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Circulation, ISSN 0009-7322, 05/2009, Volume 119, Issue 18, pp. 2471 - 2479
Background-Aldosterone stimulates cardiac collagen synthesis. Circulating biomarkers of collagen turnover provide a useful tool for the assessment of cardiac... 
Heart failure | Myocardial infarction | Extracellular matrix | Biological markers | Collagen | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | BRAIN NATRIURETIC PEPTIDE | CARBOXYTERMINAL PROPEPTIDE | TURNOVER | myocardial infarction | III PROCOLLAGEN | collagen | MARKERS | TERMINAL PROPEPTIDE | SPIRONOLACTONE | I PROCOLLAGEN | biological markers | COLLAGEN DEGRADATION | PERIPHERAL VASCULAR DISEASE | ALDOSTERONE | extracellular matrix | Predictive Value of Tests | Myocardial Infarction - mortality | Prognosis | Death, Sudden, Cardiac - epidemiology | Humans | Middle Aged | Collagen Type III - metabolism | Extracellular Matrix - metabolism | Male | Ventricular Dysfunction, Left - mortality | Placebos | Female | Spironolactone - analogs & derivatives | Spironolactone - therapeutic use | Heart Failure - mortality | Biomarkers - metabolism | Collagen Type I - metabolism | Kaplan-Meier Estimate | Proportional Hazards Models | Heart Failure - metabolism | Myocardial Infarction - metabolism | Heart Failure - drug therapy | Mineralocorticoid Receptor Antagonists - therapeutic use | Ventricular Dysfunction, Left - metabolism | Myocardial Infarction - drug therapy | Ventricular Dysfunction, Left - drug therapy | Aged | Complications and side effects | Patient outcomes | Heart ventricle, Left | Physiological aspects | Research | Heart attack | Diseases | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2016, Volume 311, Issue 5, pp. H1160 - H1169
Cardiac metabolic remodeling is a central event during heart failure (HF) development following myocardial infarction (MI). It is well known that myocardial... 
Heart failure | Myocardial infarction | Remodelling | Branched chain amino acids | Mammalian target of rapamycin | heart failure | remodeling | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | myocardial infarction | HEART-FAILURE | MOUSE | branched chain amino acids | mammalian target of rapamycin | INHIBITION | METABOLISM | DISEASE | PERIPHERAL VASCULAR DISEASE | MICE | PROTEIN PHOSPHATASE 2CM | Protein Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Protein Phosphatase 2C - metabolism | Amino Acids, Branched-Chain - metabolism | Natriuretic Peptide, Brain - metabolism | Male | Ventricular Remodeling | Ligation | Myocardium - metabolism | Fatty Acids - metabolism | Heart Failure - etiology | Real-Time Polymerase Chain Reaction | In Situ Nick-End Labeling | Echocardiography | Signal Transduction | Ventricular Dysfunction, Left - etiology | Heart Failure - metabolism | Myocardial Infarction - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Ventricular Dysfunction, Left - metabolism | Myocardial Infarction - complications | Animals | Glucose - metabolism | Coronary Vessels - surgery | Mice | 3-Methyl-2-Oxobutanoate Dehydrogenase (Lipoamide) - metabolism | Physiological aspects | Amino acids | Health aspects | Heart attack | Heart attacks | Glucose | Metabolism | Fatty acids | Index Medicus
Journal Article
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 04/2008, Volume 86, Issue 5, pp. 1125 - 1131
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 09/2012, Volume 95, Issue 4, pp. 487 - 494
Innate mechanisms of inter-organ protection underlie the phenomenon of remote ischaemic preconditioning (RPc) in which episode(s) of ischaemia and reperfusion... 
Brain | Vagus nerve | Ischaemia/reperfusion injury | Preconditioning | Ischaemia | CARDIAC & CARDIOVASCULAR SYSTEMS | K-ATP CHANNELS | LIMB ISCHEMIA | MYOCARDIAL-ISCHEMIA | BRAIN-STEM | reperfusion injury | LENTIVIRAL VECTORS | CARDIOVASCULAR CONTROL | TRANSGENE EXPRESSION | RAT-HEART | AUTONOMIC NERVOUS-SYSTEM | Myocardial Infarction - genetics | Hindlimb | Receptors, G-Protein-Coupled - metabolism | Brain Stem - metabolism | Vagus Nerve - drug effects | Male | Receptors, Neuropeptide - metabolism | Drosophila Proteins - metabolism | Rhodopsin - metabolism | Recombinant Fusion Proteins - metabolism | Autonomic Fibers, Preganglionic - metabolism | Brain Stem - drug effects | Action Potentials | Myocardial Reperfusion Injury - pathology | Time Factors | Muscarinic Antagonists - pharmacology | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Adenoviridae - genetics | Lentivirus - genetics | Myocardial Infarction - physiopathology | Neural Pathways - physiopathology | Myocardial Reperfusion Injury - genetics | Autonomic Fibers, Preganglionic - drug effects | Disease Models, Animal | Heart - innervation | Muscle, Skeletal - blood supply | Transduction, Genetic | Vagus Nerve - metabolism | Constriction | Rats | Receptors, Neuropeptide - genetics | Atropine - pharmacology | Myocardium - pathology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Vagus Nerve - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Rhodopsin - genetics | Ischemic Preconditioning, Myocardial - methods | Brain Stem - physiopathology | Myocardial Infarction - prevention & control | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Genetic Vectors | Myocardial Reperfusion Injury - prevention & control | Index Medicus | Original
Journal Article