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Journal of Neuroinflammation, ISSN 1742-2094, 01/2017, Volume 14, Issue 1, p. 10
Journal Article
FASEB Journal, ISSN 0892-6638, 01/2018, Volume 32, Issue 1, pp. 512 - 528
Neuronal inflammation is the characteristic pathologic change of acute neurologic impairment and chronic traumatic encephalopathy after traumatic brain injury... 
Extracellular vesicles | PDE4B | Polarization | MTOR signaling | Glia cells | APOPTOSIS | extracellular vesicles | BIOCHEMISTRY & MOLECULAR BIOLOGY | AKT | mTOR signaling | glia cells | CELL BIOLOGY | MESENCHYMAL STROMAL CELLS | IN-VITRO | PHOSPHODIESTERASE 4B | PATHWAY | NEUROINFLAMMATION | DISEASE | BIOLOGY | polarization | Exosomes - metabolism | Microglia - metabolism | Neurons - pathology | TOR Serine-Threonine Kinases - metabolism | Exosomes - genetics | Male | MicroRNAs - metabolism | Gene Regulatory Networks | Brain Injuries, Traumatic - genetics | Inflammation - metabolism | Microglia - pathology | Neurons - metabolism | Cyclic Nucleotide Phosphodiesterases, Type 4 - metabolism | Disease Models, Animal | Neuronal Outgrowth - physiology | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Cyclic Nucleotide Phosphodiesterases, Type 4 - genetics | Neuronal Outgrowth - genetics | Animals | Brain Injuries, Traumatic - metabolism | Brain Injuries, Traumatic - pathology | Microglia - classification | Inflammation - genetics | Inflammation - prevention & control | Mice | MicroRNAs - genetics | TOR protein | Brain | Traumatic brain injury | Encyclopedias | Inflammatory response | Genomes | Exosomes | Proteins | Signal transduction | Head injuries | Transfection | Encephalopathy | Inhibition | Neurons | Axonogenesis | MiRNA | RhoA protein | Inflammation | Rapamycin | Gene expression | Chromosome 3 | Microglia | Neurological diseases | Signaling | DNA microarrays | Tau protein | Regulatory mechanisms (biology)
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2016, Volume 22, Issue 11, pp. 1335 - 1341
At low levels, carbon monoxide (CO) has physiological roles as a second messenger and neuromodulator(1,2). Here we assess the effects of CO in a mouse model of... 
MEDICINE, RESEARCH & EXPERIMENTAL | ADULT BRAIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | CELL BIOLOGY | Immunohistochemistry | Reactive Oxygen Species - metabolism | Pericytes - drug effects | Humans | Pericytes - cytology | Nitric Oxide Synthase - antagonists & inhibitors | Male | Neurons - cytology | Carbon Monoxide - metabolism | Brain - metabolism | Human Embryonic Stem Cells - drug effects | Behavior, Animal - drug effects | Neurogenesis - drug effects | Neurons - metabolism | Nuclear Proteins - drug effects | Bromodeoxyuridine - metabolism | Neurons - drug effects | Disease Models, Animal | Human Embryonic Stem Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Mice, Inbred C57BL | Pericytes - metabolism | Cells, Cultured | Neural Stem Cells - drug effects | Nuclear Proteins - metabolism | Maze Learning - drug effects | Nerve Tissue Proteins - drug effects | Blotting, Western | Brain - drug effects | Nerve Tissue Proteins - metabolism | Animals | Brain Injuries, Traumatic - metabolism | Cell Differentiation - drug effects | Cell Proliferation - drug effects | Mice | Nitric Oxide Synthase Type I - genetics | In Vitro Techniques | Neural Stem Cells - metabolism | Organometallic Compounds - pharmacology | Brain | Care and treatment | Carbon monoxide | Health aspects | Neurogenesis | Injuries
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 04/2016, Volume 23, Issue 4, pp. 654 - 668
Journal Article