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PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0159442
Millions of mild traumatic brain injuries (TBIs) occur every year in the United States, with many people subject to multiple head injuries that can lead to chronic behavioral dysfunction... 
MILD | ASTROCYTE ACTIVATION | OPTIC-NERVE | TRAUMATIC BRAIN-INJURY | BEHAVIORAL IMPAIRMENTS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | COGNITIVE IMPAIRMENT | SPINAL-CORD-INJURY | CONCUSSION | AXONAL INJURY | Optic Tract - physiopathology | Memory Disorders - physiopathology | Neurons - pathology | Optic Nerve - pathology | Corpus Callosum - metabolism | Brain Concussion - pathology | Brain Concussion - physiopathology | Corpus Callosum - physiopathology | Neuroglia - pathology | Optic Tract - pathology | Cerebral Cortex - pathology | Memory Disorders - metabolism | Head Injuries, Closed - metabolism | Male | Gliosis - physiopathology | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Head Injuries, Closed - physiopathology | Gliosis - pathology | Neurofilament Proteins - genetics | Pyramidal Tracts - physiopathology | Pyramidal Tracts - metabolism | Neurons - metabolism | Optic Nerve - physiopathology | Pyramidal Tracts - pathology | Memory Disorders - pathology | Disease Models, Animal | Psychomotor Performance | Gene Expression | Head Injuries, Closed - pathology | Gliosis - metabolism | Mice, Inbred C57BL | Hippocampus - pathology | Optic Tract - metabolism | Hippocampus - metabolism | Animals | Corpus Callosum - pathology | Neuroglia - metabolism | Mice | Neurofilament Proteins - metabolism | Hippocampus - physiopathology | Brain Concussion - metabolism | Optic Nerve - metabolism | Complications and side effects | Care and treatment | Head injuries | Research | Memory, Disorders of | Risk factors | Intervals | Brain | Visual cortex | Traumatic brain injury | Gait | Neuronal-glial interactions | Visual system | Histopathology | Neurodegeneration | Neurofilament protein | Physiology | Acute effects | Stains | Injuries | Optic nerve | Motor ability | Abnormalities | Inflammation | Trauma | Corpus callosum | Optic tract | Medicine | Axons | Injury prevention | Brain research | Gliosis | Chronic traumatic encephalopathy | Cell death | Spinal cord injuries | Laboratory animals | Human behavior | Apoptosis | Animal cognition
Journal Article
Kidney international, ISSN 0085-2538, 2012, Volume 81, Issue 10, pp. 942 - 948
Despite the availability of renal replacement therapy, acute kidney injury (AKI) is associated with high mortality and morbidity... 
acute kidney injury | extrarenal complications | inflammation | pathophysiology | Inflammation | Pathophysiology | Extrarenal complications | Acute kidney injury | ISCHEMIA/REPERFUSION INJURY | MECHANICAL VENTILATION | ACUTE LUNG INJURY | ACUTE-RENAL-FAILURE | BILATERAL-NEPHRECTOMY | INTENSIVE-CARE | RESPIRATORY-DISTRESS-SYNDROME | ISCHEMIA-REPERFUSION INJURY | UROLOGY & NEPHROLOGY | CONGESTIVE-HEART-FAILURE | CRITICALLY-ILL PATIENTS | Brain Diseases - metabolism | Liver - pathology | Lung Diseases - metabolism | Reactive Oxygen Species - metabolism | Heart Diseases - metabolism | Kidney - pathology | Oxidative Stress | Humans | Liver Diseases - pathology | Liver - physiopathology | Brain Diseases - physiopathology | Lung Diseases - etiology | Brain - metabolism | Kidney - metabolism | Acute Kidney Injury - complications | Liver Diseases - etiology | Myocardium - metabolism | Inflammation Mediators - metabolism | Lung - metabolism | Brain Diseases - etiology | Kidney - physiopathology | Lung - pathology | Signal Transduction | Acute Kidney Injury - pathology | Heart Diseases - physiopathology | Brain - physiopathology | Liver - metabolism | Myocardium - pathology | Lung - physiopathology | Acute Kidney Injury - physiopathology | Heart Diseases - etiology | Animals | Brain Diseases - pathology | Brain - pathology | Lung Diseases - physiopathology | Liver Diseases - physiopathology | Lung Diseases - pathology | Acute Kidney Injury - metabolism | Liver Diseases - metabolism | Heart Diseases - pathology | Apoptosis
Journal Article
Annals of neurology, ISSN 0364-5134, 2015, Volume 78, Issue 1, pp. 68 - 76
Journal Article
Science translational medicine, ISSN 1946-6242, 2012, Volume 4, Issue 134, pp. 134ra60 - 134ra60
Journal Article
Journal Article
Molecular neurobiology, ISSN 1559-1182, 2008, Volume 38, Issue 1, pp. 78 - 100
...Calpain-Mediated Signaling Mechanisms in Neuronal Injury and Neurodegeneration P. S. Vosler & C. S. Brennan & J. Chen Received: 23 May 2008 /Accepted: 17 July... 
Multiple sclerosis | Calcium | Amylotrophic lateral sclerosis | Neurobiology | Calpain | Prion-related encephalopathy | Huntington’s | Alzheimer’s | Cell Biology | Parkinson’s | Life Sciences | Neurology | Cerebral ischemia | Excitotoxicity | Neurodegenerative disease | Parkinson's | Alzheimer's | Huntington's | APOPTOSIS-INDUCING FACTOR | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | CONSTITUTIVELY ACTIVE CALCINEURIN | neurodegenerative disease | calcium | SELECTIVE STRIATAL DEGENERATION | multiple sclerosis | FOCAL CEREBRAL-ISCHEMIA | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | prion-related encephalopathy | AMYOTROPHIC-LATERAL-SCLEROSIS | AMYLOID PRECURSOR PROTEIN | D-ASPARTATE RECEPTOR | NEUROSCIENCES | calpain | cerebral ischemia | amylotrophic lateral sclerosis | excitotoxicity | CYCLIN-DEPENDENT KINASE-5 | Prion Diseases - physiopathology | Alzheimer Disease - physiopathology | Parkinson Disease - pathology | Trauma, Nervous System - pathology | Calpain - metabolism | Neurodegenerative Diseases - pathology | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Huntington Disease - pathology | Brain Ischemia - physiopathology | Prion Diseases - pathology | Calpain - genetics | Parkinson Disease - physiopathology | Calpain - chemistry | Alzheimer Disease - pathology | Multiple Sclerosis - physiopathology | Amyotrophic Lateral Sclerosis - pathology | Trauma, Nervous System - physiopathology | Animals | Neurodegenerative Diseases - physiopathology | Brain Ischemia - pathology | Multiple Sclerosis - pathology | Signal Transduction - physiology | Huntington Disease - physiopathology | Brain damage | Proteases | Neurodegeneration | Cytoplasm
Journal Article