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JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2016, Volume 67, Issue 20, pp. 2395 - 2410
Abstract Hydroxy-methyl-glutaryl-coenzyme A (HMG-CoA) reductase inhibitors or statins are well tolerated, but associated with various statin-associated... 
Cardiovascular | Internal Medicine | rhabdomyolysis | skeletal muscle | interstitial lung disease | myopathy | C-REACTIVE PROTEIN | RANDOMIZED CONTROLLED-TRIALS | CARDIAC & CARDIOVASCULAR SYSTEMS | FORCE 2014 UPDATE | PRIMARY-PREVENTION TRIAL | INDUCED MUSCLE TOXICITY | INTERSTITIAL LUNG-DISEASE | CENTRAL-NERVOUS-SYSTEM | OF-THE-LITERATURE | CORONARY-HEART-DISEASE | TYPE-2 DIABETES-MELLITUS | Liver Function Tests | Sleep Wake Disorders - chemically induced | Hydroxymethylglutaryl-CoA Reductase Inhibitors - immunology | Myalgia - chemically induced | Humans | Hyperlipidemias - drug therapy | Risk Factors | Lung Diseases, Interstitial - chemically induced | Tendon Injuries - chemically induced | Necrosis - chemically induced | Brain - drug effects | Rupture - chemically induced | Hydroxymethylglutaryl-CoA Reductase Inhibitors - adverse effects | Stroke - chemically induced | Cognition Disorders - etiology | Depression - chemically induced | Rhabdomyolysis - chemically induced | Antibodies - blood | Diabetes Mellitus - chemically induced | Acute Kidney Injury - chemically induced | Muscular Diseases - chemically induced | Testosterone - blood | Autoimmune Diseases - chemically induced | Hyperlipidemias - complications | Viral antibodies | Complications and side effects | Enzymes | Thiols | Antibodies | Diabetes | Cardiology | Statins | Cardiovascular agents | Creatine kinase | Muscles | Creatine | Respiratory tract diseases | Studies | Musculoskeletal system | Genotype & phenotype | Mortality | Clinical trials | Lipids | Clinical medicine
Journal Article
Journal of Pain Research, ISSN 1178-7090, 06/2018, Volume 11, pp. 1181 - 1190
Purpose: Gabapentinoids are currently the mainstay of pharmacological treatments for patients with neuropathic pain. Little is known about the effects of this... 
Cytoskeleton | Cytotoxicity | Pregabalin | Gabapentin | Neuropathic pain | RAT | cytoskeleton | TOXICITY | RELEASE | CLINICAL NEUROLOGY | neuropathic pain | GENERAL-POPULATION | gabapentin | ANTIOXIDANT | GROWTH | pregabalin | CHRONIC BACK-PAIN | cytotoxicity | BRAIN | pregabablin
Journal Article
Lancet Haematology, The, ISSN 2352-3026, 2016, Volume 3, Issue 5, pp. e217 - e227
Summary Background Standard treatment for patients with primary CNS lymphoma remains to be defined. Active therapies are often associated with increased risk... 
Hematology, Oncology and Palliative Medicine | 1ST-LINE TREATMENT | RESPONSE CRITERIA | GUIDELINES | NON-HODGKINS-LYMPHOMA | FOLLOW-UP | CENTRAL-NERVOUS-SYSTEM | WHOLE-BRAIN RADIOTHERAPY | HIGH-DOSE CHEMOTHERAPY | STEM-CELL TRANSPLANTATION | HEMATOLOGY | Follow-Up Studies | Cytarabine - therapeutic use | Humans | Lymphoma, B-Cell - therapy | Middle Aged | Male | Comparative Effectiveness Research | Hyperglycemia - chemically induced | Radiotherapy, Adjuvant - adverse effects | Hematopoietic Stem Cell Transplantation - adverse effects | Lymphoma, B-Cell - diagnostic imaging | Transplantation Conditioning - methods | Rituximab - adverse effects | Induction Chemotherapy - methods | Anemia - chemically induced | Central Nervous System Neoplasms - therapy | Thrombocytopenia - chemically induced | Mucositis - chemically induced | Magnetic Resonance Imaging | Methotrexate - adverse effects | Dexamethasone - therapeutic use | Radiotherapy, Adjuvant - methods | Transplantation, Autologous - adverse effects | Heart Injuries - chemically induced | Italy | Hematopoietic Stem Cell Transplantation - methods | Central Nervous System Neoplasms - mortality | Germany | Thiotepa - adverse effects | Thiotepa - therapeutic use | Thrombosis - chemically induced | Intraocular Lymphoma - therapy | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Cytarabine - adverse effects | Methotrexate - therapeutic use | Infection - chemically induced | Rituximab - therapeutic use | Stroke - chemically induced | Optic Nerve Neoplasms - diagnostic imaging | Poisoning - epidemiology | Female | Remission Induction - methods | Transplantation Conditioning - adverse effects | Acute Kidney Injury - chemically induced | Neutropenia - chemically induced | Optic Nerve Neoplasms - therapy | Death, Sudden - epidemiology | Lymphoma, B-Cell - mortality | Intraocular Lymphoma - diagnostic imaging | Kaplan-Meier Estimate | Chemical and Drug Induced Liver Injury - epidemiology | Treatment Outcome | United Kingdom | Combined Modality Therapy - adverse effects | Switzerland | Induction Chemotherapy - adverse effects | Disease-Free Survival | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Central Nervous System Neoplasms - diagnostic imaging | Denmark | Combined Modality Therapy - methods | Neurotoxicity Syndromes - epidemiology | Gastrointestinal Diseases - chemically induced
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2010, Volume 30, Issue 24, pp. 8285 - 8295
Toll-like receptors play an important role in the innate immune response, although emerging evidence indicates their role in brain injury and... 
ETHANOL TREATMENT | ALZHEIMERS-DISEASE | IN-VIVO | TOLL-LIKE RECEPTORS | INJURY | NEURODEGENERATION | ENHANCES INFLAMMATORY MEDIATORS | ASTROGLIAL CELLS | ASTROCYTES | NEUROSCIENCES | CELL-DEATH | Tumor Necrosis Factor-alpha - metabolism | Rats, Wistar | Lymphocyte Antigen 96 - metabolism | Tumor Necrosis Factor-alpha - genetics | Caspase 3 - metabolism | Ethanol - administration & dosage | Green Fluorescent Proteins - genetics | Brain Injuries - metabolism | Interleukin-1beta - genetics | Lipopolysaccharides - adverse effects | Lipopolysaccharide Receptors - metabolism | Lymphocyte Antigen 96 - genetics | Ethanol - blood | Time Factors | Toll-Like Receptor 4 - deficiency | Cyclooxygenase 2 - genetics | Interleukin-1beta - metabolism | Encephalitis - metabolism | Statistics, Nonparametric | Brain Injuries - blood | Female | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Astrocytes - drug effects | Mice, Inbred C57BL | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Brain Injuries - chemically induced | Encephalitis - pathology | Rats | Mice, Knockout | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Toll-Like Receptor 4 - physiology | Animals | Microfilament Proteins | Central Nervous System Depressants - administration & dosage | Cyclooxygenase 2 - metabolism | Mice | Encephalitis - chemically induced | Central Nervous System Depressants - blood | Brain Injuries - pathology | Encephalitis - blood | Astrocytes - metabolism | Calcium-Binding Proteins - genetics
Journal Article
American Journal of Reproductive Immunology, ISSN 1046-7408, 05/2014, Volume 71, Issue 5, pp. 418 - 426
Problem Exposure to intrauterine inflammation, associated with preterm birth, has been linked to a devastating spectrum of neurobehavioral disorders.... 
mouse model | IL‐1 receptor antagonist | intrauterine inflammation | IL-1 receptor antagonist | Mouse model | Intrauterine inflammation | INTERLEUKIN-1 | REPRODUCTIVE BIOLOGY | ANTAGONIST | INFANTS | IMMUNOLOGY | CEREBRAL-PALSY | EXPRESSION | DAMAGE | Inflammation - chemically induced | Uterus - pathology | Inflammation - pathology | Premature Birth - genetics | Humans | Receptors, Interleukin-1 - genetics | Uterus - drug effects | Uterus - metabolism | Cerebral Cortex - pathology | Cerebral Cortex - metabolism | Lipopolysaccharides | Interleukin-1beta - metabolism | Premature Birth - chemically induced | Nitric Oxide Synthase Type I - antagonists & inhibitors | Female | Fetus | Cerebral Cortex - drug effects | Interleukin 1 Receptor Antagonist Protein - pharmacology | Infant, Newborn | Disease Models, Animal | Signal Transduction | Brain Injuries - chemically induced | Gene Expression Regulation | Brain Injuries - genetics | Brain Injuries - prevention & control | Receptors, Interleukin-1 - metabolism | Pregnancy | Premature Birth - pathology | Animals | Nitric Oxide Synthase Type I - metabolism | Inflammation - genetics | Inflammation - prevention & control | Mice | Nitric Oxide Synthase Type I - genetics | Receptors, Interleukin-1 - antagonists & inhibitors | Brain Injuries - pathology | Brain damage | Neurotoxicity | Premature birth | Nitric oxide | Index Medicus
Journal Article
PLOS ONE, ISSN 1932-6203, 04/2019, Volume 14, Issue 4, pp. e0214951 - e0214951
Exposure to intrauterine inflammation (IUI) is associated with short- and long-term adverse perinatal outcomes. However, little data exist on utilizing... 
BRAIN-INJURY | DOPPLER CHANGES | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | MYOCARDIAL PERFORMANCE INDEX | INSUFFICIENCY | PRETERM BIRTH | RISK | CEREBRAL-PALSY | THROMBOTIC VASCULOPATHY | DEFINING NORMAL VALUES | Inflammation - chemically induced | Inflammation - pathology | Lipopolysaccharides - toxicity | Placenta - physiology | Brain Injuries - chemically induced | Brain Injuries - metabolism | Placenta - injuries | Fetal Diseases - chemically induced | Placenta - metabolism | Pregnancy | Inflammation - metabolism | Animals | Placenta Diseases - chemically induced | Fetal Diseases - pathology | Placenta Diseases - pathology | Female | Mice | Placenta Diseases - metabolism | Brain Injuries - pathology | Fetal Diseases - metabolism | Phosphates | Brain | Fibrin | Dams | Pregnant women | Brain damage | Inflammation | Mitogens | Injuries | Enzyme-linked immunosorbent assay | Immunohistochemistry | Neuroimaging | Vimentin | Nuclear magnetic resonance--NMR | Calcium | Correlation coefficients | Doppler effect | Arteries | Lipopolysaccharides | Ultrasonic imaging | Head injuries | Uterus | Fibrinogen | Velocity measurement | Thromboembolism | Ultrasound | Public health | Velocimetry | Correlation coefficient | Medical imaging | Complications | Fetuses | Gynecology | Exposure | Obstetrics | Thrombosis | Medicine | Brain research | Placenta | Magnetic resonance imaging | Cardiac function | Brain injury | Histochemistry | Index Medicus | Nuclear magnetic resonance | NMR
Journal Article
Neuropharmacology, ISSN 0028-3908, 2011, Volume 61, Issue 5, pp. 975 - 980
Thrombin activates immunocompetent microglia and increases release of inflammatory cytokines under intracerebral hemorrhage (ICH) insults. Also, thrombin... 
Glycyrrhizin | Thrombin | High-mobility group box 1 | Intracerebral hemorrhage | MICROGLIAL ACTIVATION | CYTOKINE | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | GROUP BOX-1 PROTEIN | SLICE CULTURES | CHROMATIN PROTEIN | EDEMA | IN-VIVO | PROTEIN-KINASE PATHWAYS | PHARMACOLOGY & PHARMACY | Rats, Wistar | Brain Injuries - complications | Hemostatics - pharmacology | Male | Molecular Targeted Therapy | Anti-Inflammatory Agents - metabolism | Corpus Striatum - metabolism | Nitric Oxide - analysis | Cerebral Cortex - metabolism | Brain - metabolism | Glycyrrhizic Acid - pharmacology | Thrombin - pharmacology | Neostriatum - drug effects | Cattle | Anti-Inflammatory Agents - therapeutic use | Behavior, Animal - drug effects | Neostriatum - metabolism | Neurons - metabolism | Cell Death - drug effects | Cerebral Cortex - drug effects | Drug Evaluation, Preclinical | HMGB1 Protein - physiology | Neurons - drug effects | Corpus Striatum - pathology | HMGB1 Protein - antagonists & inhibitors | Cerebral Hemorrhage - chemically induced | Nitric Oxide - biosynthesis | Cerebral Hemorrhage - drug therapy | Brain Edema - metabolism | Anti-Inflammatory Agents - pharmacology | Tissue Culture Techniques | Brain Edema - pathology | Brain Injuries - chemically induced | Glycyrrhizic Acid - metabolism | Rats | Behavior, Animal - physiology | Collagenases - physiology | Rats, Sprague-Dawley | Brain - drug effects | Glycyrrhizic Acid - therapeutic use | Animals | Brain Edema - chemically induced | Cell Death - physiology | Corpus Striatum - drug effects | Brain Edema - drug therapy | Brain Injuries - pathology | Cerebral Hemorrhage - pathology | Methyl aspartate | Chromosomal proteins | Corticosteroids | Neurons | Nitric oxide | Dimethyl sulfoxide | Fluorescence | Universities and colleges | Protein kinases
Journal Article