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Journal of Clinical Oncology, ISSN 0732-183X, 02/2012, Volume 30, Issue 6, pp. 593 - 599
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2013, Volume 15, Issue 7, pp. 807 - 817
In a significant fraction of breast cancer patients, distant metastases emerge after years or even decades of latency. How disseminated tumour cells (DTCs) are... 
CANCER-CELLS | METASTATIC NICHE | EPITHELIAL-CELLS | HEMATOPOIETIC STEM-CELLS | ENDOTHELIAL-CELLS | SELF-RENEWAL | 3-DIMENSIONAL CULTURE | EXTRACELLULAR-MATRIX | ANGIOCRINE FACTORS | BASEMENT-MEMBRANE | CELL BIOLOGY | Neoplasm, Residual - pathology | Neoplasm, Residual - metabolism | Humans | Lung Neoplasms - metabolism | Neovascularization, Pathologic | Tumor Microenvironment | Neoplasm, Residual - blood supply | Brain Neoplasms - blood supply | Brain Neoplasms - metabolism | Breast Neoplasms - metabolism | Brain Neoplasms - secondary | Lung Neoplasms - secondary | Pericytes - pathology | Female | Tumor Cells, Cultured | Lung Neoplasms - blood supply | Pericytes - metabolism | Breast Neoplasms - blood supply | Cell Adhesion Molecules - metabolism | Zebrafish - growth & development | Bone Marrow Neoplasms - secondary | Animals | Bone Marrow Neoplasms - metabolism | Breast Neoplasms - pathology | Endothelium, Vascular - metabolism | Zebrafish - metabolism | Fluorescent Antibody Technique | Endothelium, Vascular - pathology | Stem Cell Niche - physiology | Mice | Thrombospondin 1 - metabolism | Bone Marrow Neoplasms - blood supply | Transforming Growth Factor beta - metabolism | Physiological aspects | Bone morphogenetic proteins | Genetic aspects | Breast cancer | Research | Transforming growth factors | Index Medicus
Journal Article
American Journal of Pathology, ISSN 0002-9440, 2010, Volume 177, Issue 4, pp. 1647 - 1656
Phosphatase and tensin homolog (PTEN) is a key modulator of trastuzumab sensitivity in HER2-overexpressing breast cancer. Because PTEN opposes the downstream... 
ADJUVANT CHEMOTHERAPY | CELLS | INHIBITION | THERAPY | HUMAN-BREAST-CANCER | MUTATIONS | PATHOLOGY | RECEPTORS | TRASTUZUMAB RESISTANCE | EXPRESSION | CARCINOMA | Lung Neoplasms - drug therapy | Receptors, Estrogen - metabolism | Receptor, ErbB-2 - genetics | Lung Neoplasms - mortality | Humans | Middle Aged | Bone Neoplasms - secondary | Antibodies, Monoclonal - therapeutic use | Immunoenzyme Techniques | Receptors, Progesterone - metabolism | Young Adult | Carcinoma, Ductal, Breast - drug therapy | Brain Neoplasms - mortality | Liver Neoplasms - secondary | Proto-Oncogene Proteins c-akt - metabolism | Carcinoma, Ductal, Breast - metabolism | PTEN Phosphohydrolase - genetics | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Survival Rate | Lymphatic Metastasis | Brain Neoplasms - drug therapy | Mutation - genetics | Breast Neoplasms - drug therapy | Class I Phosphatidylinositol 3-Kinases | Carcinoma, Lobular - metabolism | Trastuzumab | Bone Neoplasms - mortality | Phosphorylation | Prognosis | Receptor, ErbB-2 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Liver Neoplasms - mortality | Carcinoma, Ductal, Breast - mortality | Carcinoma, Lobular - mortality | Breast Neoplasms - metabolism | Antibodies, Monoclonal, Humanized | Brain Neoplasms - secondary | In Situ Hybridization | Lung Neoplasms - secondary | Adult | Female | Bone Neoplasms - drug therapy | Neoplasm Invasiveness | Liver Neoplasms - drug therapy | PTEN Phosphohydrolase - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - mortality | Aged | Carcinoma, Lobular - drug therapy | Index Medicus | Abridged Index Medicus | Regular
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 11/2015, Volume 6, Issue 1, pp. 8760 - 8760
Circulating tumour DNA analysis can be used to track tumour burden and analyse cancer genomes non-invasively but the extent to which it represents metastatic... 
HETEROGENEITY | ACTIVATION | VARIANTS | PATHWAY | MULTIDISCIPLINARY SCIENCES | KINASE | ACQUIRED-RESISTANCE | SEQUENCING DATA | Carcinoma, Ductal, Breast - genetics | Receptors, Estrogen - metabolism | Humans | Receptor, ErbB-2 - metabolism | Clonal Evolution - genetics | Tamoxifen - administration & dosage | Case-Control Studies | Breast Neoplasms - metabolism | Brain Neoplasms - secondary | Carcinoma, Ductal, Breast - drug therapy | Neoplasm Metastasis | Lung Neoplasms - secondary | Adult | Carcinoma, Ductal, Breast - pathology | Female | Quinazolines - administration & dosage | Liver Neoplasms - secondary | Carcinoma, Ductal, Breast - metabolism | Lung Neoplasms - genetics | Liver Neoplasms - genetics | Deoxycytidine - administration & dosage | Brain Neoplasms - genetics | Trastuzumab - administration & dosage | Breast Neoplasms - drug therapy | Sequence Analysis, DNA | Spinal Neoplasms - secondary | Breast Neoplasms - genetics | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Breast Neoplasms - pathology | Spinal Neoplasms - genetics | Bayes Theorem | DNA, Neoplasm - genetics | Mutation | Deoxycytidine - analogs & derivatives | Biological evolution | Breast cancer | Genomes | Metastasis | ErbB-2 protein | Patients | Metastases | Heterogeneity | Biopsy | Breast | Evolution | Deoxyribonucleic acid--DNA | Cancer | Tumors | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 12/2011, Volume 71, Issue 24, pp. 7360 - 7365
Journal Article
Breast Cancer Research, ISSN 1465-5411, 09/2011, Volume 13, Issue 5, pp. R87 - R87
Introduction: Some molecular subtypes of breast cancer have preferential sites of distant relapse. The protein expression pattern of the primary tumor may... 
MOLECULAR SUBTYPES | SURVIVAL | RELAPSE | CELLS | ONCOLOGY | ERBB2 | BRAIN METASTASES | MARKERS | PATTERNS | CARCINOMA | TUMORS | Nestin | Receptors, Estrogen - metabolism | Cadherins - metabolism | Follow-Up Studies | Humans | Lung Neoplasms - metabolism | Glycoproteins - metabolism | Receptor, ErbB-2 - metabolism | Bone Neoplasms - secondary | Proteins - analysis | Brain Neoplasms - metabolism | Antigens, CD - metabolism | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Receptors, Progesterone - metabolism | Brain Neoplasms - secondary | Receptor, Epidermal Growth Factor - metabolism | Peptides - metabolism | Lung Neoplasms - secondary | Female | Liver Neoplasms - secondary | Snail Family Transcription Factors | AC133 Antigen | Skin Neoplasms - metabolism | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Proteins - metabolism | Breast Neoplasms - pathology | Cyclooxygenase 2 - metabolism | Finland | Liver Neoplasms - metabolism | Skin Neoplasms - secondary | Keratin-5 - metabolism | Intermediate Filament Proteins - metabolism | Cohort Studies | Immunohistochemistry | Care and treatment | Estrogen | Development and progression | Breast cancer | Research | Gene expression | Keratin | Epidermal growth factor | Genetic aspects | Diagnosis | Progesterone | Tumor proteins | Index Medicus
Journal Article
BMC Cancer, ISSN 1471-2407, 11/2013, Volume 13, Issue 1, pp. 537 - 537
Background: Triple Negative subset of (TN) Breast Cancers (BC), a close associate of the basal-like subtype (with limited discordance) is an aggressive form of... 
Breast cancer | Microarray | Wnt | FFPE | Triple negative | GENE-EXPRESSION SIGNATURE | ANDROGEN RECEPTOR | REGULATES EXPRESSION | SUBTYPES | T-CELL-FACTOR | BETA-CATENIN | MESENCHYMAL TRANSITION | ONCOLOGY | PATHWAY | COLORECTAL-CANCER | BASAL | RNA, Small Interfering - genetics | Cell Proliferation | Prognosis | Follow-Up Studies | Oligonucleotide Array Sequence Analysis | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Bone Neoplasms - secondary | Gene Expression Profiling | Wnt Proteins - metabolism | Brain Neoplasms - metabolism | Immunoenzyme Techniques | Bone Neoplasms - metabolism | Neoplasm Recurrence, Local - pathology | Paraffin Embedding | Brain Neoplasms - secondary | Wnt Proteins - genetics | Lung Neoplasms - secondary | Triple Negative Breast Neoplasms - pathology | Biomarkers, Tumor - metabolism | Female | Bone Neoplasms - genetics | Tumor Cells, Cultured | Real-Time Polymerase Chain Reaction | Lung Neoplasms - genetics | Neoplasm Recurrence, Local - metabolism | Signal Transduction | RNA, Messenger - genetics | Brain Neoplasms - genetics | Survival Rate | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | Blotting, Western | beta Catenin - genetics | Triple Negative Breast Neoplasms - genetics | Triple Negative Breast Neoplasms - metabolism | beta Catenin - antagonists & inhibitors | Neoplasm Recurrence, Local - genetics | Biomarkers, Tumor - genetics | Neoplasm Staging | Cell Movement | Cohort Studies | Research | Metastasis | Biological markers | Analysis | Oncology, Experimental | Cancer | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2016, Volume 533, Issue 7604, pp. 493 - 498
Brain metastasis represents a substantial source of morbidity and mortality in various cancers, and is characterized by high resistance to chemotherapy. Here... 
SURVIVAL | CADHERIN SUPERFAMILY | EXPRESSION ANALYSIS | CONNEXIN | RECOGNITION | PATHWAY | BREAST-CANCER METASTASIS | MULTIDISCIPLINARY SCIENCES | GENES | CLASSIFICATION | TUMOR-CELLS | Meclofenamic Acid - pharmacology | Meclofenamic Acid - therapeutic use | Nucleotides, Cyclic - metabolism | Gap Junctions - metabolism | Benzopyrans - therapeutic use | Cadherins - metabolism | Coculture Techniques | Humans | Brain Neoplasms - pathology | Drug Resistance, Neoplasm | Lung Neoplasms - pathology | NF-kappa B - metabolism | Brain Neoplasms - metabolism | Brain Neoplasms - secondary | STAT1 Transcription Factor - metabolism | Benzamides - therapeutic use | Female | Benzamides - pharmacology | Membrane Proteins - metabolism | Benzopyrans - pharmacology | Astrocytes - cytology | Astrocytes - drug effects | Interferon-alpha - metabolism | Connexin 43 - metabolism | Tumor Necrosis Factors - metabolism | Brain Neoplasms - drug therapy | Immunity, Innate | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - pathology | Paracrine Communication - drug effects | Cell Line, Tumor | Mice | Gap Junctions - drug effects | Astrocytes - metabolism | Astrocytes | Brain cancer | Physiological aspects | Metastasis | Observations | Adenylic acid | Health aspects | Proteins | Brain | Cytokines | Cells | Tumors | Cancer | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 01/2017, Volume 541, Issue 7635, pp. 41 - 45
The fact that the identity of the cells that initiate metastasis in most human cancers is unknown hampers the development of antimetastatic therapies. Here we... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER CELLS | METABOLISM | MULTIDISCIPLINARY SCIENCES | STEM-CELL | CHEMORESISTANCE | BRAIN-METASTASIS | STROMAL CELLS | TUMOR-GROWTH | CARCINOMA | PROGRESSION | Palmitic Acid - metabolism | Palmitic Acid - pharmacology | CD36 Antigens - genetics | Cell Proliferation | Mouth Neoplasms - drug therapy | Prognosis | Neoplastic Stem Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Transcriptome | Diet, High-Fat - adverse effects | Male | Mouth Neoplasms - metabolism | Antibodies, Neutralizing - immunology | Mouth Neoplasms - diagnosis | CD36 Antigens - metabolism | Neoplasm Metastasis - drug therapy | Neoplastic Stem Cells - metabolism | Neoplasm Metastasis - prevention & control | Antibodies, Neutralizing - therapeutic use | Lymphatic Metastasis - pathology | Hyaluronan Receptors - metabolism | Neoplastic Stem Cells - pathology | Female | Lipid Metabolism - genetics | Disease Models, Animal | Palmitic Acid - administration & dosage | CD36 Antigens - immunology | Antibodies, Neutralizing - pharmacology | Penetrance | Xenograft Model Antitumor Assays | Neoplasm Metastasis - genetics | Animals | Neoplasm Metastasis - pathology | CD36 Antigens - antagonists & inhibitors | Lymphatic Metastasis - genetics | Mouth Neoplasms - pathology | Mice | Cancer cells | Genetic aspects | Metastasis | Lipid metabolism | Gene expression | Health aspects | Fatty acids | Oral cancer | Lipids | Metabolism | Cells | Tumors | Index Medicus
Journal Article