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PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e49023
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is... 
INDUCED CELL-DEATH | ISCHEMIC TOLERANCE | MICROGLIAL TOXICITY | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | PERIVENTRICULAR LEUKOMALACIA | DEVELOPING OLIGODENDROCYTES | NECROSIS-FACTOR-ALPHA | EXCITOTOXIC INJURY | INDUCED BRAIN-INJURY | BACTERIAL-ENDOTOXIN SENSITIZES | Inflammation - chemically induced | Inflammation - pathology | Microglia - metabolism | Oligodendroglia - metabolism | Rats, Wistar | Apoptosis - drug effects | Caspase 3 - metabolism | Brain - metabolism | Inflammation - metabolism | Oligodendroglia - drug effects | Nerve Fibers, Myelinated - pathology | Hyperoxia - metabolism | Microglia - pathology | Animals, Newborn | Nerve Fibers, Myelinated - metabolism | Leukoencephalopathies - pathology | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cells, Cultured | Rats | Leukoencephalopathies - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Hyperoxia - pathology | Oligodendroglia - pathology | Animals | Lipopolysaccharides - pharmacology | Brain - pathology | Apoptosis - physiology | Immunohistochemistry | Brain | Cell death | Infants (Premature) | Inflammation | Research | Mitogens | Health aspects | Injuries | Neuroimaging | Neonates | Cell culture | Pediatrics | Transcription factors | Traumatic brain injury | Premature birth | Interleukin | Superoxide dismutase | Infants | Infections | Birth | Western blotting | Lipopolysaccharides | Head injuries | Newborn babies | Ischemia | Rodents | Oligodendrocytes | Oxygen | Pain perception | Cytokines | Maturation | Incubation | Mortality | Superoxide | Gene expression | Substantia alba | Microglia | Hospitals | Placenta | Magnetic resonance imaging | Lungs | Magnetic permeability | Hyperoxia | Interleukin 10 | Brain damage | Hypoxia | Oxygen tension | Brain injury | Apoptosis | pathology | Leukoencephalopathies | Sprague-Dawley | pharmacology | Caspase 3 | Cultured | Basic Medicine | chemically induced | Myelinated | physiology | drug effects | Cells | Newborn | Wistar | Medicinska grundvetenskaper | Oligodendroglia | metabolism | Nerve Fibers
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 12/2014, Volume 371, Issue 26, pp. 2457 - 2466
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2013, Volume 33, Issue 4, pp. 769 - 776
Journal Article
International Immunopharmacology, ISSN 1567-5769, 09/2019, Volume 74, p. 105658
The acute respiratory distress syndrome (ARDS), a devastating clinical syndrome, is one of the most severe complications of acute lung injury (ALI). Despite of... 
Acute lung injury | Inflammation | DL-3-n-butylphthalide | Nuclear factor (erythroid-derived 2)-like | Sirtuin 1 | BRAIN-INJURY | ENDOTOXIN | CELL-GROWTH | IMMUNOLOGY | SIRT1 | RESPIRATORY-DISTRESS-SYNDROME | L-3-N-BUTYLPHTHALIDE PROTECTS | PHARMACOLOGY & PHARMACY | MICE | NF-KAPPA-B | EPIDEMIOLOGY | Bronchoalveolar Lavage Fluid - immunology | Benzofurans - pharmacology | Male | Sirtuin 1 - genetics | NF-E2-Related Factor 2 - immunology | Lipopolysaccharides | Anti-Inflammatory Agents - therapeutic use | Pulmonary Edema - pathology | Acute Lung Injury - drug therapy | Bronchoalveolar Lavage Fluid - cytology | Cytokines - immunology | Lung - pathology | Benzofurans - therapeutic use | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Antioxidants - pharmacology | Acute Lung Injury - pathology | Pulmonary Edema - drug therapy | Mice, Knockout | Antioxidants - therapeutic use | Pulmonary Edema - chemically induced | Animals | Acute Lung Injury - chemically induced | Lung - drug effects | Pulmonary Edema - immunology | Sirtuin 1 - immunology | Acute Lung Injury - immunology | Lung - immunology | Respiratory therapy | Edema | Oxidative stress | Cytokines | Complications | Clinical trials | SIRT1 protein | Antioxidants | Signaling | Injury prevention | Respiratory distress syndrome | Lungs | Ventilation | Attenuation | Infiltration | Mice | Chemokines
Journal Article
Neuroscience, ISSN 0306-4522, 2013, Volume 240, pp. 27 - 38
Highlights ► Systemic LPS resulted in an impairment of behavioral performance in neonatal rats. ► Systemic LPS caused brain inflammation and brain damage in... 
Neurology | lipopolysaccharide | celecoxib | microglia | astrocyte | cyclooxygenase-2 | oligodendrocyte | Lipopolysaccharide | Astrocyte | Celecoxib | Cyclooxygenase-2 | Oligodendrocyte | Microglia | CYTOKINES | COX-2 | NITRIC-OXIDE SYNTHASE | PERIVENTRICULAR LEUKOMALACIA | VULNERABILITY | NECROSIS-FACTOR-ALPHA | INTERLEUKIN-6 | NEUROSCIENCES | LATE OLIGODENDROCYTE PROGENITORS | DYSFUNCTION | Pyrazoles - therapeutic use | Age Factors | O Antigens - metabolism | Male | Glial Fibrillary Acidic Protein - metabolism | Brain - metabolism | Leukoencephalopathies - drug therapy | Forelimb - physiopathology | Leukoencephalopathies - chemically induced | Encephalitis - drug therapy | Cyclooxygenase 2 Inhibitors - therapeutic use | Female | Encephalitis - complications | Microfilament Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Cell Death - drug effects | Disease Models, Animal | Animals, Newborn | Calcium-Binding Proteins - metabolism | In Situ Nick-End Labeling | Lipopolysaccharides - toxicity | Rats | Interleukin-1beta | Rats, Sprague-Dawley | Tumor Necrosis Factor-alpha | Brain - drug effects | Pregnancy | Animals | Sulfonamides - therapeutic use | Cyclooxygenase 2 - metabolism | Brain - pathology | Leukoencephalopathies - complications | Muscle Strength - drug effects | Encephalitis - chemically induced | COX-2 inhibitors | DNA polymerases | Brain damage | Biotin | Inflammation | Angiogenesis inhibitors | Intermediate filament proteins | Mitogens | Enzyme-linked immunosorbent assay
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e68796
Journal Article
Journal Article