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Seminars in immunopathology, ISSN 1863-2300, 2011, Volume 34, Issue 1, pp. 43 - 62
... of inflammatory human diseases. In particular, a proinflammatory role for fibrinogen has been reported in vascular wall disease, stroke, spinal cord injury, brain trauma... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | ALZHEIMERS-DISEASE | IMMUNOLOGY | PATHOLOGY | GAMMA-C-DOMAIN | BINDING-SITE | CENTRAL-NERVOUS-SYSTEM | MULTIPLE-SCLEROSIS LESIONS | CRYSTAL-STRUCTURE | BLOOD-BRAIN-BARRIER | INTEGRIN ALPHA(M)BETA | PROTEASE-ACTIVATED RECEPTORS | PERIPHERAL ARTERIAL-DISEASE | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
Acta neuropathologica, ISSN 1432-0533, 2015, Volume 130, Issue 1, pp. 21 - 34
Chronic traumatic encephalopathy (CTE) is a neurodegenerative disease associated with repetitive mild traumatic brain injury... 
Pathology | Neurosciences | Medicine & Public Health | Chronic traumatic encephalopathy | Alzheimer’s disease | Beta-amyloid | Tau | Neurodegenerative disorders | Dementia | HEAD-INJURY | BRAIN-INJURY | ALZHEIMERS-DISEASE | RISK | PATHOLOGY | NEUROSCIENCES | PROTEIN DEPOSITION | CLINICAL NEUROLOGY | DIFFUSE PLAQUES | APOE | Alzheimer's disease | AGE | Apolipoprotein E4 - genetics | Age Factors | Veterans | Brain Injury, Chronic - genetics | Humans | Middle Aged | tau Proteins - metabolism | Brain - metabolism | Amyloid beta-Peptides - metabolism | Aged, 80 and over | Plaque, Amyloid - etiology | War-Related Injuries - genetics | Adult | Severity of Illness Index | War-Related Injuries - epidemiology | Athletic Injuries - metabolism | Neurodegenerative Diseases - pathology | Brain Injury, Chronic - pathology | Plaque, Amyloid - pathology | War-Related Injuries - pathology | Comorbidity | Neurodegenerative Diseases - genetics | Athletic Injuries - genetics | Neurodegenerative Diseases - metabolism | Athletes | Brain Injury, Chronic - epidemiology | Athletic Injuries - epidemiology | War-Related Injuries - metabolism | Athletic Injuries - pathology | Brain - pathology | Plaque, Amyloid - metabolism | Aged | Brain Injury, Chronic - metabolism | Neurodegenerative Diseases - epidemiology | Cohort Studies | Brain | Nervous system diseases | Encephalopathy | Postconcussional syndrome | Diagnosis | Chronic brain injury | Neurophysiology
Journal Article
Brain (London, England : 1878), ISSN 1460-2156, 2013, Volume 136, Issue 1, pp. 28 - 42
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0159442
Millions of mild traumatic brain injuries (TBIs) occur every year in the United States, with many people subject to multiple head injuries that can lead to chronic behavioral dysfunction... 
MILD | ASTROCYTE ACTIVATION | OPTIC-NERVE | TRAUMATIC BRAIN-INJURY | BEHAVIORAL IMPAIRMENTS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | COGNITIVE IMPAIRMENT | SPINAL-CORD-INJURY | CONCUSSION | AXONAL INJURY | Optic Tract - physiopathology | Memory Disorders - physiopathology | Neurons - pathology | Optic Nerve - pathology | Corpus Callosum - metabolism | Brain Concussion - pathology | Brain Concussion - physiopathology | Corpus Callosum - physiopathology | Neuroglia - pathology | Optic Tract - pathology | Cerebral Cortex - pathology | Memory Disorders - metabolism | Head Injuries, Closed - metabolism | Male | Gliosis - physiopathology | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Head Injuries, Closed - physiopathology | Gliosis - pathology | Neurofilament Proteins - genetics | Pyramidal Tracts - physiopathology | Pyramidal Tracts - metabolism | Neurons - metabolism | Optic Nerve - physiopathology | Pyramidal Tracts - pathology | Memory Disorders - pathology | Disease Models, Animal | Psychomotor Performance | Gene Expression | Head Injuries, Closed - pathology | Gliosis - metabolism | Mice, Inbred C57BL | Hippocampus - pathology | Optic Tract - metabolism | Hippocampus - metabolism | Animals | Corpus Callosum - pathology | Neuroglia - metabolism | Mice | Neurofilament Proteins - metabolism | Hippocampus - physiopathology | Brain Concussion - metabolism | Optic Nerve - metabolism | Complications and side effects | Care and treatment | Head injuries | Research | Memory, Disorders of | Risk factors | Intervals | Brain | Visual cortex | Traumatic brain injury | Gait | Neuronal-glial interactions | Visual system | Histopathology | Neurodegeneration | Neurofilament protein | Physiology | Acute effects | Stains | Injuries | Optic nerve | Motor ability | Abnormalities | Inflammation | Trauma | Corpus callosum | Optic tract | Medicine | Axons | Injury prevention | Brain research | Gliosis | Chronic traumatic encephalopathy | Cell death | Spinal cord injuries | Laboratory animals | Human behavior | Apoptosis | Animal cognition
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 9, pp. 2853 - 2858
Journal Article
Journal Article
Journal Article
Nature medicine, ISSN 1546-170X, 2017, Volume 23, Issue 6, pp. 733 - 741
.... Here we examined how vessel tone adapts to the loss of neuron-derived monoamines after spinal cord injury (SCI) in rats... 
MEDICINE, RESEARCH & EXPERIMENTAL | PIAL VESSELS | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRACE AMINES | SYNAPTIC-TRANSMISSION | HYPOXIA | CELL BIOLOGY | CENTRAL-NERVOUS-SYSTEM | LOCOMOTOR FUNCTION | CEREBRAL-ARTERIES | RECEPTORS | BRAIN | Capillaries - pathology | Serotonin 5-HT1 Receptor Antagonists - pharmacology | Receptors, Adrenergic, alpha-2 - metabolism | Transcriptome | Capillaries - drug effects | RNA, Messenger - metabolism | Oxygen - metabolism | Hypoxia - metabolism | Receptors, Serotonin, 5-HT1 - metabolism | Spinal Cord Injuries - pathology | Receptor, Serotonin, 5-HT1B - metabolism | Aromatic-L-Amino-Acid Decarboxylases - metabolism | Tyramine - metabolism | Capillaries - metabolism | Locomotion - drug effects | Injections, Spinal | Capillaries - physiopathology | Spinal Cord Injuries - metabolism | Pericytes - metabolism | Vasoconstriction | Rats | Microscopy, Interference | Oxygen Inhalation Therapy | Microscopy, Confocal | Animals | Norepinephrine - metabolism | Tryptamines - metabolism | Serotonin - metabolism | Spinal Cord Injuries - physiopathology | Biogenic Monoamines - metabolism | Locomotion - physiology | Enzymes | Care and treatment | Analysis | Tryptophan | Spinal cord injuries | Dosage and administration | Drug therapy | Health aspects | Noradrenaline | Blood flow | spinal cord injury | AADC | hypoxia | pericyte | capillary | locomotion | Trace amines | neurovascular coupling | 5-HT1B receptor | motoneurons | ischemia
Journal Article