Ventricular Pressure - genetics | Mice, Inbred C57BL | Myocardium - pathology | Stroke Volume - genetics | Gene Expression Regulation, Enzymologic | Myocardium - enzymology | Animals | Myocardial Infarction - drug therapy | Inflammation - genetics | Heart Rupture, Post-Infarction - genetics | Mice | Mice, Inbred BALB C | Matrix Metalloproteinases - metabolism | Heart Rupture, Post-Infarction - pathology | Heart Rupture, Post-Infarction - physiopathology | Inflammation - physiopathology
Journal Article
Stroke, ISSN 0039-2499, 02/2010, Volume 41, Issue 2, pp. 210 - 217
Journal Article
Glia, ISSN 0894-1491, 01/2018, Volume 66, Issue 1, pp. 126 - 144
Stimulation of Na + /H + exchanger isoform 1 (NHE1) in astrocytes causes ionic dysregulation under ischemic conditions. In this study, we created a Nhe1... 
MMP | blood–brain barrier | gliosis | cerebral edema | astrocyte end‐feet | neurovascular unit | astrocyte end-feet | blood-brain barrier | NEUROSCIENCES | Glial Fibrillary Acidic Protein - genetics | Cerebrovascular Circulation - physiology | Male | Cerebrovascular Circulation - genetics | Glial Fibrillary Acidic Protein - metabolism | Blood-Brain Barrier - ultrastructure | Gliosis - pathology | Matrix Metalloproteinase 9 - metabolism | Reperfusion | Infarction, Middle Cerebral Artery - complications | Gliosis - etiology | Disease Models, Animal | Microscopy, Electron, Transmission | Gliosis - genetics | Brain Infarction - diagnosis | Gene Expression Regulation - genetics | Gliosis - metabolism | Mice, Inbred C57BL | Sodium-Hydrogen Exchanger 1 - genetics | Mice, Transgenic | Infarction, Middle Cerebral Artery - pathology | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Brain Infarction - etiology | Motor Activity - genetics | Sodium-Hydrogen Exchanger 1 - deficiency | Animals | Neurologic Examination | Mice | Brain Infarction - genetics | Astrocytes - metabolism | Stroke (Disease) | Brain damage | Ischemia | Cytochemistry | Proteases | Analysis | Occlusion | Matrix metalloproteinases | Hydrogen | Recombinase | Gene deletion | Corn oil | Clonal deletion | Blood-brain barrier | Nhe1 gene | Cerebral blood flow | Rodents | Deletion | Cerebral infarction | Edema | Stroke | Tight junctions | Astrocytes | Na+/H+-exchanging ATPase | Inflammation | Tamoxifen | Ablation | Endothelial cells | Flox | Gelatinase B | Blood flow | Neurological diseases | Injury prevention | Gliosis | Infarction | Brain injury | Hypertrophy
Journal Article
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 10, pp. 17347 - 17359
Journal Article
Cardiovascular Research, ISSN 0008-6363, 09/2012, Volume 95, Issue 4, pp. 487 - 494
Innate mechanisms of inter-organ protection underlie the phenomenon of remote ischaemic preconditioning (RPc) in which episode(s) of ischaemia and reperfusion... 
Brain | Vagus nerve | Ischaemia/reperfusion injury | Preconditioning | Ischaemia | CARDIAC & CARDIOVASCULAR SYSTEMS | K-ATP CHANNELS | LIMB ISCHEMIA | MYOCARDIAL-ISCHEMIA | BRAIN-STEM | reperfusion injury | LENTIVIRAL VECTORS | CARDIOVASCULAR CONTROL | TRANSGENE EXPRESSION | RAT-HEART | AUTONOMIC NERVOUS-SYSTEM | Myocardial Infarction - genetics | Hindlimb | Receptors, G-Protein-Coupled - metabolism | Brain Stem - metabolism | Vagus Nerve - drug effects | Male | Receptors, Neuropeptide - metabolism | Drosophila Proteins - metabolism | Rhodopsin - metabolism | Recombinant Fusion Proteins - metabolism | Autonomic Fibers, Preganglionic - metabolism | Brain Stem - drug effects | Action Potentials | Myocardial Reperfusion Injury - pathology | Time Factors | Muscarinic Antagonists - pharmacology | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Adenoviridae - genetics | Lentivirus - genetics | Myocardial Infarction - physiopathology | Neural Pathways - physiopathology | Myocardial Reperfusion Injury - genetics | Autonomic Fibers, Preganglionic - drug effects | Disease Models, Animal | Heart - innervation | Muscle, Skeletal - blood supply | Transduction, Genetic | Vagus Nerve - metabolism | Constriction | Rats | Receptors, Neuropeptide - genetics | Atropine - pharmacology | Myocardium - pathology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Vagus Nerve - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Rhodopsin - genetics | Ischemic Preconditioning, Myocardial - methods | Brain Stem - physiopathology | Myocardial Infarction - prevention & control | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Genetic Vectors | Myocardial Reperfusion Injury - prevention & control | Original
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