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Cancer Cell, ISSN 1535-6108, 05/2012, Volume 21, Issue 5, pp. 601 - 613
The proto-oncogene is mis-expressed in various types of human brain tumors. To clarify how developmental and regional differences influence transformation, we... 
PROGENITORS | SOX9 EXPRESSION | ONCOLOGY | SONIC HEDGEHOG | MOUSE MODEL | EMBRYONIC LETHALITY | PROLIFERATION | GENERATION | MUTATIONS | PEDIATRIC MEDULLOBLASTOMA | ASTROCYTES | CELL BIOLOGY | Oncogene Proteins - genetics | Cell Proliferation | Humans | Zinc Finger Protein Gli2 | Brain Stem - metabolism | Hedgehog Proteins - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Glioma - pathology | Medulloblastoma - pathology | Cell Differentiation | N-Myc Proto-Oncogene Protein | Cerebellar Neoplasms - pathology | Prosencephalon - metabolism | Cerebellar Neoplasms - metabolism | Biomarkers - metabolism | Neuroectodermal Tumors, Primitive - metabolism | SOX9 Transcription Factor - metabolism | Transduction, Genetic | Signal Transduction | Oncogene Proteins - metabolism | Brain Neoplasms - genetics | Mice, Transgenic | Medulloblastoma - metabolism | Gestational Age | Cell Lineage | Mice, Nude | Brain Stem - embryology | Mice | Mutation | Brain Neoplasms - pathology | Brain Neoplasms - metabolism | Glioma - metabolism | Cerebellum - embryology | Hedgehog Proteins - genetics | Kruppel-Like Transcription Factors - metabolism | Female | Nuclear Proteins - genetics | Spheroids, Cellular | Proto-Oncogene Proteins - metabolism | Cerebellum - metabolism | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Neural Stem Cells - pathology | Animals | Cell Transformation, Neoplastic - pathology | Prosencephalon - embryology | Neural Stem Cells - metabolism | SOX9 Transcription Factor - genetics | Neuroectodermal Tumors, Primitive - pathology | Gliomas | Children's hospitals | Oncology, Experimental | Brain tumors | Stem cells | Transplantation | Universities and colleges | Research | Statistics | Tumors | Cancer | N-MYC | glioma | SOX9 | medulloblastoma | neural stem cells | GFAP | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Nature Communications, ISSN 2041-1723, 04/2016, Volume 7, Issue 1, p. 11185
Journal Article
Journal Article
Stem Cell Reviews and Reports, ISSN 1550-8943, 10/2017, Volume 13, Issue 5, pp. 686 - 698
Amyotrophic Lateral Sclerosis (ALS) is one of the most common adult-onset motor neuron disease causing a progressive, rapid and irreversible degeneration of... 
Life Sciences | Stem Cells | Biomedical Engineering | Motor neurons | Regenerative Medicine/Tissue Engineering | IPSCs | Pericytes | Mesenchymal stromal cells | Amyotrophic lateral sclerosis | SOD1 mice | Cell Biology | MOTOR-NEURONS | MEDICINE, RESEARCH & EXPERIMENTAL | REGRESSION-MODELS | SPINAL-CORD | AMYOTROPHIC-LATERAL-SCLEROSIS | MESENCHYMAL STEM-CELLS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | SKELETAL-MUSCLE | INFLAMMATION | MOUSE MODEL | DISEASE | CENTRAL-NERVOUS-SYSTEM | Spinal Cord - metabolism | Humans | Pericytes - cytology | Brain Stem - metabolism | Cerebral Cortex - pathology | Adipose Tissue - cytology | Male | Superoxide Dismutase-1 - deficiency | Cerebral Cortex - metabolism | Motor Neurons - pathology | Adipose Tissue - metabolism | Brain Stem - pathology | Spinal Cord - pathology | Mesenchymal Stem Cells - cytology | Pericytes - transplantation | Female | Mesenchymal Stem Cells - metabolism | Disease Models, Animal | Induced Pluripotent Stem Cells - metabolism | Induced Pluripotent Stem Cells - pathology | Gene Expression | Amyotrophic Lateral Sclerosis - therapy | Catalase - genetics | Amyotrophic Lateral Sclerosis - genetics | Pericytes - metabolism | RNA-Binding Protein FUS - genetics | Mice, Transgenic | RNA-Binding Protein FUS - metabolism | Amyotrophic Lateral Sclerosis - mortality | Blood-Brain Barrier - metabolism | Catalase - metabolism | Motor Neurons - metabolism | Blood-Brain Barrier - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Superoxide Dismutase-1 - genetics | Survival Analysis | Mice | Mutation | Antioxidants | Enzymes | Nervous system diseases | Neurons | Analysis | Stem cells | Transplantation | Gene expression | Spinal cord | Animal models | Adipose tissue | Mesenchyme | Brain stem | Central nervous system | Medical services | Clinical trials | Superoxide dismutase | Cortex (motor) | Males | FUS protein | Blood-brain barrier | Motor neuron diseases | Neurodegeneration | Degeneration | Medical research | Neurodegenerative diseases | Patients | Survival | Neurological diseases | Stromal cells | Females | In vitro methods and tests | Inhibitory postsynaptic potentials
Journal Article
Cardiovascular Research, ISSN 0008-6363, 09/2012, Volume 95, Issue 4, pp. 487 - 494
Innate mechanisms of inter-organ protection underlie the phenomenon of remote ischaemic preconditioning (RPc) in which episode(s) of ischaemia and reperfusion... 
Brain | Vagus nerve | Ischaemia/reperfusion injury | Preconditioning | Ischaemia | CARDIAC & CARDIOVASCULAR SYSTEMS | K-ATP CHANNELS | LIMB ISCHEMIA | MYOCARDIAL-ISCHEMIA | BRAIN-STEM | reperfusion injury | LENTIVIRAL VECTORS | CARDIOVASCULAR CONTROL | TRANSGENE EXPRESSION | RAT-HEART | AUTONOMIC NERVOUS-SYSTEM | Myocardial Infarction - genetics | Hindlimb | Receptors, G-Protein-Coupled - metabolism | Brain Stem - metabolism | Vagus Nerve - drug effects | Male | Receptors, Neuropeptide - metabolism | Drosophila Proteins - metabolism | Rhodopsin - metabolism | Recombinant Fusion Proteins - metabolism | Autonomic Fibers, Preganglionic - metabolism | Brain Stem - drug effects | Action Potentials | Myocardial Reperfusion Injury - pathology | Time Factors | Muscarinic Antagonists - pharmacology | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Adenoviridae - genetics | Lentivirus - genetics | Myocardial Infarction - physiopathology | Neural Pathways - physiopathology | Myocardial Reperfusion Injury - genetics | Autonomic Fibers, Preganglionic - drug effects | Disease Models, Animal | Heart - innervation | Muscle, Skeletal - blood supply | Transduction, Genetic | Vagus Nerve - metabolism | Constriction | Rats | Receptors, Neuropeptide - genetics | Atropine - pharmacology | Myocardium - pathology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Vagus Nerve - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Rhodopsin - genetics | Ischemic Preconditioning, Myocardial - methods | Brain Stem - physiopathology | Myocardial Infarction - prevention & control | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Genetic Vectors | Myocardial Reperfusion Injury - prevention & control | Original
Journal Article
Nature Communications, ISSN 2041-1723, 07/2014, Volume 5, Issue 1, p. 4381
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2009, Volume 106, Issue 38, pp. 16281 - 16286
The identification of lung tumor-initiating cells and associated markers may be useful for optimization of therapeutic approaches and for predictive and... 
Chemotherapy | Cell growth | Population growth | Lungs | Stem cells | Cell lines | Heterologous transplantation | Tumors | Lung neoplasms | Cancer | CXCR4 | ABC transporters | Cancer stem cells | Chemoresistance | Xenografts | POPULATION | xenografts | MARKER | PROSPECTIVE IDENTIFICATION | MULTIDISCIPLINARY SCIENCES | MELANOMAS | BRAIN-TUMORS | chemoresistance | GROWTH | cancer stem cells | AC133 | EXPRESSION | HEMATOPOIETIC STEM | ANTIGEN | ATP Binding Cassette Transporter, Sub-Family G, Member 2 | Immunohistochemistry | Lung Neoplasms - drug therapy | Neoplastic Stem Cells - drug effects | Humans | Lung Neoplasms - metabolism | Middle Aged | Glycoproteins - metabolism | Drug Resistance, Neoplasm | Lung Neoplasms - pathology | Male | Antineoplastic Agents - therapeutic use | Neoplasm Proteins - metabolism | Antigens, CD - metabolism | Flow Cytometry | Peptides - metabolism | Neoplastic Stem Cells - metabolism | ATP-Binding Cassette Transporters - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Female | Antineoplastic Agents - pharmacology | Tumor Cells, Cultured | Carcinoma, Non-Small-Cell Lung - pathology | Cell Survival - drug effects | Carcinoma, Non-Small-Cell Lung - metabolism | Cisplatin - pharmacology | Mice, SCID | AC133 Antigen | Receptors, CXCR4 - metabolism | Xenograft Model Antitumor Assays | Animals | Cisplatin - therapeutic use | Tumor Burden - drug effects | Survival Analysis | Cell Line, Tumor | Mice | Carcinoma, Non-Small-Cell Lung - drug therapy | Biological Sciences
Journal Article