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Critical care (London, England), ISSN 1364-8535, 2016, Volume 20, Issue 1, p. 277
Journal Article
Nature medicine, ISSN 1546-170X, 2010, Volume 16, Issue 12, pp. 1439 - 1443
.... Here we show that cerebral ischemia induces the interaction of nNOS with postsynaptic density protein-95 (PSD-95... 
MEDICINE, RESEARCH & EXPERIMENTAL | BRAIN-INJURY | NITRIC-OXIDE SYNTHASE | PDZ DOMAIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUBCELLULAR-LOCALIZATION | NEUROTOXICITY | MICE LACKING | NEUROPROTECTION | CELL BIOLOGY | EXCITOTOXICITY | PROTEIN INTERACTIONS | ANTAGONISTS | Disks Large Homolog 4 Protein | Neuroprotective Agents - therapeutic use | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Immunoprecipitation | Oligonucleotides, Antisense - metabolism | Receptors, N-Methyl-D-Aspartate - metabolism | Brain Ischemia - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Neuroprotective Agents - metabolism | Neuroprotective Agents - pharmacology | Oligonucleotides, Antisense - therapeutic use | Infarction, Middle Cerebral Artery - drug therapy | Membrane Proteins - metabolism | Molecular Structure | Cell Death - drug effects | Brain Ischemia - complications | Oligonucleotides, Antisense - pharmacology | Rats | Stroke - drug therapy | Glutamic Acid | Microscopy, Confocal | Stroke - etiology | Animals | Guanylate Kinases | Analysis of Variance | Cell Death - physiology | Signal Transduction - drug effects | Brain Ischemia - drug therapy | Nitric Oxide Synthase Type I - metabolism | Signal Transduction - physiology | Biological Transport - physiology | Mice | Nitric Oxide Synthase Type I - chemistry | Cerebral ischemia | Care and treatment | Nitric oxide | Physiological aspects | Genetic aspects | Research | Risk factors | Nerve proteins | Enzymes | Stroke | Biomedical research | Cellular biology | Neurons | Pharmacology
Journal Article
Neuroradiology, ISSN 0028-3940, 06/2015, Volume 57, Issue 9, pp. 897 - 902
Journal Article
Molecular neurobiology, ISSN 1559-1182, 2014, Volume 52, Issue 3, pp. 1430 - 1439
Oxidative stress is well known to play a pivotal role in cerebral ischemia–reperfusion injury. The nuclear factor erythroid-2-related factor 2... 
Antioxidants | MCAO | Neurology | Neurosciences | Biomedicine | Nrf2 | Neurobiology | Oxygen glucose deprivation | HO-1 | 11-Keto-β-boswellic acid | Cell Biology | BOSWELLIC ACIDS | ACTIVATING NRF2 | ARTERY OCCLUSION | ASTROCYTES | NEUROSCIENCES | 11-Keto-beta-boswellic acid | PROTECTS | IN-VITRO | NEURONS | HEME OXYGENASE-1 | RAT-BRAIN | MOLECULAR-MECHANISMS | NF-E2-Related Factor 2 - physiology | Triterpenes - toxicity | Reperfusion Injury - etiology | Triterpenes - pharmacology | Apoptosis - drug effects | Male | Infarction, Middle Cerebral Artery - metabolism | Cell Hypoxia | Boswellia - chemistry | Malondialdehyde - analysis | Neuroprotective Agents - pharmacology | Heme Oxygenase (Decyclizing) - biosynthesis | Nerve Tissue Proteins - biosynthesis | NF-E2-Related Factor 2 - genetics | Infarction, Middle Cerebral Artery - drug therapy | Phytotherapy | Drug Evaluation, Preclinical | Heme Oxygenase (Decyclizing) - genetics | Reperfusion Injury - metabolism | Superoxide Dismutase - metabolism | Astrocytes - drug effects | Heme Oxygenase (Decyclizing) - physiology | Nerve Tissue Proteins - physiology | Cells, Cultured | Rats | Neuroprotective Agents - toxicity | Glucose - pharmacology | Antioxidants - pharmacology | Infarction, Middle Cerebral Artery - pathology | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Antioxidants - therapeutic use | Brain Damage, Chronic - etiology | Animals | Reperfusion Injury - prevention & control | Signal Transduction - drug effects | Brain - pathology | Signal Transduction - physiology | Oxidative Stress - drug effects | NF-E2-Related Factor 2 - biosynthesis | Brain Damage, Chronic - prevention & control | Astrocytes - metabolism
Journal Article
Journal Article
Stroke (1970), ISSN 0039-2499, 05/2009, Volume 40, Issue 5, pp. 1590 - 1596
Journal Article